Search results for " Transition"

showing 10 items of 2751 documents

Assessing the carcinogenic potential of low-dose exposures to chemical mixtures in the environment: the challenge ahead

2015

Goodson, William H. et al.

Cancer ResearchCarcinogenesis[SDV]Life Sciences [q-bio]METHOXYCHLOR-INDUCED ALTERATIONSReviewPharmacologyMESH: Carcinogens EnvironmentalCarcinogenic synergiesChemical mixturesNeoplasmsMESH: AnimalsMESH: NeoplasmsCarcinogenesiRisk assessmentCancerACTIVATED PROTEIN-KINASESMedicine (all)Low dose1. No povertyCumulative effectsBREAST-CANCER CELLSGeneral MedicineEnvironmental exposureMESH: CarcinogenesisBIO/10 - BIOCHIMICAEPITHELIAL-MESENCHYMAL TRANSITION3. Good health[SDV] Life Sciences [q-bio]Environmental CarcinogenesisESTROGEN-RECEPTOR-ALPHARisk assessmentHumanMESH: Environmental ExposureENDOCRINE-DISRUPTING CHEMICALSTARGETING TISSUE FACTOR[SDV.CAN]Life Sciences [q-bio]/CancerBiologyPrototypical chemical disruptorsExposure[SDV.CAN] Life Sciences [q-bio]/CancerEnvironmental healthmedicine[SDV.EE.SANT] Life Sciences [q-bio]/Ecology environment/HealthCarcinogenEnvironmental carcinogenesis[SDV.EE.SANT]Life Sciences [q-bio]/Ecology environment/HealthMESH: HumansAnimalPOLYBROMINATED DIPHENYL ETHERSCancerEnvironmental Exposuremedicine.diseaseMESH: Hazardous SubstancesCarcinogens EnvironmentalMIGRATION INHIBITORY FACTORVASCULAR ENDOTHELIAL-CELLSHazardous SubstanceNeoplasmCarcinogenesis
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Abstract LB-399: Chronic inhibition of mutant EGFR in NSCLC leads to EGFR TKI resistance by TGF-β1 mediated epithelial to mesenchymal transition

2011

Abstract In NSCLC, activating EGFR mutations underlie responsiveness of NSCLCs to reversible EGFR tyrosine kinase inhibitors (TKIs), including gefitinib and erlotinib. Despite initial responses, acquired resistance invariably develops, mediated by the emergence of the secondary T790M mutation and by focal amplification of MET, in approximately 50% and 30% of patients, respectively. The resistance mechanisms for the remaining 20% of cases remain elusive. EGFR TKI-sensitive HCC827 cells were exposed to graded concentrations of erlotinib for 6 months. Approximately 70% of the isolated clones were resistant to erlotinib and harbored MET amplification, and were sensitive to dual EGFR/MET inhibit…

Cancer ResearchGene knockdownGrowth factormedicine.medical_treatmentBiologyPhenotyperespiratory tract diseasesT790MGefitinibOncologyImmunologymedicineCancer researchERBB3ErlotinibEpithelial–mesenchymal transitionmedicine.drugCancer Research
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A quest for initiating cells of head and neck cancer and their treatment.

2010

The biology of head and neck squamous cell carcinomas (HNSCC) and other cancers have been related to cancer stem-like cells (CSC). Specific markers, which vary considerably depending on tumor type or tissue of origin, characterize CSC. CSC are cancer initiating, sustaining and mostly quiescent. Compared to bulk tumors, CSC are less sensitive to chemo- and radiotherapy and may have low immunogenicity. Therapeutic targeting of CSC may improve clinical outcome. HNSCC has two main etiologies: human papillomavirus, a virus infecting epithelial stem cells, and tobacco and alcohol abuse. Here, current knowledge of HNSCC-CSC biology is reviewed and parallels to CSC of other origin are drawn where n…

Cancer ResearchPathologymedicine.medical_specialtymedicine.medical_treatmentepithelial mesenchymal transitionSox2Reviewlcsh:RC254-282NanogMetastasisstemnessSOX2RadioresistancemedicinemetastasisEpithelial–mesenchymal transitionALDH1human papillomavirusbusiness.industryHead and neck cancerCancerchemoresistancelcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseaseOct3/4Radiation therapyradioresistancestomatognathic diseasesOncologyCancer researchimmunotherapyStem cellbusinessCancers
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Tumor and its microenvironment: a synergistic interplay.

2013

The mutual and interdependent interaction between tumor and its microenvironment is a crucial topic in cancer research. Recently, it was reported that targeting stromal events could improve efficacies of current therapeutics and prevent metastatic spreading. Tumor microenvironment is a "complex network" of different cell types, soluble factors, signaling molecules and extracellular matrix components, which orchestrate the fate of tumor progression. As by definition, cancer stem cells (CSCs) are proposed to be the unique cell type able to maintain tumor mass and survive outside the primary tumor at metastatic sites. Being exposed to environmental stressors, including reactive oxygen species …

Cancer ResearchStromal cellEpithelial-Mesenchymal TransitionAngiogenesisCell SurvivalBiologyCancer stem cellCell MovementNeoplasmsmedicineTumor MicroenvironmentAnimalsHumansEpithelial–mesenchymal transitionNeoplasm MetastasisStem Cell NicheHypoxiaTumor microenvironmentNeovascularization Pathologicmedicine.diseaseAngiogenesis CAFs CAMs CRC CSCs ECM EMT GSH HIF Hypoxia MMPs ROS Tumor microenvironment VEGF cancer stem cells cancer-associated fibroblasts cancer-associated macrophages colorectal cancer epithelial mesenchymal transition extracellular matrix hypoxia-inducible factor matrix metalloproteinase reactive oxygen species reduced glutathione vascular endothelial growth factorPrimary tumorTumor progressionImmunologyCancer researchNeoplastic Stem CellsCancer-Associated FibroblastsOxidation-ReductionSignal Transduction
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Intratumoral Heterogeneity in EGFR-Mutant NSCLC Results in Divergent Resistance Mechanisms in Response to EGFR Tyrosine Kinase Inhibition

2015

Abstract Non–small cell lung cancers (NSCLC) that have developed resistance to EGF receptor (EGFR) tyrosine kinase inhibitor (TKI), including gefitinib and erlotinib, are clinically linked to an epithelial-to-mesenchymal transition (EMT) phenotype. Here, we examined whether modulating EMT maintains the responsiveness of EGFR-mutated NSCLCs to EGFR TKI therapy. Using human NSCLC cell lines harboring mutated EGFR and a transgenic mouse model of lung cancer driven by mutant EGFR (EGFR-Del19-T790M), we demonstrate that EGFR inhibition induces TGFβ secretion followed by SMAD pathway activation, an event that promotes EMT. Chronic exposure of EGFR-mutated NSCLC cells to TGFβ was sufficient to ind…

Cancer Researchmedicine.drug_classCellBiologymedicine.diseaseArticleTyrosine-kinase inhibitorrespiratory tract diseasesmedicine.anatomical_structureGefitinibOncologyProtein kinase domainImmunologymedicineCancer researchEpithelial–mesenchymal transitionErlotinibSignal transductionLung cancerneoplasmsmedicine.drugCancer Research
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Intravesical mitoxantrone in superficial bladder tumours (Ta-T1)

1993

Abstract 36 patients with histologically proven grade G1–G2, Ta-T1 transitional cell carcinoma of the bladder were introduced, after transurethral resection (TUR), into a study of intravesical chemoprophylaxis with mitoxantrone (20 mg diluted in 50 ml). After a mean follow-up of 23 months, 16 (50%) patients showed a superficial recurrence with a mean recurrence rate of 0.56 per year. In 19 patients with recurring tumours the mean recurrence rate decreased from 1.65 to 0.58 per year. 9 patients (25.7%) suffered from a chemical cystitis that in 2 cases (5.7%) required treatment interruption.

Cancer Researchmedicine.medical_specialtymedicine.medical_treatmentUrologyResectionCystitismedicineHumansCarcinoma Transitional CellMitoxantroneChemotherapyUrinary bladderbusiness.industrymedicine.diseaseSurgeryChemical cystitisAdministration IntravesicalTransitional cell carcinomamedicine.anatomical_structureUrinary Bladder NeoplasmsOncologyTreatment interruptionChemoprophylaxisMitoxantroneNeoplasm Recurrence Localbusinessmedicine.drugEuropean Journal of Cancer
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Canonical versus microcanonical analysis of first-order phase transitions

1998

Abstract I discuss the relation between canonical and microcanonical analyses of first-order phase transitions. In particular it is shown that the microcanonical Maxwell construction is equivalent to the equal-peak-height criterion often employed in canonical simulations. As a consequence the microcanonical finite-size estimators for the transition point, latent heat and interface tension are identical to standard estimators in the canonical ensemble. Special emphasis is placed on various ways for estimating interface tensions. The theoretical considerations are illustrated with numerical data for the two-dimensional 10-state Potts model.

Canonical ensembleNuclear and High Energy PhysicsPhase transitionMicrocanonical ensembleTransition pointCritical phenomenaMaxwell constructionEstimatorStatistical physicsAtomic and Molecular Physics and OpticsPotts modelMathematicsNuclear Physics B - Proceedings Supplements
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Quantum Effects and Phase Transitions in Adsorbed Molecular Layers

1998

Phase transitions in adsorbed (two dimensional) fluids and in adsorbed layers of molecules are studied with a combination of path integral Monte Carlo (PIMC), Gibbs ensemble Monte Carlo (GEMC) and finite size scaling techniques. Entropy driven phase transitions in systems with purely repulsive interactions are analyzed as well phase diagrams of fluids with internal quantum states. Adsorbed layers of H 2 molecules at a full monolayer coverage in the \(\sqrt 3 \times \sqrt 3 \) structure have a higher transition temperature to the disordered phase compared to the system with the heavier D 2 molecules, this effect is analyzed by PIMC. Linear N 2 molecules adsorbed on graphite show a transition…

Canonical ensemblePhase transitionMaterials scienceMean field theoryTricritical pointQuantum mechanicsPhase (matter)MonolayerPhysics::Chemical PhysicsPath integral Monte CarloPhase diagram
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Monte Carlo Simulation of Alloy Phase Diagrams and Short-Range Order

1986

As a prototype model for order-disorder phenomena in binary alloys, a face-centered cubic lattice is considered,the sites of which can be taken by either A-atoms or B-atoms, assuming pair-wise interactions between nearest (J) and next nearest neighbours (J). The phase diagram is constructed from Monte Carlo calculations. Some technical aspects essential for the success of such calculations are briefly mentioned (use of grand-canonical rather than canonical ensemble, how to obtain the free energy needed to locate first-order phase transitions, etc.). It is shown that the topology of the phase diagram changes when the ratio R = Jnnn/Jnn is varied, and this behaviour is discussed in the contex…

Canonical ensemblePhase transitionMean field theoryLattice (order)Monte Carlo methodBinary numberStatistical physicsRenormalization groupMathematicsPhase diagram
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CLASSIFICATION THEORY FOR PHASE TRANSITIONS

1993

A refined classification theory for phase transitions in thermodynamics and statistical mechanics in terms of their orders is introduced and analyzed. The refined thermodynamic classification is based on two independent generalizations of Ehrenfests traditional classification scheme. The statistical mechanical classification theory is based on generalized limit theorems for sums of random variables from probability theory and the newly defined block ensemble limit. The block ensemble limit combines thermodynamic and scaling limits and is similar to the finite size scaling limit. The statistical classification scheme allows for the first time a derivation of finite size scaling without reno…

Canonical ensemblePhysicsPhase transitionScaling limitProbability theoryThermodynamic limitThermodynamicsStatistical and Nonlinear PhysicsLimit (mathematics)Statistical physicsStatistical mechanicsCondensed Matter PhysicsCritical exponentInternational Journal of Modern Physics B
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