Search results for " congenital"

showing 10 items of 179 documents

Evaluation of esophageal motility and reflux in children treated for congenital diaphragmatic hernia with the use of combined multichannel intralumin…

2011

Abstract Background Gastroesophageal reflux (GER) is frequently observed in children treated for congenital diaphragmatic hernia (CDH) at birth, as well as esophageal dysmotility, that has been hypothesized to be caused by innervatory anomalies. The aim of this study is to evaluate GER and dysmotility in young patients with CDH using pH-multichannel intraluminal impedance. Methods Thirty children (17 boys and 13 girls) who underwent repair for CDH between 2002 and 2007 with a median age of 5.2 years (range, 3-10 years) were included in the study. All patients were operated on with a subcostal laparotomy incision and had a left-sided diaphragmatic defect. The defect repair required an artifi…

Malemedicine.medical_specialtymedicine.medical_treatmentMonitoring AmbulatoryCongenital diaphragmatic herniaGastroenterologyPh monitoringPostoperative ComplicationsSwallowingInternal medicineLaparotomyElectric ImpedanceHumansMedicineEsophageal Motility DisordersChildHernia Diaphragmaticbusiness.industryIncidenceEsophageal motilitySettore MED/20 - Chirurgia Pediatrica E InfantileRefluxCongenital diaphragmatic herniaGastric Acidity DeterminationGeneral MedicineHydrogen-Ion Concentrationmedicine.diseaseDeglutitionSurgeryTreatment OutcomeEsophageal motility disorderGastroesophageal refluxIntraluminal multichannel impedanceChild PreschoolAtresiaPediatrics Perinatology and Child HealthFemaleSurgeryHernias Diaphragmatic CongenitalbusinessEsophageal motility
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A new heterozygous mutation (D196N) in the Gs alpha gene as a cause for pseudohypoparathyroidism type IA in a boy who had gallstones

2011

Background Pseudohypoparathyroidism (PHP) is characterized by hypocalcemia and hyperphosphatemia in association with an increased secretion of parathyroid hormone (PTH) due to decreased target tissue responsiveness to PTH. Patients with PHP type Ia are not only resistant to PTH, but also to other hormones that bind to receptors coupled to stimulatory G protein (Gsalpha). PHP Ia and Albright hereditary osteodystrophy (AHO) are caused by a reduced activity of the Gsalpha protein. Heterozygous inactivating Gs alpha (GNAS) gene mutations have been identified in these patients. Methods We studied a boy with PHP Ia. During follow-up the patient developed elevated liver enzyme serum levels and abd…

Malemusculoskeletal diseasesHeterozygotemedicine.medical_specialtyErythrocytesFoot Deformities CongenitalEndocrinology Diabetes and MetabolismMutation MissenseParathyroid hormoneGallstonesGene mutationHyperphosphatemiaEndocrinologyInternal medicineChromograninsGTP-Binding Protein alpha Subunits GsGNAS complex locusHumansMedicineMissense mutationnatural sciencesAmino Acid SequenceChildConserved SequencePseudohypoparathyroidismBase SequenceSequence Homology Amino Acidbiologybusiness.industryDNAExonsGallstonesmedicine.diseasePedigreeCholesterolEndocrinologyAmino Acid SubstitutionPseudohypoparathyroidismPediatrics Perinatology and Child Healthbiology.proteinbusinessHand Deformities CongenitalHormoneJournal of Pediatric Endocrinology and Metabolism
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Sex reversal from functional disomy of Xp: Prenatal and post-mortem findings.

2008

Translocations involving the short arms of the X and Y chromosomes are uncommon and are often associated with anomalies in gonadal development. Segmental duplications of the X chromosome interfere with the formation of the testis in patients with a 46,XY karyotype. The gene products located within the duplicated segment, when present in double dose, may affect on male sex development. We report on a fetus with karyotype 46,XY,der (14)t(X;14) (p10;p10)dn. Attached to chromosome 14 is the entire short arm of the X chromosome. Therefore, the fetus is affected with a disomy of Xp, resulting in complete male to female sex reversal, as well as other structural defects. To the best of our knowledg…

Malesex severalDisorders of Sex DevelopmentChromosomal translocationBiologysex reversal • Xp disomy • DAX1 gene • multiple congenital anomaliesTranslocation GeneticChromosome PaintingSettore MED/38 - Pediatria Generale E SpecialisticaPregnancyPrenatal DiagnosisGene duplicationGeneticsHumansAbnormalities MultipleGenetics (clinical)X chromosomeSex Chromosome AberrationsSegmental duplicationUltrasonographyGeneticsChromosome AberrationsChromosomes Human Pair 14FetusChromosomes Human XSex ChromosomesInfant NewbornChromosomeKaryotypeSex reversalChromosome BandingPhenotypeSettore MED/03 - Genetica MedicaKaryotypingTetralogy of FallotFemaledisomy XpDandy-Walker Syndrome
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The Leber congenital amaurosis protein AIPL1 and EB proteins co-localize at the photoreceptor cilium.

2014

Purpose The aim of this study was to investigate the interaction and co-localization of novel interacting proteins with the Leber congenital amaurosis (LCA) associated protein aryl hydrocarbon receptor interacting protein-like 1 (AIPL1). Methods The CytoTrapXR yeast two-hybrid system was used to screen a bovine retinal cDNA library. A novel interaction between AIPL1 and members of the family of EB proteins was confirmed by directed yeast two-hybrid analysis and co-immunoprecipitation assays. The localization of AIPL1 and the EB proteins in cultured cells and in retinal cryosections was examined by immunofluorescence microscopy and cryo-immunogold electron microscopy. Results Yeast two-hybri…

MiceLeber Congenital AmaurosisAnimalsHumansPhotoreceptor Cellsmacromolecular substancesCarrier ProteinsEye ProteinsMicrotubule-Associated ProteinsMicrotubulesCells CulturedAdaptor Proteins Signal TransducingResearch ArticlePloS one
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Congenital \textittoxoplasma infection: monthly prenatal screening decreases transmission rate and improves clinical outcome at age 3 years

2013

Background. Toxoplasma infection during pregnancy exposes the fetus to risks of congenital infection and sequelae that depend heavily on gestational age (GA) at time of infection. Accurate risk estimates by GA are necessary to counsel parents and improve clinical decisions. Methods. We analyzed data from pregnant women diagnosed with acute Toxoplasma infection in Lyon (France) from 1987 to 2008 and assessed how the risks of congenital toxoplasmosis and of clinical signs at age 3 years vary depending on GA at the time of maternal infection. Results. Among 2048 mother-infant pairs, 93.2% of mothers received prenatal treatment and 513 (24.7%) fetuses were infected. Because of a significant red…

Microbiology (medical)AdultMalemedicine.medical_specialtyAdolescent[SDV]Life Sciences [q-bio]030231 tropical medicinePrenatal diagnosisPrenatal careToxoplasmosis CongenitalCohort Studies03 medical and health sciencesYoung Adult0302 clinical medicinePregnancyPrenatal DiagnosismedicineHumansYoung adultPregnancy Complications Infectious0303 health sciencesPregnancy030306 microbiologybusiness.industryObstetricsInfant NewbornGestational ageInfantMiddle Agedmedicine.diseaseToxoplasmosisInfectious Disease Transmission Vertical3. Good healthInfectious DiseasesChild PreschoolGestationFemaleFrancebusinessToxoplasmosisCohort study
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The Artek cup for total hip replacement of dysplastic hip joints.

2003

One specific difficulty in total hip replacement for developmental dysplasia of the hip is a shallow acetabulum with a short anteroposterior diameter. In a prospective cohort study we investigated the short-term results of the Artek cup, designed with a shallow outline and a height-reduced metal inlay for a large size 38 mm metal head, in a group of female patients with mild dysplasia of the hip.Fourteen consecutive patients with 17 dysplastic hip joints were included in the study. Their average age was 42 years. Fourteen hips had dysplasia Crowe grade I; 3 hips had dysplasia Crowe grade II. The femoral head centre was localized according to Pagnano in zone 1 in 3 cases, in zone 3 in 6 case…

Mild DysplasiaAdultReoperationmedicine.medical_specialtymedicine.medical_treatmentArthroplasty Replacement HipBone graftingProsthesis DesignFemoral headmedicineHumansOrthopedics and Sports MedicineHip Dislocation CongenitalOrthodonticsbusiness.industryGeneral MedicineMiddle Agedmedicine.diseaseAcetabulumArthroplastySurgeryRadiographymedicine.anatomical_structureTreatment OutcomeDysplasiaHarris Hip ScoreOrthopedic surgerySurgeryFemaleHip ProsthesisbusinessFollow-Up StudiesArchives of orthopaedic and trauma surgery
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Familial mixed congenital myopathy with rigid spine phenotype

1997

We describe a father and daughter with a rigid spine syndrome and proximal myopathy. The index patient was a 42-year-old man, who died from respiratory failure after a lifelong, slowly progressive proximal myopathy and a rigid spine phenotype. This was morphologically characterized by cytoplasmic bodies, increased desmin, features of reducing-body myopathy, and sarcoplasmic and intranuclear tubulofilamentous inclusions. These cases are characterized by an early onset and possibly autosomal-dominant inheritance, with associated complex structural hallmarks of both desmin-related and inclusion body myopathies. Together they may be defined as a complex mixed congenital myopathy with a rigid sp…

Mixed congenital myopathyPathologymedicine.medical_specialtyPhysiologybusiness.industryRIGID SPINE SYNDROMEAnatomymusculoskeletal systemRigid spinePhenotypeTubulofilamentous inclusionsCellular and Molecular NeuroscienceRespiratory failurePhysiology (medical)medicineDesminNeurology (clinical)medicine.symptomMyopathybusinessMuscle & Nerve
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Unilateral multicystic dysplastic kidney in infants exposed to antiepileptic drugs during pregnancy

2007

Prenatal exposure to antiepileptic drugs (AEDs) increases the risk of major congenital malformations (MCM) in the fetus. AED-related abnormalities include heart and neural tube defects, cleft palate, and urogenital abnormalities. Among the various congenital anomalies of the kidney and urinary tract (CAKUT), multicystic dysplastic kidney (MCDK) disease is one of the most severe expressions. Although prenatal ultrasound (US) examination has increased the prenatal diagnosis of MCDK, the pathogenesis is still unclear. We report on four cases of MCDK in infants of epileptic women treated with AEDs during pregnancy. From October 2003 to June 2006, we observed four infants with unilateral MCDK bo…

Nephrologymedicine.medical_specialtyTime FactorsVoiding cystourethrogramUrinary systemMulticystic dysplastic kidneyPrenatal diagnosisUltrasonography PrenatalPregnancyInternal medicineProhibitinsmedicineHumansMulticystic Dysplastic KidneyMaternal-Fetal ExchangeFetusPregnancyEpilepsymedicine.diagnostic_testbusiness.industryObstetricsInfant Newbornmedicine.diseaseSurgeryMulticystic dysplastic kidney . Antiepileptic drugs . Major congenital anomalies . InfantCarbamazepineTreatment OutcomeNephrologyPhenobarbitalPrenatal Exposure Delayed EffectsPediatrics Perinatology and Child HealthGestationAnticonvulsantsFemalebusinessFollow-Up Studies
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Protein surplus myopathies and other rare congenital myopathies.

2002

The protein surplus myopathies have emerged as a newly recognized subgroup of morphologically defined myopathies within the spectrum of congenital myopathies because of the accumulation of protein aggregates, some of them mutant proteins. Currently, nosologic, including molecular criteria include desmin-related myopathies, actinopathies, and hereditary inclusion body myopathies, whereas hyaline body myopathy is still a putative form of protein surplus myopathy because of lack of any molecular data. The congenital myopathies (CM), foremost including nemaline and myotubular myopathies, have given evidence that, despite their epidemiologic rarity, the molecular age has dawned in CM and has eve…

Pathologymedicine.medical_specialtyAdolescentInfantHyaline bodyBiologyDesminActin CytoskeletonChild PreschoolPediatrics Perinatology and Child HealthmedicineHumansPoint MutationNeurology (clinical)medicine.symptomMyopathyChildCytoskeletonMyopathies Structural CongenitalSeminars in pediatric neurology
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Transcriptome Analysis of Ullrich Congenital Muscular Dystrophy Fibroblasts Reveals a Disease Extracellular Matrix Signature and Key Molecular Regula…

2015

Background Collagen VI related myopathies encompass a range of phenotypes with involvement of skeletal muscle, skin and other connective tissues. They represent a severe and relatively common form of congenital disease for which there is no treatment. Collagen VI in skeletal muscle and skin is produced by fibroblasts. Aims & Methods In order to gain insight into the consequences of collagen VI mutations and identify key disease pathways we performed global gene expression analysis of dermal fibroblasts from patients with Ullrich Congenital Muscular Dystrophy with and without vitamin C treatment. The expression data were integrated using a range of systems biology tools. Results were validat…

Pathologymedicine.medical_specialtyUllrich congenital muscular dystrophyIntegrin alpha3Integrinlcsh:MedicineDown-RegulationAscorbic AcidBiologyMuscular DystrophiesExtracellular matrixLamininCollagen VImedicineCell AdhesionHumansGene Regulatory NetworksMuscular dystrophylcsh:ScienceWound HealingMultidisciplinarySclerosisGene Expression Profilinglcsh:RFibroblastsmedicine.diseaseMolecular biologyExtracellular MatrixUp-RegulationGene expression profilingMicroRNAsbiology.proteinlcsh:QWound healingResearch ArticleSignal Transduction
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