Search results for " STRESS"

showing 10 items of 3936 documents

Oxidative stress-mediated alterations in histone post-translational modifications

2021

Abstract Epigenetic regulation of gene expression provides a finely tuned response capacity for cells when undergoing environmental changes. However, in the context of human physiology or disease, any cellular imbalance that modulates homeostasis has the potential to trigger molecular changes that result either in physiological adaptation to a new situation or pathological conditions. These effects are partly due to alterations in the functionality of epigenetic regulators, which cause long-term and often heritable changes in cell lineages. As such, free radicals resulting from unbalanced/extended oxidative stress have been proved to act as modulators of epigenetic agents, resulting in alte…

0301 basic medicineGene ExpressionContext (language use)Biologymedicine.disease_causeBiochemistryEpigenesis GeneticHistones03 medical and health sciences0302 clinical medicinePhysiology (medical)Gene expressionmedicineHumansHistone codeEpigeneticsRegulation of gene expressionDNA MethylationChromatinCell biologyOxidative Stress030104 developmental biologyHistonebiology.proteinProtein Processing Post-Translational030217 neurology & neurosurgeryOxidative stressFree Radical Biology and Medicine
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NOX2ko Mice Show Largely Increased Expression of a Mutated NOX2 mRNA Encoding an Inactive NOX2 Protein

2020

Background: The superoxide-generating enzyme nicotinamide adenine dinucleotide phosphate (NADPH) oxidase (NOX2 or gp91phox, the phagocytic isoform) was reported as a major source of oxidative stress in various human diseases. Genetic deletion is widely used to study the impact of NOX2-derived reactive oxygen species (ROS) on disease development and progression in various animal models. Here, we investigate why NOX2 knockout mice show no NOX2 activity but express NOX2 mRNA and protein. Methods and Results: Oxidative burst (NOX2-dependent formation of ROS) was measured by L-012-based chemiluminescence and was largely absent in whole blood of NOX2 knockout mice. Protein expression was still de…

0301 basic medicineGene isoformPhysiologyClinical Biochemistrynext generation sequencing (NGS)030204 cardiovascular system & hematologymedicine.disease_causeBiochemistryArticlenicotinamide adenine dinucleotide phosphate (NADPH) oxidase (NOX2) knockout mice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineWestern blotmedicineMolecular BiologyGeneMessenger RNAmedicine.diagnostic_testurogenital systemCell BiologyMolecular biologyRespiratory burst030104 developmental biologychemistryKnockout mousecardiovascular systemoxidative stress related diseasetruncated and inactive mutanthormones hormone substitutes and hormone antagonistsOxidative stressNicotinamide adenine dinucleotide phosphatecirculatory and respiratory physiologyAntioxidants
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Antioxidant role of microvesicles, activated by genistein, in stem cells

2018

Intercellular communication is an essential hallmark of multicellular organisms. Microvesicles (MVs), which are released from and taken up by most types of cells are involved in intercellular communication. The nutrients we ingest from food contact with immune cells in the bloodstream and can promote the formation of MVs. Some foods contain molecules with regulatory activity, such as genistein, a polyphenol found in soy. We aimed to study the effects of MVs released from genistein-treated immune cells on dental pulp stem cells (DPSC). We obtained peripheral blood mononuclear cells (PBMC) from 5 young women (aged 18–25). Cells were cultured and treated with 0.5 μM genistein or 0.01% DMSO as …

0301 basic medicineGenistein030206 dentistrymedicine.disease_causeBiochemistryPeripheral blood mononuclear cellMicrovesiclesCell biology03 medical and health scienceschemistry.chemical_compound030104 developmental biology0302 clinical medicineImmune systemchemistryPhysiology (medical)Dental pulp stem cellsmedicineViability assayStem cellOxidative stressFree Radical Biology and Medicine
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RINT1 Loss Impairs Retinogenesis Through TRP53-Mediated Apoptosis

2020

Genomic instability in the central nervous system (CNS) is associated with defective neurodevelopment and neurodegeneration. Congenital human syndromes that affect the CNS development originate from mutations in genes of the DNA damage response (DDR) pathways. RINT1 (Rad50-interacting protein 1) is a partner of RAD50, that participates in the cellular responses to DNA double-strand breaks (DSB). Recently, we showed that Rint1 regulates cell survival in the developing brain and its loss led to premature lethality associated with genomic stability. To bypass the lethality of Rint1 inactivation in the embryonic brain and better understand the roles of RINT1 in CNS development, we conditionally…

0301 basic medicineGenome instabilityDNA damagereplicative stressBiologyDNA damage responseRetinal ganglionganglion cellsCell and Developmental Biology03 medical and health sciences0302 clinical medicinemedicineoptic nerve hypoplasiaProgenitor celllcsh:QH301-705.5Original ResearchNeurogenesisNeurodegenerationneurodegenerationCell BiologyCell cyclemedicine.diseaseNeural stem cellCell biologyneurogenesis030104 developmental biologylcsh:Biology (General)030220 oncology & carcinogenesisvisual system developmentDevelopmental BiologyFrontiers in Cell and Developmental Biology
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A dual role of caspase-8 in triggering and sensing proliferation-associated DNA damage, a key determinant of liver cancer development.

2017

Summary Concomitant hepatocyte apoptosis and regeneration is a hallmark of chronic liver diseases (CLDs) predisposing to hepatocellular carcinoma (HCC). Here, we mechanistically link caspase-8-dependent apoptosis to HCC development via proliferation- and replication-associated DNA damage. Proliferation-associated replication stress, DNA damage, and genetic instability are detectable in CLDs before any neoplastic changes occur. Accumulated levels of hepatocyte apoptosis determine and predict subsequent hepatocarcinogenesis. Proliferation-associated DNA damage is sensed by a complex comprising caspase-8, FADD, c-FLIP, and a kinase-dependent function of RIPK1. This platform requires a non-apop…

0301 basic medicineGenome instabilityMaleliver; Hepatocellular carcinoma; DNA damage response; replication stress; apoptosisCancer ResearchDNA RepairCarcinogenesisFas-Associated Death Domain ProteinApoptosisurologic and male genital diseasesDNA damage responseDna Damage Response ; Apoptosis ; Hepatocellular Carcinoma ; Liver ; Replication StressHistonesMice0302 clinical medicineRisk FactorsFADDPhosphorylationCellular SenescenceCaspase 8biologyLiver Neoplasmshepatocellular carcinomaLiver regeneration3. Good healthHistoneOncologyReceptors Tumor Necrosis Factor Type I030220 oncology & carcinogenesisReceptor-Interacting Protein Serine-Threonine KinasesFemalebiological phenomena cell phenomena and immunityCell agingCarcinoma HepatocellularDNA damageDNA repairreplication stressCaspase 8liverArticleGenomic Instability03 medical and health sciencesAnimalsHepatectomyHumansCrosses GeneticCell ProliferationJNK Mitogen-Activated Protein KinasesCell BiologyLiver Regeneration030104 developmental biologyImmunologyChronic Diseasebiology.proteinCancer researchHepatocytesMyeloid Cell Leukemia Sequence 1 ProteinDNA Damage
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Dicer prevents genome instability in response to replication stress

2019

Dicer, an endoribonuclease best-known for its role in microRNA biogenesis and RNA interference pathway, has been shown to play a role in the DNA damage response and repair of double-stranded DNA breaks (DSBs) in mammalian cells. However, it remains unknown whether Dicer is also important to preserve genome integrity upon replication stress. To address this question, we focused our study on common fragile sites (CFSs), which are susceptible to breakage after replication stress. We show that inhibition of the Dicer pathway leads to an increase in CFS expression upon induction of replication stress and to an accumulation of 53BP1 nuclear bodies, indicating transmission of replication-associate…

0301 basic medicineGenome instabilityreplication stressDNA damageChromosomal fragile siteBiologygenomic instabilitycommon fragile siteCell biologySettore BIO/18 - Genetica03 medical and health sciences030104 developmental biology0302 clinical medicineOncology030220 oncology & carcinogenesisFANCD2biology.proteinDicer PathwayMitosiscommon fragile sitesDroshaResearch PaperDicerDicerOncotarget
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A comparison of the performance of natural hybrids Saccharomyces cerevisiae × Saccharomyces kudriavzevii at low temperatures reveals the crucial role…

2018

Fermentation performance at low temperature is a common approach to obtain wines with better aroma, and is critical in industrial applications. Natural hybrids S. cerevisiae × S. kudriavzevii, isolated from fermentations in cold-climate European countries, have provided an understanding of the mechanisms of adaptation to grow at low temperature. In this work, we studied the performance of 23 S. cerevisiae × S. kudriavzevii hybrids at low temperature (8, 12 and 24 °C) to characterize their phenotypes. Kinetic parameters and spot tests revealed a different ability to grow at low temperature. Interestingly, the genome content of the S. kudriavzevii in hybrids was moderately correlated with a s…

0301 basic medicineGenomic contributionSaccharomyces cerevisiaeS. cerevisiaeHybridsWineSaccharomyces cerevisiaeMicrobiologySaccharomyces03 medical and health sciencesSaccharomycesS. kudriavzeviiAlleleGeneHybridGeneticsbiologyGeneral Medicinebiology.organism_classificationAdaptation PhysiologicalCold TemperatureEurope030104 developmental biologyFermentationOdorantsHybridization GeneticFermentationAdaptationCold stressSaccharomyces kudriavzeviiFood ScienceInternational journal of food microbiology
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Oxidative stress and exceptional human longevity: Systematic review.

2020

Oxidative stress (OS) has been previously linked to the aging process, as have some diseases and geriatric syndromes as frailty and sarcopenia. The aim of the present study was to perform a systematic review on oxidative stress activity and extreme longevity in humans.We conducted a systematic literature review following the PRISMA guidelines. Observational studies assessing OS-biomarkers and/or antioxidants in long-lived individuals (97 years old or over) comparing them to those of one or more age groups, (at least one of which from comprising elderly subjects) were considered for inclusion. A narrative synthesis was planned. Quality of selected studies was assessed using the Newcastle-Ott…

0301 basic medicineGerontologyAdultAgingmedia_common.quotation_subjectLongevitymedicine.disease_causeBiochemistryAntioxidants03 medical and health sciencesYoung Adult0302 clinical medicinePhysiology (medical)Plasma lipidsmedicineHumansmedia_commonAgedAged 80 and overbusiness.industrySuperoxide DismutaseLongevityMiddle Agedmedicine.diseaseOxidative Stress030104 developmental biologySystematic reviewSarcopeniaHuman longevityExtreme longevity trackingObservational studyLipid Peroxidationbusiness030217 neurology & neurosurgeryOxidative stressFree radical biologymedicine
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Targeting aging with functional food: Pasta with opuntia single-arm pilot study

2018

Interventions to extend life span represent the new perspective in aging investigation. Healthy dietary habits are important modifiable factors that can favor a healthy aging phenotype. Many studies have demonstrated benefits for metabolic syndrome and type 2 diabetes mellitus resulting from the traditional Mediterranean foods. Opuntia Ficus Indica (OFI), widespread in the Mediterranean basin, belongs to the Cactaceae family. It is known for its antioxidant and anti-inflammatory properties. Moreover, products containing extracts from OFI fruits or cladodes have been used to control obesity and other metabolic parameters, such as glycemia and lipid profile. The aim of this study was to analy…

0301 basic medicineGerontologyAdultMaleAgingMediterranean dietLongevityPsychological interventionAnti-Inflammatory AgentsPilot Projects030204 cardiovascular system & hematologyBiologyAntioxidantsOpuntia Ficus Indica03 medical and health sciencesYoung Adult0302 clinical medicineFunctional foodFunctional FoodMediterranean dietDiabetes MellitusHumansLongitudinal StudiesObesityHealthy agingAgedCell ProliferationInflammationMetabolic Syndromeage-related diseaseoxidative streLife spanTraditional medicinePlant ExtractsPerspective (graphical)food and beveragesOpuntiaMiddle AgedHealthy VolunteersOxidative Stress030104 developmental biologyPhenotypenutritionDiabetes Mellitus Type 2Cardiovascular DiseasesDietary SupplementsFemaleGeriatrics and Gerontology
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Loneliness, Social Isolation, and Cardiovascular Health

2018

Abstract Significance: Social and demographic changes have led to an increased prevalence of loneliness and social isolation in modern society. Recent Advances: Population-based studies have demonstrated that both objective social isolation and the perception of social isolation (loneliness) are correlated with a higher risk of mortality and that both are clearly risk factors for cardiovascular disease (CVD). Lonely individuals have increased peripheral vascular resistance and elevated blood pressure. Socially isolated animals develop more atherosclerosis than those housed in groups. Critical Issues: Molecular mechanisms responsible for the increased cardiovascular risk are poorly understoo…

0301 basic medicineGerontologyMaleSympathetic nervous systemHypothalamo-Hypophyseal SystemPhysiologyClinical BiochemistryPopulationDiseaseBiochemistryCardiovascular System03 medical and health sciences0302 clinical medicinecardiovascular diseaseRisk FactorsmedicineRisk of mortalityoxidative stressHumansSocial isolationeducationMolecular BiologyGeneral Environmental ScienceSocial stresseducation.field_of_studybusiness.industryLonelinessLonelinessCell BiologyForum Review Articles030104 developmental biologymedicine.anatomical_structureSocial IsolationVascular resistanceGeneral Earth and Planetary SciencesFemalemedicine.symptombusiness030217 neurology & neurosurgeryAntioxidants & Redox Signaling
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