Search results for " atherosclerotic"

showing 8 items of 58 documents

Penetrating aortic ulcer post migration of thoracic aortic endoprosthesis: Case report

2021

Introduction Thoracic endovascular aortic repair (TEVAR) is the first treatment option for many thoracic aortic pathologies. Especially after aortic dissections, it is possible to have progression during follow-up with appearance of new lesions on arterial wall. Herein, we report a case of Penetrating Aortic Ulcer (PAU) post release of Thoracic endoprosthesis. Presentation of case A 67-years-old male with hypertension and diabetes mellitus was followed at our hospital after an emergency procedure for Type B aortic dissection (TBAD) complicated by symptomatic large infrarenal AAA and treated with a proximal TEVAR plus chimney for left subclavian artery and PETTICOAT with EVAR for abdominal a…

Post releasemedicine.medical_specialtyTEVARbusiness.industrymedicine.medical_treatmentStentTreatment optionsmedicine.diseaseAortic diseaseProsthesisSettore MED/22 - Chirurgia VascolareSurgeryPenetrating atherosclerotic ulcerPenetrating atherosclerotic ulcermedicine.arteryCase reportcardiovascular systemmedicineThoracic aortaSurgeryArterial wallComplicated aortic B dissectionbusinessMigration endoprosthesis
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Phytochemical indicaxanthin suppresses 7-ketocholesterol-induced THP-1 cell apoptosis by preventing cytosolic Ca(2+) increase and oxidative stress.

2012

7-Ketocholesterol (7-KC)-induced apoptosis of macrophages is considered a key event in the development of human atheromas. In the present study, the effect of indicaxanthin (Ind), a bioactive pigment from cactus pear fruit, on 7-KC-induced apoptosis of human monocyte/macrophage THP-1 cells was investigated. A pathophysiological condition was simulated by using amounts of 7-KC that can be reached in human atheromatous plaque. Ind was assayed within a micromolar concentration range, consistent with its plasma level after dietary supplementation with cactus pear fruit. Pro-apoptotic effects of 7-KC were assessed by cell cycle arrest, exposure of phosphatidylserine at the plasma membrane, varia…

Programmed cell deathPyridinesCellMedicine (miscellaneous)Apoptosismedicine.disease_causeMonocytesCell Linechemistry.chemical_compoundCytosolmedicineHumansSulfhydryl CompoundsKetocholesterolsNutrition and DieteticsChemistryPlant ExtractsMonocyteMacrophagesNF-kappa BNADPH OxidasesOpuntiaPhosphatidylserineAtherosclerosisPlaque AtheroscleroticCell biologyBetaxanthinsMitochondriaCytosolOxidative Stressmedicine.anatomical_structureApoptosisNADPH Oxidase 4FruitDietary SupplementsCalciumReactive Oxygen SpeciesIndicaxanthinOxidative stressPhytotherapyThe British journal of nutrition
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Cardiovascular effects of air pollution

2017

Air pollution is composed of particulate matter (PM) and gaseous pollutants, such as nitrogen dioxide and ozone. PM is classified according to size into coarse particles (PM 10), fine particles (PM 2.5) and ultrafine particles. We aim to provide an original review of the scientific evidence from epidemiological and experimental studies examining the cardiovascular effects of outdoor air pollution. Pooled epidemiological studies reported that a 10 μg/m 3 increase in long-term exposure to PM 2.5 was associated with an 11% increase in cardiovascular mortality. Increased cardiovascular mortality was also related to long-term and short-term exposure to nitrogen dioxide. Exposure to air pollution…

Time FactorsCardiovascular mortalityOzoneair pollutionAir pollution030204 cardiovascular system & hematology010501 environmental sciencesmedicine.disease_causeCardiovascular SystemRisk Assessment01 natural sciencesArticleToxicology03 medical and health scienceschemistry.chemical_compound0302 clinical medicineRisk FactorsEnvironmental healthUltrafine particleAnimalsHumansoxidative stressMedicineNitrogen dioxidePlatelet activationBlood CoagulationAir quality index0105 earth and related environmental sciencesparticulate matterPollutantAir Pollutantsbusiness.industryEnvironmental ExposureGeneral MedicineParticulatesPrognosisPlaque Atheroscleroticmyocardial infarctionchemistryCardiovascular DiseasesReactive Oxygen SpeciesCardiology and Cardiovascular MedicinebusinessArchives of Cardiovascular Diseases
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Polymorphonuclear leukocyte membrane fluidity and cytosolic Ca2+ concentration in subjects with vascular atherosclerotic disease subdivided according…

2006

An abnormal activation state of polymorphonuclear leukocytes (PMN) plays a key role in organ injury induced by vascular atherosclerotic disease (VAD) and diabetes mellitus (DM). PMN membrane fluidity and cytosolic Ca2+ content can be considered markers of PMN activation. In this research we evaluated the PMN membrane fluidity and cytosolic Ca2+ content in VAD subjects with and without type 2 DM and examined the association between these parameters and the mono- or polyvascular localization. We enrolled 155 VAD subjects, including 92 non-diabetic (group A: mean age 63.6 +/- 9.2 years) and 63 diabetic patients (group B: mean age 65.4 +/- 7.8 years). Among group A 63 patients had monovascular …

Vascular atherosclerotic disease type 2 diabetes mellitus polymorphonuclear leukocyte membrane fluidity polymorphonuclear leukocyte Ca2+ contentSettore MED/09 - Medicina Interna
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Fluidity and cytosolic Ca2+ concentration of circulating polymorphonuclear leukocytes at baseline in some chronic and acute clinical conditions: revi…

2016

Abstract. Objective: In this mini-review we describe the behavior of polymorphonuclear leukocyte (PMN) membrane fluidity and of PMN cytosolic Ca2+ concentration in some chronic and acute clinical conditions. Methods: PMN membrane fluidity was evaluated employing the fluorescent probe Fura-2AM, and PMN cytosolic Ca2+ concentration was evaluated using the fluorescent probe TMA-DPH. Results: From the determination of these two parameters investigated on resting PMNs, an almost constant increase in PMN cytosolic Ca2+ concentration in chronic clinical conditions, such as vascular atherosclerotic disease with and without diabetes mellitus, essential hypertension, chronic kidney disease, and diabe…

acute ischemic strokemedicine.medical_specialtyClinical BiochemistryCell calciumEssential hypertensionBiochemistryInorganic ChemistryCell calcium; membrane fluidity; fluorescence spectroscopy; polymorphonuclear leukocyte; vascular atherosclerotic disease; diabetes mellitus; chronic kidney disease; essential hypertension; myocardial infarction; acute ischemic strokevascular atherosclerotic diseaseDiabetes mellitusInternal medicineCa2 concentrationmedicineMembrane fluidityMyocardial infarctionAcute ischemic strokePolymorphonuclear leukocytediabetes mellitubusiness.industrymembrane fluiditypolymorphonuclear leukocyteessential hypertensionfluorescence spectroscopymedicine.diseaseCytosolmyocardial infarctionCardiologybusinesschronic kidney disease
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An evaluation of RVX-208 for the treatment of atherosclerosis

2015

Introduction: RVX-208 is a first-in-class, orally active, novel small molecule in development by Resverlogix Corporation (Calgary, AB, Canada). It acts through an epigenetic mechanism by inhibiting the bromodomain and extraterminal (BET) family of proteins, increasing apolipoprotein A-I (apoA-I) and targeting high-density lipoprotein (HDL) metabolism, including generating of nascent HDL and increased larger HDL particles, resulting in the stimulation of reverse cholesterol transport. RVX-208 also has a beneficial effect on inflammatory factors known to be involved in atherosclerosis and plaque stability. New therapeutic strategies are needed for patients with atherosclerosis.Areas covered: …

medicine.medical_specialtyApolipoprotein Bapolipoprotein A-IRVX 208high-density lipoproteinPharmacologyEpigenesis Geneticchemistry.chemical_compoundatherosclerosiHigh-density lipoproteinMetabolic DiseasesInternal medicinemedicineAnimalsHumansPharmacology (medical)QuinazolinonesPharmacologybiologyAnimalCholesterolMedicine (all)Cholesterol HDLReverse cholesterol transportRVX-208QuinazolineGeneral MedicineAtherosclerosisPlaque AtheroscleroticMetabolic DiseaseBromodomainOrally activeEndocrinologyhigh-density lipoprotein particlechemistryQuinazolinesbiology.proteinlipids (amino acids peptides and proteins)HumanLipoproteinExpert Opinion on Investigational Drugs
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Visualizing the atherosclerotic plaque: a chemical perspective.

2014

Atherosclerosis is the major underlying pathologic cause of coronary artery disease. An early detection of the disease can prevent clinical sequellae such as angina, myocardial infarction, and stroke. The different imaging techniques employed to visualize the atherosclerotic plaque provide information of diagnostic and prognostic value. Furthermore, the use of contrast agents helps to improve signal-to-noise ratio providing better images. For nuclear imaging techniques and optical imaging these agents are absolutely necessary. We report on the different contrast agents that have been used, are used or may be used in future in animals, humans, or excised tissues for the distinct imaging moda…

medicine.medical_specialtyIronEarly detectionContrast MediaGadoliniumDiseaseCoronary Artery DiseaseAnginaCoronary artery diseaseCoordination ComplexesMedicineHumansMyocardial infarctionStrokeUltrasonographymedicine.diagnostic_testbusiness.industryMagnetic resonance imagingGeneral ChemistryOrganotechnetium Compoundsmedicine.diseaseMagnetic Resonance ImagingPlaque AtheroscleroticPositron emission tomographyPositron-Emission TomographyRadiologybusinessTomography X-Ray ComputedChemical Society reviews
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Myocardial infarction with non-obstructive coronary arteries (MINOCA): Intracoronary imaging-based diagnosis and management.

2021

Myocardial infarction with non-obstructive coronary arteries (MINOCA) is defined by clinical evidence of myocardial infarction (MI) with normal or near-normal coronary arteries on angiography. This condition is present in about 5% to 25% of patients presenting with acute coronary syndromes. MINOCA is a working diagnosis. Current guidelines and consensus recommend identification of underlying causes of MINOCA in order to optimize treatment, improve prognosis, and promote prevention of recurrent myocardial infarction. An accurate evaluation of patient history, symptoms and use of invasive and non-invasive imaging should lead to identification of epicardial or microvascular causes of MINOCA an…

medicine.medical_specialtyMyocarditisMyocardial InfarctionContext (language use)Coronary Artery Disease030204 cardiovascular system & hematologyCoronary AngiographyCoronary artery disease03 medical and health sciences0302 clinical medicineMyocardial infarction with non-obstructive coronary arterieRisk FactorsInternal medicineIntravascular ultrasoundmedicineHumans030212 general & internal medicineMyocardial infarctionmedicine.diagnostic_testOptical coherence tomographybusiness.industryTakotsubo syndrome.Intracoronary imagingmedicine.diseaseCoronary VesselsPlaque AtheroscleroticPulmonary embolismCoronary arteriesmedicine.anatomical_structureAngiographyCardiologyIntravascular ultrasoundCardiology and Cardiovascular MedicinebusinessJournal of cardiology
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