Search results for " monoclonal"

showing 10 items of 807 documents

Expression of cysteine proteinases cathepsins B and K and of cysteine proteinase inhibitor cystatin C in giant cell tumor of tendon sheath.

2001

The expression of cysteine proteinases cathepsins B and K and of the endogenous inhibitor of cysteine proteinases, cystatin C, was investigated in tissue specimens of patients with giant cell tumor of tendon sheath (GCTTS). Expression of both enzymes was examined by immunohistochemistry in tissue specimens of 14 patients with GCTTS. Applying double-labeling techniques, the coexpression of cathepsin B and its major endogenous inhibitor cystatin C was additionally studied. Cells expressing the respective proteins were further characterized with the macrophage markers HAM56 and anti-CD68 (clone PG-M1). Cathepsin B could be detected in numerous HAM56-positive mononuclear cells (MC), but only in…

AdultMaleCathepsin KAntigens Differentiation MyelomonocyticCathepsin ECell CountCathepsin FBiologyCysteine Proteinase InhibitorsGiant CellsCathepsin BPathology and Forensic MedicineCathepsin CCathepsin BImmunoenzyme TechniquesTendonsCathepsin OCathepsin HAntigens CDCathepsin L1HumansCystatin CCathepsin SAgedMuscle NeoplasmsGiant Cell TumorsAntibodies MonoclonalMiddle AgedMolecular biologyCathepsinsCystatinsBiochemistryLeukocytes MononuclearFemaleModern pathology : an official journal of the United States and Canadian Academy of Pathology, Inc
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Longitudinal analysis of the T-cell receptor (TCR)-VA and -VB repertoire in CD8+T cells from individuals immunized with recombinant hepatitis B surfa…

2002

SUMMARYRecent studies have suggested that vaccination induces alterations in the T cell receptor (TCR) repertoire. We investigate the diversity of the TCR repertoire after immunization with a recombinant hepatitis B surface vaccine in seven healthy subjects in CD8+ T cells in peripheral blood lymphocytes. Cellular immune responses were monitored over time by sorting CD8 T cells followed by TCR-VA and -VB complementarity determining region 3 (CDR3) analysis. Frequency of individual VB families was determined by flow cytometry. TCR-VA/VB repertoires obtained from CD8+ T cells drawn after vaccination were compared to the TCR repertoire determined prior to vaccination. Monoclonal TCR transcript…

AdultMaleDNA ComplementaryReceptors Antigen T-Cell alpha-betaT cellMolecular Sequence DataImmunologychemical and pharmacologic phenomenaCD8-Positive T-LymphocytesBiologyEpitopeInterleukin 21AntigenAntibody SpecificityClinical StudiesmedicineHumansImmunology and AllergyCytotoxic T cellHepatitis B VaccinesAmino Acid SequenceLongitudinal StudiesGene Rearrangement beta-Chain T-Cell Antigen ReceptorImmunity CellularVaccines SyntheticBase SequenceT-cell receptorAntibodies Monoclonalhemic and immune systemsT lymphocyteMiddle AgedComplementarity Determining RegionsVirologymedicine.anatomical_structureImmunologyFemaleImmunizationGene Rearrangement alpha-Chain T-Cell Antigen ReceptorCD8Clinical and Experimental Immunology
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Severe meningo-/encephalitis after daclizumab therapy for multiple sclerosis.

2019

Background: Daclizumab is a monoclonal antibody that binds the high-affinity interleukin-2 receptor and was approved for the treatment of relapsing multiple sclerosis. Due to severe inflammatory brain disorders, the approval was suspended in March 2018. Objective and Methods: This retrospective cohort study summarizes clinical, laboratory, radiological, and histological findings of seven patients who developed meningo-/encephalitis after daclizumab therapy. Results: Patients presented with encephalitis and/or meningitis and suffered from systemic symptoms such as fever (5/7), exanthema (5/7), or gastrointestinal symptoms (4/7). Secondary autoimmune diseases developed. Blood analysis reveale…

AdultMaleDaclizumabMultiple Sclerosismedicine.drug_classMonoclonal antibodyAutoimmune Diseases03 medical and health sciences0302 clinical medicineDaclizumabmedicineHumansLymphocytes030304 developmental biologyRetrospective Studies0303 health sciencesbusiness.industryMultiple sclerosisMeningoencephalitisAntibodies MonoclonalBrainMiddle Agedmedicine.disease3. Good healthNeurologyImmunologyEncephalitisFemaleNeurology (clinical)business030217 neurology & neurosurgeryEncephalitisImmunosuppressive Agentsmedicine.drugMultiple sclerosis (Houndmills, Basingstoke, England)
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Precision Medicine in Graves’ Disease: CD40 Gene Variants Predict Clinical Response to an Anti-CD40 Monoclonal Antibody

2021

BackgroundCD40, a key co-stimulatory molecule expressed on antigen-presenting cells, is genetically associated with a number of autoimmune diseases including Graves’ disease (GD). Therefore, recent therapies targeting CD40 have been developed, including the anti-CD40 monoclonal antibody Iscalimab. In a recent pilot study, Iscalimab was shown to induce clinical remission in ~ 50% of GD patients, but the reason why only 50% of GD patients responded is not known. The aim of our study was to test the hypothesis that specific CD40 single nucleotide polymorphism (SNP) genotypes and haplotypes are associated with clinical response of GD patients to Iscalimab.MethodsWe extracted genomic DNA from th…

AdultMaleGenotypemedicine.drug_classprecision medicineEndocrinology Diabetes and MetabolismGraves' diseaseSingle-nucleotide polymorphismmedicine.disease_causeMonoclonal antibodyPolymorphism Single NucleotideDiseases of the endocrine glands. Clinical endocrinologyAutoimmunityYoung AdultEndocrinologyGenotypeCD40HumansMedicineSNPRNA MessengerCD40 AntigensgeneGeneAgedOriginal Researchbusiness.industryHaplotypeAntibodies MonoclonalMiddle AgedRC648-665medicine.diseaseGraves DiseasevariantImmunologyFemaleGraves’ diseasebusinessFrontiers in Endocrinology
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Detection of Epstein-Barr virus (EBV) DNA and antigens in oral mucosa of renal transplant patients without clinical evidence of oral hairy leukoplaki…

1998

The use of the polymerase chain reaction (PCR) to detect the presence of Epstein-Barr virus (EBV) DNA in oral mucosa in the absence of specific lesions gives rise to the problem of identifying the real viral replication sites. To verify whether the detection of EBV is due to salivary contamination or its true replicative capacity in oral mucosa, saliva samples and exfoliated cells from four different oral mucosa sites were taken from 40 renal transplant patients and 20 normal subjects for examination by PCR using two pairs of primers specific for the BamHI-L and BamHI-K genomic regions. EBV-specific sequences were detected in one or more of the oral mucosa samples from 29 transplant patient…

AdultMaleHairy leukoplakiaHerpesvirus 4 HumanCancer ResearchPathologymedicine.medical_specialtySalivaLeukoplakia HairyAdolescentCD4-CD8 RatioFluorescent Antibody TechniqueGenome ViralBiologyVirus Replicationmedicine.disease_causePolymerase Chain ReactionHerpesviridaePathology and Forensic Medicinelaw.inventionImmunocompromised HostlawmedicineHumansOral mucosaSalivaAntigens ViralIn Situ HybridizationPolymerase chain reactionOral hairy leukoplakiaMouth MucosaAntibodies MonoclonalHLA-DR AntigensSequence Analysis DNAMiddle Agedmedicine.diseaseKidney TransplantationEpstein–Barr virusImmunoglobulin ATransplantationBlotting Southernmedicine.anatomical_structureOtorhinolaryngologyImmunoglobulin GDNA ViralImmunologyPeriodonticsFemaleOral SurgeryJournal of Oral Pathology & Medicine
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Preferential splenic CD8+ T-cell activation in rituximab-nonresponder patients with immune thrombocytopenia

2013

The pathogenic role of B cells in immune thrombocytopenia (ITP) has justified the therapeutic use of anti-CD20 antibodies such as rituximab (RTX). However, 60% of ITP patients do not respond to RTX. To decipher the mechanisms implicated in the failure of RTX, and because the spleen plays a well-recognized role in ITP pathogenesis, 12 spleens from ITP patients who had been nonresponders to RTX therapy were compared with 11 spleens from RTX-untreated ITP patients and 9 controls. We here demonstrate that in RTX-nonresponder ITP patients, preferential Th1 and Tc1 T lymphocyte polarizations occur, associated with an increase in splenic effector memory CD8(+) T-cell frequency. Moreover, in the RT…

AdultMaleImmunologyDrug ResistanceSpleenCD8-Positive T-LymphocytesLymphocyte ActivationReal-Time Polymerase Chain ReactionBiochemistryPathogenesisAntibodies Monoclonal Murine-DerivedYoung Adultimmune system diseaseshemic and lymphatic diseasesmedicineHumansImmunologic FactorsCytotoxic T cellAgedAged 80 and overPurpura Thrombocytopenic Idiopathicbiologybusiness.industryCell BiologyHematologyT lymphocyteMiddle AgedImmunohistochemistrymedicine.anatomical_structureImmunologyMonoclonalbiology.proteinFemaleRituximabAntibodyRituximabbusinessSpleenCD8medicine.drugBlood
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Impaired cellular immune responses in chronic renal failure: Evidence for a T cell defect

1986

Impaired cellular immune responses in chronic renal failure: Evidence for a T cell defect. Cellular immune responses in vitro were studied in 24 patients on chronic hemodialysis and 16 healthy volunteers with normal kidney function. Patients on maintenance hemodialysis had lymphopenia with diminished numbers of both T4 + and T8 + T-lymphocytes. The T4/T8 ratios were within the normal range. Peripheral blood lymphocytes (PBL) showed a diminished proliferative response upon stimulation with concanavalin A, phytohemagglutinin and poke weed mitogen. When cell surface antigens were used for stimulation (mixed lymphocyte culture) uremic lymphocytes also showed a lower proliferation rate. Although…

AdultMaleIsoantigensT-LymphocytesLymphocyteT cellLymphocyte CooperationRenal functionStimulationchemical and pharmacologic phenomenaLymphocyte ActivationLeukocyte CountImmune systemmedicineHumansLymphocytesImmunodeficiencyAgedUremiaB-LymphocytesImmunity Cellularbiologybusiness.industryAntibodies MonoclonalMiddle Agedmedicine.diseaseIn vitromedicine.anatomical_structureNephrologyConcanavalin AAntibody FormationImmunologybiology.proteinInterleukin-2Kidney Failure ChronicFemaleMitogensbusinessKidney International
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Modification of Peripheral Blood T-Lymphocyte Surface Receptors and Langerhans Cell Numbers in Hereditary Angioedema

1986

Hereditary angioedema (HAE) is a disease related to a complement disorder, namely a deficiency of C1 esterase inhibitor. Complement-split products are implicated in the regulation of the immune response, and we have compared some immunologic parameters between HAE and normal individuals. T-lymphocytes with receptors for IgG were increased in HAE, but no difference in T-cell suppressor activity for B-cells was detected. Furthermore, increased IgG receptor expression was not accompanied by any significant changes in the ratios of OKT4- and OKT8-defined antigens. Numbers of peripheral mononuclear cells (MNC) detected by alpha-naphthyl acetate esterase (ANAE) staining positivity were not signif…

AdultMaleLangerhans cellT-LymphocytesReceptor expressionReceptors Antigen T-CellFluorescent Antibody TechniqueCell CountT-Lymphocytes RegulatoryPeripheral blood mononuclear cellMonocytesLeukocyte CountImmune systemmedicineHumansAngioedemaReceptorSkinChemistryAntibodies MonoclonalGeneral MedicineMiddle Agedmedicine.diseaseComplement systemmedicine.anatomical_structureLangerhans CellsHereditary angioedemaImmunologyFemaleCell activationAmerican Journal of Clinical Pathology
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Intrathecal somatic hypermutation of IgM in multiple sclerosis and neuroinflammation

2014

Intrathecal oligoclonal bands of the cerebrospinal fluid are considered the most important immunological biomarkers of multiple sclerosis. They typically consist of clonally expanded IgG antibodies that underwent affinity maturation during sustained stimulation by largely unknown antigens. In addition, ∼40% of patients with multiple sclerosis have oligoclonal bands that consist of expanded IgM antibodies. We investigated the molecular composition of IgM- and IgG-chains from cerebrospinal fluid of 12 patients with multiple sclerosis, seven patients with other neurological diseases, and eight healthy control subjects by high-throughput deep-sequencing and single-cell PCR. Further, we studied …

AdultMaleMultiple SclerosisMolecular Sequence DataSomatic hypermutationAntibodies Monoclonal HumanizedImmunoglobulin GAffinity maturationYoung AdultmedicineHumansAmino Acid SequenceAgedCell ProliferationAged 80 and overInflammationB-LymphocytesBase SequencebiologyNatalizumabMultiple sclerosisGerminal centerCytidine deaminaseMiddle Agedmedicine.diseaseImmunoglobulin MSpinal CordImmunoglobulin class switchingImmunoglobulin MImmunoglobulin GImmunologybiology.proteinFemaleSomatic Hypermutation ImmunoglobulinNeurology (clinical)Single-Cell AnalysisBrain
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High anti-lymphoma activity of bendamustine/mitoxantrone/rituximab in rituximab pretreated relapsed or refractory indolent lymphomas and mantle cell …

2007

On the basis of a preceding phase I study, the current trial explored bendamustine in combination with mitoxantrone and rituximab (BMR) in patients with stage III/IV relapsed or refractory indolent lymphomas and mantle cell lymphoma (MCL) with or without prior rituximab containing chemo-immunotherapy (R-chemo) treatment. Therapy consisted of bendamustine 90 mg/m(2) days 1 + 2, mitoxantrone 10 mg/m(2) day 1, rituximab 375 mg/m(2) day 8. Treatment was repeated on day 29 for a total of four cycles. Between 3 April and 04 July, 57 patients were recruited from 24 participating institutions, 39% of whom had received prior R-chemo therapy. Median age was 66 years (40 - 83). Lymphoma subtypes were …

AdultMaleOncologyBendamustineCancer Researchmedicine.medical_specialtyPathologyPhases of clinical researchLymphoma Mantle-CellAntibodies Monoclonal Murine-DerivedInternal medicineAntineoplastic Combined Chemotherapy ProtocolsmedicineBendamustine HydrochlorideHumansLymphoma FollicularSurvival analysisAgedAged 80 and overSalvage TherapyMitoxantronebusiness.industryLymphoma Non-HodgkinRemission InductionAntibodies MonoclonalHematologyMiddle Agedmedicine.diseaseSurvival AnalysisLymphomaClinical trialTreatment OutcomeOncologyNitrogen Mustard CompoundsMonoclonalFemaleRituximabMitoxantroneRituximabbusinessmedicine.drugLeukemia & Lymphoma
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