Search results for " neurons"

showing 10 items of 290 documents

Neuron-type specific cannabinoid-mediated G protein signalling in mouse hippocampus

2013

Type 1 cannabinoid receptor (CB1) is expressed in different neuronal populations in the mammalian brain. In particular, CB1 on GABAergic or glutamatergic neurons exerts different functions and display different pharmacological properties in vivo. This suggests the existence of neuron-type specific signalling pathways activated by different subpopulations of CB1. In this study, we analysed CB1 expression, binding and signalling in the hippocampus of conditional mutant mice, bearing CB1 deletion in GABAergic (GABA-CB1-KO mice) or cortical glutamatergic neurons (Glu-CB1-KO mice). Compared to their wild-type littermates, Glu-CB1-KO displayed a small decrease of CB1 mRNA amount, immunoreactivity…

Cannabinoid receptorG proteinmedicine.medical_treatmentHippocampusBiologyHippocampal formationHippocampusBiochemistryMice03 medical and health sciencesCellular and Molecular NeuroscienceGlutamatergic0302 clinical medicineGTP-binding protein regulatorsReceptor Cannabinoid CB1GTP-Binding ProteinsmedicineAnimalsGABAergic Neurons030304 developmental biologyMice Knockout0303 health sciencesCannabinoidsmusculoskeletal neural and ocular physiologyfood and beveragesMice Inbred C57BLnervous systemGABAergiclipids (amino acids peptides and proteins)CannabinoidNeurosciencepsychological phenomena and processes030217 neurology & neurosurgeryProtein BindingSignal TransductionJournal of Neurochemistry
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Circuit Specific Functions of Cannabinoid CB1 Receptor in the Balance of Investigatory Drive and Exploration

2011

Well balanced novelty seeking and exploration are fundamental behaviours for survival and are found to be dysfunctional in several psychiatric disorders. Recent studies suggest that the endocannabinoid (eCB) system is an important control system for investigatory drive. Pharmacological treatment of rodents with cannabinergic drugs results in altered social and object investigation. Interestingly, contradictory results have been obtained, depending on the treatment, drug concentration and experimental conditions. The cannabinoid type 1 (CB1) receptor, a central component of the eCB system, is predominantly found at the synapses of two opposing neuronal populations, i.e. on inhibitory GABAerg…

Cannabinoid receptorMousemedicine.medical_treatmentScienceGlutamic AcidNeural HomeostasisMice TransgenicBiologyMedium spiny neuronSynaptic Transmissiongamma-Aminobutyric acidGlutamatergicBehavioral NeuroscienceMiceModel OrganismsReceptor Cannabinoid CB1medicineGeneticsAnimalsGABAergic NeuronsSocial BehaviorBiologygamma-Aminobutyric AcidPsychiatryNeuronsMultidisciplinaryBehavior AnimalMood DisordersQRAnimal ModelsNeurotransmittersEndocannabinoid systemMice Inbred C57BLMental Healthnervous systemDopamine receptorMaladjustmentExploratory BehaviorGABAergicMedicineCannabinoidNeuroscienceAnimal Geneticsmedicine.drugResearch ArticleNeurosciencePLoS ONE
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Cell type‐specific genetic reconstitution of CB1 receptor subsets to assess their role in exploratory behaviour, sociability, and memory

2021

Several studies support the notion that exploratory behaviour depends on the functionality of the cannabinoid type 1 (CB1) receptor in a cell type-specific manner. Mice lacking the CB1 receptor in forebrain GABAergic or dorsal telencephalic glutamatergic neurons have served as essential tools revealing the necessary CB1 receptor functions in these two neuronal populations. However, whether these specific CB1 receptor populations are also sufficient within the endocannabinoid system for wild-type-like exploratory behaviour has remained unknown. To evaluate cell-type-specific sufficiency of CB1 receptor signalling exclusively in dorsal telencephalic glutamatergic neurons (Glu-CB1-RS) or in fo…

Cannabinoid receptormedicine.medical_treatmentBiologyMice03 medical and health sciencesGlutamatergic0302 clinical medicineReceptor Cannabinoid CB1medicineAnimalsGABAergic NeuronsReceptorgamma-Aminobutyric Acid030304 developmental biologyMice Knockout0303 health sciencesmusculoskeletal neural and ocular physiologyGeneral NeuroscienceGlutamate receptorfood and beveragesEndocannabinoid systemMice Inbred C57BLnervous systemForebrainExploratory BehaviorGABAergiclipids (amino acids peptides and proteins)CannabinoidNeurosciencepsychological phenomena and processes030217 neurology & neurosurgeryEndocannabinoidsEuropean Journal of Neuroscience
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Heterodimer formation of wild-type and amyotrophic lateral sclerosis-causing mutant Cu/Zn-superoxide dismutase induces toxicity independent of protei…

2008

Recent studies provide evidence that wild-type Cu/Zn-superoxide dismutase (SOD1(hWT)) might be an important factor in mutant SOD1-mediated amyotrophic lateral sclerosis (ALS). In order to investigate its functional role in the pathogenesis of ALS, we designed fusion proteins of two SOD1 monomers linked by a polypeptide. We demonstrated that wild-type-like mutants, but not SOD1(G85R) homodimers, as well as mutant heterodimers including SOD1(G85R)-SOD1(hWT) display dismutase activity. Mutant homodimers showed an increased aggregation compared with the corresponding heterodimers in cell cultures and transgenic Caenorhabditis elegans, although SOD1(G85R) heterodimers are more toxic in functiona…

Cell SurvivalRecombinant Fusion Proteinsanimal diseasesSOD1MutantProtein aggregationAnimals Genetically ModifiedProtein CarbonylationSuperoxide dismutaseMicechemistry.chemical_compoundSuperoxide Dismutase-1Cell Line TumorGeneticsAnimalsHumansAmino Acid SequenceCaenorhabditis elegansMolecular BiologyGenetics (clinical)Motor NeuronsbiologySuperoxide DismutaseSuperoxideAmyotrophic Lateral SclerosisWild typenutritional and metabolic diseasesHydrogen PeroxideGeneral MedicineFusion proteinProtein Structure Tertiarynervous system diseasesCell biologyAmino Acid Substitutionnervous systemchemistryBiochemistrybiology.proteinDismutaseDimerizationHuman Molecular Genetics
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Isolation and characterization of a murine resident liver stem cell.

2008

Increasing evidence provides support that mammalian liver contains stem/progenitor cells, but their molecular phenotype, embryological derivation, biology and their role in liver cell turnover and regeneration remain to be further clarified. In this study, we report the isolation, characterization and reproducible establishment in line of a resident liver stem cell (RLSC) with immunophenotype and differentiative potentiality distinct from other previously described liver precursor/stem cells. RLSCs, derived from fetal and neonatal murine livers as well as from immortalized hepatocytic MMH lines and established in lines, are Sca+, CD34-, CD45-, alpha-fetoprotein+ and albumin-. This molecular…

Cellular differentiationLiver Stem CellCell SeparationBiologyImmunophenotypingLiver progenitor cellsMiceChondrocyteshepatocyteAnimalsCell LineageProgenitor cellLiver progenitor cells; hepatocyte; differentiationMolecular BiologyCells CulturedMultipotent Stem CellOligonucleotide Array Sequence AnalysisNeuronsOsteoblastsAnimalOligonucleotide Array Sequence AnalysiLiver cellOsteoblastGene Expression ProfilingMultipotent Stem CellsMesenchymal stem cellCell DifferentiationCell BiologydifferentiationNeuronChondrocyteMolecular biologyLiver regenerationCell biologyPhenotypeAnimals NewbornLiverMultipotent Stem CellHepatocytesStem cellAnimals; Animals Newborn; Cell Differentiation; Cell Lineage; Cell Separation; Cells Cultured; Chondrocytes; Gene Expression Profiling; Hepatocytes; Immunophenotyping; Liver; Mice; Multipotent Stem Cells; Neurons; Oligonucleotide Array Sequence Analysis; Osteoblasts; Phenotype; Molecular Biology; Cell BiologyCell death and differentiation
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Bidirectional Control between Cholesterol Shuttle and Purine Signal at the Central Nervous System.

2022

Recent studies have highlighted the mechanisms controlling the formation of cerebral cholesterol, which is synthesized in situ primarily by astrocytes, where it is loaded onto apolipoproteins and delivered to neurons and oligodendrocytes through interactions with specific lipoprotein receptors. The “cholesterol shuttle” is influenced by numerous proteins or carbohydrates, which mainly modulate the lipoprotein receptor activity, function and signaling. These molecules, provided with enzymatic/proteolytic activity leading to the formation of peptide fragments of different sizes and specific sequences, could be also responsible for machinery malfunctions, which are associated with neurological…

Central Nervous SystemNeuronsNiemann-Pick DiseasesOrganic ChemistryReceptors PurinergicLDL receptorLDL receptors; cholesterol; purinergic receptors; Cholesterol; Humans; Neurons; Purines; Receptors Purinergic; Central Nervous System; Niemann-Pick DiseasesPurinergicGeneral MedicineRECEPTORESCatalysisComputer Science ApplicationsInorganic ChemistryCholesterolPurinespurinergic receptorsReceptorsLDL receptorsHumansPhysical and Theoretical ChemistryMolecular BiologySpectroscopyInternational journal of molecular sciences
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Nitrergic modulation of gastrointestinal function during early endotoxemia.

2006

After bacterial infection, the host reacts by signalling to the central nervous system where a cascade of physiologic, neuroendocrine and behavioural processes is orchestrated, collectively termed the acute phase response. Endotoxemia following Gram-negative bacterial infection induces a wide array of effects, including fever, loss of appetite and changes in gastrointestinal function that attempt to eliminate the challenge and restore homeostasis. Systemic administration of low doses of endotoxin (5-40 microg/kg) to rats is associated with changes in gastrointestinal motor function, inhibition of gastric acid secretion and increase in the gastric mucosal resistance to damage. These changes …

Central Nervous Systemmedicine.medical_specialtyCentral nervous systemMyenteric PlexusNitric OxideNitric oxideGastric Acidchemistry.chemical_compoundInternal medicineNitrergic NeuronsDrug DiscoverymedicineAnimalsHumansPharmacologyGastrointestinal tractbiologyStomachVagus NerveEndotoxemiaRatsNitric oxide synthaseGastrointestinal Tractmedicine.anatomical_structureEndocrinologychemistryGastric MucosaRegional Blood Flowbiology.proteinGastric acidNitric Oxide SynthaseGastrointestinal functionGastrointestinal MotilityHomeostasisCurrent pharmaceutical design
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Computation of inverse functions in a model of cerebellar and reflex pathways allows to control a mobile mechanical segment.

2003

Abstract The command and control of limb movements by the cerebellar and reflex pathways are modeled by means of a circuit whose structure is deduced from functional constraints. One constraint is that fast limb movements must be accurate although they cannot be continuously controlled in closed loop by use of sensory signals. Thus, the pathways which process the motor orders must contain approximate inverse functions of the bio-mechanical functions of the limb and of the muscles. This can be achieved by means of parallel feedback loops, whose pattern turns out to be comparable to the anatomy of the cerebellar pathways. They contain neural networks able to anticipate the motor consequences …

CerebellumEfferentMovementModels NeurologicalSensory systemOlivary NucleusCerebellar CortexArtificial IntelligenceCerebellumNeural PathwaysReflexmedicineSet (psychology)Muscle SkeletalRed NucleusMotor NeuronsNeuronsArtificial neural networkGeneral NeuroscienceSupervised learningExtremitiesBiomechanical Phenomenamedicine.anatomical_structureMemory Short-TermCerebellar NucleiCerebellar cortexReflexNeural Networks ComputerPsychologyNeuroscienceAlgorithmsMuscle ContractionNeuroscience
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Making Waves: Initiation and Propagation of Corticothalamic Ca2+ Waves In Vivo

2013

Corticothalamic slow oscillations of neuronal activity determine internal brain states. At least in the cortex, the electrical activity is associated with large neuronal Ca(2+) transients. Here we implemented an optogenetic approach to explore causal features of the generation of slow oscillation-associated Ca(2+) waves in the in vivo mouse brain. We demonstrate that brief optogenetic stimulation (3-20 ms) of a local group of layer 5 cortical neurons is sufficient for the induction of global brain Ca(2+) waves. These Ca(2+) waves are evoked in an all-or-none manner, exhibit refractoriness during repetitive stimulation, and propagate over long distances. By local optogenetic stimulation, we …

Cerebral CortexRefractory periodGeneral NeuroscienceNeuroscience(all)ThalamusMice TransgenicStimulationCortical neuronsBiologyOptogeneticsCortex (botany)Mice Inbred C57BLOptogeneticsMiceThalamusIn vivoNeural PathwaysAnimalsPremovement neuronal activityCalcium SignalingNeurosciencePhotic StimulationVisual CortexNeuron
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Multiple mechanisms of secondary hyperalgesia

2000

Publisher Summary This chapter discusses the multiple mechanisms of secondary hyperalgesia. The chapter defines the minimal conditions of complexity that must be fulfilled by a model of plasticity of spinal nociceptive transmission in order to explain clinical and psychophysical observations in humans. Secondary hyperalgesia is characterized by a leftward shift of the stimulus-response function for noxious mechanical stimuli. In order to define the afferent pathways involved in inducing central sensitization and in mediating the hyperalgesia to noxious mechanical stimuli, several psychophysical experiments using selective nerve block techniques is performed. Secondary hyperalgesia is likely…

ChemistryNociceptionmedicine.anatomical_structurenervous systemSpinal Cord Dorsal HornNeuropathic painHyperalgesiamedicineNociceptive NeuronsIn patientNeuronmedicine.symptomNeuroscienceSensitization
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