Search results for " deletion"

showing 10 items of 361 documents

FISH mapping of the sex-reversal region on human chromosome 9p in two XY females and in primates

2000

Accumulating evidence suggests that haploinsufficiency of a dosage-sensitive gene(s) in human chromosome 9p24.3 is responsible for the failure of testicular development and feminisation in XY patients with monosomy for 9p. We have used molecular cytogenetic methods to characterise the sex-reversing 9p deletions in two XY females. Fluorescence in situ hybridisation (FISH) with YACs from the critical 9p region containing an evolutionarily conserved sex-determining gene, DMRT1, is a very fast and reliable assay for patient screening. Comparative YAC mapping on great ape and Old and New World monkey chromosomes demonstrated that the critical region was moved from an interstitial position on the…

MonosomyX ChromosomeDisorders of Sex DevelopmentChromosome BreakpointsChromosomal translocationBiologyY chromosomePolymerase Chain ReactionTranslocation GeneticY ChromosomeGeneticsmedicineAnimalsHumansChromosomes Artificial YeastIn Situ Hybridization FluorescenceGenetics (clinical)X chromosomeChromosomal inversionGeneticsChromosome MappingChromosomeKaryotypemedicine.diseaseCebidaeKaryotypingFemaleChromosome DeletionChromosomes Human Pair 9Transcription FactorsEuropean Journal of Human Genetics
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Reconstructing the ancestor of Mycobacterium leprae: The dynamics of gene loss and genome reduction

2007

We have reconstructed the gene content and order of the last common ancestor of the human pathogens Mycobacterium leprae and Mycobacterium tuberculosis. During the reductive evolution of M. leprae, 1537 of 2977 ancestral genes were lost, among which we found 177 previously unnoticed pseudogenes. We find evidence that a massive gene inactivation took place very recently in the M. leprae lineage, leading to the loss of hundreds of ancestral genes. A large proportion of their nucleotide content (∼89%) still remains in the genome, which allowed us to characterize and date them. The age of the pseudogenes was computed using a new methodology based on the rates and patterns of substitution in the…

Most recent common ancestorGeneticsLetterLineage (genetic)PseudogeneComputational BiologyMycobacterium tuberculosisBiologybiology.organism_classificationGenomeEvolution MolecularMycobacterium lepraeMycobacterium tuberculosisPhylogeneticsGeneticsDNA FungalMycobacterium lepraeGeneGene DeletionGenome BacterialPhylogenyGenetics (clinical)Genome Research
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The Candida albicans pH-regulated KER1 gene encodes a lysine/glutamic-acid-rich plasma-membrane protein that is involved in cell aggregation.

2004

Immunoscreening of aCandida albicanscDNA library with a polyclonal germ-tube-specific antibody (pAb anti-gt) resulted in the isolation of a gene encoding a lysine/glutamic-acid-rich protein, which was consequently designatedKER1. The nucleotide and deduced amino acid sequences of this gene displayed no significant homology with any other known sequence.KER1encodes a 134 kDa lysine (14·5 %)/glutamic acid (16·7 %) protein (Ker1p) that contains two potential transmembrane segments.KER1was expressed in a pH-conditional manner, with maximal expression at alkaline pH and lower expression at pH 4·0, and was regulated byRIM101. A Δker1/Δker1null mutant grew normally but was hyperflocculant under ge…

MutantLysineGenes FungalMolecular Sequence DataGlutamic AcidMicrobiologyFungal ProteinsMiceImmunoscreeningComplementary DNAGene Expression Regulation FungalCandida albicansAnimalsCloning MolecularCandida albicansDNA Fungalchemistry.chemical_classificationbiologyBase SequenceVirulenceLysineMembrane ProteinsHydrogen-Ion Concentrationbiology.organism_classificationMolecular biologyTransmembrane proteinAmino acidPhenotypechemistryBiochemistryPolyclonal antibodiesMice Inbred DBAbiology.proteinGene DeletionSubcellular FractionsMicrobiology (Reading, England)
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Chromosome 1 abnormalities in myeloid malignancies: a literature survey and karyotype-phenotype associations.

2009

Chromosome 1 is the largest human chromosome and contains over 1600 known genes and 1000 novel coding sequences or transcripts. It is, therefore, not surprising that recurrent chromosome 1 abnormalities are regularly encountered in both neoplastic and non-neoplastic medical conditions. The current review is focused on myeloid malignancies where we summarize the relevant published literature and discuss specific karyotype-phenotype associations. We show that chromosome 1 abnormalities are most frequent in BCR-ABL-negative classic myeloproliferative neoplasms (MPN): polycythemia vera (PV), essential thrombocythemia (ET), and primary myelofibrosis (PMF). Specific abnormalities include duplicat…

MyeloidChromosomal translocationBiologyTranslocation GeneticSettore MED/15 - Malattie Del Sanguehemic and lymphatic diseasesmedicineHumansGenes Tumor SuppressorMyelofibrosisGeneticsChromosome AberrationsMyeloproliferative DisordersEssential thrombocythemiaMyelodysplastic syndromesMyeloid leukemiaKaryotypeHematologyGeneral MedicineOncogenesmedicine.diseasemedicine.anatomical_structurePhenotypeChromosomes Human Pair 1Leukemia MyeloidKaryotypingMyelodysplastic Syndromeschomosome 1 myeloid malignancyChromosome DeletionLiterature surveyEuropean journal of haematology
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Identification of a novel recurrent 1q42.2-1qter deletion in high risk MYCN single copy 11q deleted neuroblastomas

2012

Neuroblastoma is an aggressive embryonal tumor that accounts for similar to 15% of childhood cancer deaths. Hitherto, despite the availability of comprehensive genomic data on DNA copy number changes in neuroblastoma, relatively little is known about the genes driving neuroblastoma tumorigenesis. In this study, high resolution array comparative genome hybridization (CGH) was performed on 188 primary neuroblastoma tumors and 33 neuroblastoma cell lines to search for previously undetected recurrent DNA copy number gains and losses. A new recurrent distal chromosome 1q deletion (del(1)(q42.2qter)) was detected in seven cases. Further analysis of available array CGH datasets revealed 13 additio…

Neuroblastoma/geneticsCancer ResearchProcollagen-Proline DioxygenaseMedizinGene Dosagecomparative genomic hybridizationBiologymedicine.disease_causeGene dosageN-Myc Proto-Oncogene ProteinFumarate HydrataseHypoxia-Inducible Factor-Proline DioxygenasesNeuroblastomaProcollagen-Proline Dioxygenase/geneticsCell Line TumorNeuroblastomamedicineHumansFumarate Hydratase/geneticsGeneOncogene ProteinsGeneticsN-Myc Proto-Oncogene ProteinChromosomes Human Pair 11BreakpointNuclear ProteinsChromosomemedicine.diseaseOncogene Proteins/geneticsNuclear Proteins/geneticsOncologyChromosome DeletionCarcinogenesisComparative genomic hybridization
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A nonsense mutation abrogates production of a functional enterotoxin A in Clostridium difficile toxinotype VIII strains of serogroups F and X.

1999

Clostridium difficile strains of toxinotype VIII from serogroups F and X are described as toxin B-positive, toxin A-negative (TcdB+ A-), although they harbour almost the entire tcdA gene. To identify the reason for the lack of TcdA detection, we analyzed catalytic and ligand domains of TcdA-1470 of the type strain of serogroup F, strain 1470. Using recombinant fragments, the C-terminal immunodominant ligand domain TcdA3-1470, spanning amino acid residues 1694-2711 (corresponding to VPI 10463 sequence), was detected in Western blots. Similar experiments using the recombinant N-terminal catalytic fragment TcdAc1-2-1470 (amino acid positions 1-544) failed. In addition, this fragment showed no …

Nonsense mutationBlotting WesternMutation MissenseEnterotoxinBiologymedicine.disease_causeMicrobiologyMicrobiologylaw.inventionEnterotoxinsBacterial ProteinslawCatalytic DomainGeneticsmedicineMissense mutationHumansMolecular BiologyGenechemistry.chemical_classificationMutationClostridioides difficileMolecular biologyStop codonPeptide FragmentsRecombinant ProteinsAmino acidchemistryGenes BacterialRecombinant DNAGene DeletionFEMS microbiology letters
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Illumina-based RiboMethSeq approach for mapping of 2'-O-Me residues in RNA

2016

International audience; RNA 2'-O-methylation is one of the ubiquitous nucleotide modifications found in many RNA types from Bacteria, Archaea and Eukarya. RNAs bearing 2'-O-methylations show increased resistance to degradation and enhanced stability in helices. While the exact role of each 2'-O-Me residue remained elusive, the catalytic protein Fibrillarin (Nop1 in yeast) responsible for 2'-O-methylation in eukaryotes, is associated with human pathologies. Therefore, there is an urgent need to precisely map and quantify hundreds of 2'-O-Me residues in RNA using high-throughput technologies. Here, we develop a reliable protocol using alkaline fragmentation of total RNA coupled to a commonly …

NucleotidesSequence Analysis RNAOligonucleotidesReproducibility of ResultsRNA FungalSaccharomyces cerevisiae22MethylationMethods OnlineRNA Small Nucleolar[SDV.BBM.BC]Life Sciences [q-bio]/Biochemistry Molecular Biology/Biochemistry [q-bio.BM]Gene Deletion[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyGene Library
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The loss of the inducible Aspergillus carbonarius MFS transporter MfsA leads to ochratoxin A overproduction

2014

Ochratoxin A (OTA), a nephrotoxic compound produced by certain Aspergillus and Penicillium species, is one of the most abundant mycotoxins in food commodities. Aspergillus carbonarius is the main source of OTA in wine, grape juice and dried vine fruits. Although many studies have focused on OTA production by A. carbonarius, little is known about the genes related to OTA production and transport. We have found a transporter that belongs to the major facilitator superfamily (MfsA) which is highly expressed with a 102-fold induction in an ochratoxigenic A. carbonarius strain compared to a low OTA producer strain. The encoding mfsA gene shows similarity to the multidrug efflux transporter flu1 …

Ochratoxin AOchratoxin productionGrapesGenes FungalMolecular Sequence DataWineAspergillus carbonariusBiologyMicrobiologyMicrobiologychemistry.chemical_compoundGene Expression Regulation FungalmfsAPromoter Regions GeneticOverproductionCandida albicansMycotoxinMajor facilitator superfamily transportersAspergillusBinding SitesBase Sequencefood and beveragesTransporterGeneral Medicinebiology.organism_classificationOchratoxinsMajor facilitator superfamilyOxidative StressAspergilluschemistryFruitEffluxGene DeletionFood ScienceInternational Journal of Food Microbiology
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Perfil genómico del neuroblastoma de alto riesgo mediante hibridación genómica comparada

2006

El neuroblastoma presenta alteraciones genéticas que predicen su evolución clínica. Ganancias cromosómicas completas están asociadas a estadios clínicos no avanzados y evolución favorable, mientras que pérdidas de 1p, ganancia de 17q y amplificación del gen MYCN (MNA) son indicativas de estadios clínicos avanzados y pronóstico desfavorable. Son neuroblastomas de alto riesgo (NB-HR) los presentes en niños mayores de un año: estadio 4 o MNA en cualquier estadio de enfermedad, excluyendo estadio 1. El pronóstico de estos enfermos es malo, incluso con tratamientos agresivos. Sólo MNA confiere valor pronóstico negativo. Se remitieron al Centro de Referencia Nacional del neuroblastoma 60 casos de…

OncologyComparative genomic hybridizationmedicine.medical_specialtyBiological studiesmedicine.diagnostic_testbusiness.industryFluorescence in situ hybridizationClinical courseIn situ hybridizationmedicine.diseasePediatricsRJ1-570NeuroblastomaInternal medicineNeuroblastomaMYCNPediatrics Perinatology and Child Healthmedicine1p DeletionStage (cooking)businessFluorescence in situ hybridizationComparative genomic hybridizationAnales de Pediatría
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Response to lenalidomide in myelodysplastic syndromes with del(5q): influence of cytogenetics and mutations.

2012

Summary Lenalidomide is an effective drug in low-risk myelodysplastic syndromes (MDS) with isolated del(5q), although not all patients respond. Studies have suggested a role for TP53 mutations and karyotype complexity in disease progression and outcome. In order to assess the impact of complex karyotypes on treatment response and disease progression in 52 lenalidomide-treated patients with del(5q) MDS, conventional G-banding cytogenetics (CC), single nucleotide polymorphism array (SNP-A), and genomic sequencing methods were used. SNP-A analysis (with control sample, lymphocytes CD3+, in 30 cases) revealed 5q losses in all cases. Other recurrent abnormalities were infrequent and were not ass…

OncologyMalemedicine.medical_specialtyMultivariate analysisCD3Single Nucleotide Polymorphism ArrayBiologyPolymorphism Single NucleotideInternal medicinemedicineHumansImmunologic FactorsPlateletLenalidomideIn Situ Hybridization FluorescenceLenalidomideAgedAged 80 and overMyelodysplastic syndromesCytogeneticsKaryotypeHematologyMiddle Agedmedicine.diseaseChromosome BandingThalidomideTreatment OutcomeMyelodysplastic SyndromesImmunologyMutationbiology.proteinDisease ProgressionChromosomes Human Pair 5FemaleChromosome Deletionmedicine.drugBritish journal of haematology
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