Search results for " Western"

showing 10 items of 687 documents

Effects of endocrine disruptors on genes associated with 17 beta-estradiol metabolism and excretion

2008

International audience; In order to provide a global analysis of the effects of endocrine disruptors on the hormone cellular bioavailability, we combined 17 beta-estradiol (E2) cellular flow studies with real-time PCR and Western blot expression measurements of genes involved in the hormone metabolism and excretion. Three endocrine disruptors commonly found in food were chosen for this study, which was conducted in the estrogen receptor (ER) negative hepatoblastoma HepG2 cell line: bisphenol A (BPA), genistein (GEN) and resveratrol (RES). We showed that 24h after a single dose treatment with genistein, resveratrol or bisphenol A, the expression of ATP-binding cassette transporters (the mult…

medicine.medical_specialtyATP-BINDING CASSETTE TRANSPORTERS[SDV]Life Sciences [q-bio]Clinical BiochemistryBlotting WesternEstrogen receptorGenistein010501 environmental sciencesBiologyPharmacologyResveratrol01 natural sciencesBiochemistryCell LineENDOCRINE DISRUPTORS03 medical and health scienceschemistry.chemical_compoundEndocrinologyInternal medicineUDP-GLUCURONOSYLTRANFERASEmedicineHumansHormone metabolismRNA MessengerMolecular Biology030304 developmental biology0105 earth and related environmental sciencesDNA PrimersPharmacology0303 health sciencesBase SequenceEstradiolReverse Transcriptase Polymerase Chain ReactionMultidrug resistance-associated protein 2Organic ChemistrySULFOTRANSFERASEEndocrinologyEndocrine disruptorchemistryGene Expression Regulation13. Climate actionESTRADIOL METABOLISMMultidrug Resistance-Associated Proteinshormones hormone substitutes and hormone antagonistsHormone
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The L-glutamate transporters GLAST (EAAT1) and GLT-1 (EAAT2): expression and regulation in rat lactating mammary gland.

1999

The Na(+)-dependent L-glutamate transporters GLAST (EAAT1) and GLT-1 (EAAT2), were expressed in rat lactating mammary gland, but EAAC1 (EAAT3) was not. GLT-1 expression in rat lactating mammary gland was constant in all the physiological situations studied; however, the GLAST expression is under tight regulation. Fasting for 24 h decreased the GLAST expression which returned to control values after refeeding. Weaning for 24 h produced a decrease in GLAST expression through a mechanism independent of prolactin deficiency. Resuckling for 6 h returned the expression of this transporter to control values. There is a correlation between the levels of GLAST (mRNA and protein) and the in vivo upta…

medicine.medical_specialtyAmino Acid Transport System X-AGMammary glandBlotting WesternMammary Glands AnimalIn vivoInternal medicineLactationmedicineWeaningAnimalsLactationTissue DistributionRats WistarMolecular BiologyMessenger RNAChemistryReverse Transcriptase Polymerase Chain ReactionTransporterProlactin deficiencyCell BiologyBlotting NorthernRatsBlotmedicine.anatomical_structureEndocrinologyATP-Binding Cassette TransportersFemaleMolecular membrane biology
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Analysis of molecular mechanisms and anti-tumoural effects of zoledronic acid in breast cancer cells

2012

Zoledronic acid (ZOL) is the most potent nitrogen-containing bisphosphonate (N-BPs) that strongly binds to bone mineral and acts as a powerful inhibitor of bone resorption, already clinically available for the treatment of patients with osteolytic metastases. Recent data also suggest that ZOL, used in breast cancer, may provide more than just supportive care modifying the course of the disease, though the possible molecular mechanism of action is still unclear. As breast cancer is one of the primary tumours with high propensity to metastasize to the bone, we investigated, for the first time, differential gene expression profile on Michigan Cancer Foundation-7 (MCF-7) breast cancer cells tre…

medicine.medical_specialtyAngiogenesismedicine.medical_treatmentBlotting WesternAngiogenesis InhibitorsAntineoplastic AgentsBreast NeoplasmsBiologyReal-Time Polymerase Chain ReactionZoledronic AcidZOL FN1 TGF-b1 THBS-1 invasion breast cancerBone resorptionThrombospondin 1Transforming Growth Factor beta1breast cancerBreast cancerTGF-β1Internal medicineThrombospondin 1medicineHumansBone ResorptionCell ProliferationMatrigelDiphosphonatesFN1Gene Expression ProfilingImidazolesCancerOriginal ArticlesCell BiologyZOLBisphosphonateMicroarray Analysisinvasionmedicine.diseaseFibronectinsUp-RegulationGene Expression Regulation NeoplasticEndocrinologyZoledronic acidTHBS-1MCF-7 CellsCancer researchMolecular MedicineFemalemedicine.drug
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Pentaerithrityl tetranitrate improves angiotensin II induced vascular dysfunction via induction of heme oxygenase-1

2010

The organic nitrate pentaerythritol tetranitrate is devoid of nitrate tolerance, which has been attributed to the induction of the antioxidant enzyme heme oxygenase (HO)-1. With the present study, we tested whether chronic treatment with pentaerythritol tetranitrate can improve angiotensin II–induced vascular oxidative stress and dysfunction. In contrast to isosorbide-5 mononitrate (75 mg/kg per day for 7 days), treatment with pentaerythritol tetranitrate (15 mg/kg per day for 7 days) improved the impaired endothelial and smooth muscle function and normalized vascular and cardiac reactive oxygen species production (mitochondria, NADPH oxidase activity, and uncoupled endothelial NO synthase)…

medicine.medical_specialtyAntioxidantNitric Oxide Synthase Type IIImedicine.medical_treatmentVasodilator AgentsBlotting WesternFluorescent Antibody TechniquePentaerythritol tetranitratemedicine.disease_causePentaerythritolArticlechemistry.chemical_compoundInternal medicineRats Inbred SHRInternal MedicinemedicineAnimalsPentaerythritol TetranitrateEndothelial dysfunctionchemistry.chemical_classificationReactive oxygen speciesAnalysis of VarianceAngiotensin IImedicine.diseaseAngiotensin IIMitochondriaRatsHeme oxygenaseOxidative StressEndocrinologychemistryHeminEndothelium VascularReactive Oxygen SpeciesOxidative stressHeme Oxygenase-1
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Transient focal cerebral ischemia significantly alters not only EAATs but also VGLUTs expression in rats: relevance of changes in reactive astroglia

2010

The involvement of plasma membrane glutamate transporters (EAATs - excitatory aminoacid transporters) in the pathophysiology of ischemia has been widely studied, but little is known about the role of vesicular glutamate transporters (VGLUTs) in the ischemic process. We analyzed the expression of VGLUT1-3 in the cortex and caudate-putamen of rats subjected to transient middle cerebral artery occlusion. Western blot and immunohistochemistry revealed an increase of VGLUT1 signal in cortex and caudate-putamen until 3 days of reperfusion followed by a reduction 7 days after the ischemic insult. By contrast, VGLUT2 and 3 were drastically reduced. Confocal microscopy revealed an increase in VGLUT2…

medicine.medical_specialtyBlotting WesternIschemiaFluorescent Antibody TechniqueGlutamic AcidBiologyBiochemistryBrain ischemiaGlutamate Plasma Membrane Transport ProteinsCellular and Molecular NeuroscienceCell MovementInternal medicineNeuroblast migrationCortex (anatomy)Vesicular Glutamate Transport ProteinsmedicineAnimalsCerebral CortexMicroscopy ConfocalNeuronal PlasticityCell DeathNeurogenesisPutamenGlutamate receptorInfarction Middle Cerebral Arterymedicine.diseaseImmunohistochemistryRatsEndocrinologymedicine.anatomical_structureIschemic Attack TransientAstrocytesReperfusion InjuryExcitatory postsynaptic potentialCaudate NucleusNeurogliaReperfusion injuryNeuroscienceJournal of Neurochemistry
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Expression of inhibitory glycine receptors in postnatal rat cerebral cortex.

1993

The developmental expression of inhibitory glycine receptors was analyzed in postnatal rat cerebral cortex using the specific monoclonal antibody, MAb 4a. This antibody defines an epitope common to all known glycine receptor alpha-subunits. At birth, high levels of immunoreactivity were found, which transiently increased during the second postnatal week, but subsequently declined to low adult levels. Biochemical analysis of the MAb 4a antigen from parietal areas indicates that cortical glycine receptors correspond to the neonatal receptor isoform previously identified in spinal cord of newborn animals. Immunocytochemistry showed that, within 2 weeks after birth, MAb 4a-reactive glycine rece…

medicine.medical_specialtyCentral nervous systemImmunocytochemistryBlotting WesternBiologyRats Sprague-Dawleychemistry.chemical_compoundReceptors GlycineInternal medicineCortex (anatomy)medicineAnimalsTissue DistributionReceptorMolecular BiologyGlycine receptorCerebral CortexGeneral NeuroscienceAntibodies MonoclonalNeural InhibitionStrychnineImmunohistochemistryRatsReceptors Neurotransmittermedicine.anatomical_structureEndocrinologychemistryAnimals NewbornCerebral cortexImmunologyGlycineNeurology (clinical)Developmental BiologyBrain research
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Immunohistochemical analysis of KCNQ3 potassium channels in mouse brain.

2005

KCNQ-type potassium channels generate the so-called M-current regulating excitability in many neurons. Mutations in KCNQ2/KCNQ3 channels can cause benign familial neonatal convulsions (BFNC). We describe the immunohistochemical staining of adult and developing mouse brain using an antibody directed against the N-terminus of KCNQ3 channels (KCNQ3N). A widespread KCNQ3N immunoreactivity predominantly of neuropil but also of somata was detected in different regions of the adult mouse brain, in particular in the hippocampus, cortex, thalamus and cerebellum. This staining pattern appeared gradually and became more intense during development. In the pyramidal cell layer of the hippocampus, the im…

medicine.medical_specialtyCerebellumPathologyCentral nervous systemThalamusBlotting WesternHippocampusBiologyKCNQ3 Potassium ChannelMiceCortex (anatomy)Internal medicinemedicineNeuropilAnimalsGeneral NeuroscienceBrainGene Expression Regulation DevelopmentalImmunohistochemistryPotassium channelMice Inbred C57BLEndocrinologymedicine.anatomical_structureParvalbuminsnervous systemAnimals Newbornsense organsPyramidal cellNeuroscience letters
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Aromatase and amphiregulin are correspondingly expressed in human liver cancer cells

2009

Human hepatocellular carcinoma (HCC) is associated with high mortality rates, being the third most common cause of cancer death worldwide. Although estrogens have been implicated in HCC, their potential role in development and/or progression of this malignancy remains unclear. In this study we investigated mRNA and protein expression of aromatase (Aro) and amphiregulin (AREG) in relation to estrogen receptors (ERs), in HepG2, Huh7, and HA22T human malignant liver cell lines, using RT-PCR and Western blot analyses. Aro expression was significantly higher (approximately 13-fold, P= 0.003) in HepG2 cells than in Huh7 cells, while no Aro expression could be detected in HA22T cells. Interestingl…

medicine.medical_specialtyEGF Family of ProteinsBlotting WesternEstrogen receptorAmphiregulinGeneral Biochemistry Genetics and Molecular BiologyAromataseHistory and Philosophy of ScienceAmphiregulinWestern blotInternal medicineCell Line TumormedicineHumansEstrogen receptors hepatocellular carcinoma amphiregulinAromataseDNA PrimersGlycoproteinsbiologymedicine.diagnostic_testBase SequenceReverse Transcriptase Polymerase Chain ReactionGeneral NeuroscienceLiver cellLiver NeoplasmsEstrogen Receptor alphamedicine.diseasedigestive system diseasesBlotEndocrinologyCell cultureHepatocellular carcinomabiology.proteinCancer researchIntercellular Signaling Peptides and Proteins
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Cigarette smoke extract activates human bronchial epithelial cells affecting non-neuronal cholinergic system signalling in vitro.

2010

Abstract Aims Acetylcholine (ACh) is synthesized by Choline Acetyl-Transferase (ChAT) that exerts its physiological effects in airway epithelial cells via muscarinic receptor (MR) activation. We evaluate the effect of ACh stimulation on human bronchial epithelial cells (16-HBE) and test whether cigarette smoke extract (CSE) can modify the basal cellular response to ACh affecting the non-neuronal cholinergic system signalling. Main methods ACh stimulated 16-HBE were tested for ACh-binding, Leukotriene B 4 (LTB 4 ) release and ERK1/2 and NFkB pathway activation. Additionally, we investigated all the aforementioned parameters as well as ChAT and MR proteins and mRNA expression and endogenous A…

medicine.medical_specialtyLeukotriene B4Blotting WesternEndogenyStimulationBronchiPharmacologyBiologyComplex MixturesIn Vitro TechniquesLeukotriene B4General Biochemistry Genetics and Molecular BiologyCell LineCholine O-Acetyltransferasechemistry.chemical_compoundInternal medicineSmokeparasitic diseasesMuscarinic acetylcholine receptorTobaccomedicineHumansGeneral Pharmacology Toxicology and PharmaceuticsReceptorExtracellular Signal-Regulated MAP KinasesAnalysis of VarianceReverse Transcriptase Polymerase Chain ReactionNF-kappa BCholine Acetyl-TransferaseAcetylcholine muscarinic receptorhuman bronchial epithelial cellsGeneral MedicineFlow CytometryCholine acetyltransferaseReceptors MuscarinicAcetylcholineEndocrinologychemistryGene Expression RegulationTelenzepineAcetylcholinemedicine.drugSignal Transduction
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IRS-2 deficiency impairs NMDA receptor-dependent long-term potentiation

2011

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License.-- et al.

medicine.medical_specialtyPatch-Clamp TechniquesCognitive Neurosciencemedicine.medical_treatmentBlotting WesterneducationHippocampusComputingMilieux_LEGALASPECTSOFCOMPUTINGNeurotransmissionBiologyHippocampusReceptors N-Methyl-D-AspartateSynaptic TransmissionMiceCellular and Molecular NeuroscienceInternal medicinemedicineAnimalsImmunoprecipitationlong-term potentiationMice Knockoutsynaptic plasticitydiabetesInsulinDiabetesLong-term potentiationArticlesNMDA receptorIRS2insulin receptor signalingSynaptic fatigueEndocrinologynervous systemSynaptic plasticityInsulin Receptor Substrate ProteinsNMDA receptorFemaleNeuroscienceCerebral Cortex
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