Search results for " T-Cell"

showing 10 items of 320 documents

Immunological Markers for PML Prediction in MS Patients Treated with Natalizumab

2015

International audience; Natalizumab (NTZ), a monoclonal antibody recognizing the alpha4 integrin chain, has been approved for the treatment of active multiple sclerosis, but expose to the onset of a rare side effect, progressive multifocal leukoencephalopathy (PML). Estimating the individual risk of PML in NTZ-treated patients is a major challenge, and therapeutic strategies are mainly guided by the overall PML risk assessed by identified risk factors: JC virus (JCV) seropositivity, treatment duration (with peak incidence after 24 months), and the previous use of immunosuppressive therapies. Given that this stratification does not yet allow a precise individual prediction of PML, other pred…

lcsh:Immunologic diseases. Allergy[SDV.IMM] Life Sciences [q-bio]/ImmunologySide effectmedicine.drug_classvirusesImmunologyJC virusReview Articlerisk stratificationCD11aJC virusmultiple sclerosismedicine.disease_causeMonoclonal antibodyCD49dprogressive multifocal leukoencephalopathyNatalizumabeffector memory T-cellst effector memory cellsImmunology and AllergyMedicineselectinPMLbusiness.industryMultiple sclerosisProgressive multifocal leukoencephalopathyvirus diseasesmedicine.disease3. Good healthJCVImmunologySelectins[SDV.IMM]Life Sciences [q-bio]/Immunologylcsh:RC581-607businessmedicine.drugFrontiers in Immunology
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The deubiquitinating enzyme CYLD regulates the differentiation and maturation of thymic medullary epithelial cells.

2014

The cross talk between thymocytes and the thymic epithelium is critical for T-cell development and the establishment of central tolerance. Medullary thymic epithelial cells (mTECs) are located in the thymic medulla and mediate the elimination of self-reactive thymocytes, thereby preventing the onset of autoimmunity. Previous studies identified the deubiquitinating enzyme CYLD as a critical regulator of T-cell development by activating proximal T-cell receptor signaling during the transition of double-positive to single-positive thymocytes. Here we evaluated the impact of the naturally occurring short-splice variant of the cyld gene (sCYLD) on the development and maturation of mTECs. We foun…

medicine.medical_specialtyMedullary cavityImmunologyReceptors Antigen T-CellCell CountThymus GlandBiologyDeubiquitinating Enzyme CYLDImmunophenotypingMiceInternal medicinemedicineImmunology and AllergyAnimalsMice KnockoutThymocytesUbiquitinationCell DifferentiationEpithelial CellsCell BiologyCell biologyDeubiquitinating Enzyme CYLDCysteine EndopeptidasesEndocrinologyPhenotypeAntigens SurfaceMutationFemaleSignal TransductionImmunology and cell biology
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Comparison of pegylated interferon α-2b plus psoralen PUVA versus standard interferon α-2a plus PUVA in patients with cutaneous T-cell lymphoma

2011

Objective  The aim of this study was to evaluate the safety and efficacy profile of pegylated interferon α-2b (PEG-IFN α-2b) in combination with photochemotherapy (PUVA) in the treatment of cutaneous T-cell lymphoma (CTCL) in comparison with standard IFN α plus PUVA. Design  Retrospective cohort study over a period of 7 years. Patients and interventions  A total of 17 consecutive CTCL patients (stage IA–IV) were retrospectively analysed for toxicity and response rates associated with PEG-IFN α-2b (1.5 μg/kg weekly) plus PUVA (n = 9) or standard IFN α-2a (9 MIU 3×/week) plus PUVA (n = 8). Main outcome measures  Differences of response rates (complete/partial remission), progression-free surv…

medicine.medical_specialtybusiness.industrymedicine.medical_treatmentCutaneous T-cell lymphomaDermatologymedicine.diseaseDermatologyGastroenterologyDiscontinuationchemistry.chemical_compoundInfectious DiseaseschemistryPegylated interferonInternal medicinePUVA therapyToxicitymedicinebusinessProspective cohort studyAdverse effectPsoralenmedicine.drugJournal of the European Academy of Dermatology and Venereology
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Reversible Myelofibrose bei angioimmunoblastischer Lymphadenopathie

2008

Ankle oedema and abdominal swelling suddenly developed in a 55-year-old woman who also had lymphadenopathy in the neck, axillae and groin. Ultrasonography demonstrated hepatosplenomegaly, ascites and pleural effusions. Histological examination of some lymph-nodes from the axilla and groin revealed angioimmunoblastic lymphadenopathy (low-malignant peripheral T cell lymphoma). Bone-marrow biopsy was undertaken because of a normocytic anaemia (haemoglobin 4.9 g/dl) requiring blood transfusion, thrombocytopenia (5000/microliters) and monoclonal IgG gammopathy. This showed lymphoma-associated secondary myelofibrosis. Treatment with prednisone (2 mg/kg daily for 8 weeks) and vincristine (1 mg/m2 …

medicine.medical_specialtymedicine.diagnostic_testGroinbusiness.industryHepatosplenomegalyGeneral Medicinemedicine.diseaseGastroenterologyPeripheral T-cell lymphomamedicine.anatomical_structurePrednisoneGammopathyInternal medicineBiopsyAscitesmedicinemedicine.symptomMyelofibrosisbusinessmedicine.drugDMW - Deutsche Medizinische Wochenschrift
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Immune Modulating Effects of NKT Cells in a Physiologically Low Dose Leishmania major Infection Model after αGalCer Analog PBS57 Stimulation

2014

Leishmaniasis is a parasitic infection affecting ∼12 million people worldwide, mostly in developing countries. Treatment options are limited and no effective vaccines exist to date. Natural Killer T (NKT) cells are a conserved innate-like lymphocyte population with immunomodulating effects in various settings. A number of reports state a role of NKT cells in different models of Leishmania infection. Here, we investigated the effect of NKT cells in a physiologically relevant, intradermal low dose infection model. After inoculation of 103 infectious-stage L. major, comparable numbers of skin-immigrating NKT cells in both susceptible BALB/c mice and resistant C57BL/6 mice were noted. Compared …

medicine.medical_treatmentLymphocyteMedizinPathogenesisNK cellsProtozoologyPathology and Laboratory MedicineCellular typesMedicine and Health SciencesLymphoid OrgansLeishmania majorImmune ResponseLeishmania majorSkinProtozoansMice Inbred BALB Ceducation.field_of_studybiologylcsh:Public aspects of medicineNatural killer T cellInfectious Diseasesmedicine.anatomical_structureCytokineMedical MicrobiologyHost-Pathogen InteractionsWhite blood cellsCytokinesAnatomyResearch ArticleCell biologyBlood cellslcsh:Arctic medicine. Tropical medicinelcsh:RC955-962Immune CellsImmunologyPopulationT cellsLeishmaniasis CutaneousGalactosylceramidesSpleenImmunopathologyMicrobiologyLymphatic SystemImmunomodulationImmune ActivationImmune systemImmunityMicrobial ControlmedicineAnimalsImmunologic FactorseducationImmunity to InfectionsMicrobial PathogensBiology and life sciencesImmunityOrganismsPublic Health Environmental and Occupational HealthImmunoregulationlcsh:RA1-1270Molecular Developmentbiology.organism_classificationAcquired Immune SystemParasitic ProtozoansMice Inbred C57BLDisease Models AnimalAnimal cellsImmune SystemImmunologyNatural Killer T-CellsClinical ImmunologyParasitologyDevelopmental Biology
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Differential requirements for antigen or homeostatic cytokines for proliferation and differentiation of human Vgamma9Vdelta2 naive, memory and effect…

2005

We have compared four human subsets of Vgamma9Vdelta2 T cells, naive (T(naive), CD45RA(+)CD27(+)), central memory (T(CM), CD45RA(-)CD27(+)), effector memory (T(EM), CD45RA(-)CD27(-)) and terminally differentiated (T(EMRA), CD45RA(+)CD27(-)), for their capacity to proliferate and differentiate in response to antigen or homeostatic cytokines. Cytokine responsiveness and IL-15R expression were low in T(naive) cells and progressively increased from T(CM) to T(EM) and T(EMRA) cells. In contrast, the capacity to expand in response to antigen or cytokine stimulation showed a reciprocal pattern and was associated with resistance to cell death and Bcl-2 expression. Whereas antigen-stimulated cells a…

medicine.medical_treatmentT cellCellular differentiationImmunologychemical and pharmacologic phenomenaBiologyLymphocyte ActivationAntigenimmune system diseasesT-Lymphocyte SubsetsmedicineImmunology and AllergyHomeostasisHumansAntigensReceptorCells CulturedInterleukin-15Receptors Interleukin-15virus diseaseshemic and immune systemsCell DifferentiationReceptors Antigen T-Cell gamma-deltaReceptors Interleukin-2In vitroCell biologyTumor Necrosis Factor Receptor Superfamily Member 7Cytokinemedicine.anatomical_structureInterleukin 15CytokinesLeukocyte Common AntigensImmunologic MemoryEx vivoEuropean journal of immunology
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HLA-B27-restricted T cells from patients with ankylosing spondylitis recognize peptides from B*2705 that are similar to bacteria-derived peptides

2003

SUMMARY Ankylosing spondylitis (AS) is an inflammatory systemic disease affecting the spine, sacroiliacal and peripheral joints. Although the aetiology of AS remains unknown, the strong association with the HLA-B27 allele might reflect directly a detrimental effect of the HLA-B27 molecule itself, resulting from its potential capability to present ‘arthritogenic’ peptides to CD8+ T cells. Because some forms of SpA are triggered by enterobacterial infection, such arthritogenic peptides might originate from autologous and/or bacterial proteins triggering cross-reactive CD8+ T cell clones. Intriguingly, two peptides from the second extracellular domain of HLA-B*2705 share sequence homologies wi…

musculoskeletal diseasesAdultCytotoxicity ImmunologicMaleT cellReceptors Antigen T-Cell alpha-betaImmunologyComplementarity determining regionCD8-Positive T-LymphocytesAutoantigensEpitopeCell LineEpitopesAntigenClinical StudiesImmunology and AllergyMedicineHumansSpondylitis AnkylosingCells CulturedHLA-B27 AntigenAgedAged 80 and overHLA-B27Antigens Bacterialbusiness.industryTumor Necrosis Factor-alphaELISPOTT lymphocyteMiddle AgedComplementarity Determining Regionsmedicine.anatomical_structureImmunologyFemalebusinessPeptidesCD8
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HLA-B27-restricted cytotoxic T lymphocyte responses to arthritogenic enterobacteria or self-antigens are dominated by closely related TCRBV gene segm…

1996

Identification of the T-cell receptors (TCR) used by synovial cytotoxic T lymphocytes (CTL) of patients with reactive arthritis (ReA) may be crucial to better understanding the pathogenetic mechanism underlying the HLA-B27 association of spondylarthropathies. The authors, therefore, sequenced 25 TCRB chains from HLA-B27-restricted CD8+ CTL clones and two clonal lines specific for self- or Yersinia enterocolitica antigen isolated from synovial fluids of 3 HLA-B27+ patients with ReA and PBL of one healthy HLA-B27+ individual. Fourteen non-HLA-B27-restricted CTL served as controls. Both autoreactive and Y. enterocolitica specific HLA-B27-restricted CTL used a highly limited set of VB genes wit…

musculoskeletal diseasesAdultMaleSalmonella typhimuriumYersinia InfectionsReceptors Antigen T-Cell alpha-betaImmunologyMolecular Sequence Datachemical and pharmacologic phenomenaChlamydia trachomatisBiologyCD8-Positive T-LymphocytesArthritis ReactiveAutoantigensPolymerase Chain ReactionProhibitinsSynovial FluidCytotoxic T cellHumansAmino Acid SequenceGene Rearrangement beta-Chain T-Cell Antigen Receptorskin and connective tissue diseasesReceptorSpondylarthropathiesGeneHLA-B27 AntigenYersinia enterocoliticaHLA-B27Antigens BacterialT-cell receptorhemic and immune systemsGeneral MedicineDNAChlamydia InfectionsCTL*ImmunologySalmonella InfectionsCD8T-Lymphocytes CytotoxicScandinavian journal of immunology
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Detection of natural killer T cells in mice infected with Rickettsia conorii.

2013

Little information is available regarding the role of natural killer T (NKT) cells during the early stage of Rickettsia conorii infection. Herein, C3H/HeN mice were infected with the Malish 7 strain of R. conorii. Splenocytes from these mice were analysed in the early stage of the infection by flow cytometry and compared with uninfected controls. Our results showed an increase in NKT cells in infected mice. Additionally, NKT interleukin (IL)-17(+) cells increased three days after infection, together with a concurrent decrease in the relative amount of NKT interferon (IFN)-γ(+) cells. We also confirmed a higher amount of NK IFN-γ(+) cells in infected mice. Taken together, our data showed tha…

rickettsiosis; interleukin 17; interferon-γchemical and pharmacologic phenomenaSpleenrickettsiosisBiologyBoutonneuse FeverFlow cytometryMiceInterferonmedicineAnimalsCells CulturedImmunity CellularMice Inbred C3HGeneral VeterinaryGeneral Immunology and Microbiologymedicine.diagnostic_testInterleukinGeneral Medicinemedicine.diseaseNatural killer T cellbiology.organism_classificationInterferon-γ; interleukin 17; rickettsiosisVirologyRickettsia conoriiRickettsiosismedicine.anatomical_structureImmunologyNatural Killer T-CellsInterferon-γInterleukin 17Rickettsia conoriiinterleukin 17Spleenmedicine.drugTransboundary and emerging diseases
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Metabolic Changes in Tumor Microenvironment: How Could They Affect γδ T Cells Functions?

2021

The metabolic changes that occur in tumor microenvironment (TME) can influence not only the biological activity of tumor cells, which become more aggressive and auto sustained, but also the immune response against tumor cells, either producing ineffective responses or polarizing the response toward protumor activity. γδ T cells are a subset of T cells characterized by a plasticity that confers them the ability to differentiate towards different cell subsets according to the microenvironment conditions. On this basis, we here review the more recent studies focused on altered tumor metabolism and γδ T cells, considering their already known antitumor role and the possibility of manipulating th…

tumoral metabolismQH301-705.5T-LymphocytesPopulationReviewMajor histocompatibility complexγδ T cellsImmune systemAntigens NeoplasmIn vivomedicineAnimalsHumanstumor microenvironmentBiology (General)educationtumoral metabolism; ?? T cells; tumor microenvironmentClinical Trials as Topiceducation.field_of_studyTumor microenvironmentbiologyReceptors Antigen T-Cell gamma-deltaBiological activityGeneral MedicineHypoxia (medical)Lipid MetabolismIn vitroCell biologybiology.proteinmedicine.symptom
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