Search results for " genomi"

showing 10 items of 572 documents

Lrp4, a Novel Receptor for Dickkopf 1 and Sclerostin, Is Expressed by Osteoblasts and Regulates Bone Growth and Turnover In Vivo

2009

Lrp4 is a multifunctional member of the low density lipoprotein-receptor gene family and a modulator of extracellular cell signaling pathways in development. For example, Lrp4 binds Wise, a secreted Wnt modulator and BMP antagonist. Lrp4 shares structural elements within the extracellular ligand binding domain with Lrp5 and Lrp6, two established Wnt co-receptors with important roles in osteogenesis. Sclerostin is a potent osteocyte secreted inhibitor of bone formation that directly binds Lrp5 and Lrp6 and modulates both BMP and Wnt signaling. The anti-osteogenic effect of sclerostin is thought to be mediated mainly by inhibition of Wnt signaling through Lrp5/6 within osteoblasts. Dickkopf1 …

Genetic Markersmusculoskeletal diseasesmedicine.medical_specialtylcsh:MedicineBiologyBone morphogenetic proteinBone and BonesCell LineMicechemistry.chemical_compoundInternal medicineBiochemistry/Cell Signaling and Trafficking StructuresmedicineAnimalsHumanslcsh:ScienceLDL-Receptor Related ProteinsAdaptor Proteins Signal TransducingGlycoproteinsBone growthBone DevelopmentOsteoblastsMultidisciplinarylcsh:RWnt signaling pathwayLRP6Rheumatology/Bone and Mineral MetabolismLRP5OsteoblastPhenotypemedicine.anatomical_structureEndocrinologyGene Expression RegulationReceptors LDLGenetics and Genomics/Disease ModelschemistryOsteocyteBone Morphogenetic ProteinsIntercellular Signaling Peptides and ProteinsSclerostinlcsh:QSignal TransductionResearch ArticlePLoS ONE
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Improved models for animal research

2008

Experimental animal models are critical to understand gene function and human disease. Many rodent models are presently available providing avenues to elucidate gene function and/or to recapitulate specific pathological conditions. To a large extent, successful translation of clinical evidence or analytical data into appropriate mouse models is possible through progress in transgenic or gene deletion technology. Despite these significant improvements, major limitations still exist in manipulating the mouse genome. For this reason and to maximize success, the design and planning of mouse models need good knowledge concerning the requirements and limitations of commonly used strategies and em…

Genetically modified mouseHuman diseaseEmerging technologiesComputer sciencemedia_common.quotation_subjectKnockout mouseExperimental Animal ModelsComputational biologyFunction (engineering)GenomeFunctional genomicsmedia_common
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Chromosomal Dynamics in Cercopithecini Studied by Williams-Beuren probe mapping

2010

Chromosomal evolution in the tribe Cercopithecini and in related taxa is highly debated. Uncertainty in reconstruction is mainly related to the great genetic variability and polymorphism of the taxa. Recent molecular and karyological efforts individuate a split between genetically conservative, arbitrarily defi ned, “terrestrial” guenons and vervets, and genetically variable and derived “arboreal” guenons. We performed the mapping of WS loci probe (7q11.23) in order to verify the chromosomal dynamics of the genomes of three tree-dwelling species of the tribe: C. neglectus, C. mitis mitis, C. albogularis labiatus. Data from literature for the “ground-dwelling” Chlorocebus aethiops sabaeus ha…

GeneticsArboreal locomotionCercopithecinibiologySettore BIO/08 - AntropologiaTribe (biology)biology.organism_classificationCercopithecus Genomic Evolution Human Chromosome 7 Williams-Beuren lociGenomePongo pygmaeusTaxonGeneticsGenetic variabilityGeneral Agricultural and Biological SciencesSyntenyCaryologia
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The cis-regulatory sequences required for expression of the Drosophila melanogaster adult cuticle gene ACP65A.

2009

Post-embryonic development in insects requires successive molts. Molts are triggered by ecdysteroids, and the nature of the molt (larval, pupal or adult) is determined by juvenile hormones. The genes encoding cuticle proteins are targets of both classes of hormones, and therefore are interesting models to study hormone action at the molecular level. The Drosophila ACP65A cuticle gene is expressed exclusively during the synthesis of the adult exoskeleton, in epidermal domains synthesising flexible cuticle. We have examined the cis -regulatory sequences of ACP65A using phylogenetic comparisons and functional analysis, and find that only about 180 bp are essential, including an 81 bp intron. T…

GeneticsBase SequenceCuticlefungiMolecular Sequence DataIntronBiologyRegulatory Sequences Nucleic Acidbiology.organism_classificationDrosophila melanogasterGene Expression RegulationRegulatory sequenceInsect ScienceJuvenile hormoneGene expressionGeneticsAnimalsInsect ProteinsRegulatory Elements TranscriptionalDrosophila melanogasterMolecular BiologyGeneFunctional genomicsPhylogenyInsect molecular biology
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A ceRNA approach may unveil unexpected contributors to deletion syndromes, the model of 5q- syndrome.

2015

In genomic deletions, gene haploinsufficiency might directly configure a specific disease phenotype. Nevertheless, in some cases no functional association can be identified between haploinsufficient genes and the deletion-associated phenotype. Transcripts can act as microRNA sponges. The reduction of transcripts from the hemizygous region may increase the availability of specific microRNAs, which in turn may exert in-trans regulation of target genes outside the deleted region, eventually contributing to the phenotype. Here we prospect a competing endogenous RNA (ceRNA) approach for the identification of candidate genes target of epigenetic regulation in deletion syndromes. As a model, we an…

GeneticsCancer ResearchCandidate gene5q- syndromeCompeting endogenous RNAgenomic deletionsSettore BIO/11 - Biologia MolecolareBiologySettore MED/08 - Anatomia PatologicaPhenotypemyelodysplastic syndromeTranscriptomecompeting endogenous RNAsOncologymicroRNAResearch PerspectiveCeRNAcompeting endogenous RNAEpigeneticsgenomic deletion5q- syndrome; CeRNA; competing endogenous RNAs; genomic deletions; myelodysplastic syndromeHaploinsufficiencyGeneOncoscience
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Molecular and clinical characterization of a small duplication Xp in a human female with psychiatric disorders

2011

CGH techniques allow us to detect small duplications thatoccur in humans with phenotypic manifestations and demon-strate the importance of these duplications in the etiologyof neurodevelopmental impairment. As in the case of otherX-linked disorders, X-inactivation plays a major role in theclinical expression of such X chromosomal imbalances withusually milder symptoms in females than in males. Mostmale patients carrying Xp duplication have mental retarda-tion (X-linked mental retardation) and variable facial dys-morphic features (Gimelli

GeneticsChromosomes Human XComparative Genomic HybridizationMental Disordershuman geneticsBiologyPhenotypeHuman geneticspsychiatric disorderfunctional Xp disomySettore MED/38 - Pediatria Generale E SpecialisticaSettore MED/03 - Genetica MedicaX Chromosome InactivationChild PreschoolGene duplicationChromosome DuplicationGeneticsMental Retardation X-LinkedHumansarray CGHFemaleChildfunctional Xp disomy; array CGH; psychiatric disorders; human geneticsGenetic Association StudiesSex Chromosome Aberrations
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Evolution of small prokaryotic genomes

2015

As revealed by genome sequencing, the biology of prokaryotes with reduced genomes is strikingly diverse. These include free-living prokaryotes with ∼800 genes as well as endosymbiotic bacteria with as few as ∼140 genes. Comparative genomics is revealing the evolutionary mechanisms that led to these small genomes. In the case of free-living prokaryotes, natural selection directly favored genome reduction, while in the case of endosymbiotic prokaryotes neutral processes played a more prominent role. However, new experimental data suggest that selective processes may be at operation as well for endosymbiotic prokaryotes at least during the first stages of genome reduction. Endosymbiotic prokar…

GeneticsComparative genomicsMicrobiology (medical)Natural selectionendosymbiosisEndosymbiosisMuller’s ratchetminimal genome sizelcsh:QR1-502Muller's ratchetReview ArticleBiologyreductive genome evolutionrobustness-based selective reductionGenomeMicrobiologyDNA sequencinglcsh:Microbiologysymbionellestreamlining evolutionEvolutionary biologyGeneBlack Queen HypothesisSyntenyFrontiers in Microbiology
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Structural analyses of a hypothetical minimal metabolism

2007

By integrating data from comparative genomics and large-scale deletion studies, we previously proposed a minimal gene set comprising 206 protein-coding genes. To evaluate the consistency of the metabolism encoded by such a minimal genome, we have carried out a series of computational analyses. Firstly, the topology of the minimal metabolism was compared with that of the reconstructed networks from natural bacterial genomes. Secondly, the robustness of the metabolic network was evaluated by simulated mutagenesis and, finally, the stoichiometric consistency was assessed by automatically deriving the steady-state solutions from the reaction set. The results indicated that the proposed minimal …

GeneticsComparative genomicsModels StatisticalCellsScale-free networkMetabolic networkRobustness (evolution)Computational biologyMetabolismBacterial genome sizeBiologyNetwork topologyModels BiologicalGeneral Biochemistry Genetics and Molecular BiologyCell Physiological PhenomenaCluster AnalysisComputer SimulationMinimal genomeGeneral Agricultural and Biological SciencesMetabolic Networks and PathwaysResearch ArticlePhilosophical Transactions of the Royal Society B: Biological Sciences
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Comparative Genomics of the RBR Family, Including the Parkinson's Disease–Related Gene Parkin and the Genes of the Ariadne Subfamily

2002

Genes of the RBR family are characterized by the RBR signature (two RING finger domains separated by an IBR/DRIL domain). The RBR family is widespread in eukaryotes, with numerous members in animals (mammals, Drosophila, Caenorhabditis) and plants (Arabidopsis). But yeasts, such as Saccharomyces cerevisiae or Schizosaccharomyces pombe, contain only two RBR genes. We determined the phylogenetic relationships and the most likely orthologs in different species of several family members for which functional data are available. These include: (1) parkin, whose mutations are involved in forms of familial Parkinson's disease; (2) the ariadne genes, recently characterized in Drosophila and mammals;…

GeneticsComparative genomicsSubfamilyUbiquitin-Protein LigasesGenomicsBiologybiology.organism_classificationParkinLigasesCaenorhabditismedicine.anatomical_structureSchizosaccharomyces pombeGeneticsRing fingermedicinebiology.proteinAnimalsHumansButterfliesMolecular BiologyGenePhylogenyEcology Evolution Behavior and SystematicsCullinMolecular Biology and Evolution
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Parkin and relatives: the RBR family of ubiquitin ligases

2004

Mutations in the parkin gene cause autosomal-recessive juvenile parkinsonism. Parkin encodes a ubiquitinprotein ligase characterized by having the RBR domain, composed of two RING fingers plus an IBR/DRIL domain. The RBR family is defined as the group of genes whose products contain an RBR domain. RBR family members exist in all eukaryotic species for which significant sequence data is available, including animals, plants, fungi, and several protists. The integration of comparative genomics with structural and functional data allows us to conclude that RBR proteins have multiple roles, not only in protein quality control mechanisms, but also as indirect regulators of transcription. A recent…

GeneticsComparative genomicschemistry.chemical_classificationDNA ligasebiologyPhysiologyUbiquitin-Protein LigasesParkinson DiseaseGenomicsParkinProtein Structure TertiaryUbiquitin ligaseProtein structureUbiquitinchemistryGeneticsbiology.proteinTranscriptional regulationAnimalsGenePhylogenyPhysiological Genomics
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