Search results for " STRESS"

showing 10 items of 3936 documents

Effect of tea consumption on oxidative stress and expression of DNA repair genes among metal press workers exposed to occupational noise

2020

Abstract Several studies have shown that tea consumption is associated with beneficial effects on human health, which is mainly explained by the antioxidant properties of tea. However, evidence on the effect of nutrition interventions on oxidative stress in an occupational setting is limited. Therefore, the present study aimed to investigate the effect of tea consumption on oxidative stress in noise-exposed metal press workers. The study sample comprised 24 metal press workers and 24 age-matched control subjects. Metal press workers were assigned to the intervention group consisting of a glass of jujube tea and a portion of raisins per day for 4 weeks. Full-shift noise dosimetry was perform…

Paperchemistry.chemical_classification0303 health sciencesAntioxidantDNA repairbusiness.industryHealth Toxicology and Mutagenesismedicine.medical_treatmentOxidative phosphorylationToxicologyMalondialdehydemedicine.disease_causeControl subjects03 medical and health scienceschemistry.chemical_compound0302 clinical medicinechemistry030220 oncology & carcinogenesismedicineThiolTea consumptionFood sciencebusinessOxidative stress030304 developmental biologyToxicology Research
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An in vitro procedure for evaluation of early stage oxidative stress in an established fish cell line applied to investigation of PHAH and pesticide …

2004

Oxidative stress by increased production of reactive oxygen species such as superoxide has been implicated in the toxicity of PCB's and non-target toxicity of many pesticides. We report the development of a microplate-based method for determination of early stage oxidative stress using an established cell line (EPC) from a skin tumour of carp Cyprinus carpio L. and 2',7'-dichlorodihydrofluorescein diacetate (H(2)-DCFDA) as a fluorescent probe for detection of reactive oxygen species (ROS) formation. Sublethal concentrations of the herbicide Paraquat, an established redox cycling agent and a crude PCB mixture, Arochlor 1254 elicited a linear increase in ROS formation over 2 h exposure which …

ParaquatAroclorsCarpsTime FactorsAquatic ScienceBiologyIn Vitro TechniquesOceanographymedicine.disease_causePesticide toxicityToxicologychemistry.chemical_compoundParaquatToxicity TestsmedicineTumor Cells CulturedAnimalsPesticidesFluorescent Dyeschemistry.chemical_classificationReactive oxygen speciesSuperoxideGeneral MedicineFluoresceinsPollutionpeople.cause_of_deathAcute toxicityOxidative StresschemistryBiochemistryToxicityMalathionpeopleReactive Oxygen SpeciesOxidative stressMarine environmental research
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Effects of pharmacological agents on the lifespan phenotype of Drosophila DJ-1beta mutants.

2010

Mutations in the DJ-1 gene cause autosomal recessive, early-onset Parkinsonism. The DJ-1 protein exerts a protective role against oxidative stress damage, working as a cellular oxidative stress sensor, and it seems to regulate gene expression at different levels. In Drosophila, two DJ-1 orthologs have been identified: DJ-1β and DJ-1β. Several studies have shown that loss of DJ-1β function causes Parkinson's disease (PD)-like phenotypes in flies such as age-dependent locomotor defects, reduced lifespan, and enhanced sensitivity to toxins that induce oxidative stress, like the herbicide paraquat. However, no dopaminergic neurodegeneration is observed. These results suggested that both locomot…

ParaquatDopamineMutantOxidative phosphorylationBiologymedicine.disease_causeAntioxidantsLipid peroxidationchemistry.chemical_compoundParkinsonian DisordersGeneticsmedicineAnimalsGeneticschemistry.chemical_classificationReactive oxygen speciesNeurodegenerationDopaminergicBrainParkinson DiseaseGeneral Medicinemedicine.diseasePhenotypeCell biologyOxidative StressPhenotypechemistryMutationDrosophilaReactive Oxygen SpeciesOxidative stressGene
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Neuroglobin and cytoglobin overexpression protects human SH-SY5Y neuroblastoma cells against oxidative stress-induced cell death

2006

Although reactive oxygen species (ROS) at physiological concentrations are required for normal cell function, excessive production of ROS is detrimental to cells. Neuroglobin and cytoglobin are two globins, whose functions are still a matter of debate. A potential role in the detoxification of ROS is suggested. The influence of neuroglobin and cytoglobin on cell death after oxidative stress in human neuroblastoma SH-SY5Y cells was evaluated. Exposure of SH-SY5Y cells to paraquat or H(2)O(2) resulted in a concentration- and time-dependent induction of apoptotic and necrotic cell death. H(2)O(2) was 16 times more potent to induce cell death as compared to paraquat. SH-SY5Y cells transfected w…

ParaquatProgrammed cell deathTime FactorsBlotting WesternGene ExpressionNeuroglobinNerve Tissue ProteinsBiologymedicine.disease_causeNeuroblastomaCell Line TumormedicineHumansGlobinCell DeathDose-Response Relationship DrugHerbicidesGeneral NeuroscienceCytoglobinCytoglobinHydrogen PeroxideTransfectionFlow CytometryOxidantsMolecular biologyGlobinsOxidative StressApoptosisCell cultureNeuroglobinOxidative stressNeuroscience letters
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Expression of a vegetative-storage-protein gene from Arabidopsis is regulated by copper, senescence and ozone

2001

Emerging data suggest that the mechanisms regulating plant copper homeostasis could be implicated in stress and senescence signal transduction pathways. To gain insight into copper-modulated patterns of gene expression, copper-treated Arabidopsis thaliana (L.) Heynh. plants were analysed by mRNA differential display. The experimental conditions were selected using aggregation of ribulose-1,5-bisphosphate carboxylase-oxygenase (Rubisco) as a molecular sensor to monitor copper-induced oxidative stress. Two copper-induced messengers encoding a vegetative storage protein (VSP2) were isolated by this technique. Both clones differed in the length of their 3'-untranslated region according to the p…

ParaquatSenescenceCopper SulfateRibulose-Bisphosphate CarboxylaseMolecular Sequence DataArabidopsisPlant Sciencemedicine.disease_causeOzoneGene Expression Regulation PlantArabidopsisGene expressionGeneticsmedicineStorage proteinArabidopsis thalianaRNA MessengerCloning Molecularchemistry.chemical_classificationBase SequencebiologyArabidopsis ProteinsRuBisCOGene Expression Regulation DevelopmentalHydrogen Peroxidebiology.organism_classificationOxidative StresschemistryBiochemistrybiology.proteinSignal transductionSequence AnalysisCopperOxidative stressPlanta
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Demographic and Parental Factors Associated With Developmental Outcomes in Children With Intellectual Disabilities

2019

The aim of the study was to examine the relation between demographic variables, parental characteristics, and cognitive, language and motor skills development in children with intellectual disabilities (ID). A sample of 89 children with ID, aged 20 to 47 months, completed the Bayley Scales of Infant Development to measure cognitive, motor and linguistic development. Parents were administered questionnaires about demographic information and parental anxiety, depression, parental stress, conjugality and familial functioning. Parenting behaviors (affection, responsiveness, encouragement, and teaching) were observed using the Spanish version of PICCOLO (Parenting Interactions with Children: Che…

Parent and childlcsh:BF1-990parental anxietyPeople with mental disabilitiesparental stressBayley Scales of Infant Development050105 experimental psychologyDevelopmental psychology03 medical and health sciences0302 clinical medicinefamily functioningCognitive developmentmedicinePsychology0501 psychology and cognitive sciencesDemografiaparental depressionChild developmentMotor skillGeneral PsychologyOriginal ResearchDemographychild development05 social sciencesCognitionChild developmentChecklistLanguage developmentchildren with intellectual disabilitiesDesenvolupament infantillcsh:PsychologyPares i fillspositive parentingDiscapacitats mentalsAnxietymedicine.symptomPsychology030217 neurology & neurosurgery
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Pilot study on stress and resilience in families with premature newborns

2014

Introduction: Prematurity is associated with severe clinical conditions, long hospital stays, and uncertainty about patient outcomes. These circumstances lead to a stressful situation that may affect family functioning. The aim of this study was to study risk and protection factors affecting family functioning in preterm as compared to healthy term infants. Population and methods: Preterm infants with and without pathological conditions (n = 40) were recruited at 24 months post-conception age, together with a control group of healthy term newborn infants (n = 31). Parents or usual caregivers responded to the Inventory of Family Protection Factors and Parental Stress scales. The results were…

ParentsPediatricsmedicine.medical_specialtyFactores de protecciónmedia_common.quotation_subjectPopulationHospitalizaciónDysfunctional familyPilot ProjectsAffect (psychology)Protection factorsPediatricsRJ1-57003 medical and health sciences0302 clinical medicineReward030225 pediatricsManagement of Technology and InnovationAdaptation PsychologicalmedicineHumansFamilyParental stress030212 general & internal medicineRisk factoreducationPathologicalmedia_commoneducation.field_of_studyResiliencebusiness.industryInfant NewbornResilience PsychologicalPrematuridadHospitalizationCase-Control StudiesFamily resilienceResilienciaFamiliaPsychological resilienceAnalysis of varianceEstrés parentalbusinessPrematurityInfant PrematureStress Psychological
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Small Extracellular Vesicles Secreted by Region-specific Astrocytes Ameliorate the Mitochondrial Function in a Cellular Model of Parkinson’s Disease

2021

AbstractExtracellular vesicles (EVs) are emerging as powerful players in cell-to-cell communication both in health and diseased brain. In Parkinson’s disease (PD) – characterized by selective dopaminergic (DAergic) neuron death in ventral midbrain (VMB) and degeneration of DAergic terminals in striatum (STR) – astrocytes (AS) exert dual harmful/protective functions. When activated by chemokine CCL3, AS promote a robust DAergic neuroprotection both in cellular and pre-clinical models of PD, with mechanisms not fully elucidated. Here we used a combination of techniques to characterize AS-EVs derived from VMB and STR, and investigated their potential to exert neuroprotection. First, we show th…

Parkinson's diseaseChemistryDopaminergicmedicineSecretionStriatumCellular modelmedicine.disease_causeNeuron deathmedicine.diseaseNeuroprotectionOxidative stressCell biology
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Drosophila DJ-1 mutants are sensitive to oxidative stress and show reduced lifespan and motor deficits.

2007

Parkinson's disease (PD) is a progressive movement disorder caused by the selective and massive loss of dopaminergic neurons (DA) in the substantia nigra pars compacta (SNc). DJ-1 loss-of-function mutations are involved in inherited early-onset PD forms and result in dysfunction of the oxidative stress response. In mice models, DJ-1 loss provokes sensitivity to oxidative insults but does not produce neurodegeneration. Similar results have been found when analyzing Drosophila mutants for the DJ-1 orthologous genes, DJ-1alpha and DJ-1beta. Here, we report the analysis of two new mutations for the Drosophila DJ-1 genes. Both ubiquitous induction of DJ-1alpha knockdown by RNAi and loss of funct…

Parkinson's diseaseDopamineProtein Deglycase DJ-1Substantia nigraNerve Tissue ProteinsBiologyMotor Activitymedicine.disease_causeLife ExpectancyGeneticsmedicineAnimalsDrosophila ProteinsLoss functionNeuronsGene knockdownPars compactaNeurodegenerationAge FactorsGeneral MedicineAnatomymedicine.diseaseCell biologyOxidative Stressmedicine.anatomical_structureMutationRNA InterferenceNeuronOxidative stressGene
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The HDAC inhibitor SAHA synergistically stimulates the cytotoxic effect induced by Parthenolide in MDA-MB231 cells

2014

We showed that the sesquiterpene lactone Parthenolide (PN) exerts strong cytotoxic effects on triple negative breast cancer MDA-MB231 cells. Our recent results suggest that PN exerts in these cells a cytoprotective effect, which is due to the activation of mTOR pathway. To inhibit this protective response we employ the HDAC inhibitor SAHA, which is known to prevent AKT/mTOR pathway. We show that PN activates Akt, mTOR, p70S6kinase and NRF2 while SAHA abolishes these effects. Further cell pretreatment with SAHA synergistically sensitizes the cells to the cytotoxic effect of PN. Moreover SAHA alone activates the autophagic process. The addition of PN to SAHA reduces this effect and induces ap…

Parthenolide cellular Stress apoptosis autophagy triple negative breast cancer cells HDAC inhibitor.
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