Search results for " Tolerance"

showing 10 items of 760 documents

IL-10 signaling prevents gluten-dependent intraepithelial CD4(+) cytotoxic T lymphocyte infiltration and epithelial damage in the small intestine

2019

Breach of tolerance to gluten leads to the chronic small intestinal enteropathy celiac disease. A key event in celiac disease development is gluten-dependent infiltration of activated cytotoxic intraepithelial lymphocytes (IELs), which cytolyze epithelial cells causing crypt hyperplasia and villous atrophy. The mechanisms leading to gluten-dependent small intestinal IEL infiltration and activation remain elusive. We have demonstrated that under homeostatic conditions in mice, gluten drives the differentiation of anti-inflammatory T cells producing large amounts of the immunosuppressive cytokine interleukin-10 (IL-10). Here we addressed whether this dominant IL-10 axis prevents gluten-depend…

0301 basic medicineeducation.field_of_studyChemistryImmunologyPopulationnutritional and metabolic diseasesmedicine.diseasedigestive systemdigestive system diseasesImmune toleranceGranzyme BEpithelial Damage03 medical and health sciences030104 developmental biology0302 clinical medicinemedicineCancer researchImmunology and AllergyIntraepithelial lymphocyteCytotoxic T cellEnteropathyeducationInfiltration (medical)030215 immunologyMucosal Immunology
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Cyclic AMP Represents a Crucial Component of Treg Cell-Mediated Immune Regulation

2016

T regulatory (Treg) cells are one of the key players in the immune tolerance network, and a plethora of manuscripts have described their development and function in the course of the last two decades. Nevertheless, it is still a matter of debate as to which mechanisms and agents are employed by Treg cells, providing the basis of their suppressive potency. One of the important candidates is cyclic AMP (cAMP), which is long known as a potent suppressor at least of T cell activation and function. While this suppressive function by itself is widely accepted, the source and the mechanism of action of cAMP are less clear, and a multitude of seemingly contradictory data allow for, in principle, tw…

0301 basic medicinelcsh:Immunologic diseases. AllergyFOXP3Mini ReviewT cellImmunologyimmune tolerance networkAdenylate kinaseBiologyregulatory T cellsImmune tolerance03 medical and health sciencesmedicineImmunology and Allergycyclic AMPReceptorEffectorimmune regulationFOXP3suppressionAdenosineCell biology030104 developmental biologymedicine.anatomical_structureadenosineImmunologylcsh:RC581-607Intracellularmedicine.drugFrontiers in Immunology
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Enhanced protection of C57 BL/6 vs Balb/c mice to melanoma liver metastasis is mediated by NK cells.

2017

ABSTRACT The B16F10 murine melanoma cell line displays a low expression of MHC class I molecules favoring immune evasion and metastases in immunocompetent C57 BL/6 wild-type mice. Here, we generated metastases to the liver, an organ that is skewed towards immune tolerance, by intrasplenic injection of B16F10 cells in syngeneic C57 BL/6 compared to allogeneic Balb/c mice. Surprisingly, Balb/c mice, which usually display a pronounced M2 macrophage and Th2 T cell polarization, were ∼3 times more susceptible to metastasis than C57 BL/6 mice, despite a much higher M1 and Th1 T cell immune response. The anti-metastatic advantage of C57 BL/6 mice could be attributed to a more potent NK-cell mediat…

0301 basic medicinelcsh:Immunologic diseases. AllergyImmunologyNK cellsMajor histocompatibility complexcancer immunologyliverlcsh:RC254-282BALB/cImmune toleranceMetastasis03 medical and health sciencesImmune systemMHC class ImedicineImmunology and Allergymetastasisinnate immunityOriginal ResearchInnate immune systembiologybiology.organism_classificationmedicine.diseaselcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmacrophages030104 developmental biologyOncologyCancer cellCancer researchbiology.proteinlcsh:RC581-607Oncoimmunology
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Peripherally Induced Regulatory T Cells: Recruited Protectors of the Central Nervous System against Autoimmune Neuroinflammation

2017

Defects in regulatory T cells (Treg cells) aggravate multiple sclerosis (MS) after its onset and the absence of Treg cell functions can also exacerbate the course of disease in an animal model of MS. However, autoimmune neuroinflammation in many MS models can be acutely provoked in healthy animals leading to an activation of encephalitogenic T cells despite the normal induction of immune tolerance in the thymus including thymically-produced (t)Treg cells. In contrast, neuroinflammation can be ameliorated or even completely prevented by the antigen-specific Treg cells formed extrathymically in the peripheral immune system (pTreg cells) during tolerogenic responses to relevant neuronal antige…

0301 basic medicinelcsh:Immunologic diseases. AllergyMini ReviewImmunologychemical and pharmacologic phenomenaBiologyImmune toleranceneuroinflammation03 medical and health sciences0302 clinical medicineAntigenmedicineImmunology and AllergyIL-2 receptordendritic cellsNeuroinflammationtoleranceexperimental autoimmune encephalomyelitis/multiple sclerosisMultiple sclerosisPeripheral toleranceHOPXmedicine.diseaseCD5Tolerance induction030104 developmental biologypTreg cellsImmunologyCD5lcsh:RC581-607Treg cells030215 immunologyFrontiers in Immunology
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Editorial: Current concepts of cellular and biological drugs to modulate regulatory T cell activity in the clinic

2016

The Editorial on the Research Topic Current Concepts of Cellular and Biological Drugs to Modulate Regulatory T Cell Activity in the Clinic Regulatory T (Treg) cells are essential for the maintenance of peripheral tolerance and prevent the development of autoimmunity and allergy. While on the one hand being indispensable for the perpetuation of tolerance to harmless antigens or self-antigens, Treg cells contribute to cancer pathogenesis and progression (1). Hence, the potential to treat a multitude of different human diseases by pharmacological modulation of Treg cells is enormous. Consequently, this T cell population is in the focus of biomedical research and development. Currently, isolate…

0301 basic medicinelcsh:Immunologic diseases. AllergyRegulatory T cellT cellImmunologyPopulationAutoimmunitymedicine.disease_causeregulatory T cellsAutoimmunityAutoimmune Diseases03 medical and health sciencesmedicineImmunology and Allergyclinical studieseducationeducation.field_of_studybusiness.industryPeripheral toleranceImmunoregulationT helper cellDendritic CellsTolerance inductionEditorial030104 developmental biologymedicine.anatomical_structureHumanized mouseImmunologybusinesslcsh:RC581-607Frontiers in Immunology
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Novel anti-GARP antibody DS-1055a augments anti-tumor immunity by depleting highly suppressive GARP+ regulatory T cells

2021

Abstract Regulatory T (Treg) cells, which are essential for maintaining self-tolerance, inhibit anti-tumor immunity, consequently hindering protective cancer immunosurveillance, and hampering effective anti-tumor immune responses in tumor-bearing hosts. Here, we show that depletion of Treg cells via targeting glycoprotein A repetitions predominant (GARP) induces effective anti-tumor immune responses. GARP was specifically expressed by highly suppressive Treg cells in the tumor microenvironment (TME) of multiple cancer types in humans. In the periphery, GARP was selectively induced in Treg cells, but not in effector T cells, by polyclonal stimulation. DS-1055a, a novel afucosylated anti-huma…

0301 basic medicinemedicine.drug_classmedicine.medical_treatmentImmunologychemical and pharmacologic phenomenaMice SCIDBiologyMonoclonal antibodyT-Lymphocytes RegulatoryMice03 medical and health sciences0302 clinical medicineImmune systemCancer immunotherapyMice Inbred NODImmunityNeoplasmsImmune ToleranceTumor MicroenvironmentmedicineAnimalsHumansImmunology and AllergyMice KnockoutTumor microenvironmentImmunityAntibodies MonoclonalMembrane ProteinsFOXP3General MedicineImmunosurveillance030104 developmental biology030220 oncology & carcinogenesisLeukocytes MononuclearCancer researchbiology.proteinFemaleImmunotherapyAntibodyInternational Immunology
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Glycometabolic Alterations in Secondary Adrenal Insufficiency: Does Replacement Therapy Play a Role?

2018

Secondary adrenal insufficiency (SAI) is a potentially life-threatening endocrine disorder due to an impairment of corticotropin (ACTH) secretion from any process affecting the hypothalamus or pituitary gland. ACTH deficit can be isolated or associated with other pituitary failures (hypopituitarism). An increased mortality due to cardiovascular, metabolic, and infectious diseases has been described in both primary and secondary adrenal insufficiency. However, few studies have provided compelling evidences on the underlying mechanism in SAI, because of the heterogeneity of the condition. Recently, some studies suggested that inappropriate glucocorticoid (GCs) replacement therapy, as for dose…

0301 basic medicinemedicine.medical_specialtyEndocrinology Diabetes and MetabolismMini Reviewbody mass index; diabetes mellitus; glucocorticoids; impaired glucose tolerance; insulin resistance; metabolic syndrome; secondary adrenal insufficiency; weight gain030209 endocrinology & metabolismbody mass indexHypopituitarismlcsh:Diseases of the endocrine glands. Clinical endocrinologymetabolic syndromeGrowth hormone deficiencyImpaired glucose tolerance03 medical and health sciences0302 clinical medicineInsulin resistanceEndocrinologyInternal medicineDiabetes mellitusinsulin resistanceMedicineEndocrine systemlcsh:RC648-665glucocorticoidsbusiness.industryweight gainmedicine.disease030104 developmental biologyEndocrinologyimpaired glucose tolerancediabetes mellitussecondary adrenal insufficiencyMetabolic syndromebusinessGlucocorticoidmedicine.drug
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Decreases in Circulating Concentrations of Long-Chain Acylcarnitines and Free Fatty Acids During the Glucose Tolerance Test Represent Tissue-Specific…

2019

Background: Insulin plays a pivotal role in the regulation of both carbohydrate and lipid intermediate turnover and metabolism. In the transition from a fasted to fed state, insulin action inhibits lipolysis in adipocytes, and acylcarnitine synthesis in the muscles and heart. The aim of this study was to measure free fatty acid (FFA) and acylcarnitine levels during the glucose tolerance test as indicators of tissue-specific insulin resistance. Results: Insulin release in response to glucose administration decreased both FFA and long-chain acylcarnitine levels in plasma in healthy control animals by 30% (120 min). The glucose tolerance test and [3H]-deoxy-D-glucose uptake in tissues revealed…

0301 basic medicinemedicine.medical_specialtyGlucose uptakemedicine.medical_treatmentEndocrinology Diabetes and MetabolismAdipose tissuelong-chain acylcarnitines030209 endocrinology & metabolismType 2 diabetesglucose tolerance testlcsh:Diseases of the endocrine glands. Clinical endocrinology03 medical and health sciences0302 clinical medicineInsulin resistanceEndocrinologyInternal medicineinsulin resistancemedicineLipolysisOriginal ResearchGlucose tolerance testlcsh:RC648-665medicine.diagnostic_testChemistryInsulinfree fatty acidsmedicine.disease030104 developmental biologyEndocrinologyPostprandialtype 2 diabetesFrontiers in Endocrinology
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Does high sugar consumption exacerbate cardiometabolic risk factors and increase the risk of type 2 diabetes and cardiovascular disease?

2012

Consumption of sugar has been relatively high in the Nordic countries; the impact of sugar intake onmetabolic risk factors and related diseases has been debated. The objectives were to assess the effect of sugarintake (sugar-sweetened beverages, sucrose and fructose) on association with type 2 diabetes, cardiovasculardisease and related metabolic risk factors (impaired glucose tolerance, insulin sensitivity, dyslipidemia, bloodpressure, uric acid, inflammation markers), and on all-cause mortality, through a systematic review ofprospective cohort studies and randomised controlled intervention studies published between January 2000and search dates. The methods adopted were as follows: the fir…

0301 basic medicinemedicine.medical_specialtylcsh:TX341-641030209 endocrinology & metabolismType 2 diabetesDiseaseReview ArticleVDP::Medical disciplines: 700::Health sciences: 800::Nutrition: 811fructoseImpaired glucose tolerance03 medical and health scienceschemistry.chemical_compound0302 clinical medicineNordic nutrition recommendationssystematic reviewInternal medicinemedicineProspective cohort study030109 nutrition & dieteticsNutrition and Dieteticsbusiness.industryPublic Health Environmental and Occupational Healthmedicine.diseaseBlood pressurechemistrysugarUric acidbusinesssugar-sweetened beverageslcsh:Nutrition. Foods and food supplyDyslipidemiaFood ScienceCohort studyFoodnutrition research
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Molecular Pathways Mediating Immunosuppression in Response to Prolonged Intensive Physical Training, Low-Energy Availability, and Intensive Weight Lo…

2019

Exercise and exercise-induced weight loss have a beneficial effect on overall health, including positive effects on molecular pathways associated with immune function, especially in overweight individuals. The main aim of our study was to assess how energy deprivation (i.e., "semi-starvation") leading to substantial fat mass loss affects the immune system and immunosuppression in previously normal weight individuals. Thus, to address this hypothesis, we applied a high-throughput systems biology approach to better characterize potential key pathways associated with immune system modulation during intensive weight loss and subsequent weight regain. We examined 42 healthy female physique athle…

0301 basic medicinemedicine.medical_treatmentPhysiologyliikuntaOverweightSystemic inflammationLeukocyte Countphysical training0302 clinical medicineWeight lossLeukocytesImmunology and AllergyMedicineOXIDATIVE STRESSta315DIETARY RESTRICTIONSport and Fitness SciencesOriginal Research2. Zero hungerimmunosuppressionIdrottsvetenskapbioinformatiikkaImmunosuppressionbioinformaticslow energy availability3. Good healthimmuunivasteIMMUNE FUNCTIONOBESITYChemokine secretionFemalemedicine.symptomfyysinen aktiivisuusAdultlcsh:Immunologic diseases. AllergyImmunologyEXERCISEInflammationYoung Adult03 medical and health sciencesLEPTINImmune systemINFLAMMATIONImmune ToleranceHumansimmunosuppression ; low energy availability ; physical training ; bioinformatics ; weight lossCell Proliferationbusiness.industrylaihdutusCYTOKINESmedicine.diseaseObesityDietenergiansaanti030104 developmental biologyHEMATOPOIETIC STEMImmunoglobulin G3121 General medicine internal medicine and other clinical medicineCELLS3111 Biomedicineweight lossEnergy IntakeTranscriptomelcsh:RC581-607business030215 immunologyFrontiers in Immunology
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