Search results for " Smoke"

showing 10 items of 127 documents

Cigarette smoke increases BLT2 receptor functions in bronchial epithelial cells: in vitro and ex vivo evidence

2013

Summary Leukotriene B4 (LTB4) is a neutrophil chemotactic molecule with important involvement in the inflammatory responses of chronic obstructive pulmonary disease (COPD). Airway epithelium is emerging as a regulator of innate immune responses to a variety of insults including cigarette smoke, the major risk factor for COPD. In this study we have explored whether cigarette smoke extracts (CSE) or soluble mediators present in distal lung fluid samples (mini-bronchoalveolar lavages) from smokers alter the expression of the LTB4 receptor 2 (BLT2) and peroxisome proliferator-activated receptor-α (PPAR-α) in bronchial epithelial cells. We also evaluated the effects of CSE on the expression of i…

Leukotriene B4NeutrophilsImmunologyIntercellular Adhesion Molecule-1Blotting WesternReceptors Leukotriene B4Peroxisome proliferator-activated receptorSettore MED/41 - AnestesiologiaInflammationBronchiBiologychronic obstructive pulmonary diseasechemistry.chemical_compoundTobaccoacute lung injiurybronchial epithelial cellleukotriene B4.medicineCell AdhesionImmunology and AllergyHumansPPAR alphaReceptorPromoter Regions GeneticCells Culturedchemistry.chemical_classificationInnate immune systemPlant Extractscigarette smokeSmokingEpithelial CellsOriginal Articlesrespiratory systemFlow CytometryIntercellular Adhesion Molecule-1Neutrophiliarespiratory tract diseasesacute lung injiury; bronchial epithelial cells; cigarette smoke; chronic obstructive pulmonary disease; inflammation; leukotriene B4.STAT1 Transcription FactorchemistryinflammationImmunologyRespiratory epitheliumRNA Interferencemedicine.symptomBronchoalveolar Lavage FluidProtein Binding
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Exposure to cigarette smoke extract and lipopolysaccharide modifies cytoskeleton organization in bronchial epithelial cells

2017

The integrity of the respiratory epithelium is crucial for airway homeostasis. Tobacco smoke exposure and recurrent infections of the airways play a crucial role in the progression and in the decline of the respiratory function in chronic obstructive pulmonary disease (COPD). The aim of this study was to detect differentially expressed proteins in a bronchial epithelial cell line (16-HBE) stimulated with cigarette smoke extract (CSE) and lipopolysaccharide (LPS), a constituent of gram-negative bacteria, alone and/or in combination, by using two-dimensional electrophoresis (2DE) analysis coupled with matrix-assisted laser desorption/ionization time-of-flight mass spectrometry. Western blot a…

Lipopolysaccharides0301 basic medicinePulmonary and Respiratory Medicinebronchial epithelial cells; cigarette smoke; cytoskeleton; Molecular Biology; Pulmonary and Respiratory Medicine; Clinical BiochemistryProteomeLipopolysaccharideCytoskeleton organizationClinical BiochemistryRespiratory MucosaCell Line03 medical and health scienceschemistry.chemical_compound0302 clinical medicineSmokebronchial epithelial cellHumansCigarette smokeMedicineCytoskeletonMolecular Biologybronchial epithelial cellsCytoskeletonbusiness.industrycigarette smokeTobacco smoke exposureEpithelial CellsTobacco Productsrespiratory systemrespiratory tract diseases030104 developmental biologyGene Expression Regulationchemistry030220 oncology & carcinogenesisImmunologyRespiratory epitheliumAirwaybusinessHomeostasisExperimental Lung Research
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Oxidative stress and innate immunity responses in cigarette smoke stimulated nasal epithelial cells

2013

Cigarette smoke extracts (CSE) may play a significant role in diseases of the upper airway including chronic rhinosinusitis. Even short term exposure of cigarette smoke has adverse effects on mitochondrial functions and redox homeostasis in tissues which may progress to further complications associated with chronic smoking. Cigarette smoke alters toll-like receptor 4 (TLR4) expression and activation in bronchial epithelial cells. Carbocysteine is an anti-oxidant and mucolytic agent. The effects of carbocysteine on CSE induced oxidative stress and on associated innate immune and inflammatory responses in nasal epithelial cells are largely unknown. The present study was aimed to assess in CSE…

LipopolysaccharidesNecrosisNeutrophilsPhalloidineCARB CSE Cigarette smoke LPS Nasal epithelial cells ROS Reactive oxygen species TLR4 carbocysteine cigarette smoke extracts lipolysaccharide reactive oxygen species toll like receptor 4Fluorescent Antibody TechniqueApoptosisMucous membrane of noseCell SeparationBiologyToxicologymedicine.disease_causeCell LineNecrosisSmokeTobaccomedicineHumansExpectorantschemistry.chemical_classificationReactive oxygen speciesInnate immune systemCarbocysteineEpithelial CellsCarbocysteineTobacco ProductsGeneral MedicineActinsImmunity InnateToll-Like Receptor 4Nasal MucosaOxidative StresschemistryApoptosisImmunologyTLR4medicine.symptomReactive Oxygen SpeciesOxidative stressToxicology in Vitro
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Sociodemographic and behavioural correlates of lifetime number of sexual partners: findings from the English Longitudinal Study of Ageing.

2018

BackgroundNo current data are available on correlates of lifetime sexual partners at older ages. This study aimed to explore correlates of the lifetime number of sexual partners in a sample of older adults.MethodData were from 3054 men and 3867 women aged ≥50 years participating in the English Longitudinal Study of Ageing. Participants reported their lifetime number of sexual partners and a range of sociodemographic characteristics and health behaviours. Multivariable multinomial logistic regression was used to examine correlates of lifetime number of sexual partners, with analyses performed separately for men and women and weighted for non-response.ResultsYounger age, being separated/divor…

Longitudinal studyYounger ageSocial Sciences and HumanitiesSocial Sciences (SOC)Sosyal Bilimler ve Beşeri BilimlerEthnic groupOBSTETRICS & GYNECOLOGYSağlık BilimleriDoğum ve JinekolojiClinical Medicine (MED)SOCIAL SCIENCES BIOMEDICAL0302 clinical medicineSociologyKADIN HASTALIKLARI & DOĞUMSurgery Medicine SciencesKlinik Tıp (MED)030212 general & internal medicineHomosexuality1506older adultsmedia_commonMultinomial logistic regressionKlinik TıpObstetrics and GynecologyGeneral Social SciencesKadın Hastalıkları ve DoğumTıpnumber of sexual partnersCerrahi Tıp BilimleriLife course approachMedicineAİLE ÇALIŞMALARISosyal Bilimler (SOC)0305 other medical sciencePsychologySOSYAL BİLİMLER BİYOMEDİKSELmedia_common.quotation_subjectSOCIAL SCIENCES GENERALnumber of sexual partnerHome Economics03 medical and health sciencesfindings from the English Longitudinal Study of Ageing- BMJ Sex Reprod Health cilt.4 ss.2018-2020 2019 [Jackson S. E. YANG L. López Sánchez G. Veronese N. KOYANAGI A. Grabovac I. SOYSAL P. Smith L. -Sociodemographic and behavioural correlates of lifetime number of sexual partners]Health SciencesGenel Sosyal BilimlerSosyal ve Beşeri BilimlerSocial Sciences & Humanitiessolder adultSosyoloji030505 public healthEv EkonomisiResearchSosyal Bilimler GenelCLINICAL MEDICINEFormer SmokerpredictorsReproductive MedicineAgeingFAMILY STUDIESDemographyBMJ sexualreproductive health
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Loss of life expectancy from air pollution compared to other risk factors: a worldwide perspective

2020

Abstract Aims Long-term exposure of humans to air pollution enhances the risk of cardiovascular and respiratory diseases. A novel Global Exposure Mortality Model (GEMM) has been derived from many cohort studies, providing much-improved coverage of the exposure to fine particulate matter (PM2.5). We applied the GEMM to assess excess mortality attributable to ambient air pollution on a global scale and compare to other risk factors. Methods and results We used a data-informed atmospheric model to calculate worldwide exposure to PM2.5 and ozone pollution, which was combined with the GEMM to estimate disease-specific excess mortality and loss of life expectancy (LLE) in 2015. Using this model, …

Lung DiseasesMaleFine particulate matterTime Factors010504 meteorology & atmospheric sciencesPhysiologyAnthropogenic emissionsFossil fuel emissionsAir pollution010501 environmental sciencesGlobal Healthmedicine.disease_cause01 natural sciencesRisk FactorsGlobal healthAcademicSubjects/MED00200Childmedia_commonAged 80 and overExposure to ViolenceExpectancy theoryAir PollutantsMortality rateMiddle AgedParticulatesCardiovascular DiseasesChild PreschoolPublic health risksFemaleCardiology and Cardiovascular MedicineLoss of lifeAdultPollutionAdolescentRisk in Cardiovascular Diseasemedia_common.quotation_subjectAir pollutionViolenceRisk AssessmentYoung AdultOzoneLife ExpectancyPhysiology (medical)Environmental healthTobacco SmokingmedicineHumansReview Series from the Naples 2019 Joint Meeting of the ESC Working Groups on Myocardial Function and Cellular Biology of the HeartAged0105 earth and related environmental sciencesInfant NewbornLoss of life expectancyInfantEnvironmental ExposureOriginal ArticlesNatural emissionsEditor's ChoiceLife expectancyEnvironmental scienceParticulate MatterTobacco Smoke PollutionCardiovascular Research
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Environmental risk factors and lung diseases in children: From guidelines to health effects

2013

Abstract During the last decades research all over the world has highlighted the deleterious effects of outdoor and indoor pollution on respiratory health of adults and children. The World Health Organization (WHO) “ Air quality guidelines for Europe ” played a fundamental role in providing information and guidance to authorities involved in the air pollution field and they are considered the key source on which the European Commission's directive on air quality is based. Children appear to be most vulnerable to the harmful effects of outdoor pollutants, which can cause both acute exacerbations, as well as chronic respiratory symptoms and diseases. Possible mechanisms include the induction …

Lung Diseasesmedicine.medical_specialtyAir pollutionmedicine.disease_causelaw.inventionAllergic sensitizationSettore MED/38 - Pediatria Generale E SpecialisticachildrenRisk FactorslawEnvironmental healthEpidemiologymedicineHumanspollutionChildAir quality indexPollutantAir PollutantsLungbusiness.industryMortality rateInfantObstetrics and Gynecologyhealthpollution health childrenVentilationmedicine.anatomical_structureChild PreschoolPediatrics Perinatology and Child HealthVentilation (architecture)Tobacco Smoke Pollutionbusiness
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Investigating the Relationship between Parental Education, Asthma and Rhinitis in Children Using Path Analysis.

2022

Parental socioeconomic position (SEP) is a known determinant of a child’s health. We aimed to investigate whether a low parental education, as proxy of SEP, has a direct effect on physician-diagnosed asthma, current asthma and current allergic rhinitis in children, or whether associations are mediated by exposure to other personal or environmental risk factors. This study was a secondary data analysis of two cross-sectional studies conducted in Italy in 2006. Data from 2687 adolescents (10–14 years) were analyzed by a path analysis model using generalized structural equation modelling. Significant direct effects were found between parental education and family characteristics (n…

MaleAdolescentHealth Toxicology and MutagenesisPublic Health Environmental and Occupational Healthprenatal educationstructural equation modellingRhinitis AllergicAsthmaFathersCross-Sectional Studiesasthma; children; prenatal education; rhinitis; structural equation modellingchildrenPregnancyRisk FactorsHumansFemaleTobacco Smoke Pollutionasthma; rhinitis; structural equation modelling; prenatal education; childrenChildRhinitisInternational journal of environmental research and public health
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Tobacco smoke and risk of childhood acute non-lymphocytic leukemia: findings from the SETIL study.

2014

Background Parental smoking and exposure of the mother or the child to environmental tobacco smoke (ETS) as risk factors for Acute non-Lymphocytic Leukemia (AnLL) were investigated. Methods Incident cases of childhood AnLL were enrolled in 14 Italian Regions during 1998–2001. We estimated odds ratios (OR) and 95% confidence intervals (95%CI) conducting logistic regression models including 82 cases of AnLL and 1,044 controls. Inverse probability weighting was applied adjusting for: age; sex; provenience; birth order; birth weight; breastfeeding; parental educational level age, birth year, and occupational exposure to benzene. Results Paternal smoke in the conception period was associated wit…

MaleLifestyle Causes of CancerPediatricslcsh:MedicineSettore MED/42 - Igiene Generale E ApplicataTobacco smokeEconomicaPregnancySurveys and Questionnaireshemic and lymphatic diseasesOdds RatioMedicine and Health SciencesPublic Health SurveillanceChildlcsh:ScienceMultidisciplinaryparental smokingCancer Risk Factorschildhood leukemiaSmokingBirth orderLeukemia Myeloid AcuteOncologyMaternal ExposureChild PreschoolPrenatal Exposure Delayed EffectsFemaleAETIOLOGYResearch Articlemedicine.medical_specialtyBirth weightSocio-culturalemedicineHumansTobacco smoke childhoodPregnancybusiness.industrylcsh:RCase-control studyPregnancy and CancerAmbientaleOdds ratiomedicine.diseaseConfidence intervalPediatric OncologyCase-Control StudiesTobacco Smoke Pollutionlcsh:QbusinessBreast feedingDemographyPLoS ONE
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Is physical activity associated with low-risk health behaviours among 15-year-old adolescents in Finland?

2012

Aims: To investigate the associations between physical activity and the pattern of risk health behaviour consisting of smoking, alcohol consumption, snuff (snus), cannabis, and condom use among 15-year-old adolescents, taking their educational aspirations and family affluence into account. Methods: The data were collected in the Health Behaviour in School-aged Children (HBSC) study in Finland in 2006. Standardised questionnaires were issued at schools to a 15-year-old nationally representative sample, of which 84.5% (1710 pupils) participated. Logistic regression analysis was used to explore the associations between physical activity, pattern of risk health behaviour, family affluence, and…

MaleMarijuana AbuseCross-sectional studyHealth Behaviorphysical activityfamily affluencelaw.inventionCondomsnuoretlawMedicineFinlandeducation.field_of_studybiologyexerciseSmokingAge Factorsta3141General MedicineTobacco Use Disorderriskitekijärisk factorSnusEducational StatusFemaleFamily Relationsmedicine.medical_specialtyTobacco SmokelessAlcohol DrinkingAdolescentPopulationMotor Activityterveyden edistäminenRisk-TakingSex FactorsCondomliikunta-aktiivisuusEnvironmental healthHumansSnuffRisk factorperheen koettu varallisuusPsychiatryeducationbusiness.industryPublic Health Environmental and Occupational Healthbiology.organism_classificationfyysinen harjoitteluHealth promotionCross-Sectional StudiesAdolescent BehaviorCannabisbusiness
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Gender-Dependent Effect of GSTM1 Genotype on Childhood Asthma Associated with Prenatal Tobacco Smoke Exposure

2014

It remains unclear whether the GSTM1 genotype interacts with tobacco smoke exposure (TSE) in asthma development. This study aimed to investigate the interactions among GSTM1 genotype, gender, and prenatal TSE with regard to childhood asthma development. In a longitudinal birth cohort in Taiwan, 756 newborns completed a 6-year follow-up, and 591 children with DNA samples available for GSTM1 genotyping were included in the study,and the interactive influences of gender-GSTM1 genotyping-prenatal TSE on childhood asthma development were analyzed. Among these 591 children, 138 (23.4%) hadphysician-diagnosed asthmaat 6 years of age, and 347 (58.7%) werenull-GSTM1. Prenatal TSE significantly incre…

MalePediatricsmedicine.medical_specialtyArticle Subjectanimal diseasesTaiwanlcsh:MedicineComorbidityLower riskPolymorphism Single NucleotideGeneral Biochemistry Genetics and Molecular BiologyPregnancyRisk FactorsGenotypePrevalencemedicineHumansGenetic Predisposition to DiseaseSex DistributionChildneoplasmsGenotypingGlutathione TransferaseAsthmaChildhood asthmaintegumentary systemGeneral Immunology and Microbiologybusiness.industrylcsh:RTobacco smoke exposureInfant NewbornInfantTotal igeGeneral Medicinemedicine.diseaseAsthmanervous system diseasesCausalityChild PreschoolPrenatal Exposure Delayed EffectsFemaleTobacco Smoke PollutionbusinessBirth cohortResearch ArticleBioMed Research International
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