Search results for " endothelial cell"

showing 10 items of 177 documents

Oxidative stress-driven expression of myeloperoxidase in endothelial cells results in accumulation of markers of oxidative and nitrosative stress in …

2009

Endothelial cells (ECs), are able to manage with higher concentrations of ROS as well as reactive nitrogen species, respect to the other cell types. The unbalance between oxidizing species and antioxidant cellular defences has been implicated in the pathogenesis of cardiovascular diseases. While the role of MPO in the oxidative burst is well established, the effects of this enzyme on endothelial biology have only recently come to light. MPO can interact with endothelial –derived NO causing its depletion and favouring its conversion to nitrogen dioxide radical, a compound responsible of protein nitration generating 3-nitrotyrosine. Moreover, MPO is the only enzyme capable to form hypoclorous…

Settore BIO/16 - Anatomia Umanamyeloperoxidase endothelial cells endocardium oxidative stress nitrosative stress hydrogen peroxide 3-chlorotyrosine 3-nitrotyrosine
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eNOS Activation by HDL Is Impaired in Genetic CETP Deficiency.

2014

Mutations in the CETP gene resulting in defective CETP activity have been shown to cause remarkable elevations of plasma HDL-C levels, with the accumulation in plasma of large, buoyant HDL particles enriched in apolipoprotein E. Genetic CETP deficiency thus represents a unique tool to evaluate how structural alterations of HDL impact on HDL atheroprotective functions. Aim of the present study was to assess the ability of HDL obtained from CETP-deficient subjects to protect endothelial cells from the development of endothelial dysfunction. HDL isolated from one homozygous and seven heterozygous carriers of CETP null mutations were evaluated for their ability to down-regulate cytokine-induced…

Settore MED/09 - Medicina InternaCHOLESTEROL EFFLUXApolipoprotein BEpidemiologylcsh:MedicineANTIINFLAMMATORY PROPERTIESmedicine.disease_causeBiochemistryVascular Medicinechemistry.chemical_compoundHigh-density lipoproteinEnosMedicine and Health SciencesEndothelial dysfunctionlcsh:ScienceMutationMultidisciplinarybiologyHomozygoteCETP; eNOS; HDL;NeurochemistryLipidsGenetic EpidemiologyeNOSlipids (amino acids peptides and proteins)AnatomyNeurochemicalsLipoproteins HDLResearch Articlemedicine.medical_specialtyDrug Research and DevelopmentHDLNitric Oxide Synthase Type IIILipoproteinsENDOTHELIAL FUNCTIONINHIBITIONCardiologyDown-RegulationVascular Cell Adhesion Molecule-1Nitric OxideCELL-ADHESION MOLECULE-1Lipid Metabolism Inborn ErrorsESTER TRANSFER PROTEINInternal medicineCETPCholesterylester transfer proteinHuman Umbilical Vein Endothelial CellsmedicineHumansNITRIC-OXIDE SYNTHASEInflammationClinical GeneticsPharmacologyCholesterollcsh:RTorcetrapibEndothelial CellsBiology and Life SciencesProteinsnutritional and metabolic diseasesLipid MetabolismAtherosclerosismedicine.diseasebiology.organism_classificationCholesterol Ester Transfer Proteinscarbohydrates (lipids)MetabolismEndocrinologychemistryOther Clinical MedicineMutationImmunologyCardiovascular Anatomybiology.proteinlcsh:QTORCETRAPIBClinical MedicineHIGH-DENSITY-LIPOPROTEINSCAVENGER RECEPTOR BI
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LC–ESI–FT–MSn Metabolite Profiling of Symphytum officinale L. Roots Leads to Isolation of Comfreyn A, an Unusual Arylnaphthalene Lignan

2020

Preparations of comfrey (Symphytum officinale L.) roots are used topically to reduce inflammation. Comfrey anti-inflammatory and analgesic properties have been proven in clinical studies. However, the bioactive compounds associated with these therapeutic activities are yet to be identified. An LC&ndash

Spectrometry Mass Electrospray Ionizationcomfrey rootsMetaboliteAnti-Inflammatory AgentsComfreySymphytum officinalePlant Roots01 natural sciencescomfreyn AArticleCatalysisUmbilical veinInorganic Chemistrylcsh:Chemistrychemistry.chemical_compoundLC–ESI–Orbitrap–MSComfreyHuman Umbilical Vein Endothelial CellsSymphytum officinaleHumans<i>Symphytum officinale</i>Physical and Theoretical ChemistryGloboidnan AMolecular Biologylcsh:QH301-705.5SpectroscopyLignanPlants MedicinalChromatographyMolecular StructurebiologyChemistry010401 analytical chemistryOrganic ChemistryComfrey roots; Comfreyn A; LC–ESI–Orbitrap–MS; Phenylpropanoids; Symphytum officinaleGeneral Medicinebiology.organism_classification0104 chemical sciencesComputer Science Applications010404 medicinal & biomolecular chemistrylcsh:Biology (General)lcsh:QD1-999Metabolite profilingTwo-dimensional nuclear magnetic resonance spectroscopyChromatography LiquidphenylpropanoidsInternational Journal of Molecular Sciences
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Paracrine effect of membrane vesicles released by mouse mesoangioblast stem cells on non correlated cell types

2016

Introduction Mouse mesoangioblasts are vessel-associated multipotent progenitor stem cells, which are able to differentiate into different mesodermal cell types. In our previous paper we have demonstrated that these cells are able to shed in the extracellular environment membrane vesicles (EV), which contain both structural proteins and biological factors such as FGF2 and the two gelatinases MMP2/9. EV represent an important mediator of cell-to-cell communication and are involved in both autocrine and paracrine signalling. Interestingly, there is a bidirectional signalling exchange between stem cell EV and damaged cells. In particular, EV from injured cells can reprogram stem cells to acqui…

Stem cells mesoangioblasts membrane vesicles migration. macrophages endothelial cells.
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Gold(I) Biscarbene Complexes Derived from Vascular-Disrupting Combretastatin A-4 Address Different Targets and Show Antimetastatic Potential

2014

Gold N-heterocyclic carbene (NHC) complexes are an emerging class of anticancer drugs. We present a series of gold(I) biscarbene complexes with NHC ligands derived from the plant metabolite combretastatin A-4 (CA-4) that retain its vascular-disrupting effect, yet address different cellular and protein targets. Unlike CA-4, these complexes did not interfere with tubulin, but with the actin cytoskeleton of endothelial and cancer cells. For the highly metastatic 518A2 melanoma cell line this effect was accompanied by a marked accumulation of cells in the G1 phase of the cell cycle and a suppression of active prometastatic matrix metalloproteinase-2. Despite these mechanistic differences the co…

StereochemistryNeovascularization PhysiologicAntineoplastic AgentsBiologyBiochemistryMicechemistry.chemical_compoundCoordination ComplexesTubulinCell Line TumorBibenzylsDrug DiscoveryHuman Umbilical Vein Endothelial CellsAnimalsHumansGeneral Pharmacology Toxicology and PharmaceuticsMelanomaCell ProliferationPharmacologyCombretastatin A-4Tube formationCombretastatinMice Inbred BALB COrganic ChemistryCell cycleActin cytoskeletonG2 Phase Cell Cycle CheckpointsActin CytoskeletonChorioallantoic membranechemistryDrug Resistance NeoplasmCell cultureCancer cellMCF-7 CellsCancer researchMolecular MedicineGoldHT29 CellsMethaneChemMedChem
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Permeability properties of a three-cell type in vitro model of blood-brain barrier.

2005

We previously found that RBE4.B brain capillary endothelial cells (BCECs) form a layer with blood‐brain barrier (BBB) properties if co‐cultured with neurons for at least one week. As astrocytes are known to modulate BBB functions, we further set a culture system that included RBE4.B BCECs, neurons and astrocytes. In order to test formation of BBB, we measured the amount of (3)H‐sucrose able to cross the BCEC layer in this three‐cell type model of BBB. Herein we report that both neurons and astrocytes induce a decrease in the permeability of the BCEC layer to sucrose. These effects are synergic as if BCECs are cultured with both neurons and astrocytes for 5 days, permeability to sucrose decr…

SucroseCell typeTime FactorsBlotting WesternVascular permeabilityBiologyBlood–brain barrierOccludinArticleCapillary PermeabilityOccludinmedicineAnimalsRats WistarCell Line TransformedNeuronsBrainEndothelial CellsMembrane ProteinsCell BiologyPermeationblood-brain barrier cortical neurons astrocytes brain capillary endothelial cells RBE4.B occludin.Coculture TechniquesRatsmedicine.anatomical_structurenervous systemMembrane proteinBiochemistryBlood-Brain BarrierPermeability (electromagnetism)Astrocytescardiovascular systembiology.proteinBiophysicsMolecular MedicineAntibody
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Gold nanoparticle interactions with endothelial cells cultured under physiological conditions

2017

PEGylated gold nanoparticles (AuNPs) have an extended circulation time after intravenous injection in vivo and exhibit favorable properties for biosensing, diagnostic imaging, and cancer treatment. No impact of PEGylated AuNPs on the barrier forming properties of endothelial cells (ECs) has been reported, but recent studies demonstrated that unexpected effects on erythrocytes are observed. Almost all studies to date have been with static-cultured ECs. Herein, ECs maintained under physiological cyclic stretch and flow conditions and used to generate a blood-brain barrier model were exposed to 20 nm PEGylated AuNPs. An evaluation of toxic effects, cell stress, the release profile of pro-infla…

SwineBiomedical EngineeringNanoparticleNanotechnology02 engineering and technology010402 general chemistryBlood–brain barrier01 natural sciencesPolyethylene GlycolsIn vivoHuman Umbilical Vein Endothelial CellsMedicineAnimalsHumansGeneral Materials ScienceParticle SizeCells Culturedbusiness.industryEndothelial Cells021001 nanoscience & nanotechnologyQPR10104 chemical sciencesCancer treatmentCell stressmedicine.anatomical_structureColloidal goldBlood-Brain BarrierBiophysicsNanoparticlesCirculation timeGold0210 nano-technologybusinessBlood stream
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Indicaxanthin from

2018

Oxidized low-density lipoproteins (oxLDL) play a pivotal role in the etiopathogenesis of atherosclerosis through the activation of inflammatory signaling events eventually leading to endothelial dysfunction and senescence. In the present work, we investigated the effects of indicaxanthin, a bioavailable, redox-modulating phytochemical from Opuntia ficus indica fruits, with anti-inflammatory activity, against oxLDL-induced endothelial dysfunction. Human umbilical vein cord cells (HUVEC) were stimulated with human oxLDL, and the effects of indicaxanthin were evaluated in a range between 5 and 20 μM, consistent with its plasma level after a fruit meal (7 μM). Pretreatment with indicaxanthin si…

Transcription GeneticCell SurvivalPyridinesNF-kappa BOpuntiaHydrogen PeroxideReactive Nitrogen SpeciesThiobarbituric Acid Reactive SubstancesBetaxanthinsUp-RegulationLipoproteins LDLHuman Umbilical Vein Endothelial CellsHumansRNA MessengerReactive Oxygen SpeciesCell Adhesion MoleculesOxidation-ReductionATP Binding Cassette Transporter 1Research ArticleOxidative medicine and cellular longevity
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Inflammation Determines the Pro-Adhesive Properties of High Extracellular D-Glucose in Human Endothelial Cells In Vitro and Rat Microvessels In Vivo

2010

BACKGROUND: Hyperglycemia is acknowledged as an independent risk factor for developing diabetes-associated atherosclerosis. At present, most therapeutic approaches are targeted at a tight glycemic control in diabetic patients, although this fails to prevent macrovascular complications of the disease. Indeed, it remains highly controversial whether or not the mere elevation of extracellular D-glucose can directly promote vascular inflammation, which favors early pro-atherosclerotic events. METHODS AND FINDINGS: In the present work, increasing extracellular D-glucose from 5.5 to 22 mmol/L was neither sufficient to induce intercellular adhesion molecule-1 (ICAM-1) and vascular cell adhesion mo…

Umbilical VeinsEndotheliumCardiovascular Disorders/Coronary Artery Diseaselcsh:MedicineInflammationIn vivoDiabetes mellitusCell AdhesionmedicineExtracellularAnimalsHumansLeukocyte RollingCardiovascular Disorders/Vascular BiologyUNESCO::CIENCIAS DE LA VIDA::Microbiología ::Otraslcsh:ScienceCell adhesionInflammationMultidisciplinaryInflammation; Pro-adhesive properties; High extracellular D-glucose; Human endothelial cells In Vitro; Rat microvessels In VivoDose-Response Relationship DrugChemistrylcsh:RCardiovascular Disorders/Peripheral Vascular DiseaseAdhesivenessEndothelial CellsChemotaxismedicine.diseaseIn vitroRatsChemotaxis LeukocyteDiabetes and EndocrinologyCell Biology/Cell AdhesionGlucosemedicine.anatomical_structureHyperglycemiaMicrovesselsImmunologyCancer researchlcsh:QEndothelium Vascularmedicine.symptomResearch Article
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Enzymatically modified, nonoxidized LDL induces selective adhesion and transmigration of monocytes and T-lymphocytes through human endothelial cell m…

1999

Abstract —Circulating monocytes and T lymphocytes extravasate through the endothelium at sites of developing atheromatous lesions, where they tend to accumulate and mediate the progression of the disease. We have previously demonstrated the presence of an enzymatically degraded, nonoxidized form of LDL (E-LDL) in early human fatty streaks, which possesses major biological properties of an atherogenic lipoprotein. The effects of E-LDL on human endothelial cells have now been studied with respect to adhesion and transmigration of monocytes and T lymphocytes. E-LDL induced a rapid and dose-dependent selective adhesion of monocytes and T lymphocytes to endothelial cell monolayers within 30 min…

Umbilical VeinsP-selectinArteriosclerosisT-LymphocytesIntercellular Adhesion Molecule-1HL-60 CellsBiologyMonocytesMuscle Smooth VascularCell MovementE-selectinmedicineCell AdhesionHumansLymphocyte homing receptorCell adhesionDose-Response Relationship DrugMonocyteT lymphocyteCholesterol LDLIntercellular Adhesion Molecule-1Molecular biologyEndothelial stem cellLipoproteins LDLPlatelet Endothelial Cell Adhesion Molecule-1KineticsP-Selectinmedicine.anatomical_structureImmunologybiology.proteinlipids (amino acids peptides and proteins)Endothelium VascularCardiology and Cardiovascular MedicineE-SelectinArteriosclerosis, thrombosis, and vascular biology
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