Search results for " epigenetic"

showing 10 items of 162 documents

A new epigenetic cross-talk links effete and iswi in determining chromatin structure

2011

effete ISWI epigenetics
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The Epigenetics of the Endocannabinoid System

2020

The endocannabinoid system (ES) is a cell-signalling system widely distributed in biological tissues that includes endogenous ligands, receptors, and biosynthetic and hydrolysing machineries. The impairment of the ES has been associated to several pathological conditions like behavioural, neurological, or metabolic disorders and infertility, suggesting that the modulation of this system may be critical for the maintenance of health status and disease treatment. Lifestyle and environmental factors can exert long-term effects on gene expression without any change in the nucleotide sequence of DNA, affecting health maintenance and influencing both disease load and resistance. This potentially …

endocannabinoids; endocannabinoid system; epigenetics; Δ9THC; DNA methylation; histone modifications; non coding RNA; reproduction; spermatozoaReviewDiseaseEnvironmentnon coding RNACatalysisEpigenesis Geneticlcsh:ChemistryreproductionInorganic ChemistryStress PhysiologicalΔ9THCspermatozoaGene expressionAnimalsHumansEpigeneticsendocannabinoidsendocannabinoid systemPhysical and Theoretical ChemistryReceptors Cannabinoidlcsh:QH301-705.5Molecular BiologySpectroscopyDNA methylationepigeneticsbiologyhistone modificationsOrganic ChemistryCannabinoid Receptor AgonistsGeneral MedicineNon-coding RNAEndocannabinoid systemComputer Science ApplicationsCell biologyHistonelcsh:Biology (General)lcsh:QD1-999DNA methylationδ<sup>9</sup>thcbiology.proteinInternational Journal of Molecular Sciences
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Resveratrol post-transcriptionally regulates pro-inflammatory gene expression via regulation of KSRP RNA binding activity

2014

Resveratrol shows beneficial effects in inflammation-based diseases like cancer, cardiovascular and chronic inflammatory diseases. Therefore, the molecular mechanisms of the anti-inflammatory resveratrol effects deserve more attention. In human epithelial DLD-1 and monocytic Mono Mac 6 cells resveratrol decreased the expression of iNOS, IL-8 and TNF-α by reducing mRNA stability without inhibition of the promoter activity. Shown by pharmacological and siRNA-mediated inhibition, the observed effects are SIRT1-independent. Target-fishing and drug responsive target stability experiments showed selective binding of resveratrol to the RNA-binding protein KSRP, a central post-transcriptional regul…

endocrine system diseasesMRNA destabilizationRNA Stabilityp38 mitogen-activated protein kinasesGene ExpressionRNA-binding proteinResveratrolBiologyp38 Mitogen-Activated Protein KinasesMicechemistry.chemical_compoundCell Line TumorStilbenesGene expressionGeneticsAnimalsHumansddc:610RNA Messengerskin and connective tissue diseasesMice KnockoutMessenger RNAGene knockdownExosome Multienzyme Ribonuclease Complexorganic chemicalsAnti-Inflammatory Agents Non-SteroidalGene regulation Chromatin and EpigeneticsRNA-Binding Proteinsfood and beveragesMolecular biology3. Good healthCell biologychemistryResveratrolMutationTrans-ActivatorsPhosphorylationInflammation Mediatorshormones hormone substitutes and hormone antagonistsNucleic Acids Research
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Does Arsenic Contamination Affect DNA Methylation Patterns in a Wild Bird Population? : An Experimental Approach

2021

Pollutants, such as toxic metals, negatively influence organismal health and performance, even leading to population collapses. Studies in model organisms have shown that epigenetic marks, such as DNA methylation, can be modulated by various environmental factors, including pollutants, influencing gene expression, and various organismal traits. Yet experimental data on the effects of pollution on DNA methylation from wild animal populations are largely lacking. We here experimentally investigated for the first time the effects of early-life exposure to environmentally relevant levels of a key pollutant, arsenic (As), on genome-wide DNA methylation in a wild bird population. We experimentall…

environmental epigeneticsPopulationAnimals Wild010501 environmental sciencesBiology01 natural sciencesArticleArsenicecotoxicologyarseeniBirdsParus majorGene expressionEnvironmental ChemistryAnimalspollutionhaitalliset aineetEpigeneticseducationGene0105 earth and related environmental sciencesZinc fingerGeneticseducation.field_of_studyPlan_S-Compliant-TAympäristön saastuminenGeneral ChemistrytalitiainenDNA Methylation3. Good healthDNA-metylaatioArsenic contamination of groundwaterekotoksikologiaCpG site13. Climate actionepigenetiikkavillieläimetinternationalDNA methylationecological epigeneticsEnvironmental Pollutants
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FRUIT INHIBITS FLOWERING IN ALTERNATE BEARING CITRUS VARIETIES. HORMONAL, GENETIC AND EPIGENETIC REGULATION

In mature citrus trees, flowering is regulated by exogenous signals (i.e., low/high temperature, water shortage/water supply) that control flowering time, and by endogenous signals that control flower intensity. Actually, the ratio of endogenous promoters to inhibitors is considered as responsible for flowering; however, there is evidence suggesting that the inhibitors and their metabolism are the sole factors controlling flowering in Citrus. All the meristems have the information and ability to flower unless a negative factor hampers the process. The main endogenous factor controlling flower intensity is the fruit. Attending to the aforementioned, in this PhD. thesis, the following hypothe…

genetic; epigenetic;flowering ; GA; ABA; CiFT2; FLC TEM1 SVP [Citrus]Settore AGR/03 - Arboricoltura Generale E Coltivazioni ArboreefloweringABAGACitrus: geneticCiFT2FLC TEM1 SVPepigenetic
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Possible mechanisms of Raf-1 Kinase Inhibitor Protein down-regulation in hepatocellular carcinoma

2011

hepatocellular carcinoma HDACi epigenetic changes.RKIP (Raf-1 Kinase Inhibitor Protein)Settore BIO/14 - Farmacologia
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Quinoline anticancer agents active on DNA and DNA-interacting proteins: From classical to emerging therapeutic targets.

2021

Quinoline is one of the most important and versatile nitrogen heterocycles embodied in several biologically active molecules. Within the numerous quinolines developed as antiproliferative agents, this review is focused on compounds interfering with DNA structure or with proteins/enzymes involved in the regulation of double helix functional processes. In this light, a special focus is given to the quinoline compounds, acting with classical/well-known mechanisms of action (DNA intercalators or Topoisomerase inhibitors). In particular, the quinoline drugs amsacrine and camptothecin (CPT) have been studied as key lead compounds for the development of new agents with improved PK and tolerability…

medicine.drug_classAntineoplastic Agents01 natural sciences03 medical and health scienceschemistry.chemical_compoundDrug DiscoverymedicineHumansAmsacrine030304 developmental biologyCell ProliferationPharmacology0303 health sciencesDNA Intercalators G-quadruplex Topoisomerase Epigenetic targets Antiproliferative compounds SAR studiesbiologyMolecular Structure010405 organic chemistryTopoisomeraseOrganic ChemistryQuinolineGeneral MedicineDNA NeoplasmSettore CHIM/08 - Chimica Farmaceutica0104 chemical sciencesDNA-Binding ProteinsG-QuadruplexesHistonechemistryBiochemistrybiology.proteinQuinolinesHistone deacetylaseCamptothecinDNATopoisomerase inhibitormedicine.drugEuropean journal of medicinal chemistry
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RNA memory

2011

In the last decade non-coding RNAs (ncRNAs) have emerged as cellular key regulators. The attention of the scientific community has focused on ncRNAs with repressive features on eukaryotic transcriptional regulation. Many experimental evidences suggest that ncRNAs could also positively regulate transcription. The RNA-Memory Model (Arancio W. Rejuvenation Res. 2010 Apr-Jun;13(2-3):365-72.) gives possible explanations to several biological phenomena via trans-acting ncRNAs (memRNAs) able to orchestrate chromatin remodelling and in turn enhance transcription. memRNAs assert their functions especially during the post-mitotic chromatin remodelling. memRNAs can mark the genes transcribed in the mo…

ncRNAs epigenetics
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Epigenetic siRNA and Chemical Screens Identify SETD8 Inhibition as a Therapeutic Strategy for p53 Activation in High-Risk Neuroblastoma

2017

Given the paucity of druggable mutations in high-risk neuroblastoma (NB), we undertook chromatin-focused small interfering RNA and chemical screens to uncover epigenetic regulators critical for the differentiation block in high-risk NB. High-content Opera imaging identified 53 genes whose loss of expression led to a decrease in NB cell proliferation and 16 also induced differentiation. From these, the secondary chemical screen identified SETD8, the H4K20me1 methyltransferase, as a druggable NB target. Functional studies revealed that SETD8 ablation rescued the pro-apoptotic and cell-cycle arrest functions of p53 by decreasing p53K382me1, leading to activation of the p53 canonical pathway. I…

p530301 basic medicineCancer ResearchSmall interfering RNAMethyltransferaseCellular differentiationDruggabilityBiologyArticleEpigenesis GeneticNeuroblastoma03 medical and health sciences0302 clinical medicineNeuroblastomamedicineHumansEpigeneticsRNA Small InterferingGeneCell ProliferationsiRNA screenCell growthQuinazolineCell DifferentiationdifferentiationHistone-Lysine N-Methyltransferasemedicine.diseaseSETD8030104 developmental biologyOncology030220 oncology & carcinogenesisQuinazolinesCancer researchdifferentiation; epigenetics; neuroblastoma; p53; SETD8; siRNA screen; Oncology; Cell Biology; Cancer ResearchTumor Suppressor Protein p53epigeneticHuman
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High-SETD8 inactivates p53 in neuroblastoma

2017

p53Cancer Researchepigeneticsbusiness.industryMEDLINEneuroblastoma SETD8 p53 epigeneticsBiologymedicine.diseaseSETD8neuroblastomaText miningEditorialOncologyNeuroblastomaCancer researchmedicineEpigeneticsbusinessOncoscience
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