Search results for " inflammation."

showing 10 items of 623 documents

A new p65 isoform that bind the glucocorticoid hormone and is expressed in inflammation liver diseases and COVID-19

2021

AbstractInflammation is a physiological process whose deregulation causes some diseases including cancer. Nuclear Factor kB (NF-kB) is a family of ubiquitous and inducible transcription factors, in which the p65/p50 heterodimer is the most abundant complex, that play critical roles mainly in inflammation. Glucocorticoid Receptor (GR) is a ligand-activated transcription factor and acts as an anti-inflammatory agent and immunosuppressant. Thus, NF-kB and GR are physiological antagonists in the inflammation process. Here we show that in mice and humans there is a spliced variant of p65, named p65 iso5, which binds the corticosteroid hormone dexamethasone amplifying the effect of the glucocorti…

MaleCOVID-19.Molecular biologyGene ExpressionDexamethasoneHepatitisMiceGlucocorticoid receptorAdrenal Cortex HormonesProtein IsoformsNF-kBMultidisciplinaryLiver DiseasesQLiver NeoplasmsRNF-kappa BMiddle AgedLiverMedicineFemalemedicine.symptomliver diseaseTranscriptionNFKB P65iso5 inflammationGlucocorticoidmedicine.drugAdultGene isoformCarcinoma HepatocellularScienceImmunologyInflammationBiologyGlucocorticoid ReceptorArticleReceptors GlucocorticoidmedicineAnimalsHumansGlucocorticoidsTranscription factorDexamethasoneInflammationSARS-CoV-2p65 isoformTranscription Factor RelAWild typeCOVID-19Mice Inbred C57BLAlternative SplicingSettore BIO/18 - GeneticaGene Expression RegulationLeukocytes MononuclearCancer researchHormone
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A Pilot Study on Prostate Cancer Risk and Pro-Inflammatory Genotypes: Pathophysiology and Therapeutic Implications

2010

Host genetic factors are crucial risk determinants for many human cancers. In this framework, an interesting model is represented by prostate cancer (PC), which is featured by a complex pathophysiology with a strong genetic component. Multiple genes seem to influence PC risk and several single nucleotide polymorphisms (SNPs) of candidate genes modifying PC susceptibility have been identified. It is noteworthy the potential association of common SNPs in pro-inflammatory genes with PC risk, since chronic inflammation is assumed to play a key role in prostate carcinogenesis. With the aim to identify candidate genes as an experimental basis to develop new strategies for both prevention and trea…

MaleCandidate geneGenotypePilot ProjectsSingle-nucleotide polymorphismBioinformaticsPolymorphism Single NucleotideProstate cancerRisk FactorsDrug DiscoveryGenotypemedicineHumansSNPSettore MED/05 - Patologia ClinicaGeneAgedAged 80 and overInflammationPharmacologySettore MED/04 - Patologia Generalebusiness.industryProstatic NeoplasmsCancerMiddle AgedProstate cancer (PC) inflammation genetics TLR4 TLR2 PTGS2 5-LO SNP.medicine.diseaseImmunologyTLR4Inflammation Mediatorsbusiness
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Peripheral cytokine and chemokine alterations in depression: a meta-analysis of 82 studies

2017

Objective: To conduct a systematic review and meta-analysis of studies that measured cytokine and chemokine levels in individuals with major depressive disorder (MDD) compared to healthy controls (HCs). Method: The PubMed/MEDLINE, EMBASE, and PsycINFO databases were searched up until May 30, 2016. Effect sizes were estimated with random-effects models. Result: Eighty-two studies comprising 3212 participants with MDD and 2798 HCs met inclusion criteria. Peripheral levels of interleukin-6 (IL-6), tumor necrosis factor (TNF)-alpha, IL-10, the soluble IL-2 receptor, C-C chemokine ligand 2, IL-13, IL-18, IL-12, the IL-1 receptor antagonist, and the soluble TNF receptor 2 were elevated in patient…

MaleChemokinePeripheral cytokinemedicine.drug_classmedicine.medical_treatmentInflammationchemokinesbehavioral disciplines and activities03 medical and health sciences0302 clinical medicinemental disordersHumansMedicineReceptorDepressive Disorder Majorbiologybusiness.industrychemokines; cytokines; depression; inflammation; meta-analysis; Psychiatry and Mental HealthReceptor antagonistmedicine.diseasecytokines030227 psychiatrymeta-analysisCytokineinflammationPsychiatry and Mental HealthMeta-analysisImmunologydepressionbiology.proteinMajor depressive disorderFemaleTumor necrosis factor alphamedicine.symptombusiness030217 neurology & neurosurgery
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Angiotensin II Induces Neutrophil Accumulation In Vivo Through Generation and Release of CXC Chemokines

2004

Background—Angiotensin II (Ang II) is implicated in the development of cardiac ischemic disorders in which prominent neutrophil accumulation occurs. Ang II can be generated intravascularly by the renin-angiotensin system or extravascularly by mast cell chymase. In this study, we characterized the ability of Ang II to induce neutrophil accumulation.Methods and Results—Intraperitoneal administration of Ang II (1 nmol/L) induced significant neutrophil recruitment within 4 hours (13.3±2.3×106neutrophils per rat versus 0.7±0.5×106in control animals), which disappeared by 24 hours. Maximal levels of CXC chemokines were detected 1 hour after Ang II injection (577±224 pmol/L cytokine-inducible neut…

MaleChemokinemedicine.medical_specialtyEndotheliumCellsInflammationAngiotensin ; Interleukins ; Cells ; Endothelium ; InflammationPulmonary ArteryUmbilical CordRats Sprague-DawleyAngiotensin:CIENCIAS MÉDICAS ::Medicina interna [UNESCO]Physiology (medical)Internal medicineRenin–angiotensin systemCell AdhesionLeukocytesAnimalsHumansMedicineMesenteryRNA MessengerEndotheliumPeritoneal CavityMacrophage inflammatory proteinCells CulturedUNESCO::CIENCIAS MÉDICAS ::Medicina internaInflammationbiologybusiness.industryAngiotensin IIMicrocirculationInterleukinsInterleukin-8Endothelial CellsChemotaxis:CIENCIAS MÉDICAS [UNESCO]Angiotensin IIRatsmedicine.anatomical_structureEndocrinologyNeutrophil InfiltrationUNESCO::CIENCIAS MÉDICASbiology.proteinmedicine.symptomCardiology and Cardiovascular MedicinebusinessChemokines CXCIntravital microscopy
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Silibinin improves hepatic and myocardial injury in mice with nonalcoholic steatohepatitis

2012

Abstract Background Nonalcoholic fatty liver disease is a chronic metabolic disorder with significant impact on cardiovascular and liver mortality. Aims In this study, we examined the effects of silibinin on liver and myocardium injury in an experimental model of nonalcoholic fatty liver disease. Methods A four-week daily dose of silibinin (20 mg/kg i.p.) was administrated to db/db mice fed a methionine–choline deficient diet. Hepatic and myocardial histology, oxidative stress and inflammatory cytokines were evaluated. Results Silibinin administration decreased HOMA-IR, serum ALT and markedly improved hepatic and myocardial damage. Silibinin reduced isoprostanes, 8-deoxyguanosine and nitrit…

MaleGene ExpressionIsoprostanesmedicine.disease_causeGastroenterologyAntioxidantsMicechemistry.chemical_compoundMethionineNon-alcoholic Fatty Liver DiseaseNonalcoholic fatty liver diseasePhosphorylationGastroenterologyAlanine TransaminaseGlutathioneCholine DeficiencyMitochondrial respiratory chainLiverHeart Inflammation NAFLD Oxidative stress SilibininCytokinesmedicine.symptomSilymarinmedicine.medical_specialtySilibininInflammationStatistics NonparametricProinflammatory cytokineInsulin resistanceInternal medicinemedicineAnimalsNitritesAnalysis of VarianceNitratesHepatologySettore BIO/16 - Anatomia Umanabusiness.industryMyocardiumJNK Mitogen-Activated Protein KinasesGlutathionemedicine.diseaseDietFatty LiverOxidative StressEndocrinologychemistrySilybinInsulin ResistancebusinessOxidative stressDigestive and Liver Disease
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Impact of pathogen burden in patients with coronary artery disease in relation to systemic inflammation and variation in genes encoding cytokines.

2003

The number of infectious pathogens to which an individual has been exposed (pathogen burden) has been linked to the development and the prognosis of coronary artery disease (CAD). The interaction among infection, genetic host susceptibility, and CAD remains unclear. This study was aimed at evaluating the modulation of the association between CAD and pathogen burden, by serum levels of inflammatory markers and polymorphisms of the interleukin (IL)-6 and tumor necrosis factor (TNF)-alpha genes. Immmunoglobulin (Ig) G or IgA antibodies to 8 pathogens were determined in 991 patients with CAD and 333 control subjects. Serum levels of high-sensitivity C-reactive protein, fibrinogen, IL-6, and TNF…

MaleGenotypemedicine.medical_treatmentInflammationCoronary Artery DiseaseSystemic inflammationInfectionsRisk FactorsmedicinePrevalenceHumansGenetic Predisposition to DiseasePathogenAgedInflammationChlamydiaPolymorphism Geneticbiologybusiness.industryInterleukin-6Tumor Necrosis Factor-alphaCholesterol HDLInterleukinFibrinogenEnvironmental ExposureHelicobacter pyloriMiddle Agedmedicine.diseasebiology.organism_classificationPrognosisImmunoglobulin ACytokineC-Reactive ProteinCase-Control StudiesImmunoglobulin GImmunologyCytokinesTumor necrosis factor alphaFemalemedicine.symptomCardiology and Cardiovascular MedicinebusinessBiomarkersThe American journal of cardiology
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Gender-Related Immune-Inflammatory Factors, Age-Related Diseases, and Longevity

2010

This review discusses the role of estrogens as pro- or antiinflammatory players in immune-inflammatory responses. In particular, their role in Alzheimer’s disease (AD), an example of immune-inflammatory disease, is discussed briefly. AD is a progressive neurodegenerative disease, which in Western societies accounts for the majority of cases of clinical senile dementia. However, sexual dimorphism of diseases may also depend on factors independent of sex hormones (i.e., a gender effect), as demonstrated by our data on differential longevity in females and males. In fact, differences in mortality between men and women are not only a question of sex that refers to biological differences, but ra…

MaleGerontologyAgingmedia_common.quotation_subjectLongevityDiseasegender inflammation age-related diseases longevityImmune systemAlzheimer DiseaseAnimalsHumansImmunologic FactorsSettore MED/05 - Patologia ClinicaDiseasemedia_commonSettore MED/04 - Patologia GeneraleSex CharacteristicsEstrogen Replacement TherapyLongevitySocial constructionismGender psychologySexual dimorphismImmune SystemFemaleInflammation MediatorsGeriatrics and GerontologyPsychologySex characteristicsHormoneClinical psychologyRejuvenation Research
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Immunogenetics, Gender, and Longevity

2007

In this article we discuss relevant data on aging, longevity, and gender with particular focus on inflammation gene polymorphisms which could affect an individual's chance to reach the extreme limit of human life. The present review is not an extensive revision of the literature, but rather an expert opinion based on selected data from the authors' laboratories. In 2000-2005 in the more developed regions, the life expectancy at birth is 71.9 years for men (78.3 in Japan) and 79.3 years for women (86.3 in Japan). Indeed, gender accounts for important differences in the prevalence of a variety of age-related diseases. Considering people of far-advanced age, demographic data document a clear-c…

MaleGerontologyAgingmedia_common.quotation_subjectLongevityPopulationDiseaseGeneral Biochemistry Genetics and Molecular BiologyHistory and Philosophy of ScienceHLA AntigensImmunogeneticsHumansMedicineSex Ratioeducationmedia_commonInflammationeducation.field_of_studySuccessful agingbusiness.industryGeneral NeuroscienceMortality rateAging Immune response Inflammation LongevityLongevityInfectious disease (medical specialty)Life expectancyFemalebusinessDeveloped countryAnnals of the New York Academy of Sciences
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The relevance of nutrition for the concept of cognitive frailty

2016

Purpose of review Physical and cognitive frailty are interrelated and synergistic syndromes more frequently seen in old age, which represent intermediate stages between aging successfully and disability. Poor nutrition is a fundamental determinant for both conditions, while various dietary components are proposed to prevent and/or improve them. This updated review discusses the possible influence of nutritional factors on cognitive frailty and its potential mediators. Recent findings Oxidative stress, low-grade systemic inflammation, neuroinflammation, and altered autophagy, all associated with obesity, metabolic syndrome and insulin resistance, are proposed mechanisms to explain the influe…

MaleGerontologySettore MED/09 - Medicina InternaMediterranean dietFrail ElderlyPsychological interventionNutritional StatusMedicine (miscellaneous)Affect (psychology)03 medical and health sciences0302 clinical medicineAutophagyHumansMental exerciseMedicineCognitive DysfunctionObesity030212 general & internal medicineOvereatingCognitive declineAgedAged 80 and overInflammationaging cognition frailty inflammation nutritionNutrition and DieteticsFrailtybusiness.industrymedicine.diseaseObesityMiddle ageOxidative StressCognitive AgingFemalebusiness030217 neurology & neurosurgeryCurrent Opinion in Clinical Nutrition & Metabolic Care
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Protein oxidation in chronic kidney disease.

2013

An imbalance between oxidative processes and antioxidant systems has been widely demonstrated in chronic kidney diseases (CKD). In this study we enrolled 26 healthy subjects, 27 patients with CKD on conservative treatment (CT-CKD) with various degrees of renal failure, and 31 CKD subjects in haemodialysis treatment (HD-CKD), evaluated before and after a standard haemodialysis session. In each group we measured protein carbonyl groups (PC) as an index of protein oxidation, lipid peroxidation (TBARS) and two plasma markers of leukocyte activation, elastase and myeloperoxidase (MPO). In CT-CKD subjects the PC level was significantly higher than in normal controls, and it was negatively correla…

MaleHEMODIALYSISmedicine.medical_specialtySettore MED/09 - Medicina InternaPhysiologyBIOMARKERSRenal functionurologic and male genital diseasesProtein oxidationThiobarbituric Acid Reactive SubstancesLipid peroxidationDiabetes Complicationschemistry.chemical_compoundCARBONYL STRESSMARKERSINFLAMMATIONGlycationRenal DialysisPhysiology (medical)Internal medicinemedicineTBARSHumansRenal Insufficiency ChronicPeroxidasebiologyPancreatic Elastasebusiness.industryNITRIC-OXIDE METABOLITESElastaseHematologyMiddle Agedmedicine.diseasefemale genital diseases and pregnancy complicationsOxidative StressEndocrinologychemistryMyeloperoxidaseNITRIC-OXIDE METABOLITES; CHRONIC-RENAL-FAILURE; CARBONYL STRESS; HEMODIALYSIS; BIOMARKERS; MARKERS; INFLAMMATIONImmunologybiology.proteinFemaleCHRONIC-RENAL-FAILURELipid PeroxidationCardiology and Cardiovascular MedicinebusinessOxidation-ReductionKidney diseaseClinical hemorheology and microcirculation
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