Search results for " Toxicity"

showing 10 items of 346 documents

Synthesis and pharmacological evaluation of 1-methyl-5- [substituted-4(3H)-oxo-1,2,3-benzotriazin-3-yl]-1H-pyrazole-4-acetic acid derivatives

1998

Several new 1-methyl-5-[substituted-4-oxo-1,2,3-benzotriazin-3-yl] -1H-pyrazole-4-acetic acids and their ethyl ester derivatives were prepared. The compounds were tested for analgesic and antiinflammatory activities, acute toxicity, ulcerogenic effect, and as in vitro inhibitors of 3 alpha-hydroxysteroid dehydrogenase (3 alpha-HSD), since it is claimed that the inhibition of such an enzyme predicts in vivo antiinflammatory activity. Some compounds were more active than phenylbutazone in the phenylbenzoquinone and acetic acid peritonitis tests, and equiactive to the same drug in the carrageenin paw edema test. All the compounds inhibited the 3 alpha-HSD, but no correlation was observed with …

Male3-Hydroxysteroid DehydrogenasesStereochemistryAnti-Inflammatory AgentsPharmaceutical SciencePyrazoleChemical synthesisMicechemistry.chemical_compoundAcetic acidIn vivoDrug DiscoveryPhenylbutazonemedicineAnimalsEnzyme InhibitorsAnalgesicsbiology3-alpha-Hydroxysteroid Dehydrogenase (B-Specific)Acute toxicityRatschemistryEnzyme inhibitorToxicitybiology.proteinPyrazolesmedicine.drugIl Farmaco
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Effect of gender on mitochondrial toxicity of Alzheimer's Abeta peptide.

2007

The aim of this article is to review the role of mitochondria in the pathogenesis of Alzheimer's disease. Additionally, the effect of gender on the incidence of Alzheimer's disease and the pathophysiological mechanisms involved will be discussed. Mitochondria, in the presence of Alzheimer's amyloid-beta peptide, increase the formation of reactive oxygen species which act both as damaging agents and also as signaling molecules. These radicals, in fact, unleash a mechanism involving the liberation of cytochrome c that leads to neuronal apoptosis. Notably, young females appear protected against the mitochondrial toxicity of amyloid-beta, likely due to the upregulation of antioxidant enzymes wh…

MaleAntioxidantPhysiologymedicine.medical_treatmentClinical BiochemistryPharmacologyMitochondrionBiologymedicine.disease_causeBiochemistryp38 Mitogen-Activated Protein Kinaseschemistry.chemical_compoundDownregulation and upregulationAlzheimer DiseasemedicineHumansMolecular BiologyGeneral Environmental Sciencechemistry.chemical_classificationReactive oxygen speciesAmyloid beta-PeptidesEstrogensCell Biologymedicine.diseaseOxidantsMitochondriaEnzyme ActivationMitochondrial toxicitychemistryBiochemistryToxicityGeneral Earth and Planetary SciencesPhytoestrogensFemaleOxidative stressAntioxidantsredox signaling
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Biocompatibility of a chlorhexidine local delivery system in a subcutaneous mouse model

2011

Made available in DSpace on 2013-08-12T19:11:10Z (GMT). No. of bitstreams: 0 Previous issue date: 2011-03-01 Made available in DSpace on 2013-09-30T18:34:50Z (GMT). No. of bitstreams: 0 Previous issue date: 2011-03-01 Submitted by Vitor Silverio Rodrigues (vitorsrodrigues@reitoria.unesp.br) on 2014-05-20T14:04:50Z No. of bitstreams: 0 Made available in DSpace on 2014-05-20T14:04:50Z (GMT). No. of bitstreams: 0 Previous issue date: 2011-03-01 Objective: This study aimed evaluating histologically and histomorphometrically the response of the conjunctive tissue face to the implant of chlorhexidine chips in the subcutaneous tissues of rats. Study Design: In this research 35 male rats Wistar wer…

MaleBiocompatibilityDentistryBiocompatible Materialschlorhexidine toxicityMiceDrug Delivery SystemsSubcutaneous Tissueparasitic diseasesmedicinedrug delivery systemAnimalsRats WistarperiodontitisGeneral DentistryPeriodontitisbusiness.industryChlorhexidineChlorhexidine:CIENCIAS MÉDICAS [UNESCO]medicine.diseaseBiocompatible materialRatsOtorhinolaryngologyModels AnimalUNESCO::CIENCIAS MÉDICASAnti-Infective Agents LocalBiocompatibilitySurgeryDelivery systembusinessmedicine.drug
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Early Skin Toxicity as a Predictive Factor for Tumor Control in Hepatocellular Carcinoma Patients Treated with Sorafenib.

2010

Abstract Introduction. Sorafenib is an oral multikinase inhibitor that targets Raf kinase and receptor tyrosine kinases and has led to a longer median overall survival (OS) time and time to progression (TTP) in patients with advanced hepatocellular carcinoma (HCC). This study was conducted to assess the link between the antitumor efficacy of sorafenib and its early cutaneous side effects in advanced HCC patients. Materials and Methods. All patients received 800 mg daily of sorafenib until progression or unacceptable toxicities. We retrospectively analyzed the incidence of rash and hand–foot skin reactions (HFSR) during the first month of treatment, comparing tumor control (partial response …

MaleCancer ResearchPyridinesSettore MED/06 - Oncologia MedicaKaplan-Meier EstimateGastroenterologySkin Toxicity Hepatocellular CarcinomaSorafenib.Aged 80 and overintegumentary systemIncidence (epidemiology)BenzenesulfonatesLiver NeoplasmsMiddle AgedSorafenibRashhumanitiesOncologyHepatocellular carcinomaToxicityDisease ProgressionFemaleDrug Eruptionsmedicine.symptommedicine.drugAdultNiacinamideSorafenibmedicine.medical_specialtyCarcinoma HepatocellularAntineoplastic AgentsInternal medicinemedicineCarcinomaHumansneoplasmsSurvival analysisAgedRetrospective StudiesSurrogate endpointbusiness.industryPhenylurea CompoundsExanthemamedicine.diseaseSurvival Analysisdigestive system diseasesSurgerybody regionsMultivariate AnalysisHepatobiliarybusiness
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Optimal local control and tolerability of three-dimensional conformal radiation therapy in prostate cancer: A single institutional experience of dose…

2013

Aims To evaluate long-term late side effects, clinical and biochemical relapse in non-metastatic prostate cancer patients treated with dose escalation, from 74 to 78 Gy, by means of three dimensional conformal radiation therapy. Materials and Methods Clinical data of 125 patients with prostate cancer who underwent three-dimensional conformal radiation therapy were retrospectively evaluated. All patients were stratified, according to the NCCN classification, in low, intermediate and high risk, and all of them showed histologically proven adenocarcinoma stage T1–T3 with at least 2 years of follow-up. Late toxicity was analyzed using a modified Radiation Therapy Oncology Group toxicity scale. …

MaleCancer ResearchThree dimensional conformal radiation therapyUrinary BladderUrogenital SystemKaplan-Meier EstimateAdenocarcinomaDisease-Free SurvivalBiomarkers Tumor80 and overHumansLate toxicity; Local control; Prostate cancer; Radiation therapy; Three dimensional conformal radiation therapy; Adenocarcinoma; Aged; Aged 80 and over; Biomarkers Tumor; Disease-Free Survival; Gastrointestinal Tract; Humans; Italy; Kaplan-Meier Estimate; Male; Middle Aged; Neoplasm Staging; Prostate-Specific Antigen; Prostatic Neoplasms; Radiation Injuries; Radiotherapy Dosage; Radiotherapy Conformal; Rectum; Retrospective Studies; Urinary Bladder; Urogenital System; Medicine (all); Oncology; Cancer ResearchRadiation InjuriesAgedNeoplasm StagingRetrospective StudiesAged 80 and overProstate cancerTumorRadiotherapyConformalMedicine (all)RectumProstatic NeoplasmsRadiotherapy DosageGeneral MedicineMiddle AgedProstate-Specific AntigenRadiation therapyGastrointestinal TractItalyOncologyLocal controlRadiotherapy ConformalLate toxicityBiomarkers
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Acute ammonia neurotoxicity in vivo involves increase in cytoplasmic protein P53 without alterations in other markers of apoptosis.

2007

Acute intoxication with large ammonia doses leads to activation of NMDA receptors in the brain, resulting in oxidative stress and disturbance of mitochondrial function. Altered mitochondrial function is a crucial step in some mechanisms of cellular apoptosis. This study assesses whether ammonia intoxication in vivo leads to induction of apoptotic markers such as permeability transition pore (PTP) formation, caspase-3, and caspase-9 activation, changes in p53 protein, or cytochrome c release. Acute ammonia intoxication did not affect caspase-9 or caspase-3 activities. The mitochondrial membrane potential also remained unaltered in non-synaptic brain mitochondria after injection of ammonia, i…

MaleCytoplasmApoptosisMitochondrionmedicine.disease_causeCellular and Molecular NeuroscienceIn vivoAmmoniamedicineAnimalsRats WistarbiologyCaspase 3brain mitochondriaCytochrome capoptosisNeurotoxicityBrainCytochromes cammonia toxicitybrain nucleimedicine.diseaseCaspase 9Cell biologyMitochondriaRatsEnzyme ActivationCytosolcytochrome cCytoplasmApoptosisbiology.proteinTumor Suppressor Protein p53Oxidative stressJournal of neuroscience research
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N-Valproyl-L-Phenylalanine as new potential antiepileptic drug: Synthesis, characterization and in vitro studies on stability, toxicity and anticonvu…

2013

Valproic acid (VPA) is considered first-line drug in treatment of generalized idiopathic seizures such as absence, generalized tonic-clonic and myoclonic seizures. Among major antiepileptic drugs, VPA is also considered effective in childhood epilepsies and infantile spasms. Due to its broad activity, VPA acts as a mood stabilizer in bipolar disorder and it is useful in migraine prophylaxis. Despite its long-standing usage, severe reactions to VPA, such as liver toxicity and teratogenicity, are reported. To circumvent side effects due to structural characteristics of VPA, we synthesized in good yield a new VPA-aminoacid conjugate, the N-valproyl-L-Phenylalanine, and characterized by FT-IR, …

MaleDrugCell Membrane PermeabilityAminoacidic derivative Astrocytes toxicity CNS-Targeting Enzymatic Stability Hippocampal epilepsy Valproic acid.Cell Survivalmedicine.drug_classPhenylalaninemedicine.medical_treatmentmedia_common.quotation_subjectPrimary Cell CulturePhenylalaninePharmacologySettore BIO/09 - FisiologiaHippocampusTissue Culture TechniquesDrug StabilityDrug DiscoverymedicineAnimalsRats WistarEvoked Potentialsmedia_commonValproic AcidChemistryHydrolysisValproic AcidBiological TransportMood stabilizerMicrotomyHydrogen-Ion ConcentrationIn vitroRatsAnticonvulsantSettore CHIM/09 - Farmaceutico Tecnologico ApplicativoAstrocytesToxicityAnticonvulsantslipids (amino acids peptides and proteins)Conjugatemedicine.drug
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Developmental and Reproductive Effects of Low Cadmium Concentration onArtemiaparthenogenetica

2003

Hatching, reproductive, and lifespan characteristics of an Artemia parthenogenetica population from La Mata (Alicante, Spain) exposed to cadmium were studied. No effects on percentage of cyst hatched nor time of hatching were observed on cysts exposed from 0.01 to 5 mg Cd/L. Cohorts of Artemia were chronically exposed to cadmium in life-table experiments to test whether ecologically relevant impacts may occur after several generations of exposure to a very low concentration of cadmium. Chronic toxicity tests were performed using neonates of a third generation of shrimps exposed to 0.08 mg Cd/L (1/1000 of the LOEC for 24 h acute toxicity test). No significant reduction in the intrinsic rate …

MaleEnvironmental EngineeringPopulation DynamicsPopulationArtemia parthenogeneticachemistry.chemical_elementBrine shrimpBiologyToxicologyAnimal scienceAnimalsWater PollutantseducationChronic toxicityCadmiumeducation.field_of_studyHatchingEnvironmental ExposureGeneral Medicinebiology.organism_classificationFecundityAcute toxicityFertilitychemistryFemaleArtemiaCadmiumJournal of Environmental Science and Health, Part A
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Cross-species transcriptomic analysis elucidates constitutive aryl hydrocarbon receptor activity

2014

Background Research on the aryl hydrocarbon receptor (AHR) has largely focused on variations in toxic outcomes resulting from its activation by halogenated aromatic hydrocarbons. But the AHR also plays key roles in regulating pathways critical for development, and after decades of research the mechanisms underlying physiological regulation by the AHR remain poorly characterized. Previous studies identified several core genes that respond to xenobiotic AHR ligands across a broad range of species and tissues. However, only limited inferences have been made regarding its role in regulating constitutive gene activity, i.e. in the absence of exogenous ligands. To address this, we profiled transc…

MaleHEPATIC GENE-EXPRESSION413 Veterinary scienceMedical and Health SciencesTranscriptomeDIOXIN RECEPTORMice0302 clinical medicineTCDD-induced toxicityReceptorsTranscriptional regulationABNORMAL LIVER DEVELOPMENT2.1 Biological and endogenous factorsCluster AnalysisAetiologyReceptorAH RECEPTORIN-VIVOAryl hydrocarbon receptorGeneticsRegulation of gene expression0303 health sciencesBiological Sciencesrespiratory systemCore-gene batteryAryl HydrocarbonOrgan Specificity030220 oncology & carcinogenesisAHR endogenous ligands2378-TETRACHLORODIBENZO-P-DIOXIN TCDDSignal transductionResearch ArticleBiotechnologySignal TransductionProtein BindingBioinformatics1.1 Normal biological development and functioningeducationRAT-LIVERConstitutive gene expressionBiologyMICE LACKING03 medical and health sciencesSpecies SpecificityUnderpinning researchInformation and Computing SciencesGeneticsAnimals030304 developmental biologyAryl hydrocarbon receptor activityGene Expression ProfilingComputational BiologyAryl hydrocarbon receptorCELL-CYCLE CONTROLRatsrespiratory tract diseasesGene expression profilingReceptors Aryl HydrocarbonGene Expression RegulationSUBCHRONIC EXPOSUREbiology.proteinDigestive DiseasesTranscriptome
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Impact of rectal distension on prostate CBCT-based positioning assessed with 6 degrees-of-freedom couch

2018

International audience; Introduction: Prostate requires a daily correction of its 3-dimensional position in relation with rectal distension. In this study, we sought to determine whether rectal distension with respect to the rectal behavior might have an impact on prostate translations and/or rotations during prostate image guided radiation therapy using a 6 degrees-of-freedom (DOF) couch.Methods and materials: We reviewed the data from 39 patients with localized prostate cancer treated with protracted external radiation therapy using a 6 DOF couch. Before each fraction, a kilovoltage cone beam computed tomography (kV-CBCT) scan was performed. The automatic fusion algorithm was set to fuse …

MaleKilovoltage Cone Beam Computed Tomography[SDV.IB.IMA]Life Sciences [q-bio]/Bioengineering/Imagingmedicine.medical_treatment78 GyFailureRectum[SDV.CAN]Life Sciences [q-bio]/Cancer[SDV.IB.MN]Life Sciences [q-bio]/Bioengineering/Nuclear medicineIncreased RiskStandard deviationPatient PositioningTrial030218 nuclear medicine & medical imaging03 medical and health sciencesProstate cancer0302 clinical medicineProstatemedicineHumansRadiology Nuclear Medicine and imagingConformal Radiation-TherapyImage-guided radiation therapyCancerContouringbusiness.industryImage-GuidanceRadiotherapy Planning Computer-AssistedAcute ToxicityProstateRectumProstatic NeoplasmsRadiotherapy DosageExternal-Beam RadiotherapyCone-Beam Computed Tomographymedicine.diseaseRadiation therapymedicine.anatomical_structureOncology030220 oncology & carcinogenesisFeasibility StudiesRadiotherapy Intensity-ModulatedStrategiesbusinessNuclear medicineTomography X-Ray ComputedRadiotherapy Image-Guided
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