Search results for " Traumatic"

showing 10 items of 77 documents

Correction to: Depletion of regulatory T cells increases T cell brain infiltration, reactive astrogliosis, and interferon-γ gene expression in acute …

2019

Traumatic brain injury (TBI) is a major cause of death and disability. T cells were shown to infiltrate the brain during the first days after injury and to exacerbate tissue damage. The objective of this study was to investigate the hitherto unresolved role of immunosuppressive, regulatory T cells (Tregs) in experimental TBI."Depletion of regulatory T cell" (DEREG) and wild type (WT) C57Bl/6 mice, treated with diphtheria toxin (DTx) to deplete Tregs or to serve as control, were subjected to the controlled cortical impact (CCI) model of TBI. Neurological and motor deficits were examined until 5 days post-injury (dpi). At the 5 dpi endpoint, (immuno-) histological, protein, and gene expressio…

InflammationGeneral NeuroscienceImmunologyCorrectionBrainT-Lymphocytes Regulatorylcsh:RC346-429Lymphocyte DepletionCellular and Molecular NeuroscienceDisease Models AnimalInterferon-gammaMiceNeurologyAstrocytesBrain Injuries TraumaticAnimalsGliosislcsh:Neurology. Diseases of the nervous systemJournal of Neuroinflammation
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Comparison of speed-vacuum method and heat-drying method to measure brain water content of small brain samples

2016

Abstract Background A reliable measurement of brain water content (wet-to-dry ratio) is an important prerequisite for conducting research on mechanisms of brain edema formation. The conventionally used oven-drying method suffers from several limitations, especially in small samples. A technically demanding and time-consuming alternative is freeze-drying. New method Centrifugal vacuum concentrators (e.g. SpeedVac/speed-vacuum drying) are a combination of vacuum-drying and centrifugation, used to reduce the boiling temperature. These concentrators have the key advantages of improving the freeze-drying speed and maintaining the integrity of dried samples, thus, allowing e.g. DNA analyses. In t…

Male0301 basic medicineHot TemperatureTime FactorsGenotyping TechniquesVacuumNeuroscience(all)Analytical chemistrySmall brainBrain EdemaCentrifugationBrain water03 medical and health sciences0302 clinical medicineBrain Injuries TraumaticAnimalsDesiccationWater contentBrain ChemistryMoistureChemistryBrain edemaGeneral Neurosciencetechnology industry and agricultureArea under the curveWaterMice Inbred C57BLDisease Models AnimalBoiling pointNeuroprotective AgentsSodium Bicarbonate030104 developmental biologyROC CurveArea Under CurveContent (measure theory)Feasibility Studies030217 neurology & neurosurgeryBiomedical engineeringJournal of Neuroscience Methods
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Normalization with Corresponding Naïve Tissue Minimizes Bias Caused by Commercial Reverse Transcription Kits on Quantitative Real-Time PCR Results

2016

Real-time reverse transcription polymerase chain reaction (PCR) is the gold standard for expression analysis. Designed to improve reproducibility and sensitivity, commercial kits are commonly used for the critical step of cDNA synthesis. The present study was designed to determine the impact of these kits. mRNA from mouse brains were pooled to create serial dilutions ranging from 0.0625 μg to 2 μg, which were transcribed into cDNA using four different commercial reverse-transcription kits. Next, we transcribed mRNA from brain tissue after acute brain injury and naïve mice into cDNA for qPCR. Depending on tested genes, some kits failed to show linear results in dilution series and revealed s…

Male0301 basic medicineSerial dilutionlcsh:MedicineGene ExpressioncDNA synthesisArtificial Gene Amplification and ExtensionBioinformaticsBiochemistryPolymerase Chain ReactionMice0302 clinical medicineBrain Injuries Traumaticlcsh:ScienceGenes EssentialMultidisciplinaryReverse Transcriptase Polymerase Chain ReactionMessenger RNAComplementary DNAHousekeeping geneNucleic acidsReverse transcription polymerase chain reactionResearch ArticleNormalization (statistics)DNA ComplementaryForms of DNANucleic acid synthesisBiologyReal-Time Polymerase Chain ReactionResearch and Analysis Methods03 medical and health sciencesExtraction techniquesComplementary DNAGeneticsAnimalsRNA MessengerChemical synthesisRNA synthesisMolecular Biology TechniquesMolecular BiologyGeneMessenger RNABiology and life scienceslcsh:RDNAReverse TranscriptionMolecular biologyRNA extractionReverse transcriptaseMice Inbred C57BLBiosynthetic techniques030104 developmental biologyRNAlcsh:Q030217 neurology & neurosurgeryPLOS ONE
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Administration of all‐ trans retinoic acid after experimental traumatic brain injury is brain protective

2020

BACKGROUND AND PURPOSE: All‐trans retinoic acid (ATRA) is a vitamin A metabolite, important in the developing and mature brain. Pre‐injury ATRA administration ameliorates ischaemic brain insults in rodents. This study examined the effects of post‐traumatic ATRA treatment in experimental traumatic brain injury (TBI). EXPERIMENTAL APPROACH: Male adult mice were subjected to the controlled cortical impact model of TBI or sham procedure and killed at 7 or 30 days post‐injury (dpi). ATRA (10 mg kg−1, i.p.) was given immediately after the injury and 1, 2 and 3 dpi. Neurological function and sensorimotor coordination were evaluated. Brains were processed for (immuno‐) histological, mRNA and protei…

Male0301 basic medicineTraumatic brain injuryRetinoic acidTretinoinPharmacologyHippocampal formationHMGB1Mice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineBrain Injuries TraumaticmedicineAnimalsInflammationPharmacologyMicrogliabiologybusiness.industryBrainmedicine.diseaseGranule cellResearch PapersAstrogliosis030104 developmental biologymedicine.anatomical_structurechemistryBlood-Brain BarrierApoptosisbiology.proteinbusiness030217 neurology & neurosurgeryBritish Journal of Pharmacology
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Plasminogen activator inhibitor‐1 augments damage by impairing fibrinolysis after traumatic brain injury

2019

Objective Plasminogen activator inhibitor-1 (PAI-1) is the key endogenous inhibitor of fibrinolysis, and enhances clot formation after injury. In traumatic brain injury, dysregulation of fibrinolysis may lead to sustained microthrombosis and accelerated lesion expansion. In the present study, we hypothesized that PAI-1 mediates post-traumatic malfunction of coagulation, with inhibition or genetic depletion of PAI-1 attenuating clot formation and lesion expansion after brain trauma. Methods We evaluated PAI-1 as a possible new target in a mouse controlled cortical impact (CCI) model of traumatic brain injury. We performed the pharmacological inhibition of PAI-1 with PAI-039 and stimulation b…

Male0301 basic medicineTraumatic brain injurymedicine.medical_treatmentBrain damagePharmacologyLesionMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineBrain Injuries TraumaticSerpin E2FibrinolysisAnimalsMedicineThrombusResearch ArticlesIndoleacetic Acidsbusiness.industryFibrinolysisBrainmedicine.diseaseMice Inbred C57BL030104 developmental biologyNeurologychemistryPlasminogen activator inhibitor-1Neurology (clinical)medicine.symptombusinessPlasminogen activator030217 neurology & neurosurgeryIntravital microscopyResearch ArticleAnnals of Neurology
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Single intracerebroventricular progranulin injection adversely affects the blood–brain barrier in experimental traumatic brain injury

2021

Progranulin (PGRN) is a neurotrophic and anti-inflammatory factor with protective effects in animal models of ischemic stroke, subarachnoid hemorrhage, and traumatic brain injury (TBI). Administration of recombinant (r) PGRN prevents exaggerated brain pathology after TBI in Grn-deficient mice, suggesting that local injection of recombinant progranulin (rPGRN) provides therapeutic benefit in the acute phase of TBI. To test this hypothesis, we subjected adult male C57Bl/6N mice to the controlled cortical impact model of TBI, administered a single dose of rPGRN intracerebroventricularly (ICV) shortly before the injury, and examined behavioral and biological effects up to 5 days post injury (dp…

Male0301 basic medicinemedicine.medical_specialtySubarachnoid hemorrhageTraumatic brain injuryPrimary Cell Culture610 MedizinBlood–brain barrierOccludinBiochemistryNeuroprotectionMice03 medical and health sciencesCellular and Molecular NeuroscienceProgranulins0302 clinical medicineInternal medicine610 Medical sciencesBrain Injuries TraumaticmedicineAnimalsNeuroinflammationInjections IntraventricularTight Junction ProteinsBehavior AnimalMicrogliabiologybusiness.industrymedicine.diseaseRecombinant ProteinsMice Inbred C57BL030104 developmental biologymedicine.anatomical_structureEndocrinologyAnimals NewbornBlood-Brain BarrierAstrocytesbiology.proteinEncephalitisMicrogliabusiness030217 neurology & neurosurgeryNeurotrophin
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RS1 (Rsc1A1) deficiency limits cerebral SGLT1 expression and delays brain damage after experimental traumatic brain injury

2018

Acute cerebral lesions are associated with dysregulation of brain glucose homeostasis. Previous studies showed that knockdown of Na+ -D-glucose cotransporter SGLT1 impaired outcome after middle cerebral artery occlusion and that widely expressed intracellular RS1 (RSC1A1) is involved in transcriptional and post-translational down-regulation of SGLT1. In the present study, we investigated whether SGLT1 is up-regulated during traumatic brain injury (TBI) and whether removal of RS1 in mice (RS1-KO) influences SGLT1 expression and outcome. Unexpectedly, brain SGLT1 mRNA in RS1-KO was similar to wild-type whereas it was increased in small intestine and decreased in kidney. One day after TBI, SGL…

Male0301 basic medicinemedicine.medical_specialtyTraumatic brain injuryGene ExpressionBrain EdemaBrain damageBiochemistryProinflammatory cytokineMice03 medical and health sciencesCellular and Molecular NeuroscienceSodium-Glucose Transporter 10302 clinical medicineInternal medicineCortex (anatomy)Brain Injuries TraumaticmedicineAnimalsGlucose homeostasisEye ProteinsBrain ChemistryCerebral CortexMice KnockoutGene knockdownKidneyMovement DisordersMicrogliabusiness.industrydigestive oral and skin physiologyBrainmedicine.diseaseUp-RegulationMice Inbred C57BL030104 developmental biologymedicine.anatomical_structureEndocrinologyCytokinesMicrogliamedicine.symptombusinessCell Adhesion Molecules030217 neurology & neurosurgeryJournal of Neurochemistry
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Deficiency of Plasminogen Activator Inhibitor Type 2 Limits Brain Edema Formation after Traumatic Brain Injury

2019

Plasminogen activator inhibitor-2 (PAI-2/SerpinB2) inhibits extracellular urokinase plasminogen activator (uPA). Under physiological conditions, PAI-2 is expressed at low levels but is rapidly induced by inflammatory triggers. It is a negative regulator of fibrinolysis and serves to stabilize clots. In the present study, PAI-2 expression is upregulated 25-fold in pericontusional brain tissue at 6 h after traumatic brain injury (TBI), with a maximum increase of 87-fold at 12 h. To investigate a potentially detrimental influence of PAI-2 on secondary post-traumatic processes, male PAI-2-deficient (PAI-2-KO) and wild-type mice (WT) were subjected to TBI by controlled cortical impact injury. Br…

Male030506 rehabilitationmedicine.medical_specialtyTraumatic brain injuryBrain EdemaInflammationBlood–brain barrierMice03 medical and health sciences0302 clinical medicineInternal medicineBrain Injuries TraumaticPlasminogen Activator Inhibitor 2medicineExtracellularAnimalsMice KnockoutBrain edemaUrokinase Plasminogen Activatorbusiness.industrymedicine.diseaseMice Inbred C57BLEndocrinologymedicine.anatomical_structureNeurology (clinical)medicine.symptom0305 other medical sciencebusinessPlasminogen activator030217 neurology & neurosurgeryJournal of Neurotrauma
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Skull Fractures Induce Neuroinflammation and Worsen Outcomes after Closed Head Injury in Mice

2020

The weight-drop model is used widely to replicate closed-head injuries in mice; however, the histopathological and functional outcomes may vary significantly between laboratories. Because skull fractures are reported to occur in this model, we aimed to evaluate whether these breaks may influence the variability of the weight-drop (WD) model. Male Swiss Webster mice underwent WD injury with either a 2 or 5 mm cone tip, and behavior was assessed at 2 h and 24 h thereafter using the neurological severity score. The expression of interleukin (IL)-6, IL-1β, tumor necrosis factor-α, matrix metalloproteinase-9, and tissue inhibitor of metalloproteinase-1 genes was m…

Male030506 rehabilitationmedicine.medical_specialtyTraumatic brain injurySkull fractureMice03 medical and health sciencesTraumatic brain injury0302 clinical medicineNeuroinflammationSkull fractureHead Injuries ClosedBrain Injuries TraumaticWeight-drop modelmedicineAnimalsNeuroinflammationInflammationSkull Fracturesbusiness.industryOriginal Articlesmedicine.diseaseSurgeryDisease Models AnimalSkullmedicine.anatomical_structureClosed head injuryNeurology (clinical)0305 other medical sciencebusiness030217 neurology & neurosurgeryJournal of Neurotrauma
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Efficacy and safety of decompressive craniectomy with non-suture duraplasty in patients with traumatic brain injury.

2020

AbstractBackgroundDecompressive craniectomy is an important surgical treatment for patients with severe traumatic brain injury (TBI). Several reports have been published on the efficacy of non-watertight sutures in duraplasty performed in decompressive craniectomy. This study aims to evaluate the effectiveness of dura closure without sutures (non-suture duraplasty) in decompressive craniectomy for TBI.MethodsOne hundred and six patients were enrolled at a single trauma center between January 2017 and December 2018. We retrospectively collected the data and classified the patients into non-suture and suture duraplasty craniectomy groups. We compared the characteristics of patients and their …

MaleCritical Care and Emergency MedicineTraumatic Brain InjuryPhysiologymedicine.medical_treatmentBlood Loss SurgicalGlasgow Outcome ScaleNervous SystemVascular MedicineDiagnostic RadiologyNervous System Procedures0302 clinical medicineSuture (anatomy)AntibioticsBrain Injuries TraumaticMedicine and Health SciencesSurgical treatmentTomographyTrauma MedicineCerebrospinal FluidMultidisciplinaryAntimicrobialsRadiology and ImagingGlasgow Outcome ScaleTrauma centerQRDrugsMiddle AgedSevere Blood LossCranioplastySutureless Surgical ProceduresBody FluidsTreatment OutcomeMedicineFemaleDecompressive craniectomyAnatomyTraumatic InjuryResearch ArticleAdultDecompressive Craniectomymedicine.medical_specialtyImaging TechniquesTraumatic brain injuryScienceOperative TimeTrauma SurgerySurgical and Invasive Medical ProceduresNeuroimagingHemorrhageResearch and Analysis MethodsMicrobiologyCranioplasty03 medical and health sciencesSigns and SymptomsBlood lossDiagnostic MedicineMicrobial ControlmedicineHumansIn patientRetrospective StudiesPharmacologybusiness.industryBiology and Life Sciences030208 emergency & critical care medicinemedicine.diseaseComputed Axial TomographySurgeryDura MaterClinical MedicinebusinessNeurotraumaTrauma surgery030217 neurology & neurosurgeryNeurosciencePLoS ONE
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