Search results for " epithelium"

showing 10 items of 249 documents

Sphingosine-1-phosphate increases human alveolar epithelial IL-8 secretion, proliferation and neutrophil chemotaxis

2009

Sphingosine-1-phosphate (S1P) has been presented recently as a pro-inflammatory agent in the airway epithelium since S1P levels are increased in bronchoalveolar lavage fluid of human asthmatics. However, the effects of S1P over the alveolar epithelium and neutrophil interactions are poorly understood. Here, we show that S1P increased interleukin 8 (IL-8) gene expression and protein secretion and proliferation in alveolar epithelial cells A549 at physiological concentrations (1 microM). At the same time, S1P increased intracellular Ca2+ concentration (potency 17.91 microM, measured by epifluorescence microscopy), phospholipase D (PLD) activity (measured by chemiluminiscence method) and extra…

LuminescenceNeutrophilsIntercellular Adhesion Molecule-1Gene ExpressionBiologyPertussis toxinReceptors G-Protein-Coupled1-ButanolSphingosineCell Line TumorPhospholipase DHumansInterleukin 8PhosphorylationExtracellular Signal-Regulated MAP KinasesEgtazic AcidCell ProliferationFlavonoidsPharmacologyA549 cellCell adhesion moleculeInterleukin-8Epithelial CellsChemotaxisIntercellular Adhesion Molecule-1Intercellular adhesion moleculeMolecular biologyPulmonary AlveoliChemotaxis LeukocytePertussis ToxinBiochemistryRespiratory epitheliumCalciumlipids (amino acids peptides and proteins)LysophospholipidsEuropean Journal of Pharmacology
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Can PBDEs affect the pathophysiologic complex of epithelium in lung diseases?

2020

Brominated flame-retardant (BFRs) exposure promotes multiple adverse health outcomes involved in oxidative stress, inflammation, and tissues damage. We investigated BFR effects, known as polybrominated diphenyl ethers (PBDEs) (47, 99 and 209) in an air-liquid-interface (ALI) airway tissue derived from A549 cell line, and compared with ALI culture of primary human bronchial epithelial cells (pHBEC). The cells, exposed to PBDEs (47, 99 and 209) (0.01-1 mu M) for 24 h, were studied for IL-8, Muc5AC and Muc5B (mRNAs and proteins) production, as well as NOX-4 (mRNA) expression. Furthermore, we evaluated tight junction (TJ) integrity by Trans-Epithelial Electrical Resistance (TEER) measurements, …

Lung DiseasesHealth Toxicology and Mutagenesis0208 environmental biotechnology02 engineering and technology010501 environmental sciencesMucin 5ACBROMINATED FLAME RETARDANTSmedicine.disease_cause01 natural sciencesPolybrominated diphenyl ethersPARTICULATE MATTERElectric ImpedanceHalogenated Diphenyl EthersFlame RetardantsInhalationTight junctionAIRWAY MUCUSChemistryGeneral Medicinerespiratory systemPollutionMucin-5BINTRACELLULAR GLUTATHIONEPolybrominated diphenyl ethers; Inflammation; Oxidative stress; Mucins; Epithelial barrier integrity; Rheological propertiesmedicine.anatomical_structureNADPH Oxidase 4medicine.symptomEnvironmental EngineeringInflammationBronchiEXPOSURE SYSTEMTight JunctionsAndrologymedicineEnvironmental ChemistryHumansRheological propertiesPolybrominated diphenyl ether0105 earth and related environmental sciencesAgedInflammationEpithelial barrier integrityPOLYBROMINATED DIPHENYL ETHERSMucinInterleukin-8MucinsPublic Health Environmental and Occupational HealthEpithelial CellsGeneral ChemistryN-ACETYLCYSTEINEEpithelium020801 environmental engineeringrespiratory tract diseasesOxidative StressA549 CellsMucinEX-VIVO MODELOxidative streRespiratory epitheliumAEROSOL-PARTICLESOxidative stressChemosphere
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Compromised integrity of excised porcine intestinal epithelium obtained from the abattoir affects the outcome of in vitro particle uptake studies

2002

Excised porcine intestinal tissue obtained from the local abattoir was studied for its suitability to examine the uptake and transport of poly(lactic-co-glycolic acid) (PLGA) nanoparticles in Peyer's (PP) and non-Peyer's patch (NPP) tissue in vitro. Incubation of such tissue with fluorescent PLGA and polystyrene particles revealed negligible uptake into the intercellular space with no noticeable difference between PP and NPP tissue. Similarly, yeast cells, which were used as a positive control for selective uptake into PP tissue, were found in the subepithelial area of both PP and NPP tissue. Therefore we examined the morphological integrity of the tissue for the duration of the experiments…

LysisCell SurvivalPolymersSwinePharmaceutical ScienceBiocompatible MaterialsSaccharomyces cerevisiaeAndrologyPeyer's Patcheschemistry.chemical_compoundPolylactic Acid-Polyglycolic Acid CopolymermedicineAnimalsLactic AcidIntestinal MucosaParticle SizeFluorescent DyesMicroscopy ConfocalTissue PreservationChemistrytechnology industry and agricultureIntestinal epitheliumSmall intestineEpitheliumIn vitroPeyer PatchPLGAmedicine.anatomical_structureMicroscopy FluorescenceBiochemistryTissue PreservationAbattoirsPolyglycolic AcidEuropean Journal of Pharmaceutical Sciences
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Cigarette smoke increases Toll-like receptor 4 and modifies lipopolysaccharide-mediated responses in airway epithelial cells.

2008

Airway epithelium is emerging as a regulator of innate immune responses to a variety of insults including cigarette smoke. The main goal of this study was to explore the effects of cigarette smoke extracts (CSE) on Toll-like receptor (TLR) expression and activation in a human bronchial epithelial cell line (16-HBE). The CSE increased the expression of TLR4 and the lipopolysaccharide (LPS) binding, the nuclear factor-kappaB (NF-kappaB) activation, the release of interleukin-8 (IL-8) and the chemotactic activity toward neutrophils. It did not induce TLR2 expression or extracellular signal-regulated signal kinase 1/2 (ERK1/2) activation. The LPS increased the expression of TLR4 and induced bot…

MAPK/ERK pathwayLipopolysaccharidesLipopolysaccharideNeutrophilsImmunologyBronchiRespiratory Mucosachemistry.chemical_compoundSmokeTobaccoImmunology and AllergyHumansImmunity MucosalCell Line TransformedMitogen-Activated Protein Kinase 1Toll-like receptorMitogen-Activated Protein Kinase 3Interleukin-8NF-kappa BChemotaxisEpithelial CellsOriginal ArticlesCell biologyChemokine CXCL10Toll-Like Receptor 4TLR2Chemotaxis LeukocytechemistryImmunologyTLR4Respiratory epitheliumSignal transductionSignal TransductionImmunology
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Early mitochondrial dysfunction, superoxide anion production, and DNA degradation are associated with non-apoptotic death of human airway epithelial …

2002

It has been shown that bacterial exoproducts may induce airway epithelium injury. During the epithelial repair process, the respiratory epithelial cells no more establish tight junctional intercellular complexes and may be particularly susceptible to bacterial virulence factors. In this study, we analyzed the effect of Pseudomonas aeruginosa exotoxin A (ETA) at different periods of time and concentrations on 16 HBE 14o(-) human bronchial epithelial cells in culture conditions inducing a phenotype of repairing cells. ETA treatment for 24 and 48 h led to the killing of 40.0 +/- 5.7% and 79.0 +/- 1.4% of the cells, respectively, as determined by the dimethylthiazole 2,5 diphenyl tetrazolium br…

MESH: Cell DeathMESH: ADP Ribose TransferasesMESH : DNAClinical BiochemistryCellApoptosisMESH : Dose-Response Relationship DrugMitochondrion[SDV.MHEP.PSR]Life Sciences [q-bio]/Human health and pathology/Pulmonology and respiratory tractMembrane PotentialsMESH: Dose-Response Relationship Drugchemistry.chemical_compoundSuperoxidesMESH: Intracellular MembraneMESH : DNA FragmentationRespiratory systemEnzyme InhibitorsCells CulturedADP Ribose TransferasesMESH : Cell SurvivalCell DeathSuperoxideMESH: DNAMESH: BronchiCaspase InhibitorsMESH : BronchiMitochondriaMESH : Epithelial Cellsmedicine.anatomical_structureMESH: Cell SurvivalMESH: Enzyme InhibitorsMESH: Epithelial CellsMESH : ADP Ribose TransferasesIntracellularMESH: Cells CulturedPulmonary and Respiratory MedicineProgrammed cell deathCell SurvivalVirulence FactorsBacterial ToxinsExotoxinsBronchiDNA FragmentationRespiratory MucosaBiologyMicrobiologyNecrosisNasal PolypsMESH : Cells CulturedmedicineHumansMESH: DNA FragmentationMESH : Intracellular MembraneMolecular BiologyMESH : Enzyme InhibitorsMESH: HumansMESH: CaspasesDose-Response Relationship DrugMESH: ApoptosisMESH : HumansEpithelial CellsCell BiologyDNAIntracellular MembranesMESH: ExotoxinschemistryMESH: Bacterial ToxinsApoptosisMESH : ExotoxinsMESH : Cell DeathMESH : Bacterial ToxinsRespiratory epithelium[SDV.MHEP.PSR] Life Sciences [q-bio]/Human health and pathology/Pulmonology and respiratory tractMESH : CaspasesMESH : Apoptosis[ SDV.MHEP.PSR ] Life Sciences [q-bio]/Human health and pathology/Pulmonology and respiratory tract
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IC3D Classification of Corneal Dystrophies—Edition 2

2015

To update the 2008 International Classification of Corneal Dystrophies (IC3D) incorporating new clinical, histopathologic, and genetic information.The IC3D reviewed worldwide peer-reviewed articles for new information on corneal dystrophies published between 2008 and 2014. Using this information, corneal dystrophy templates and anatomic classification were updated. New clinical, histopathologic, and confocal photographs were added.On the basis of revisiting the cellular origin of corneal dystrophy, a modified anatomic classification is proposed consisting of (1) epithelial and subepithelial dystrophies, (2) epithelial-stromal TGFBI dystrophies, (3) stromal dystrophies, and (4) endothelial d…

Macular corneal dystrophygenetic structuresEndothelial dystrophiesGenetic diseaseStromaEpitheliumGelatinousdrop-like corneal dystrophyCorneaLisch Epithelial Corneal DystrophyCornea pathologyPosteror polymorphous corneal dystrophyCorneal Dystrophies HereditaryPosterior amorphous corneal dystrophyEpithelial-stromal TGFBI dystrophiesMacular corneal dystrophyFleck corneal dystrophyLattice corneal dystrophyPre-Descemet corneal dystrophyCongenital stromal corneal dystrophySubepithelialmucinous corneal dystrophySchnyder corneal dystrophyThiel-Behnke corneal dystrophyPosterior polymorphous corneal dystrophyEpithelial and subepithelial dystrophiesFuchsendothelial corneal dystrophyFleck corneal dystrophyReis-Bücklers corneal dystrophyCongenital hereditary endothelial dystrophyCentralcloudy dystrophy of FrançoisCongenital stromal corneal dystrophyPosterior amorphous corneal dystrophymedicine.medical_specialtyHistologyeducationHereditary diseaseHistopathologyBiologyKeratoconusLisch epithelial corneal dystrophyMeesmann dystrophyNOBowman membraneDescemetmembraneInternational Classification of DiseasesTerminology as TopicOphthalmologyGeneticsmedicineHumansBowman membrane; Centralcloudy dystrophy of François; Confocal microscopy; Confocal microscopy; Congenital corneal endothelial dystrophy and X-linked endothelialdystrophy; Congenital stromal corneal dystrophy; Cornea; Cornea; Cornea dystrophy; Cornea pathology; Descemetmembrane; Endothelial dystrophies; Endothelium; Epithelial and subepithelial dystrophies; Epithelial basement membranedystrophy; Epithelial recurrent erosion dystrophies; Epithelial-stromal TGFBI dystrophies; Epithelium; Fleck corneal dystrophy; Fuchsendothelial corneal dystrophy; Gelatinousdrop-like corneal dystrophy; Genetic disease; Genetics; Granular corneal dystrophy type 1; Granular corneal dystrophy type 2; Hereditary disease; Histology; Histopathology; Keratoconus; Lattice corneal dystrophy; Lisch epithelial corneal dystrophy; Macular corneal dystrophy; Meesmann dystrophy; Posterior amorphous corneal dystrophy; Posteror polymorphous corneal dystrophy; Pre-Descemet corneal dystrophy; Reis-Bücklers corneal dystrophy; Schnyder corneal dystrophy; Stroma; Stromal dystrophies; Subepithelialmucinous corneal dystrophy; TGFBI; Thiel-Behnke corneal dystrophy; OphthalmologyEndotheliumEpithelial basement membranedystrophyCornea dystrophyCongenital corneal endothelial dystrophy and X-linked endothelialdystrophymedicine.diseaseeye diseasesConfocal microscopyOphthalmologyGranular corneal dystrophy type 2Granular corneal dystrophy type 1Stromal dystrophiesLattice corneal dystrophysense organsTGFBIEpithelial recurrent erosion dystrophiesCornea
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Aged Mice Devoid of the M3 Muscarinic Acetylcholine Receptor Develop Mild Dry Eye Disease

2021

The parasympathetic nervous system is critically involved in the regulation of tear secretion by activating muscarinic acetylcholine receptors. Hence, various animal models targeting parasympathetic signaling have been developed to induce dry eye disease (DED). However, the muscarinic receptor subtype (M1–M5) mediating tear secretion remains to be determined. This study was conducted to test the hypothesis that the M3 receptor subtype regulates tear secretion and to evaluate the ocular surface phenotype of mice with targeted disruption of the M3 receptor (M3R−/−). The experimental techniques included quantification of tear production, fluorescein staining of the ocular surface, environmenta…

Male0301 basic medicineM3medicine.disease_causeMiceM<sub>3</sub>0302 clinical medicineMuscarinic acetylcholine receptorTear secretionBiology (General)SpectroscopyCorneal epitheliumMice Knockouttear secretionChemistryEpithelium CornealMuscarinic acetylcholine receptor M3General Medicinedry eye diseaseComputer Science ApplicationsChemistrymedicine.anatomical_structureDry Eye SyndromesGoblet CellsConjunctivamedicine.medical_specialtyConjunctivaQH301-705.5ArticleCatalysisProinflammatory cytokineInorganic Chemistry03 medical and health sciencesmuscarinic receptorInternal medicinecorneamedicineAnimalsPhysical and Theoretical ChemistryQD1-999Molecular BiologyReceptor Muscarinic M3Goblet cellOrganic Chemistryeye diseasesMice Inbred C57BLDisease Models Animal030104 developmental biologyEndocrinologyTears030221 ophthalmology & optometrysense organsReactive Oxygen SpeciesOxidative stressInternational Journal of Molecular Sciences
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Functional evidence of multidrug resistance transporters (MDR) in rodent olfactory epithelium.

2012

WOS: 000305340700029; International audience; BACKGROUND: P-glycoprotein (Pgp) and multidrug resistance-associated protein (MRP1) are membrane transporter proteins which function as efflux pumps at cell membranes and are considered to exert a protective function against the entry of xenobiotics. While evidence for Pgp and MRP transporter activity is reported for olfactory tissue, their possible interaction and participation in the olfactory response has not been investigated. PRINCIPAL FINDINGS: Functional activity of putative MDR transporters was assessed by means of the fluorometric calcein acetoxymethyl ester (calcein-AM) accumulation assay on acute rat and mouse olfactory tissue slices.…

MaleAnatomy and Physiology[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionGene Expressionlcsh:MedicineATP-binding cassette transporterPharmacologyMicechemistry.chemical_compoundMolecular Cell Biologypolycyclic compoundslcsh:ScienceMice Inbred BALB CMultidisciplinaryNeuromodulationProbenecidReverse Transcriptase Polymerase Chain ReactionNeurochemistryFluoresceinsSensory SystemsCell biologyElectrophysiologymedicine.anatomical_structureAlimentation et NutritionCyclosporineQuinolinesMedicineFemaleEffluxCellular TypesMultidrug Resistance-Associated Proteinsproduct p-glycoprotein;blood-brain-barrier;receptor neurons;cyclic-nucleotides;tumor-cells;expression;localization;protein;gene;tissuesMultidrug Resistance-Associated ProteinsResearch ArticleATP Binding Cassette Transporter Subfamily BNeurophysiologyBiologyOlfactory Receptor NeuronsOlfactory mucosaPsychologie (Sciences cognitives)Olfactory MucosaPeripheral Nervous SystemmedicineAnimalsFood and NutritionRats WistarBiologyOlfactory SystemOlfactory receptorlcsh:RNeurosciencesEpithelial CellsBiological TransportTransporterRatsCalceinMicroscopy FluorescenceVerapamilchemistryNeurons and Cognitionlcsh:QPropionates[SDV.AEN]Life Sciences [q-bio]/Food and NutritionOlfactory epitheliumNeuroscience
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Odorant metabolism catalyzed by olfactory mucosal enzymes influences peripheral olfactory responses in rats.

2013

International audience; A large set of xenobiotic-metabolizing enzymes (XMEs), such as the cytochrome P450 monooxygenases (CYPs), esterases and transferases, are highly expressed in mammalian olfactory mucosa (OM). These enzymes are known to catalyze the biotransformation of exogenous compounds to facilitate elimination. However, the functions of these enzymes in the olfactory epithelium are not clearly understood. In addition to protecting against inhaled toxic compounds, these enzymes could also metabolize odorant molecules, and thus modify their stimulating properties or inactivate them. In the present study, we investigated the in vitro biotransformation of odorant molecules in the rat …

MaleAnatomy and Physiology[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionSensory PhysiologyEnzyme Metabolismlcsh:MedicineQuinolonesBiochemistryCarboxylesterasechemistry.chemical_compoundPentanols0302 clinical medicineCoumarinsEnzyme Inhibitorslcsh:Sciencechemistry.chemical_classification0303 health sciencesMultidisciplinaryEnzyme ClassesEsterasesSensory SystemsEnzymes3. Good healthElectrophysiologyProtein Transportmedicine.anatomical_structureBiochemistryMedicineSensory PerceptionMetabolic PathwaysResearch ArticleIsoamyl acetateBiologyNeurological SystemXenobiotics03 medical and health sciencesOlfactory mucosaOlfactory MucosaTransferasesmedicineAnimalsRats WistarBiology030304 developmental biologyOlfactory Systemlcsh:RGlycosyltransferasesCytochrome P450MonooxygenaseOlfactory PerceptionRatsMetabolismEnzymechemistryOdorantsBiocatalysisbiology.proteinlcsh:Q[SDV.AEN]Life Sciences [q-bio]/Food and NutritionOlfactory epithelium030217 neurology & neurosurgeryDrug metabolismNeuroscience
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Human airway epithelial extracellular vesicle miRNA signature is altered upon asthma development

2020

Background: miRNAs are master regulators of signaling pathways critically involved in asthma and are transferred between cells in extracellular vesicles (EV). We aimed to investigate whether the miRNA content of EV secreted by primary normal human bronchial epithelial cells (NHBE) is altered upon asthma development. Methods: NHBE cells were cultured at air-liquid interface and treated with interleukin (IL)-13 to induce an asthma-like phenotype. EV isolations by precipitation from basal culture medium or apical surface wash were characterized by nanoparticle tracking analysis, transmission electron microscopy, and Western blot, and EV-associated miRNAs were identified by a RT-qPCR-based prof…

MaleEXPRESSIONMECHANISMAdolescentMICRORNASImmunologyRespiratory MucosaBiologyDENDRITIC CELLSTh2 CellsWestern blotmicroRNAmedicineImmunology and AllergyHumansSecretionChildCells CulturedmiRNASUPPRESSIONInterleukin-13LAVAGE FLUID EXOSOMESmedicine.diagnostic_testInterleukinCell PolarityCell DifferentiationEpithelial Cellsairway epitheliumDendritic cellExtracellular vesiclePROFILESrespiratory systemasthmaDYSFUNCTIONCell biologyddc:Th2 polarizationNasal LavageRespiratory epitheliumFemaleSignal transductionTranscriptomeextracellular vesiclesSignal Transduction
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