Search results for " neurons"

showing 10 items of 290 documents

Probabilistic Contextuality in EPR/Bohm-type Systems with Signaling Allowed

2014

In this chapter, we review a principled way of defining and measuring contextuality in systems with deterministic inputs and random outputs, recently proposed and developed in \citep{KujalaDzhafarovLarsson2015,DKL2015FooP}.

Quantum PhysicsFOS: Biological sciencesQuantitative Biology - Neurons and CognitionProbability (math.PR)FOS: MathematicsFOS: Physical sciencesNeurons and Cognition (q-bio.NC)Quantum Physics (quant-ph)81P13 81Q99 60A99Mathematics - Probability
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The neurobiological bases for the pharmacotherapy of nicotine addiction.

2007

Nicotine, the major psychoactive agent present in tobacco, acts as a potent addictive drug both in humans and laboratory animals, whose locomotor activity is also stimulated. A large body of evidence indicates that the locomotor activation and the reinforcing effects of nicotine may be related to its stimulatory effects on the mesolimbic dopaminergic function. Thus, it is now well established that nicotine can increase in vivo DA outflow in the nucleus accumbens and the corpus striatum. The stimulatory effect of nicotine on DA release most probably results from its ability to excite the neuronal firing rate and to increase the bursting activity of DA neurons in the substantia nigra pars com…

RAT STRIATAL SYNAPTOSOMESNicotineINDUCED BEHAVIORAL SENSITIZATIONmedia_common.quotation_subjectSubstantia nigraStriatumNicotinic AntagonistsBiologyNucleus accumbensPharmacologyReceptors NicotinicNicotineDrug DiscoverySUSTAINED-RELEASE BUPROPIONmedicineLOCOMOTOR STIMULANT ACTIONAnimalsHumansNicotinic Agonistsmedia_commonPharmacologyMIDBRAIN DOPAMINE NEURONSPars compactaAddictionNIGRA PARS COMPACTAFACILITATES SMOKING CESSATIONTobacco Use DisorderSUBUNIT MESSENGER-RNAAntidepressive AgentsVentral tegmental areaVENTRAL TEGMENTAL AREANicotinic agonistmedicine.anatomical_structurenervous systemmedicine.drugSEROTONIN(2C) RECEPTORS BLOCKSCurrent pharmaceutical design
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Reelin and CXCL12 regulate distinct migratory behaviors during the development of the dopaminergic system.

2014

The proper functioning of the dopaminergic system requires the coordinated formation of projections extending from dopaminergic neurons in the substantia nigra (SN), ventral tegmental area (VTA) and retrorubral field to a wide array of forebrain targets including the striatum, nucleus accumbens and prefrontal cortex. The mechanisms controlling the assembly of these distinct dopaminergic cell clusters are not well understood. Here, we have investigated in detail the migratory behavior of dopaminergic neurons giving rise to either the SN or the medial VTA using genetic inducible fate mapping, ultramicroscopy, time-lapse imaging, slice culture and analysis of mouse mutants. We demonstrate that…

Receptors CXCR4Cell Adhesion Molecules NeuronalDopamineEmbryonic DevelopmentSubstantia nigraNerve Tissue ProteinsStriatumBiologyNucleus accumbensLigandsModels BiologicalTime-Lapse ImagingMiceCell MovementDopaminergic CellmedicineAnimalsCell LineageReelinMolecular BiologyMice KnockoutExtracellular Matrix ProteinsDopaminergic NeuronsDopaminergicSerine EndopeptidasesVentral Tegmental AreaAnatomyChemokine CXCL12Ventral tegmental areaSubstantia NigraReelin Proteinmedicine.anatomical_structurenervous systemForebrainbiology.proteinNeuroscienceDevelopmental BiologySignal TransductionDevelopment (Cambridge, England)
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Role of nitric oxide synthase isoforms for ophthalmic artery reactivity in mice.

2014

Abstract Nitric oxide synthases (NOS) are involved in regulation of ocular vascular tone and blood flow. While endothelial NOS (eNOS) has recently been shown to mediate endothelium-dependent vasodilation in mouse retinal arterioles, the contribution of individual NOS isoforms to vascular responses is unknown in the retrobulbar vasculature. Moreover, it is unknown whether the lack of a single NOS isoform affects neuron survival in the retina. Thus, the goal of the present study was to examine the hypothesis that the lack of individual nitric oxide synthase (NOS) isoforms affects the reactivity of mouse ophthalmic arteries and neuron density in the retinal ganglion cell (RGC) layer. Mice defi…

Retinal Ganglion CellsVasodilator AgentsNitric Oxide Synthase Type IIVideo microscopyVasodilationCell CountNitric Oxide Synthase Type IMuscle Smooth Vascularchemistry.chemical_compoundMiceOphthalmic ArteryPhenylephrineEnosEnzyme InhibitorsMice KnockoutbiologyAnatomySensory SystemsNitric oxide synthaseIsoenzymesVasodilationmedicine.anatomical_structureNG-Nitroarginine Methyl EsterRetinal ganglion cellKnockout mouseRetinal NeuronsNitroprussidemedicine.medical_specialtyNitric Oxide Synthase Type IIIEndothelial NOSNitric oxideCellular and Molecular NeuroscienceTonometry OcularInternal medicinemedicineAnimalsNitric Oxide DonorsIntraocular Pressurebusiness.industrybiology.organism_classificationAcetylcholineMice Inbred C57BLOphthalmologyEndocrinologychemistryVasoconstrictionbiology.proteinAdrenergic alpha-1 Receptor AgonistsEndothelium VascularbusinessExperimental eye research
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Seeing with the visual cortex

1984

A short analysis of the input-output organization of the primary visual cortical areas in the cat and monkey is followed by a description of the salient microelectrophysiological properties of retino-geniculo-cortical system neurons. It is concluded that a strict hierarchical model of cortical processing of visual information is no longer tenable.

Retinal Ganglion Cellsgenetic structuresMotion PerceptionDermatologyVisual systemCortical processingmedicineAnimalsVisual PathwaysBinocular neuronsVisual CortexBrain MappingOrientation columnGeneral NeuroscienceGeniculate BodiesHaplorhiniGeneral MedicineForm PerceptionPsychiatry and Mental healthVisual cortexmedicine.anatomical_structureSalientSynapsesCatsVisual PerceptionNeurology (clinical)Visual FieldsPsychologyNeuroscienceThe Italian Journal of Neurological Sciences
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A novel automated segmentation method for retinal layers in OCT images proves retinal degeneration after optic neuritis.

2015

Aim The evaluation of inner retinal layer thickness can serve as a direct biomarker for monitoring the course of inflammatory diseases of the central nervous system such as multiple sclerosis (MS). Using optical coherence tomography (OCT), thinning of the retinal nerve fibre layer and changes in deeper retinal layers have been observed in patients with MS. Here, we first compare a novel method for automated segmentation of OCT images with manual segmentation using two cohorts of patients with MS. Using this method, we also aimed to reproduce previous findings showing retinal degeneration following optic neuritis (ON) in MS. Methods Based on a 5×5 expansion of the Prewitt operator to efficie…

Retinal degenerationAdultMalePathologymedicine.medical_specialtyMultiple SclerosisOptic Neuritisgenetic structuresDiagnostic Techniques Ophthalmological03 medical and health sciencesCellular and Molecular Neurosciencechemistry.chemical_compoundYoung Adult0302 clinical medicineNerve FibersOptical coherence tomographyOphthalmologyMedicineHumansSegmentationOptic neuritisGanglion cell layerRetinamedicine.diagnostic_testbusiness.industryRetinal DegenerationReproducibility of ResultsRetinalMiddle Agedmedicine.diseaseeye diseasesSensory SystemsOphthalmologymedicine.anatomical_structurechemistry030221 ophthalmology & optometryOptic nerveFemalesense organsbusiness030217 neurology & neurosurgeryAlgorithmsBiomarkersTomography Optical CoherenceRetinal NeuronsThe British journal of ophthalmology
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The existence of FGFR1-5-HT1A receptor heterocomplexes in midbrain 5-HT neurons of the rat: relevance for neuroplasticity.

2012

The ascending midbrain 5-HT neurons to the forebrain may be dysregulated in depression and have a reduced trophic support. Within situproximity ligation assay (PLA) and supported by coimmunoprecipitation and colocation of the FGFR1 and 5-HT1A immunoreactivities in the midbrain raphe cells, evidence for the existence of FGFR1–5-HT1A receptor heterocomplexes in the dorsal and median raphe nuclei of the Sprague Dawley rat as well as in the rat medullary raphe RN33B cells has been obtained. Especially after combined FGF-2 and 8-OH-DPAT treatment, a marked and significant increase in PLA clusters was found in the RN33B cells. Similar results were reached with the FRET technique in HEK293T cells,…

Retractedmedicine.medical_specialtySerotonin receptorsEncèfalSettore BIO/11 - Biologia MolecolareBiologySettore BIO/09 - FisiologiaReceptors de serotoninaMidbrainInternal medicineRatesmedicineReceptor5-HT receptorNeuronal Plasticity Receptor Fibroblast Growth Factor Receptor Serotonin 5-HT1A Serotonergic Neurons SerotoninRapheGeneral NeuroscienceEncephalonFibroblastsRatsEndocrinologymedicine.anatomical_structurenervous systemForebrainAutoreceptor5-HT1A receptorNeuron
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The effect of neurodegenerative lesions on the mechanism of motor resonance induced by action observation

2018

The concept of “embodied cognition” considers that the classical Perception-Cognition-Action architecture proposing a sequential flow of processing with clean cuts between all modules is not appropriate to understand the behavioral effect of neurodegenerative disorders and to find innovative therapeutic solutions. In the last decades, the discovery of the mirror neurons (MN) has given a biological substrate to this theoretical perspective: the MN are now thought linking together knowledge about actions and perceptions not only to integrate perception in action planning and execution but also as a neural mechanism supporting a wide range of cognitive functions, e.g. empathy and language. At …

Résonance motrice[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyMirror neuronsNeurodegenerative diseasesMaladies neurodégénérativesMotor resonance[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyNeurones miroirsEmbodied cognition
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Rot1 plays an antagonistic role to Clb2 in actin cytoskeleton dynamics throughout the cell cycle.

2007

ROT1 is an essential gene whose inactivation causes defects in cell cycle progression and morphogenesis in budding yeast. Rot1 affects the actin cytoskeleton during the cell cycle at two levels. First, it is required for the maintenance of apical growth during bud growth. Second, Rot1 is necessary to polarize actin cytoskeleton to the neck region at the end of mitosis; because of this defect, rot1 cells do not properly form a septum to complete cell division. The inability to polarize the actin cytoskeleton at the end of mitosis is not due to a defect in the recruitment of the polarisome scaffold protein Spa2 or the actin cytoskeleton regulators Cdc42 and Cdc24 in the neck region. Previous …

Saccharomyces cerevisiae ProteinsGenes FungalArp2/3 complexmacromolecular substancesSaccharomyces cerevisiaeCyclin BActin remodeling of neuronsGene Expression Regulation FungalCDC2-CDC28 KinasesCytoskeletonCytoskeletonPolarisomebiologyCell CycleActin remodelingCell PolarityMembrane ProteinsCell BiologyActin cytoskeletonActinsCell biologyProfilinParacytophagyMutationbiology.proteinMolecular ChaperonesJournal of cell science
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Nicotine-induced FGF-2 mRNA in rat brain is preserved during aging

2004

Indirect trophic actions of nicotine on brain during aging are suggested from observations describing nicotine as a cognitive enhancer, increasing vigilance and improving learning and memory, and both in vitro and in vivo models have demonstrated neuroprotective effects of nAChR agonists. Previously, we have reported that an acute intermittent (-)nicotine treatment significantly increases fibroblast growth factor-2 (FGF-2) mRNA and protein in several brain regions of rat brain. The present study was designed to analyse if nicotine-induced FGF-2 expression in the rat brain was preserved during aging. Using in situ hybridization and quantitative RNase protection assay the present paper report…

SenescenceNicotineAgingmedicine.medical_specialtyCentral nervous systemStimulationIn situ hybridizationBiologynAChRNeuroprotectionNicotine treatmentStriatumNicotineHippocampuInternal medicineGliamedicineSubstantia nigraAnimalsTissue DistributionRNA MessengerAcetylcholine receptorGeneral NeuroscienceBrainNeuronFGF-2 expression; nicotine treatment; hippocampus; substantia nigra; striatum; brain; neurons; glia; nAChR; neurotrophism; neuroprotectionNeuroprotectionRatsmedicine.anatomical_structureEndocrinologyGene Expression RegulationFGF-2 expressionNeurotrophismFibroblast Growth Factor 2Neurology (clinical)NeuronGeriatrics and GerontologyDevelopmental Biologymedicine.drug
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