Search results for " intraperitoneal"

showing 10 items of 55 documents

Deep seroma after incisional hernia repair. Case reports and review of the literature.

2015

Wound-related complications are common after incisional hernia repair with mesh; seroma formation is the most frequent problem. The formation of a deep seroma has been rarely reported in the literature.In one year, September 2012-2013, 136 patients underwent surgery for incisional hernia repair, both elective and urgent.The following complications were observed: one dislocation of polypropylene prosthesis, a massive relapsed seroma and two deep seromas described in this article. A 63- years-old female underwent open incisional hernia repair with an intraperitoneal PTFE patch. She developed recurrent seroma under the mesh drained percutaneously, and finally the prosthesis was removed. A 72- …

MaleDeep serum incisional hernia intraperitoneal meshMiddle AgedSurgical MeshPolypropylenesImaging Three-DimensionalPostoperative ComplicationsSeromaHumansIncisional HerniaSurgical Wound InfectionFemaleTomography X-Ray ComputedPolytetrafluoroethyleneDevice RemovalHerniorrhaphyAged
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PSA-NCAM expression in the piriform cortex of the adult rat. Modulation by NMDA receptor antagonist administration.

2002

Administration of NMDA receptor antagonists upregulates the expression of the polysialylated form of the neural cell adhesion molecule (PSA-NCAM) in the adult hippocampus. Since the piriform cortex is also populated by PSA-NCAM immunoreactive neurons during adulthood, we sought to characterize them in detail and to test whether NMDA receptor antagonists also modulate PSA-NCAM in this cortical region. PSA-NCAM immunoreactivity is located mainly in layer II, where many neurogliaform and some pyramidal-semilunar transitional neurons are labeled. Many large neurons in layer III and endopiriform nucleus also express PSA-NCAM. Interestingly, some small labeled cells resembling migratory neuroblas…

MaleDoublecortin ProteinSynaptogenesisHippocampusNeural Cell Adhesion Molecule L1Receptors N-Methyl-D-AspartateRats Sprague-DawleyNeuroblastCell MovementPiriform cortexmedicineAnimalsMolecular BiologyNeural Cell Adhesion MoleculesNeuronsbiologyGeneral NeuroscienceOlfactory PathwaysDoublecortinRatsmedicine.anatomical_structurenervous systembiology.proteinSialic AcidsNeural cell adhesion moleculeNeurology (clinical)NeuNNeuroscienceNucleusExcitatory Amino Acid AntagonistsInjections IntraperitonealDevelopmental BiologyBrain research
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Inhibition of clastogenicity of benzo[a]pyrene and of its trans-7,8-dihydrodiol in mice in vivo by fruits, vegetables, and flavonoids.

2003

In the in vivo mouse bone marrow micronucleus assay, homogenates of spinach, artichoke, peaches, and blue grapes as well as commercial concentrates of these vegetables and fruits reduced induction of micronuclei by benzo[a]pyrene (BaP) by 43-50%. Concentrates of strawberries (31% reduction) and of cauliflower (20% reduction) were less potent. Inhibition of genotoxicity by spinach and peaches was not caused by any delay in maturation of micronucleated erythrocytes as shown by experiments with sampling times of 24, 48, and 72 h after dosing of BaP. Pre-treatment of the mice with spinach 48, 24, and 12h before application of BaP resulted in a 44% reduction of micronuclei while peaches generate…

MaleHealth Toxicology and MutagenesisFlavonoidAdministration OralBone Marrow CellsMice Inbred Strainsmedicine.disease_causecomplex mixturesDihydroxydihydrobenzopyreneschemistry.chemical_compoundClastogenMiceVegetablesGeneticsmedicineBenzo(a)pyreneCytochrome P-450 CYP1A1AnimalsFood scienceMicronuclei Chromosome-Defectivechemistry.chemical_classificationMicronucleus TestsbiologyDose-Response Relationship DrugPlant Extractsfood and beveragesAntimutagenic Agentsbiology.organism_classificationDose–response relationshipBenzo(a)pyrenechemistryBiochemistryLiverFruitMicronucleus testCytochrome P-450 CYP2B1SpinachDrug Therapy CombinationQuercetinQuercetinGenotoxicityInjections IntraperitonealMutagensMutation research
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IL-5 Enhances in Vitro and in Vivo Antigen-Specific IgA Production in MHC Genetically Determined Low IL-5 Responder Mice

1995

Lymphonode cells from BALB/k mice, but not from BALB/c mice, immunized with picryl chloride (PCl) produce IL-5 when stimulated with the specific antigen in vitro and this correlates with picryl-specific IgA levels in vivo, which are 6 to 10 times higher in BALB/k mice. B lymphocytes from BALB/k mice cultured with PCl-immune T cells from BALB/k produce in vivo anti-PCl-IgA, while B lymphocytes from BALB/c mice, cultured with T cells from BALB/c mice, fail to produce appreciable amounts of anti-PCl IgA, unless IL-5 is added to cultures. B lymphocytes from both strains of mice produce similar amounts of total IgA antibodies when stimulated in vitro with lipopolysaccharide. In vivo administrati…

MaleLipopolysaccharideImmunologyPicryl ChlorideMajor histocompatibility complexMajor Histocompatibility ComplexPicryl chlorideEpitopesMicechemistry.chemical_compoundAntigenIn vivoAnimalsInterleukin 5Cells CulturedMice Inbred BALB CbiologyMolecular biologyMice Mutant StrainsRecombinant ProteinsIn vitroImmunoglobulin Achemistrybiology.proteinInterleukin-5AntibodyInjections IntraperitonealCellular Immunology
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Reinstatement of Morphine-Induced Conditioned Place Preference in Mice by Priming Injections

2004

To construct a model of relapse of drug abuse in mice, the induction, we evaluated the extinction and reinstatement of morphine-induced place preference. In Experiment 1, we examined the effects of morphine (0, 2, 3, 5, 10, 20 and 40 mg/kg) in the conditioned place preference (CPP) paradigm. Mice showed CPP with 5, 10, 20 and 40 mg/kg. In Experiment 2, we evaluated the effects of two different extinction procedures. After conditioning with 40 mg/kg of morphine, the mice underwent daily extinction sessions of 60 or 15 min of duration. CPP was extinguished after seven and nine sessions, respectively. In Experiment 3, we tested the reinstating effects of several priming doses of morphine. Mice…

MaleNarcoticsReinforcement SchedulePharmacologyArticleExtinction Psychologicallcsh:RC321-571MiceRewardmedicineAnimalslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryDose-Response Relationship DrugMorphineExtinction (psychology)Conditioned place preferenceDose–response relationshipNeurologyAnesthesiaMorphineConditioning OperantConditioningNeurology (clinical)PsychologyReinforcement PsychologyPriming (psychology)Injections Intraperitonealmedicine.drugNeural Plasticity
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Effects of acute and chronic maprotiline administration on inhibitory avoidance in male mice

2000

The effects of acute and chronic administration of maprotiline (5, 10 or 20 mg/kg, intraperitoneally) were assessed on inhibitory avoidance in male mice. Acute administration of maprotiline before training did not effect training phase latencies, but impaired performance (i.e. produced shorter latencies) in the test at doses of 5 and 20 mg/kg. When given after training, the drug did not modify test latencies at any of the doses used. Chronic administration for 21 days (interrupted 24 h before training) also shortened latencies in the test but not in training. An experiment on the acute effects of maprotiline on analgesia (determination of flinch and jump thresholds for increasing electric f…

MalePain ThresholdAnterograde amnesiaRatónInhibitory postsynaptic potentialDrug Administration ScheduleDevelopmental psychologyNorepinephrine (medication)MiceBehavioral NeuroscienceDrug toleranceThreshold of painAvoidance LearningReaction TimemedicineAnimalsMaprotilineDose-Response Relationship DrugBrainNeural InhibitionDrug ToleranceMaprotilineAnesthesiaMental RecallAntidepressive Agents Second-Generationmedicine.symptomPsychologyReuptake inhibitorInjections Intraperitonealmedicine.drugBehavioural Brain Research
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Chronic administration of fluoxetine impairs inhibitory avoidance in male but not female mice

2002

The effects of chronic administration of fluoxetine (20 mg/kg/day i.p.) on a one-trial step-through inhibitory avoidance task were investigated in male and female CD1 mice. In Experiment 1, treatment was administered for 21 days before the training session, whereas in Experiment 2, other subjects were subjected to the same treatment starting 24 h after the training session. The comparison of test versus training latencies showed memory deterioration with pre-training administration of fluoxetine (Experiment 1), which affected males but not females. Sex differences in this task were also observed in Experiment 1, with females showing a better performance. Sex differences were evident in cont…

MaleSex CharacteristicsFluoxetineRatónMemoriaPhysiologyClassical conditioningMotor ActivityInhibitory postsynaptic potentialLocomotor activityDevelopmental psychologySexual dimorphismMiceBehavioral NeuroscienceNon specificMemoryFluoxetineAvoidance LearningmedicineAnimalsFemalePsychologyInjections IntraperitonealSelective Serotonin Reuptake Inhibitorsmedicine.drugBehavioural Brain Research
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2-Methoxyestradiol confers neuroprotection and inhibits a maladaptive HIF-1α response after traumatic brain injury in mice

2014

HIF-1α is pivotal for cellular homeostasis in response to cerebral ischemia. Pharmacological inhibition of HIF-1α may reduce secondary brain damage by targeting post-translational mechanisms associated with its proteasomal degradation and nuclear translocation. This study examined the neuroprotective effects of 2-methoxyestradiol (2ME2), the involved HIF-1α-dependent response, and alternative splicing in exon 14 of HIF-1α (HIF-1α∆Ex14) after traumatic brain injury (TBI) in mice. Intraperitoneal 2ME2 administration 30 min after TBI caused a dose-dependent reduction in secondary brain damage after 24 h. 2ME2 was physiologically tolerated, showed no effects on immune cell brain migration, and …

MaleTraumatic brain injuryBlotting WesternIschemiaCellular homeostasisBrain damagePharmacologyBiologyBiochemistryNeuroprotectionBrain IschemiaMitochondrial ProteinsMiceCellular and Molecular Neurosciencechemistry.chemical_compoundPlasminogen Activator Inhibitor 1medicineAnimalsCell NucleusNeuronsEstradiolTumor Necrosis Factor-alphaAlternative splicingMembrane ProteinsExonsHypoxia-Inducible Factor 1 alpha Subunitmedicine.diseaseImmunohistochemistryUp-RegulationMice Inbred C57BLAlternative SplicingProtein TransportNeuroprotective AgentsGene Expression RegulationchemistryBrain InjuriesPlasminogen activator inhibitor-1Tumor necrosis factor alphamedicine.symptomNeuroscienceInjections IntraperitonealSubcellular FractionsJournal of Neurochemistry
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A critical role for TNFα in the selective attachment of mononuclear leukocytes to angiotensin-II-stimulated arterioles

2007

Abstract Angiotensin II (Ang-II) exerts inflammatory activity and is involved in different cardiovascular disorders. This study has evaluated the involvement of tumor necrosis factor alpha (TNFα) in the leukocyte accumulation elicited by Ang-II. Ang-II (1 nM intraperitoneally in rats) induced TNFα release at 1 hour followed by neutrophil and mononuclear cell recruitment. The administration of an antirat TNFα antiserum had no effect on Ang-IIinduced neutrophil accumulation but inhibited the infiltration of mononuclear cells and reduced CC chemokine content in the peritoneal exudate. Pretreatment with either an anti-TNFα or an anti-IL-4 antiserum decreased Ang-II-induced arteriolar mononuclea…

MaleUmbilical Veinsmedicine.medical_specialtyEndotheliummedicine.medical_treatmentImmunologyBiologyBiochemistryPeripheral blood mononuclear cellMicrocirculationRats Sprague-DawleyInternal medicineCell AdhesionmedicineAnimalsHumansVasoconstrictor AgentsRNA MessengerVenuleReverse Transcriptase Polymerase Chain ReactionTumor Necrosis Factor-alphaAngiotensin IICell BiologyHematologyAngiotensin IIRatsArteriolesMononuclear cell infiltrationmedicine.anatomical_structureCytokineEndocrinologyLeukocytes MononuclearTumor necrosis factor alphaEndothelium VascularInterleukin-4ChemokinesInjections IntraperitonealBlood
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Cannabidiol prevents several of the behavioral alterations related to cocaine addiction in mice.

2021

Abstract Cocaine dependence is a highly prevalent disease in modern society and lacks an effective treatment. Cannabidiol (CBD), a major non-psychoactive constituent of Cannabis sativa, has been shown to be a promising tool in the management of some neuropsychiatric disorders, including cocaine abuse. However, its therapeutic effects on the behavioral outcomes related to cocaine addiction remain unclear. The present research evaluates the effects of CBD (30, 60 and 120 mg/kg; injected intraperitoneally) on the acquisition, expression, extinction and reinstatement of cocaine (10 mg/kg)-induced conditioned place preference (CPP; Study 1); cocaine (25 mg/kg)-induced locomotor stimulation (Stud…

Malemedia_common.quotation_subjectConditioning ClassicalPharmacologyHyperkinesisCocaine dependenceExtinction Psychological03 medical and health sciencesCocaine-Related DisordersMice0302 clinical medicineCocainemedicineAnimalsCannabidiolBiological Psychiatrymedia_commonPharmacologyDose-Response Relationship Drugbusiness.industryAddictionTherapeutic effectExtinction (psychology)Abstinencemedicine.diseaseConditioned place preferenceTail suspension test030227 psychiatrySubstance Withdrawal SyndromeMice Inbred C57BLbusinessCannabidiolInjections IntraperitonealLocomotionmedicine.drugProgress in neuro-psychopharmacologybiological psychiatry
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