Search results for " cell transfer"

showing 10 items of 116 documents

Platelet, Not Endothelial, P-Selectin Expression Contributes to Generation of Immunity in Cutaneous Contact Hypersensitivity

2010

Leukocyte extravasation is a prerequisite for host defense and autoimmunity alike. Detailed understanding of the tightly controlled and overlapping sequences of leukocyte extravasation might aid development of novel therapeutic strategies. Leukocyte extravasation is initiated by interaction of selectins with appropriate carbohydrate ligands. Lack of P-selectin expression leads to decreased contact hypersensitivity responses. Yet, it remains unclear if this is due to inhibition of leukocyte extravasation to the skin or due to interference with initial immune activation in lymph nodes. In line with previous data, we here report a decreased contact hypersensitivity response, induced by 2,4,-di…

Blood PlateletsAdoptive cell transferP-selectinInflammationDermatitis ContactPathology and Forensic MedicineMiceImmunityMedicineAnimalsBlood Platelets/*metabolismCell ShapeSkinInflammationbusiness.industryImmunityEndothelial CellsSkin/immunology/*pathologyDermatitis Contact/complications/*immunology/*pathologyLeukocyte extravasationAdoptive TransferInflammation/complications/immunology/pathologyEndothelial stem cellMice Inbred C57BLP-Selectinmedicine.anatomical_structureImmunologyEndothelial Cells/*metabolismDinitrofluorobenzeneBone marrowmedicine.symptomImmunity/*immunologybusinessSelectinP-Selectin/*metabolismRegular Articles
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Low zone tolerance induced by systemic application of allergens inhibits TC1-mediated skin inflammation

2005

Background The induction of tolerance may be a promising target of strategies aimed at preventing harmful allergic diseases. Low zone tolerance (LZT), induced by epicutaneous application of low doses of contact allergens, inhibits the development of T C 1-mediated contact hypersensitivity (CHS). Objective We evaluated the effect of systemic (oral, intravenous) administration of low amounts of haptens on specific immune reactions and tolerance induction. Methods By using the mouse model of LZT, we analyzed immune reactions in vivo (skin inflammation) and T-cell responses in vitro after oral, intravenous, or epicutaneous application of low amounts of the contact allergen 2,4,6-trinitro-1-chlo…

CD4-Positive T-LymphocytesAdoptive cell transferAllergymedicine.medical_treatmentImmunologyDose-Response Relationship ImmunologicAdministration OralInflammationPicryl ChlorideAdministration CutaneousDermatitis ContactT-Lymphocytes RegulatoryImmune toleranceMiceImmune systemImmune TolerancemedicineAnimalsImmunology and AllergyMice KnockoutChemistryCell DifferentiationImmunotherapyAllergensmedicine.diseaseMice Inbred C57BLTolerance inductionTrinitrobenzenesulfonic AcidOrgan SpecificityInjections IntravenousImmunologymedicine.symptomCD8T-Lymphocytes CytotoxicJournal of Allergy and Clinical Immunology
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Critical role of IL-10 in the induction of low zone tolerance to contact allergens

2003

The development and mechanisms of tolerance to allergens are poorly understood. Using the murine low zone tolerance (LZT) model, where contact hypersensitivity (CHS) is prevented by repeated topical low-dose applications of contact allergens, we show that LZT induction is IL-10 dependent. IL-10 is required for the generation of LZT effector cells, that is, CD8+ regulatory T cells. Only T cells from tolerized IL-10+/+ mice or IL-10-/- mice reconstituted with IL-10 during LZT induction adoptively transferred LZT to naive mice and prevented CHS, whereas T cells from IL-10-/- mice failed to do so. The IL-10 required for normal LZT development is derived from lymph node CD4+ T cells, the only sk…

CD4-Positive T-LymphocytesAdoptive cell transferPopulationPicryl ChlorideBiologyCD8-Positive T-LymphocytesDermatitis ContactArticleImmune tolerancePicryl chloridechemistry.chemical_compoundMiceImmune systemmedicineImmune ToleranceAnimalseducationLymph nodeMice Knockouteducation.field_of_studyGeneral MedicineAllergensAdoptive TransferInterleukin-10Mice Inbred C57BLInterleukin 10medicine.anatomical_structurechemistryImmunologyCD8
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T-cell receptor transfer into human T cells with ecotropic retroviral vectors

2014

Adoptive T-cell transfer for cancer immunotherapy requires genetic modification of T cells with recombinant T-cell receptors (TCRs). Amphotropic retroviral vectors (RVs) used for TCR transduction for this purpose are considered safe in principle. Despite this, TCR-coding and packaging vectors could theoretically recombine to produce replication competent vectors (RCVs), and transduced T-cell preparations must be proven free of RCV. To eliminate the need for RCV testing, we transduced human T cells with ecotropic RVs so potential RCV would be non-infectious for human cells. We show that transfection of synthetic messenger RNA encoding murine cationic amino-acid transporter 1 (mCAT-1), the re…

CD4-Positive T-LymphocytesAdoptive cell transfermedicine.medical_treatmentGenetic enhancementGenetic VectorsReceptors Antigen T-CellCD8-Positive T-LymphocytesBiologyImmunotherapy AdoptiveJurkat cellsVesicular stomatitis Indiana virusCell LineJurkat CellsMiceTransduction (genetics)Viral Envelope ProteinsCancer immunotherapyTransduction GeneticGeneticsmedicineAnimalsHumansRNA MessengerMolecular BiologyCationic Amino Acid Transporter 1Membrane GlycoproteinsHEK 293 cellsT-cell receptorTransfectionAdoptive TransferVirologyElectroporationHEK293 CellsRetroviridaeLeukemia Virus Gibbon ApeMolecular MedicinePlasmidsGene Therapy
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Adoptive transfer of ex vivo expanded SARS‐CoV‐2‐specific cytotoxic lymphocytes: A viable strategy for COVID‐19 immunosuppressed patients?

2021

Cellular and humoral response to acute respiratory syndrome coronavirus 2 (SARS‐CoV‐2) infections is on focus of research. We evaluate herein the feasibility of expanding virus‐specific T cells (VST) against SARS‐CoV‐2 ex vivo through a standard protocol proven effective for other viruses. The experiment was performed in three different donors' scenarios: (a) SARS‐CoV‐2 asymptomatic infection/negative serology, (b) SARS‐CoV‐2 symptomatic infection/positive serology, and (c) no history of SARS‐CoV‐2 infection/negative serology. We were able to obtain an expanded VST product from donors 1 and 2 (1.6x and 1.8x increase of baseline VST count, respectively) consisting in CD3 + cells (80.3% and 6…

CD4-Positive T-LymphocytesAdoptive cell transferviruses030230 surgerymedicine.disease_causevirus-specific T cellsAsymptomaticSARS‐CoV‐2Serology03 medical and health sciences0302 clinical medicineCOVID‐19medicineCytotoxic T cellHumansRespiratory systemthird‐party donorsCoronavirusTransplantationbusiness.industrySARS-CoV-2COVID-19Original Articlesvirus‐specific T cellsAdoptive Transferlymphocyte expansionrespiratory virusInfectious DiseasesImmunologyRespiratory virus030211 gastroenterology & hepatologyOriginal Articlethird-party donorsmedicine.symptombusinessadoptive immunotherapyEx vivo
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A Key Regulatory Role of the Transcription Factor NFATc2 in Bronchial Adenocarcinoma via CD8+ T Lymphocytes

2009

AbstractThe Ca2+-regulated calcineurin/nuclear factor of activated T cells (NFAT) cascade controls alternative pathways of T-cell activation and peripheral tolerance. Here, we describe reduction of NFATc2 mRNA expression in the lungs of patients with bronchial adenocarcinoma. In a murine model of bronchoalveolar adenocarcinoma, mice lacking NFATc2 developed more and larger solid tumors than wild-type littermates. The extent of central tumor necrosis was decreased in the tumors in NFATc2(−/−) mice, and this finding was associated with reduced tumor necrosis factor-α and interleukin-2 (IL-2) production by CD8+ T cells. Adoptive transfer of CD8+ T cells of NFATc2(−/−) mice induced transforming…

CD4-Positive T-LymphocytesCancer ResearchAdoptive cell transferTranscription GeneticTransplantation HeterologousMice TransgenicReceptors Nerve Growth FactorAdenocarcinomaCD8-Positive T-LymphocytesBiologyReceptors Tumor Necrosis FactorTransforming Growth Factor beta1Interferon-gammaMiceGlucocorticoid-Induced TNFR-Related ProteinAnimalsHumansIL-2 receptorInterleukin-7 receptorMice Inbred BALB CReceptors Interleukin-7NFATC Transcription FactorsTumor Necrosis Factor-alphaBronchial NeoplasmsInterleukin-2 Receptor alpha SubunitPeripheral toleranceForkhead Transcription FactorsNFATCalcineurinDisease Models AnimalOncologyCancer researchInterleukin-2Tumor necrosis factor alphaCD8Cancer Research
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Uptake and presentation of exogenous antigen and presentation of endogenously produced antigen by skin dendritic cells represent equivalent pathways …

2008

Gene gun-mediated biolistic DNA vaccination with beta-galactosidase (betaGal)-encoding plasmid vectors efficiently modulated antigen-induced immune responses in an animal model of type I allergy, including the inhibition of immunoglobulin E (IgE) production. Here we show that CD4(+) as well as CD8(+) T cells from mice biolistically transfected with a plasmid encoding betaGal under the control of the fascin promoter (pFascin-betaGal) are capable of inhibiting betaGal-specific IgE production after adoptive transfer into naïve recipients. Moreover, suppression of IgE production was dependent on interferon (IFN)-gamma. To analyse the modalities of activation of CD4(+) and CD8(+) T cells regardi…

CD4-Positive T-LymphocytesCytotoxicity ImmunologicKeratinocytesAdoptive cell transferGenetic VectorsImmunologyAntigen presentationPriming (immunology)CD8-Positive T-LymphocytesBiologyImmunoglobulin GDNA vaccinationInterferon-gammaMiceCross-PrimingImmune systemAntigenHypersensitivityVaccines DNAAnimalsImmunology and AllergyCytotoxic T cellPromoter Regions GeneticMice KnockoutAntigen PresentationInterleukin-12 Subunit p40Keratin-15VaccinationT-Lymphocytes Helper-InducerOriginal ArticlesBiolisticsImmunoglobulin Ebeta-GalactosidaseAdoptive TransferMolecular biologyImmunoglobulin GLangerhans CellsImmunologybiology.proteinKeratin-5FemaleImmunology
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Epicutaneous and Oral Low-Zone Tolerance Protects from Colitis in Mice

2016

Tolerance to environmental antigens that encounter the organism at interfaces like skin or gut prevents deleterious systemic immune responses. The aim of this study was to analyze whether and how low doses of haptens, by entry through the skin or gastrointestinal tract, affect the outcome of the predominantly Th1/Th17-mediated 2,4,6-trinitro-benzenesulfonic acid-induced colitis, which mimics an autoimmune bowl disease in man. Epicutaneous and oral applications of low doses of the allergen resulted in the induction of low-zone tolerance (LZT) and protected from colitis development, demonstrated by a significantly reduced inflammatory response of the gut in vivo. In line with this observation…

CD4-Positive T-LymphocytesMale0301 basic medicineAdoptive cell transferT cellAdministration Oralchemical and pharmacologic phenomenaDermatologyAdministration CutaneousDermatitis ContactT-Lymphocytes RegulatoryBiochemistryImmune toleranceMice03 medical and health sciences0302 clinical medicineImmune systemAntigenImmune TolerancemedicineAnimalsHumansIL-2 receptorColitisMolecular Biologybusiness.industryInterleukin-2 Receptor alpha SubunitFOXP3Cell BiologyAllergensColitismedicine.diseaseAdoptive TransferInterleukin-10Disease Models Animal030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisImmunologyFemalebusinessJournal of Investigative Dermatology
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Adoptive transfer of protective immunity from Cryptosporidium parvum-infected interferon-gamma and interleukin-12-deficient mice to naive recipients.

2008

We investigated the possibility of transfer immunity from Cryptosporidium parvum-infected interferon-gamma (GKO) and interleukin-12p40 (IL-12KO) deficient C57BL/6 mice to naive mice by transfer of intraepithelial lymphocytes (IELs) and CD4(+) T cells from spleen and mesenteric lymph nodes (MLNs). Three days after the transfer recipients were infected with C. parvum. IELs isolated from GKO donor mice after resolution of infection (day 15) but not at the peak of infection (day 8) significantly reduced the parasite load in recipient mice. In IL-12KO mice, IELs and also CD4(+) T cells isolated from the spleen and MLNs of donor mice at the peak of infection (day 5) and after resolution (day 15) …

CD4-Positive T-LymphocytesMaleAdoptive cell transferCryptosporidiosisSpleenHost-Parasite InteractionsInterferon-gammaMiceImmunityCell Movementparasitic diseasesmedicineMesenteric lymph nodesAnimalsImmunity MucosalCryptosporidium parvumMice KnockoutGeneral VeterinaryGeneral Immunology and MicrobiologybiologyImmunomagnetic SeparationPublic Health Environmental and Occupational Healthbiology.organism_classificationAdoptive TransferInterleukin-12Mice Inbred C57BLInfectious Diseasesmedicine.anatomical_structureCryptosporidium parvumAdoptive immunityImmunologyInterleukin 12Molecular MedicineIntraepithelial lymphocyteFemaleLymph NodesSpleenVaccine
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Induction of regulatory T cells by leflunomide in a murine model of contact allergen sensitivity.

2006

Allergic contact dermatitis and contact hypersensitivity (CHS) are characterized by allergen-specific activation of CD8 + and CD4 + T cells and the production of cytokines resulting in an inflammatory response and tissue damage. We show here that the immunosuppressive compound leflunomide ( N -[4-trifluoro-methylphenyl]-5-methylisoxazol-4 carboxamide, HWA 486) (LF) inhibited the contact allergic response induced in mice by epicutaneous application of the haptens dinitrofluorobenzene (DNFB) and oxazolone. The extent of ear swelling remained significantly reduced following repeated challenge with DNFB for up to 18 weeks. LF and DNFB had to be applied simultaneously for inhibition to occur. Th…

CD4-Positive T-LymphocytesMaleAdoptive cell transferDermatologyCD8-Positive T-Lymphocytesmedicine.disease_causeLymphocyte ActivationBiochemistryOxazolone03 medical and health scienceschemistry.chemical_compoundMice0302 clinical medicineAllergenDinitrofluorobenzenemedicineAnimalsRNA MessengerAllergic contact dermatitisMolecular Biology030304 developmental biology0303 health sciencesMice Inbred BALB Cintegumentary systemChemistryOxazoloneCell BiologyIsoxazolesAllergensmedicine.diseaseMolecular biology3. Good healthInterleukin-10Disease Models AnimalAllergic responseImmunologyDermatitis Allergic ContactCytokinesDinitrofluorobenzeneFemaleHaptenCD8Immunosuppressive AgentsLeflunomide030215 immunologyThe Journal of investigative dermatology
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