Search results for " cell transfer"

showing 10 items of 116 documents

Safe and Effective Adoptive T-Cell Receptor Transfer with a High Affinity Single Chain p53(264–272)-Specific TCR

2012

Abstract Abstract 4226 Several studies have demonstrated the clinical efficacy of adoptive T cell therapy for targeting cancer. Using HLA-A2.1 transgenic mice, we have demonstrated the feasibility of T-cell receptor (TCR) gene transfer into T cells to circumvent self-tolerance to the widely expressed human p53(264–272) tumor-associated antigen and developed approaches to generate high-affinity CD8-independent TCR. A safety concern of TCR gene transfer is the pairing of endogenous and introduced TCR chains resulting in the potential generation of self-reactive T cells (off-target autoimmunity). Several strategies to favor matched TCR chains pairing and thus enhancing TCR cell surface express…

Genetically modified mouseAdoptive cell transferGenetic enhancementT cellCD3ImmunologyT-cell receptorCell BiologyHematologyBiologyBiochemistryCell biologymedicine.anatomical_structureAntigenHumanized mouseImmunologymedicinebiology.proteinBlood
researchProduct

Cutting Edge: TGF-β Signaling Is Required for the In Vivo Expansion and Immunosuppressive Capacity of Regulatory CD4+CD25+ T Cells

2004

Abstract Data regarding the role of TGF-β for the in vivo function of regulatory CD4+CD25+ T cells (Treg) are controversial. A transgenic mouse model with impaired TGF-β signaling specifically in T cells was used to assess the role of endogenous TGF-β for the in vivo function of CD4+CD25+ Treg in a murine model of colitis induced by dextran sulfate. Transfer of wild-type, but not transgenic CD4+CD25+ Treg was found to suppress colitis in wild-type mice. In addition, by transferring CFSE-labeled CD4+CD25+ Treg we could demonstrate that endogenous TGF-β promotes the expansion of CD4+CD25+ Treg in vivo. Transgenic mice themselves developed reduced numbers of peripheral CD4+CD25+ Treg and were …

Genetically modified mouseAdoptive cell transferTransgeneImmunologychemical and pharmacologic phenomenaEndogenyBiologyT-Lymphocytes RegulatoryMiceTransforming Growth Factor betaIn vivomedicineAnimalsHumansImmunology and AllergyGenetic Predisposition to DiseaseLymphocyte CountIL-2 receptorColitisReceptorCell DifferentiationReceptors Interleukin-2hemic and immune systemsColitismedicine.diseaseAdoptive TransferCell biologyImmunologySignal TransductionThe Journal of Immunology
researchProduct

A Potent Tumor-Reactive p53-Specific Single-Chain TCR without On- or Off-Target Autoimmunity In Vivo

2018

Genetic engineering of T cells with a T cell receptor (TCR) targeting tumor antigen is a promising strategy for cancer immunotherapy. Inefficient expression of the introduced TCR due to TCR mispairing may limit the efficacy and adversely affect the safety of TCR gene therapy. Here, we evaluated the safety and therapeutic efficiency of an optimized single-chain TCR (scTCR) specific for an HLA-A2.1-restricted (non-mutated) p53(264–272) peptide in adoptive T cell transfer (ACT) models using our unique transgenic mice expressing human p53 and HLA-A2.1 that closely mimic the human setting. Specifically, we showed that adoptive transfer of optimized scTCR-redirected T cells does not induce on-tar…

Genetically modified mouseAdoptive cell transfermedicine.medical_treatmentGenetic enhancementT-LymphocytesReceptors Antigen T-CellAutoimmunityBiology03 medical and health sciencesMice0302 clinical medicineAntigenCancer immunotherapyDrug DiscoveryHLA-A2 AntigenGeneticsmedicineAnimalsHumansMolecular Biology030304 developmental biologyPharmacology0303 health sciencesT-cell receptorImmunotherapyGenetic TherapyTumor antigen030220 oncology & carcinogenesisCancer researchMolecular MedicineOriginal ArticleTumor Suppressor Protein p53
researchProduct

Facilitating matched pairing and expression of TCR chains introduced into human T cells.

2006

AbstractAdoptive transfer of T lymphocytes is a promising treatment for a variety of malignancies but often not feasible due to difficulties generating T cells that are reactive with the targeted antigen from patients. To facilitate rapid generation of cells for therapy, T cells can be programmed with genes encoding the α and β chains of an antigen-specific T-cell receptor (TCR). However, such exogenous α and β chains can potentially assemble as pairs not only with each other but also with endogenous TCR α and β chains, thereby generating αβTCR pairs of unknown specificity as well as reducing the number of exogenous matched αβTCR pairs at the cell surface. We demonstrate that introducing cy…

GeneticsAdoptive cell transferTranscription GeneticCD3T-LymphocytesImmunologyGenetic transferT-cell receptorReceptors Antigen T-CellCell BiologyHematologyT lymphocyteGene TherapyBiologyBiochemistryCell biologyCell LineAntigenCell cultureProtein Biosynthesisbiology.proteinHumansCysteineReceptorBlood
researchProduct

γδT cells elicited by CMV reactivation after allo-SCT cross-recognize CMV and leukemia.

2013

Human cytomegalovirus (CMV) infections and relapse of disease remain major problems after allogeneic stem cell transplantation (allo-SCT), in particular in combination with CMV-negative donors or cordblood transplantations. Recent data suggest a paradoxical association between CMV reactivation after allo-SCT and reduced leukemic relapse. Given the potential of Vδ2-negative γδT cells to recognize CMV-infected cells and tumor cells, the molecular biology of distinct γδT-cell subsets expanding during CMV reactivation after allo-SCT was investigated. Vδ2(neg) γδT-cell expansions after CMV reactivation were observed not only with conventional but also cordblood donors. Expanded γδT cells were ca…

Human cytomegalovirusCancer ResearchAdoptive cell transferT cellT-LymphocytesCytomegalovirusBiologyAntigenT-Lymphocyte SubsetsmedicineHomologous chromosomeHumansTransplantation HomologousLeukemiavirus diseasesReceptors Antigen T-Cell gamma-deltaHematologymedicine.diseaseTransplantationLeukemiamedicine.anatomical_structureOncologyImmunologyVirus ActivationStem cellStem Cell TransplantationLeukemia
researchProduct

Adoptive Transfer of T-Cell-Receptor Engineered Human T Cells Specifically Reduces Viral Titers in HLA-Transgenic NSG Mice Infected with a Humanized …

2014

Abstract Reactivation of latent human cytomegalovirus (HCMV) infection is a frequent complication in patients after allogeneic hematopoietic stem cell transplantation (HSCT). Preclinical research in murine models as well as clinical phase I/II trials have shown that the adoptive transfer of virus-specific CD8+ T cells is a therapeutic option for preventing HCMV disease. However, the feasibility of HCMV-specific immunotherapy is currently limited in clinical routine due to technical restrictions. It has also limitations, if the donor is HCMV-seronegative or carries only low numbers of HCMV-specific memory T cells. In this situation, grafting non-reactive T cells by virus-antigen specific T-c…

Human cytomegalovirusSevere combined immunodeficiencyAdoptive cell transfervirusesT cellmedicine.medical_treatmentImmunologyCell BiologyHematologyImmunotherapyBiologymedicine.diseaseBiochemistryVirologyCell therapymedicine.anatomical_structureImmune systemImmunologymedicineCD8Blood
researchProduct

Maintenance and Function of Human CD8+ T Cells and NK Cells in Humanized Mice

2014

Human CD8+ T lymphocytes and NK cells can be successfully engrafted in highly immuno-deficient mouse strains such as NOD/shi-SCID/γgcnull (NOG), NOD/SCID/IL2Rγnull (NSG), NOD/Rag1KO/γcnull (NRG), and BALB/c-Rag2KO/γcnull (BRG) mice following reconstitution with human CD34+ hematopoietic stem cells (HSCs) or, alternatively, upon adoptive transfer of peripheral blood mononuclear cells (PBMC). These humanized immune system (HIS) mice have evolved as a promising tool to study human CD8+ T cell and NK cell-mediated immune responses to cancer and infectious diseases and to explore new approaches in adoptive immunotherapy and vaccination. However, long-term generation of CD8+ T lymphocytes and NK …

Interleukin 21Adoptive cell transfermedicine.anatomical_structureJanus kinase 3T cellImmunologyInterleukin 12medicineCytotoxic T cellIL-2 receptorBiologyAntigen-presenting cell
researchProduct

Control of cytomegalovirus in bone marrow transplantation chimeras lacking the prevailing antigen-presenting molecule in recipient tissues rests prim…

1998

ABSTRACT Cytomegalovirus (CMV) infection during the transient immunodeficiency after bone marrow transplantation (BMT) develops into disease unless antiviral CD8 T cells are restored in due course. Histoincompatibility between donor and recipient is associated with increased risk. Complications may include a rejection response against the foreign major histocompatibility complex (MHC) antigens and a lack of antiviral control resulting from a misfit between donor-derived T cells and the antigenic viral peptides presented in recipient tissues. Here we have established a murine model of CMV disease after experimental BMT performed across a single MHC class I disparity. Specifically, BALB/c bon…

Lung DiseasesAdoptive cell transferImmunologyAntigen-Presenting CellsViral Pathogenesis and ImmunityCD8-Positive T-LymphocytesMajor histocompatibility complexMicrobiologyMajor Histocompatibility ComplexChimera (genetics)MiceAntigenVirologyMHC class ImedicineCytotoxic T cellAnimalsAntigen-presenting cellMice Inbred BALB CBone TransplantationbiologyChimeraVirologymedicine.anatomical_structureInsect ScienceImmunologyCytomegalovirus Infectionsbiology.proteinBone marrow
researchProduct

An immune escape screen reveals Cdc42 as regulator of cancer susceptibility to lymphocyte-mediated tumor suppression.

2007

Abstract Adoptive cellular immunotherapy inducing a graft-versus-tumor (GVT) effect is the therapeutic mainstay of allogeneic hematopoietic stem cell transplantation (ASCT) for high-risk leukemias. Autologous immunotherapies using vaccines or adoptive transfer of ex vivo–manipulated lymphocytes are clinically explored in patients with various cancer entities. Main reason for failure of ASCT and cancer immunotherapy is progression of the underlying malignancy, which is more prevalent in patients with advanced disease. Elucidating the molecular mechanisms contributing to immune escape will help to develop strategies for the improvement of immunologic cancer treatment. To this end, we have und…

MAPK/ERK pathwayCytotoxicity ImmunologicAdoptive cell transferTranscription GeneticMAP Kinase Signaling Systemmedicine.medical_treatmentImmunologyMolecular Sequence DataApoptosisBiologyBiochemistryMiceImmune systemCancer immunotherapyNeoplasmsmedicineCytotoxic T cellAnimalsHumansLymphocytescdc42 GTP-Binding ProteinCells CulturedBase SequenceCancerCell BiologyHematologymedicine.diseaseGene Expression Regulation NeoplasticMice Inbred C57BLCdc42 GTP-Binding ProteinProto-Oncogene Proteins c-bcl-2ImmunologyCancer cellCancer researchDisease SusceptibilityNeoplasm TransplantationBlood
researchProduct

Impact of immunosuppressive drugs on the therapeutic efficacy of ex vivo expanded human regulatory T cells

2015

Immunosuppressive drugs in clinical transplantation are necessary to inhibit the immune response to donor antigens. Although they are effective in controlling acute rejection, they do not prevent long-term transplant loss from chronic rejection. In addition, immunosuppressive drugs have adverse side effects, including increased rate of infections and malignancies. Adoptive cell therapy with human Tregs represents a promising strategy for the induction of transplantation tolerance. Phase I/II clinical trials in transplanted patients are already underway, involving the infusion of Tregs alongside concurrent immunosuppressive drugs. However, it remains to be determined whether the presence of …

Male0301 basic medicineAdoptive cell transferchemical and pharmacologic phenomenaPharmacologyBiologyT-Lymphocytes RegulatoryArticleCell therapyImmunosuppressive AgentMice03 medical and health sciences0302 clinical medicineImmune systemIn vivoAnimalsHumansCells CulturedMice KnockoutMice Inbred BALB CAnimalHematologyAdoptive TransferTacrolimusInterleukin-10TransplantationSettore MED/16 - Reumatologia030104 developmental biologyGene Expression RegulationHumanized mouseImmunologyFemaleReceptors ChemokineImmunosuppressive AgentsEx vivoHuman030215 immunologyHaematologica
researchProduct