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showing 10 items of 1985 documents

Metabolic adaptation in the human gut microbiota during pregnancy and the first year of life

2018

Abstract Background The relationship between the gut microbiome and the human host is dynamic and we may expect adjustments in microbiome function if host physiology changes. Metatranscriptomic approaches should be key in unraveling how such adjustments occur. Methods We employ metatranscriptomic sequencing analyses to study gene expression in the gut microbiota of infants through their first year of life, and of their mothers days before delivery and one year afterwards. Findings In infants, hallmarks of aerobic metabolism disappear from the microbial metatranscriptome as development proceeds, while the expression of functions related to carbohydrate transport and metabolism increases and …

AdultMale0301 basic medicineResearch paperCarbohydrate transportPregnancy Trimester ThirdPhysiologyFirst year of lifeButyrateGut floraGeneral Biochemistry Genetics and Molecular BiologyFeces03 medical and health sciences0302 clinical medicineBacterial ProteinsPregnancymedicineHumansGutMicrobiomeMetatranscriptomicsPregnancyBacteriabiologySequence Analysis RNAGene Expression ProfilingMicrobiotaInfant NewbornInfantGene Expression Regulation BacterialGeneral MedicineMetabolismLipid Metabolismbiology.organism_classificationmedicine.diseaseIntestinal epitheliumGastrointestinal MicrobiomeButyratesMetabolism030104 developmental biology030220 oncology & carcinogenesisFemaleMaternal AgeEBioMedicine
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Chlorinated Hydrocarbon Residues (PCB and DDT) in Human Liver, Adipose Tissue and Brain in Finland

2009

In this work the concentration of chlorinated hydrocarbons (PCB and DDT) was analyzed from 73 autopsy samples. Half of the material came from the coastal area of the Gulf of Bothnia and the other half from Central Finland. The correlation of the concentration of the drug to age, sex and the locality was studied as well as the occupation of the dead subjects.

AdultMaleAdolescentAdipose tissueToxicologyDDTSex Factorsparasitic diseasesHumansOccupationsChildFinlandAgedPharmacologychemistry.chemical_classificationHuman liverAge FactorsInfant NewbornBrainInfantMiddle AgedPolychlorinated BiphenylsHydrocarbonAdipose TissueLiverchemistryChild PreschoolEnvironmental chemistryFemalegeographic locationsActa Pharmacologica et Toxicologica
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PORCN mutations in focal dermal hypoplasia: coping with lethality.

2009

Contains fulltext : 81709.pdf (Publisher’s version ) (Closed access) The X-linked dominant trait focal dermal hypoplasia (FDH, Goltz syndrome) is a developmental defect with focal distribution of affected tissues due to a block of Wnt signal transmission from cells carrying a detrimental PORCN mutation on an active X-chromosome. Molecular characterization of 24 unrelated patients from different ethnic backgrounds revealed 23 different mutations of the PORCN gene in Xp11.23. Three were microdeletions eliminating PORCN and encompassing neighboring genes such as EBP, the gene associated with Conradi-Hunermann-Happle syndrome (CDPX2). 12/24 patients carried nonsense mutations resulting in loss …

AdultMaleAdolescentBase SequenceDNA Mutational AnalysisMolecular Sequence DataInfant NewbornInfantMembrane ProteinsGenomic disorders and inherited multi-system disorders [IGMD 3]Focal Dermal HypoplasiaSettore MED/38 - Pediatria Generale E SpecialisticaSettore MED/03 - Genetica MedicaChild PreschoolMutationGoltz syndrome FDH PORCN WNT skewed X-inactivation postzygotic mosaicHumansProtein IsoformsFemaleAmino Acid SequenceChildAcyltransferasesHuman Mutation
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Functional characterization of hepatocytes for cell transplantation: customized cell preparation for each receptor.

2009

The first indication of hepatocyte transplantation is inborn liver-based metabolic disorders. Among these, urea cycle disorders leading to the impairment to detoxify ammonia and Crigler-Najjar Syndrome type I, a deficiency in the hepatic UDP-glucuronosyltransferase 1A1 present the highest incidence. Metabolically qualified human hepatocytes are required for clinical infusion. We proposed fast and sensitive procedures to determine their suitability for transplantation. For this purpose, viability, attachment efficiency, and metabolic functionality (ureogenic capability, cytochrome P450, and phase II activities) are assayed prior to clinical cell infusion to determine the quality of hepatocyt…

AdultMaleAdolescentCell SurvivalCell TransplantationCellBiomedical Engineeringlcsh:MedicineReceptors Cell SurfaceCell SeparationPharmacologyCold Ischemia TimeDonor Selectionchemistry.chemical_compoundYoung AdultmedicineHumansUreaGlucuronosyltransferaseReceptorChildUrea Cycle Disorders InbornCells CulturedAgedCrigler-Najjar SyndromeAged 80 and overTransplantationLiver DiseasesMetabolic disorderlcsh:RCold IschemiaGraft SurvivalInfant NewbornInfantCell BiologyMiddle Agedmedicine.diseaseTransplantationmedicine.anatomical_structurechemistryUrea cycleChild PreschoolUreaHepatocytesBiological AssayFemaleSteatosisCell transplantation
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Social factors related to the clinical severity of influenza cases in Spain during the A (H1N1) 2009 virus pandemic

2013

Abstract Background During the 2009 influenza pandemic, a change in the type of patients most often affected by influenza was observed. The objective of this study was to assess the role of individual and social determinants in hospitalizations due to influenza A (H1N1) 2009 infection. Methods We studied hospitalized patients (cases) and outpatients (controls) with confirmed influenza A (H1N1) 2009 infection. A standardized questionnaire was used to collect data. Variables that might be related to the hospitalization of influenza cases were compared by estimation of the odds ratio (OR) and 95% confidence intervals (CI) and the variables entered into binomial logistic regression models. Resu…

AdultMaleAdolescentComorbidityInfluenza A Virus H1N1 SubtypeOccupational ExposureInfluenza HumanHumansMass ScreeningChildPandemicsPandemiclcsh:Public aspects of medicineInfant NewbornPublic Health Environmental and Occupational HealthInfantvirus diseaseslcsh:RA1-1270Confounding Factors EpidemiologicHealth Status DisparitiesHospitalizationInfluenza A (H1N1) 2009Social ClassSocial factorsSpainCase-Control StudiesChild PreschoolFemaleResearch ArticleBMC Public Health
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Characterization of 14 novel deletions underlying Rubinstein–Taybi syndrome: an update of the CREBBP deletion repertoire

2015

Rubinstein–Taybi syndrome (RSTS) is a rare, clinically heterogeneous disorder characterized by cognitive impairment and several multiple congenital anomalies. The syndrome is caused by almost private point mutations in the CREBBP (~55 % of cases) and EP300 (~8 %) genes. The CREBBP mutational spectrum is variegated and characterized by point mutations (30–50 %) and deletions (~10 %). The latter are diverse in size and genomic position and remove either the whole CREBBP gene and its flanking regions or only an intragenic portion. Here, we report 14 novel CREBBP deletions ranging from single exons to the whole gene and flanking regions which were identified by applying complementary cytomolecu…

AdultMaleAdolescentContiguous gene syndromeCohort StudiesExonGeneticmedicineGeneticsHumansPoint MutationCREB-binding proteinEP300ChildPreschoolGenetics (clinical)Sequence DeletionGeneticsRubinstein-Taybi Syndromebiologymedicine.diagnostic_testRubinstein–Taybi syndromeBase SequencePoint mutationMedicine (all)Infant NewbornInfantMiddle Agedmedicine.diseaseNewbornCREB-Binding ProteinHuman geneticsAdolescent; Adult; CREB-Binding Protein; Child; Child Preschool; Cohort Studies; Female; Humans; Infant; Infant Newborn; Male; Middle Aged; Rubinstein-Taybi Syndrome; Base Sequence; Point Mutation; Sequence Deletion; Genetics (clinical); Genetics; Medicine (all)Child Preschoolbiology.proteinFemaleCohort StudieAdolescent; Adult; CREB-Binding Protein; Child; Child Preschool; Cohort Studies; Female; Humans; Infant; Infant Newborn; Male; Middle Aged; Rubinstein-Taybi Syndrome; Base Sequence; Point Mutation; Sequence Deletion; Medicine (all); Genetics; Genetics (clinical)Fluorescence in situ hybridizationHuman
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Calcification patterns of the internal elastic membrane

1969

Calcification patterns of the internal elastic membrane of the main pelvic arteries, lower limb arteries, brachial, splenic and renal arteries were demonstrated grossly by a modified von Kossa technique. In the elastic segment in the common and internal iliac arteries, the membrane calcification appeared as groups of roundish or polygonal incrustations. They were found frequently in newborns, and were always present in infants of more than three months, as well as in adults. In the muscular arteries, the calcified parts of the internal elastic membrane appeared grossly as pairs of bands (“calcific bands”) along the edges of the pre-existing gaps in this membrane. When calcification was pron…

AdultMaleAdolescentEndocrinology Diabetes and MetabolismLower limbEndocrinologyAge groupsmedicineHumansOrthopedics and Sports MedicineInternal Elastic MembraneChildVon Kossa stainMedial surfaceMembranesChemistryAge FactorsInfant NewbornCalcinosisInfantArteriesGeneral MedicineAnatomyMiddle AgedElastic Tissuemedicine.diseaseChild PreschoolFemaleCalcificationCalcified Tissue Research
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Establishment of standardised SLA/LP immunoassays: specificity for autoimmune hepatitis, worldwide occurrence, and clinical characteristics

2002

Background: Antibodies to soluble liver antigen/liver pancreas (SLA/LP) are specific markers of autoimmune hepatitis. Their target antigen has recently been cloned. Aims: To establish standardised immunoassays using the recombinant antigen, and to assess the frequency and significance of seropositivity in patients from different countries. Methods: An enzyme linked immunoassay was developed using purified recombinant antigen and validated by testing sera from 200 healthy blood donors and 1026 patients with various liver and non-liver diseases. The assay was then applied to 454 sera from 419 patients with autoimmune hepatitis from different countries. All sera were also tested by inhibition …

AdultMaleAdolescentEnzyme-Linked Immunosorbent AssayAutoimmune hepatitisSensitivity and Specificitylaw.inventionJapanMaintenance therapyAntigenRecurrencelawGermanymedicineHumansChildAutoimmune diseaseHepatitisbiologymedicine.diagnostic_testbusiness.industryLiver DiseasefungiHistocompatibility Antigens Class IHistocompatibility Antigens Class IIInfant NewbornGastroenterologyAntibodies MonoclonalInfantmedicine.diseaseRecombinant ProteinsUnited StatesHepatitis AutoimmuneTreatment OutcomeChild PreschoolImmunoassayImmunologybiology.proteinRecombinant DNAFemaleAntibodybusinessBiomarkersBrazilGut
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Translocations Disrupting PHF21A in the Potocki-Shaffer-Syndrome Region Are Associated with Intellectual Disability and Craniofacial Anomalies

2012

Contains fulltext : 110038.pdf (Publisher’s version ) (Closed access) Potocki-Shaffer syndrome (PSS) is a contiguous gene disorder due to the interstitial deletion of band p11.2 of chromosome 11 and is characterized by multiple exostoses, parietal foramina, intellectual disability (ID), and craniofacial anomalies (CFAs). Despite the identification of individual genes responsible for multiple exostoses and parietal foramina in PSS, the identity of the gene(s) associated with the ID and CFA phenotypes has remained elusive. Through characterization of independent subjects with balanced translocations and supportive comparative deletion mapping of PSS subjects, we have uncovered evidence that t…

AdultMaleAdolescentGenotypePotocki–Shaffer syndromeChromosome DisordersHaploinsufficiencyBiologyHistone DeacetylasesSodium ChannelsTranslocation GeneticArticleChromatin remodelingCraniofacial Abnormalities03 medical and health sciencesSCN3A0302 clinical medicineIntellectual DisabilityNAV1.3 Voltage-Gated Sodium ChannelmedicineTranscriptional regulationGeneticsAnimalsHumansDeletion mappingGenetics(clinical)CraniofacialZebrafishGenetics (clinical)030304 developmental biologyGenetics0303 health sciencesChromosomes Human Pair 11Infant Newbornmedicine.diseaseGenetics and epigenetic pathways of disease DCN MP - Plasticity and memory [NCMLS 6]Child PreschoolHomeoboxFemaleChromosome DeletionHaploinsufficiencyExostoses Multiple Hereditary030217 neurology & neurosurgeryThe American Journal of Human Genetics
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Single-Nucleotide Polymorphism Array-Based Characterization of Ring Chromosome 18

2012

Objective To study genotype–phenotype correlation of ring chromosome 18 [r(18)] in 9 patients with 46,XN karyotype. Study design In 9 patients with a de novo 46,XN,r(18) karyotype (7 females, 2 males), we performed high-resolution single-nucleotide polymorphism array analysis (Illumina Human Omni1-QuadV1 array in 6 patients, Affymetrix 6.0 array in 3 patients), investigation of parental origin, and genotype–phenotype correlation. Results No breakpoint was recurrent. Single metaphases with loss of the ring, double rings, or secondarily rearranged rings were found in some cases, but true mosaicism was present in none of these cases. In 3 patients, additional duplications in 18p (of 1.4 Mb, 2 …

AdultMaleAdolescentRing chromosomeSingle-nucleotide polymorphismBiologyPolymorphism Single NucleotideCROMOSSOMOS HUMANOS (ANORMALIDADES;COMPLICAÇÕES)Young AdultMeiosisPolymorphism (computer science)SNPBody SizeHumansRing ChromosomesChildGenetic Association StudiesOligonucleotide Array Sequence AnalysisGeneticsBreakpointInfant NewbornInfantKaryotypeMiddle AgedPhenotypeChild PreschoolKaryotypingPediatrics Perinatology and Child HealthFemaleChromosome DeletionChromosomes Human Pair 18HeadMaternal AgeMicrosatellite Repeats
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