Search results for " STRESS"

showing 10 items of 3936 documents

GLP 1 receptor agonists, glycemic variability, oxidative stress and acute coronary syndrome

2020

Abstract Glycemic variability (GV) has been recently described as an independent cardiovascular risk factor in patients with acute coronary syndrome (ACS). Also, new findings suggest that GV plays an important role in the development of complications related to impaired glucose metabolism and oxidative stress. On the other hand, although treatment for diabetes can reduce HbA1c in a similar extent their effectiveness over GV can be radically different. Patients in the acute setting of a coronary syndrome should be strictly monitorized to maintain glucose within recommended values; avoiding values lower than 90 mg/dL (5 mmol/L) and higher than 180 mg/dL (10 mmol/L). Also, evidence suggests th…

Blood Glucose0301 basic medicinemedicine.medical_specialtyAcute coronary syndromeCarbohydrate metabolismmedicine.disease_causeGlucagon-Like Peptide-1 Receptor03 medical and health sciences0302 clinical medicineRisk FactorsInternal medicineDiabetes mellitusHumansInsulinMedicineAcute Coronary SyndromeRisk factorGlucagon-like peptide 1 receptorGlycemicbusiness.industryGeneral Medicinemedicine.diseaseOxidative StressGlucose030104 developmental biologyEndocrinologyDiabetes Mellitus Type 2business030217 neurology & neurosurgeryOxidative stressMaceMedical Hypotheses
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Exercise and Metformin Intervention Prevents Lipotoxicity-Induced Hepatocyte Apoptosis by Alleviating Oxidative and ER Stress and Activating the AMPK…

2022

Objective. Nonalcoholic fatty liver disease (NAFLD) and type 2 diabetes (T2DM) commonly coexist and act synergistically to drive adverse clinical outcomes. This study is aimed at investigating the effects of exercise intervention and oral hypoglycaemic drug of metformin (MET) alone or combined on hepatic lipid accumulation. To investigate if oxidative stress and endoplasmic reticulum stress (ERS) are involved in lipotoxicity-induced hepatocyte apoptosis in diabetic mice and whether exercise and/or MET alleviated oxidative stress or ERS-apoptosis by AMPK-Nrf2-HO-1 signaling pathway. Methods. Forty db/db mice with diabetes ( random   blood   glucose ≥ 250   mg / dL ) were randomly allocated i…

Blood GlucoseAgingArticle SubjectNF-E2-Related Factor 2metformiiniApoptosisAMP-Activated Protein KinasesBiochemistryAntioxidantsDiabetes Mellitus ExperimentalMiceohjelmoitunut solukuolemaSuperoxide Dismutase-1aineenvaihduntahäiriötAnimalsHypoglycemic AgentsHematoxylinoksidatiivinen stressibcl-2-Associated X ProteinCaspase 3Cell BiologyGeneral MedicineEndoplasmic Reticulum StressLipidsMetforminOxidative StressDiabetes Mellitus Type 2ei-alkoholiperäinen rasvamaksasairausHepatocyteslääkehoitoEosine Yellowish-(YS)koe-eläinmallitaikuistyypin diabetesSignal TransductionliikuntahoitoOxidative Medicine and Cellular Longevity
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Postnatal Overfeeding Causes Early Shifts in Gene Expression in the Heart and Long-Term Alterations in Cardiometabolic and Oxidative Parameters

2013

International audience; Background: Postnatal overfeeding (OF) in rodents induces a permanent moderate increase in body weight in adulthood. However, the repercussions of postnatal OF on cardiac gene expression, cardiac metabolism and nitro-oxidative stress are less well known. Methodology/Principal Findings: Immediately after birth, litters of C57BL/6 mice were either maintained at 10 (normal-fed group, NF), or reduced to 3 in order to induce OF. At weaning, mice of both groups received a standard diet. The cardiac gene expression profile was determined at weaning and cardiac metabolism and oxidative stress were assessed at 7 months. The cardiac expression of several genes, including membe…

Blood GlucoseAnatomy and PhysiologyTime FactorsMouseMicroarrays[SDV]Life Sciences [q-bio]Myocardial InfarctionGene Expressionlcsh:Medicine030204 cardiovascular system & hematologyCardiovascularmedicine.disease_causeCardiovascular SystemMiceOvernutrition0302 clinical medicineBlood plasmaInsulinlcsh:Science2. Zero hungerRegulation of gene expression0303 health sciencesMultidisciplinaryEjection fractionVentricular RemodelingHeartAnimal ModelsReactive Nitrogen Species[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemApelin[SDV] Life Sciences [q-bio]Body CompositionMedicineFemaleDisease SusceptibilityOxidation-ReductionResearch ArticlePhysiogenomicsmedicine.medical_specialtyDiastoleEndocrine SystemMyocardial Reperfusion InjuryBiology03 medical and health sciencesModel Organisms[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemInternal medicinemedicineAnimalsWeaningVentricular remodelingBiology030304 developmental biologyEndocrine Physiology[ SDV ] Life Sciences [q-bio]Gene Expression ProfilingMyocardiumBody Weightlcsh:RComputational Biologymedicine.diseaseOxidative StressEndocrinologyGene Expression Regulationlcsh:QOxidative stress
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Health benefits of pistachios consumption

2017

The health benefits of nuts, mainly in relation to the improvement of dysmetabolic conditions such as obesity, type 2 diabetes mellitus and the related cardiovascular diseases, have been widely demonstrated. Compared to other nuts, pistachios have a lower fat and caloric content, and contain the highest levels of unsaturated fatty acids, potassium, γ-tocopherol, phytosterols and xanthophyll carotenoids, all substances that are well known for their antioxidant and anti-inflammatory actions. This variety of nutrients contributes to the growing body of evidence that the consumption of pistachios improves health, leading to a greater potential of healthy antioxidant and anti-inflammatory activi…

Blood GlucoseAntioxidantmedicine.medical_treatmentPhytochemicalsPlant ScienceHealth benefits01 natural sciencesBiochemistrymetabolic syndromeAnalytical ChemistryNutrientRisk FactorsmedicineHumansNutsObesityFood sciencePistachioanti-inflammatory activityGlycemicInflammationConsumption (economics)010405 organic chemistrybusiness.industryBody WeightOrganic ChemistryType 2 Diabetes Mellitusmedicine.diseaseLipidsObesityDiet0104 chemical sciencesOxidative Stress010404 medicinal & biomolecular chemistrypolyphenolDiabetes Mellitus Type 2Cardiovascular DiseasesPistaciaMetabolic syndromebusinessNutritive Valueunsaturated fatty acids
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Physiological and agonistic behavioural response of Procambarus clarkii to an acoustic stimulus

2012

Summary This study examined the effects of an acoustic stimulus on the haemolymph and agonistic behaviour of the red swamp crayfish Procambarus clarkii. The experiment was conducted in a tank equipped with a video recording system using 6 groups (3 control and 3 test groups) of five adult crayfish (30 specimens in total). After one hour of habituation, the behaviour of the crayfish was monitored for two hours. During the second hour, the animals in the test groups were exposed to a linear sweep (frequency range 0.1-25 kHz; peak amplitude 148 dBrms re 1 µPa at 12 kHz) acoustic stimulus for 30 minutes. Exposure to the noise produced significant variations in haemato-immunological parameters a…

Blood GlucoseFish ProteinsMaleHemocytesSound SpectrographyPhysiologyVideo RecordingCell CountAstacoideaAquatic ScienceStimulus (physiology)Animal scienceHemolymphAgonistic behaviourAnimalsHSP70 Heat-Shock ProteinsHabituationMolecular BiologyEcology Evolution Behavior and SystematicsVideo recordingProcambarus clarkiibiologyHemagglutinationOsmolar ConcentrationAnatomyacoustic stress agonistic behaviour physiological response red swamp crayfish.biology.organism_classificationCrayfishAcoustic StimulationInsect ScienceFemaleAnimal Science and ZoologyRabbitsAgonistic BehaviorJournal of Experimental Biology
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Differential expression of the corticosteroid receptors GR1, GR2 and MR in rainbow trout organs with slow release cortisol implants

2012

The present study describes the transcriptional levels of the corticosteroid receptors (CRs) GR1, GR2 and MR in the different organs of the rainbow trout (Oncorhynchus mykiss) in response to a slow release of cortisol, throughout a 10-day period. We show that after short term (1 day after cortisol implantation), when the plasma levels of cortisol emulate an acute stress, the GR2 and MR expression levels were upregulated in the brain and head kidney tissues. This result reflects the role of these organs as regulators of the stress response. In general, the rest of the organs, especially gills, intestine, liver, muscle and spleen, showed decreased transcriptional levels of GR1, GR2 and MR, al…

Blood GlucoseFish Proteinsendocrine systemmedicine.medical_specialtyHydrocortisoneTranscription GeneticPhysiologymedicine.drug_classSpleenBiologyHematocritCarbohydrate metabolismBiochemistryReceptors GlucocorticoidDownregulation and upregulationStress PhysiologicalInternal medicinemedicineAnimalsChronic stressLactic AcidReceptorMolecular BiologyDrug Implantsmedicine.diagnostic_testOxygen transportBrainHead KidneyUp-RegulationReceptors Mineralocorticoidmedicine.anatomical_structureEndocrinologyOrgan SpecificityOncorhynchus mykissCorticosteroidhormones hormone substitutes and hormone antagonistsComparative Biochemistry and Physiology Part A: Molecular & Integrative Physiology
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Early adaptive response of the retina to a pro-diabetogenic diet: Impairment of cone response and gene expression changes in high-fructose fed rats

2015

The lack of plasticity of neurons to respond to dietary changes, such as high fat and high fructose diets, by modulating gene and protein expression has been associated with functional and behavioral impairments that can have detrimental consequences. The inhibition of high fat-induced rewiring of hypothalamic neurons induced obesity. Feeding rodents with high fructose is a recognized and widely used model to trigger obesity and metabolic syndrome. However the adaptive response of the retina to short term feeding with high fructose is poorly documented. We therefore aimed to characterize both the functional and gene expression changes in the neurosensory retina of Brown Norway rats fed duri…

Blood GlucoseLeptinMalemedicine.medical_specialtyretinamedicine.medical_treatment[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionEukaryotic Initiation Factor-2BiologyDiabetes Mellitus ExperimentalfructoseCellular and Molecular Neurosciencechemistry.chemical_compoundDownregulation and upregulationInternal medicineGene expressionDietary CarbohydratesmedicineAnimalsInsulin[SDV.MHEP.OS]Life Sciences [q-bio]/Human health and pathology/Sensory Organs2. Zero hungerRetinamedicine.diagnostic_testGene Expression ProfilingLeptinInsulinFructoseEndoplasmic Reticulum Stressmedicine.diseaseCrystallinsSensory SystemsRatsOphthalmologyCholesterolmedicine.anatomical_structureEndocrinologyGene Expression Regulationchemistry[ SDV.MHEP.OS ] Life Sciences [q-bio]/Human health and pathology/Sensory OrgansFructosamineRetinal Cone Photoreceptor Cellsgene expressionsense organsMetabolic syndromeelectroretinographydiet[SDV.AEN]Life Sciences [q-bio]/Food and NutritionElectroretinography
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Postnatal overfeeding in rats leads to moderate overweight and to cardiometabolic and oxidative alterations in adulthood.

2011

In contrast to the masses of data on obesity, few data are available concerning the cardiometabolic and oxidative consequences of moderate overweight. The model of postnatal overfeeding (OF) induces an increase in body weight at weaning that remains during adult life. Litters of Wistar rats were either maintained at 12 pups (normal-fed group, NF), or reduced to 3 pups at birth in order to induce OF. At 6 months of age, metabolic parameters, circulating oxidative stress and aortic and coronary vasoreactivity were assessed. Cardiac susceptibility to ischemia-reperfusion injury was also evaluated ex vivo as were markers of cardiac remodeling. OF led to an increase in body weight at weaning (+5…

Blood GlucoseLeptinleft ventricular end-systolic pressuremedicine.medical_treatment030204 cardiovascular system & hematologyOverweight+dP/dtmedicine.disease_causeBiochemistryCardiovascular System0302 clinical medicineOvernutritionHRleft ventricular developed pressureheart rateInsulinhydroperoxidesworking modeComputingMilieux_MISCELLANEOUSmatrix metallo-proteinase-2W0303 health sciencesANOVAMMP-2OFLeptinROOHinternational unitsGeneral MedicineLsuperoxide dismutase[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemleft ventricular maximal pressure developmentFemalemedicine.symptomleft ventricular end-diastolic pressureanalysis of variancemedicine.medical_specialtyLDHNFleft ventricular minimal pressure developmentIschemiaSNPbody mass indexheartReal-Time Polymerase Chain Reactionoxidative stress AchBMI03 medical and health sciences[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemLangendorff modeoverfeedingInternal medicineRLUBKmedicineWeaningAnimalsLVEDPSODRats WistarVentricular remodeling030304 developmental biologyDNA PrimersPostnatal overfeedingBase Sequencebusiness.industryInsulinsodium nitroprussiatelactate dehydrogenaseLVDPLVESPOverweightrelative light unitsmedicine.diseaseacetylcholinearbitrary unitsRatsIUOxidative StressEndocrinology−dP/dtAUnormal-fedbradykininbusinessEx vivoOxidative stressBiochimie
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Does Glycemic Control Modulate the Impairment of NLRP3 Inflammasome Activation in Type 2 Diabetes?

2019

Since mitochondrial dysfunction is associated with NOD-like receptor family protein 3 (NLRP3) activation in type 2 diabetes (T2D), which can eventually lead to an impaired immune response, we set out to determine if glycemic control modulates the effects of T2D on the NLRP3 inflammasome. We have studied leukocytes from 61 diabetic patients [25 with glycated hemoglobin (HbA(1c)) 7% and 36 with HbA(1c) 8%] and 40 healthy controls. Total and mitochondrial reactive oxygen species (ROS) production was enhanced in T2D patients, and mitochondrial ROS was more pronounced in those with poor glycemic control. Levels of gene and protein expression of NLRP3 were decreased in both diabetic groups and mo…

Blood GlucoseMale0301 basic medicineMitochondrial ROSendocrine system diseasesInflammasomesPhysiologyClinical BiochemistryType 2 diabetesmedicine.disease_causeBiochemistrychemistry.chemical_compoundGene expressionoxidative stressGeneral Environmental Scienceintegumentary systemInterleukinInflammasomeMiddle AgedMitochondriaglycaemic controlCytokinesFemaletype 2 diabetesInflammation MediatorsSignal Transductionmedicine.drugmedicine.medical_specialty03 medical and health sciencesmitochondrial functionInternal medicineNLR Family Pyrin Domain-Containing 3 ProteinmedicineHumansBody Weights and MeasuresMolecular BiologyAgedGlycemicGlycated Hemoglobin030102 biochemistry & molecular biologybusiness.industrynutritional and metabolic diseasesCell Biologymedicine.diseaseNLRP3 inflammasome030104 developmental biologyEndocrinologyDiabetes Mellitus Type 2chemistryGeneral Earth and Planetary SciencesGlycated hemoglobinReactive Oxygen SpeciesbusinessBiomarkersOxidative stressAntioxidants & Redox Signaling
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Antioxidant treatment for impaired hypoxic ventilatory responses in experimental diabetes in the rat

2018

Inflammation, tissue hypoxia, and impaired hypoxic ventilatory response (HVR) are the intricately entwined features of diabetes which perpetuate the disease and its sequelae. Hyperglycemia, notably, is an oxygen consuming process due to enhanced cellular metabolism. Oxidative stress underlies diabetic pathogenesis and also is a crucial modulator of the hypoxic chemoreflex. The present study seeks to determine if suppressed ventilation in diabetes could be improved by antioxidant treatment. The study was performed in streptozotocin-induced diabetes in awake rats. Two weeks into full-fledged diabetes, the rats were divided into mangiferin (potent natural antioxidant)-treated and untreated, wi…

Blood GlucoseMale0301 basic medicinePulmonary and Respiratory MedicineAntioxidantPhysiologyXanthonesmedicine.medical_treatmentInflammationHypoxic ventilatory responsePharmacologymedicine.disease_causeThiobarbituric Acid Reactive SubstancesAntioxidantsDiabetes Mellitus ExperimentalSuperoxide dismutase03 medical and health scienceschemistry.chemical_compoundAntioxidant treatment; Diabetes; Hypoxic ventilatory response; Inflammation; Mangiferin; Oxidative stress.Diabetes mellitusmedicineAnimalsRats WistarHypoxiaMangiferinInflammationbiologySuperoxide DismutaseTumor Necrosis Factor-alphabusiness.industryRespirationGeneral NeuroscienceBrainmedicine.diseaseOxidative Stress030104 developmental biologychemistrybiology.proteinTumor necrosis factor alphaLipid Peroxidationmedicine.symptombusinessOxidative stressRespiratory Physiology & Neurobiology
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