Search results for " oxidative"

showing 10 items of 234 documents

Distribution of betalain pigments in red blood cells after consumption of cactus pear fruits and increased resistance of the cells to ex vivo induced…

2005

Betalain pigments are bioavailable phytochemicals recently acknowledged as natural radical scavengers. This work, which extends previous research on the postabsorbitive fate of dietary betalains, investigated the distribution of betanin and indicaxanthin in red blood cells (RBCs) isolated from healthy volunteers (n = 8), before and during the 1-8 h interval after a cactus pear fruit meal, and the potential antioxidative activity of the pigments in these cells. A peak concentration of indicaxanthin (1.03 +/- 0.2 microM) was observed in RBCs isolated at 3 h after fruit feeding, whereas the concentration at 5 h was about half, and even smaller amounts were measured at 8 h. Indicaxanthin was no…

AdultCactaceaeMaleAntioxidantErythrocytesIndolesPyridinesmedicine.medical_treatmentBetalainsindicaxanthinred blood cellBiologyHemolysischemistry.chemical_compoundBetalainBotanymedicineHumansFood sciencecactus pear; betalains; betanin; indicaxanthin; red blood cell; oxidative hemolysis; bioavailable phytochemicalsBetaninbioavailable phytochemicalsbetaninbetalainGeneral Chemistrymedicine.diseaseHemolysisBetaxanthinsDietQuaternary Ammonium CompoundsRed blood celloxidative hemolysiKineticsmedicine.anatomical_structurechemistryCumene hydroperoxideFruitcactus pearFemaleBetacyaninsGeneral Agricultural and Biological SciencesIndicaxanthinEx vivoJournal of agricultural and food chemistry
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Differences in the behavior of advanced glycation end products and advanced oxidation protein products in patients with allergic rhinitis

2013

BACKGROUND: The presence of oxidative stress in patients with asthma is well documented; however, the role of oxidative stress in allergic rhinitis has received less attention, although it is likely to be similar to that observed in patients with asthma. Advanced glycation end products (AGEs) and advanced oxidation protein products (AOPPs) are compounds formed by the transformation of macromolecules, including proteins, which can serve as densitometric markers of oxidative stress and inflammation in several diseases. OBJECTIVE: The aim of this study was to investigate the role of AGEs and AOPPs as new markers of oxidative stress and inflammation in patients affected by allergic rhinitis. ME…

AdultGlycation End Products AdvancedMaleRhinitis Allergic PerennialSettore MED/09 - Medicina InternaAllergyAllergic rhinitisAllergic rhinitiHumansAdvanced glycation end productsAgedPeroxidaseAdvanced oxidation protein productImmunoglobulin EMiddle AgedAdvanced glycation end products.; Advanced oxidation protein products; Allergic rhinitis; Oxidative stress.; AllergyRhinitis AllergicOxidative StressSpectrometry FluorescenceAdvanced Oxidation Protein ProductsSpectrophotometryCase-Control StudiesOxidative stress: AllergyFemaleAdvanced glycation end productOxidation-ReductionBiomarkers
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Assessment of the cytotoxic potential of an aqueous-ethanolic extract from Thalassia testudinum angiosperm marine grown in the Caribbean Sea

2018

Abstract Objectives Reported antioxidant, anti-inflammatory and neuroprotective properties for one aqueous-ethanolic extract from Thalassia testudinum which grows in the Caribbean Sea compelled us to explore about extract cytotoxic effects. Methods Cell viability was assayed on tumour (HepG2, PC12, Caco-2 and 4T1) and non-tumour (VERO, 3T3, CHO, MCDK and BHK2) cell lines. The extract effects upon primary cultures of rat and human hepatocytes and human lymphocytes were assayed. Key findings The extract exhibited cytotoxicity against cancer cells compared to normal cells, and the IC50 values were 102 μg/ml for HepG2, 135 μg/ml for PC12, 165 μg/ml for Caco-2 and 129 μg/ml for 4T1 cells after 4…

AdultMale0301 basic medicineCell SurvivalDNA damagePrimary Cell CulturePharmaceutical ScienceHydrocharitaceaePC12 CellsRats Sprague-DawleyInhibitory Concentration 5003 medical and health sciencesNeoplasmsAnimalsHumansCytotoxic T cellLymphocytesViability assayCytotoxicityPharmacologyDose-Response Relationship DrugEthanolbiologyPlant ExtractsChemistryPharmacology. TherapyWaterHep G2 Cellsbiology.organism_classificationAntineoplastic Agents PhytogenicMolecular biologyRatsComet assay030104 developmental biologyCaribbean RegionCell cultureThalassia testudinumCancer cellSolventsCaco-2 CellsThalassia testudinum DNA damage cytotoxicity oxidative stressJournal of pharmacy and pharmacology
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Impaired plasma nitric oxide availability and extracellular superoxide dismutase activity in healthy humans with advancing age

2006

This study is aimed to verify the modifications of extracellular superoxide dismutase (EC-SOD) activity and its potential involvement on the mechanism responsible for the impairment of plasma nitric oxide (NO) availability occurring with advancing age in healthy humans. For this purpose, plasma samples were drawn from 40 healthy men, aged 20-92 years, in fasting state and used for measurements of stable end-product nitrite/nitrate (NOx), as expression of NO availability, EC-SOD activity, thiobarbituric acid reactive substances (TBARS) as marker of lipid peroxidation, Trolox equivalent antioxidant capacity (TEAC) as a measure of plasma total antioxidant capacity, and in vitro susceptibility …

AdultMaleAgingmedicine.medical_specialtyAntioxidantThiobarbituric acidextracellular superoxide dismutasemedicine.medical_treatmentTrolox equivalent antioxidant capacitymedicine.disease_causeAntioxidantsGeneral Biochemistry Genetics and Molecular BiologyNitric oxideLipid peroxidationchemistry.chemical_compoundNitric oxide Extracellular superoxide dismutase ; Oxidative stress; Advancing ageadvancing agenitric oxideInternal medicinemedicineTBARSAnimalsHumansoxidative stressGeneral Pharmacology Toxicology and PharmaceuticsAgedAged 80 and overSuperoxide DismutaseFastingGeneral MedicineMiddle AgedLipidsEndocrinologychemistryBiochemistryLow-density lipoproteinLipid PeroxidationExtracellular SpaceOxidative stressLife Sciences
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Oxidative stress markers at birth: Analyses of a neonatal population

2015

In order to further understand neonatal stress and, thus, control it efficaciously, there is a need for more information on the manifestations of stress at the molecular level in the newborn, with particular regard to oxidants, and anti-oxidant and anti-stress mechanisms, including mitochondrial heat shock protein-chaperones such as Hsp60. We investigated patterns of anti-oxidants, biomarkers of oxidative stress, and Hsp60 levels in sera from newborns and found significant associations between glutathione (GSH) levels and gestational age, delivery modality, and lipid hydroperoxydes (LOOH) level. LOOH levels and spontaneous (vaginal) delivery were independently associated with increased GSH …

AdultMaleLipid Peroxidesanimal structuresHistologyNeonatal stressPopulationNeonatal strePhysiologyOxidative-stress markerDiseaseBiologymedicine.disease_causeMitochondrial Proteinschemistry.chemical_compoundLipid hydroperoxydemedicineHumanseducationOxidative-stress markerseducation.field_of_studyfungiInfant NewbornAnti-stress moleculeGestational ageChaperonin 60Cell BiologyGeneral MedicineGlutathioneHsp60GlutathioneNeonatal stress; Oxidative-stress markers; Lipid hydroperoxydes; Anti-stress molecules; Glutathione; Hsp60Oxidative StressAdult lifeLipid hydroperoxydeschemistryAnti-stress moleculesImmunologyFemaleHSP60BiomarkersOxidative stressNeonatal stressActa Histochemica
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Analysis of the correlations between oxidative stress, gelatinases and their tissue inhibitors in the human subjects with obstructive sleep apnea syn…

2015

Obstructive sleep apnea syndrome (OSAS) is commonly associated with endothelial dysfunction, atherosclerosis and cardiovascular disorders. On the basis of this observation, our aim was to examine the oxidative status and the matrix metalloproteases (MMP) profile in a group of subjects with OSAS. We enrolled 48 subjects with OSAS defined after a 1-night cardiorespiratory sleep study, who were subsequently subdivided in two subgroups according to the severity of OSAS (low grade = L-OSAS; high grade= H-OSAS). We measured the parameters of oxidative stress, such as lipid peroxidation, protein oxidation, total antioxidant status (TAS), nitric oxide metabolites (NOx), and the plasma concentration…

AdultMaleSleep Apnea ObstructiveTissue Inhibitor of Metalloproteinase-2Tissue Inhibitor of Metalloproteinase-1Settore MED/09 - Medicina InternaMiddle AgedNitric OxideThiobarbituric Acid Reactive SubstancesOxidative StressMatrix Metalloproteinase 9Thiobarbituric Acid Reactive SubstanceHumansMatrix Metalloproteinase 2FemaleSettore MED/36 - Diagnostica Per Immagini E RadioterapiaAdult; Aged; Female; Humans; Male; Matrix Metalloproteinase 2; Matrix Metalloproteinase 9; Middle Aged; Nitric Oxide; Sleep Apnea Obstructive; Thiobarbituric Acid Reactive Substances; Tissue Inhibitor of Metalloproteinase-1; Tissue Inhibitor of Metalloproteinase-2; Oxidative StressAgedHuman
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Glutathione levels in blood from ataxia telangiectasia patients suggest in vivo adaptive mechanisms to oxidative stress

2007

Objective: To evaluate an in vivo pro-oxidant state in patients with ataxia telangiectasia (AT). Methods: A set of oxidative stress endpoints were measured in 9 AT homozygotes, 16 AT heterozygotes (parents) and 83 controls (grouped in age ranges as for patients and parents, respectively). The following analytes were measured: (a) leukocyte 8-hydroxy-2-deoxyguanosine (8-OHdG); (b) blood glutathione (GSSG and GSH); and (c) plasma levels of glyoxal (Glx) and methylglyoxal (MGlx). Results: AT patients displayed a significant decrease in blood GSSG (p=0.012) and in MGlx plasma concentrations (P=0.012). A nonsignificant decrease in the GSSG:GSH ratio (p = 0.1) and a non-significant increase in 8-…

AdultMalemedicine.medical_specialtyAdolescentglyoxalClinical Biochemistryataxia telengiectasiamedicine.disease_causeAtaxia Telangiectasiachemistry.chemical_compoundIn vivoInternal medicinemethylglyoxalmedicineHumansataxia telangectasiaoxidative stressglutathioneChildoxidative streMethylglyoxalDeoxyguanosine8-Hydroxy-2'-deoxyguanosineHeterozygote advantageGeneral MedicineGlutathionePyruvaldehydemedicine.diseaseAdaptation Physiological8-Hydroxy-2'-deoxyguanosineEndocrinologychemistryBiochemistryChild PreschoolAtaxia-telangiectasiaFemaleataxia telangectasia; oxidative stress; glutathione; glyoxal; methylglyoxal; 8-Hydroxy-2'-deoxyguanosineOxidative stressTarget organDNA DamageClinical Biochemistry
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Age, homocysteine, and oxidative stress: Relation to hypertension and type 2 diabetes mellitus

2010

Abstract OBJECTIVES: Hyperhomocysteinemia and oxidative stress are independent risk factors for cardiovascular events, which occur more frequently in old age. We evaluated these parameters in relation to age and the presence of hypertension and type 2 diabetes mellitus. METHODS: Two hundred eighty-two subjects (female/male: 142/140; 141 were >65 years and 141 were <65 years; mean age 73.9 +/- 6.6 years and 52.5 +/- 8.2 years, respectively) were randomly recruited from those attending our institution. Blood pressure, anthropometric parameters, oxidative stress parameters (reactive oxygen species [ROS] and malondialdehyde [MDA]), and homocysteine levels were evaluated in participants. RESULTS…

AdultMalemedicine.medical_specialtyHyperhomocysteinemiaAgingSettore MED/09 - Medicina InternaHomocysteineMedicine (miscellaneous)Type 2 diabetesmedicine.disease_causechemistry.chemical_compoundInternal medicineDiabetes mellitusMalondialdehydeMedicineHumansHomocysteineAgedNutrition and Dieteticsbusiness.industryAge FactorsType 2 Diabetes MellitusMiddle Agedmedicine.diseaseMalondialdehydeaging homocysteine oxidative stress hypertension type 2 diabetesOxidative StressBlood pressureEndocrinologychemistryDiabetes Mellitus Type 2HypertensionFemalebusinessReactive Oxygen SpeciesOxidative stress
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Inverse correlation between plasma oxysterol and LDL-cholesterol levels in hepatitis C virus-infected patients

2012

Background: Hepatitis C virus infection is characterised by enhanced oxidative stress, which can be measured quantitatively by plasma oxysterol concentration. These molecules may affect lipid metabolism through the activation of Liver X Receptors. Hepatitis C virus exploits host lipid metabolism to facilitate its replication and diffusion. In our study we aimed to evaluate and highlight the potential pathogenetic role of oxysterols, 7-ketocholesterol and 7-β-hydroxycholesterol, in hepatitis C virus-related lipid dysmetabolism. Methods: The study was performed in 42 patients with chronic hepatitis C (93% genotype 1b) and 38 non-alcoholic fatty liver disease patients. Plasma oxysterols 7-keto…

AdultMalemedicine.medical_specialtyOxysterolHepatitis C virusHepacivirusIsotope dilutionmedicine.disease_causechronic hepatitis c infection; lipid metabolism; non-alcoholic fatty liver disease; oxidative stressNon-alcoholic Fatty Liver DiseaseInternal medicinemedicinepolycyclic compoundsoxysterol hcv nafldHumansoxysterols sterols cholesterol mass spectrometry metabolomicsLiver X receptorKetocholesterolsHepatologybusiness.industryFatty liverGastroenterologyLipid metabolismHepatitis CCholesterol LDLMiddle Agedmedicine.diseaseHepatitis CChronic hepatitis C infectionHydroxycholesterolsFatty LiverOxidative StressEndocrinologyLipid metabolismBiochemistryMultivariate AnalysisLinear Modelslipids (amino acids peptides and proteins)FemalebusinessOxidative stress
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Analysis of thiamine transporter genes in sporadic beriberi

2014

Abstract Objective Thiamine or vitamin B 1 deficiency diminishes thiamine-dependent enzymatic activity, alters mitochondrial function, impairs oxidative metabolism, and causes selective neuronal death. We analyzed for the first time, the role of all known mutations within three specific thiamine carrier genes, SLC19 A2, SLC19 A3 , and SLC25 A19 , in a patient with atrophic beriberi, a multiorgan nutritional disease caused by thiamine deficiency. Methods A 44-year-old male alcoholic patient from Morocco developed massive bilateral leg edema, a subacute sensorimotor neuropathy, and incontinence. Despite normal vitamin B 1 serum levels, his clinical picture was rapidly reverted by high-dose in…

AdultMalemedicine.medical_specialtySLC19 A- SLC25 A19SLC19 AEndocrinology Diabetes and MetabolismGene mutationBeriberimedicine.disease_causeMitochondrial Membrane Transport Proteinslaw.inventionBeriberilawInternal medicineGenotypemedicineThiamine transporterObjective: Thiamine or vitamin B1 deficiency diminishes thiamine-dependent enzymatic activity alters mitochondrial function impairs oxidative metabolism and causes selective neuronal death. We analyzed for the first time the role of all known mutations within three specific thiamine carrier genes SLC19 A2 SLC19 A3 and SLC25 A19 in a patient with atrophic beriberi a multiorgan nutritional disease caused by thiamine deficiency. Methods: A 44-year-old male alcoholic patient from Morocco developed massive bilateral leg edema a subacute sensorimotor neuropathy and incontinence. Despite normal vitamin B1 serum levels his clinical picture was rapidly reverted by high-dose intramuscular thiamine treatment suggesting a possible genetic resistance. We used polymerase chain reaction followed by amplicon sequencing to study all the known thiamine-related gene mutations identified within the Human Gene Mutation Database. Results: Thirty-seven mutations were tested: 29 in SLC19 A2 6 in SLC19 A3 and 2 in SLC25 A19. Mutational analyses showed a wild-type genotype for all sequences investigated. Conclusion: This is the first genetic study in beriberi disease. We did not detect any known mutation in any of the three genes in a sporadic dry beriberi patient. We cannot exclude a role for other known or unknown mutations in the same genes or in other thiamine-associated genes in the occurrence of this nutritional neuropathy.HumansThiamineGenePolymerase chain reactionGeneticsMutationNutrition and DieteticsbiologyMembrane Transport ProteinsThiamine Deficiencymedicine.diseaseAlcoholismEndocrinologyMutationbiology.proteinThiamineMutations
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