Search results for " polymorphism"

showing 10 items of 1028 documents

Different Transcriptional Activity and In Vitro TNF-α Production in Psoriasis Patients Carrying the TNF-α 238A Promoter Polymorphism

2000

Genes encoded on chromosome 6 within the major histocompatibility complex region are thought to play an important role in the pathogenesis of psoriasis. A potential candidate gene is tumor necrosis factor alpha. The tumor necrosis factor alpha promoter contains several polymorphisms including two G--A transitions at position -308 and -238, which are the most common in Caucasian populations. The TNF238.2 (-238A) allele has been strongly associated with psoriasis. We have investigated the effect of the -238 and -308 variants on transcription of the tumor necrosis factor alpha gene in luciferase reporter gene assays. In addition, peripheral blood mononuclear cells of 47 patients with psoriasis…

AdultMaleTranscription Geneticmedicine.medical_treatmentT cellDermatologyBiologyBiochemistryPeripheral blood mononuclear cellAntigenPsoriasisTNFαmedicineSuperantigenHumansPsoriasisPromoter Regions GeneticMolecular Biologytranscriptional activityAgedAged 80 and overPBMGPolymorphism GeneticTumor Necrosis Factor-alphaPromoterCell BiologyMiddle Agedmedicine.diseaseMolecular biologypromoter polymorphismCytokinemedicine.anatomical_structureImmunologyLeukocytes MononuclearFemaleTumor necrosis factor alphaJournal of Investigative Dermatology
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Vascular endothelial growth factor gene polymorphisms in Behçet's disease

2004

Objective. To evaluate potential associations of vascular endothelial growth factor (VEGF) gene polymorphisms with Behçet's disease (BD) and disease expression. Methods. Case patients were 122 consecutive Italian patients with BD followed at the Rheumatology, Ophthalmology, and Neurology Units in Bologna, Ferrara, Milano, Palermo, Potenza, Prato, Reggio Emilia, and Trento over a 3-year period (1997-99) and who satisfied the International Study Group criteria for BD. Also selected as a control group were 200 healthy age and sex matched blood donors. All patients with BD and controls were genotyped by polymerase chain reaction and allele-specific oligonucleotide techniques for +936 C/T (rs302…

AdultMaleVascular Endothelial Growth Factor Aclinical manifestationsPolymorphism GeneticAdolescentGenotypeBehcet SyndromeVEGF productionBehcet's diseaseVEGF polymorphismGene FrequencyLeukocytes MononuclearHumansAdolescent; Adult; Behcet Syndrome; Cells Cultured; Female; Gene Frequency; Genetic Predisposition to Disease; Genotype; Humans; Leukocytes Mononuclear; Male; Vascular Endothelial Growth Factor A; Polymorphism GeneticFemaleGenetic Predisposition to DiseaseCells Cultured
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Lack of association between ubiquitin carboxy-terminal hydrolase L1 gene polymorphism and PD.

2001

In 1998, an IL93Met mutation in the ubiquitin carboxy-terminal hydrolase L1 ( UCH-L1 ) gene was identified in a German family affected by PD.1 Recently, others2-4⇓⇓ found that the S18Y polymorphism in exon 3 of UCH-L1 is associated with a low risk of PD. To verify these interesting results, we decided to design a case-control study on the S18Y polymorphism of the UCH-L1 gene using sporadic PD cases. In the meantime, as we were analyzing our samples, a case-control study5 on 142 patients with PD and 142 age- and sex-matched control subjects did not confirm the protective effect found by Maraganore et al.2 In view of these conflicting findings, we reasoned that our contribution may have some …

AdultMaleallele frequenciesParkinson's diseasegenotypepolymorphismlaw.inventionExonDegenerative diseaseUbiquitinlawHydrolasemedicineHumansGeneNeural Cell Adhesion MoleculesPolymerase chain reactionAgedAged 80 and overNeuroscience (all)Membrane GlycoproteinsPolymorphism GeneticbiologyUCH-L1 geneParkinson DiseaseMiddle Agedmedicine.diseaseUbiquitin carboxy-terminal hydrolase L1Settore BIO/18 - GeneticaImmunologybiology.proteinSettore MED/26 - NeurologiaFemaleNeurology (clinical)Gene polymorphismThiolester HydrolasesLeukocyte L1 Antigen ComplexUbiquitin ThiolesteraseNeurology
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Distribution, genetic and cardiovascular determinants of FVIII:c - Data from the population-based Gutenberg Health Study

2015

Background: Elevated levels of FVIII:c are associated with risk for both venous and arterial thromboembolism. However, no population-based study on the sex-specific distribution and reference ranges of plasma FVIII: c and its cardiovascular determinants is available. Methods: FVIII:c was analyzed in a randomly selected sample of 2533 males and 2440 females from the Gutenberg Health Study in Germany. Multivariable regression analyses for FVIII:c were performed under adjustment for genetic determinants, cardiovascular risk factors and cardiovascular disease. Results and conclusions: Females (126.6% (95% CI: 125.2/128)) showed higher FVIII:c levels than males (121.2% (119.8/122.7)). FVIII:c le…

AdultMalecongenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyLinkage disequilibriumGenotypeanimal diseasesPopulationFVIII:c reference valuesSingle-nucleotide polymorphismDiseaseAge DistributionVon Willebrand factorGermanyThromboembolismhemic and lymphatic diseasesInternal medicineVenous thrombosisHumansMedicineGenetic Predisposition to DiseaseProspective StudiesSex DistributioneducationAgededucation.field_of_studyEpidemiological studiesFactor VIIIPolymorphism Geneticbiologybusiness.industryIncidenceC-reactive proteinArterial thrombosisDNAMiddle AgedNomogrammedicine.diseaseVenous thrombosisPopulation SurveillanceImmunologybiology.proteinFemaleCardiology and Cardiovascular MedicinebusinessFollow-Up Studies
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Allelic variants of IL1R1gene associate with severe hand osteoarthritis

2010

Background In search for genes predisposing to osteoarthritis (OA), several genome wide scans have provided evidence for linkage on 2q. In this study we targeted a 470 kb region on 2q11.2 presenting the locus with most evidence for linkage to severe OA of distal interphalangeal joints (DIP) in our genome wide scan families. Methods We genotyped 32 single nucleotide polymorphisms (SNPs) in this 470 kb region comprising six genes belonging to the interleukin 1 superfamily and monitored for association with individual SNPs and SNP haplotypes among severe familial hand OA cases (material extended from our previous linkage study; n = 134), unrelated end-stage bilateral primary knee OA cases (n =…

AdultMalelcsh:Internal medicineLinkage disequilibriumLINKAGE DISEQUILIBRIUMlcsh:QH426-470Hand JointsNF-KAPPA-BSingle-nucleotide polymorphismLocus (genetics)KNEE OSTEOARTHRITISBiologyRADIOGRAPHIC SIGNSPolymorphism Single NucleotideSeverity of Illness IndexGenomeCHROMOSOME 2QINTERLEUKIN-1 RECEPTOR ANTAGONIST03 medical and health sciences0302 clinical medicineOsteoarthritisGeneticsHumansSNPGenetic Predisposition to DiseaseGenetics(clinical)Allelelcsh:RC31-1245GeneAllelesPOLYMORPHISMSGenetics (clinical)AgedHAPLOTYPE RECONSTRUCTION030304 developmental biologyReceptors Interleukin-1 Type I030203 arthritis & rheumatologyGenetics0303 health sciencesHaplotypeCLUSTERMiddle Aged314 Health sciences3. Good healthlcsh:GeneticsCase-Control StudiesDISC DEGENERATIONFemaleResearch ArticleBMC Medical Genetics
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Role of proinflammatory alleles in longevity and atherosclerosis: results of studies performed on -1562C/T MMP-9 in centenarians and myocardial infar…

2007

Centenarians are characterized by marked delay or escape from age-associated diseases that cause mortality at earlier ages. Jointly, atherosclerosis and its complications, such as myocardial infarction (AMI), significantly contribute to mortality in the elderly. Inflammation is a key component of atherosclerosis and inflammatory genes are good candidates for the risk of the development of atherosclerosis. Genetic traits contribute to the risk of AMI and allelic variations in inflammatory genes should boost the risk of disease. If proinflammatory genotypes significantly contribute to the risk of AMI, alleles associated with disease susceptibility should not be included in the genetic backgro…

AdultMalemedia_common.quotation_subjectLongevityMyocardial InfarctionInfarctionInflammationSingle-nucleotide polymorphismDiseaseCoronary Artery DiseaseBiologyPolymorphism Single NucleotideGeneral Biochemistry Genetics and Molecular BiologyProinflammatory cytokineCohort StudiesMetalloproteaseHistory and Philosophy of ScienceGene FrequencymedicineSNPHumansAllelePolymorphismSicilyAllelesmedia_commonAged 80 and overInflammationGeneral NeuroscienceLongevityMiddle Agedmedicine.diseaseMatrix Metalloproteinase 9InfarctionImmunologyFemalemedicine.symptomAnnals of the New York Academy of Sciences
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Influence of CYP3A5 and ABCB1 gene polymorphisms and other factors on tacrolimus dosing in Caucasian liver and kidney transplant patients

2011

Tacrolimus is a substrate of cytochrome P4503A (CYP3A) enzymes as well as of the drug transporter ABCB1. We have investigated the possible influence of CYP3A5 and ABCB1 single nucleotide polymorphisms (SNPs) and other factors (e.g. albumin, hematocrit and steroids) on tacrolimus blood levels achieved in a population of Caucasian liver (n=51) and kidney (n=50) transplant recipients. At 1, 3 and 6 months after transplantation, tacrolimus doses (mg/kg/day) and trough blood levels (C0) were recorded and the weight-adjusted tacrolimus dosage (mg/kg/day) was calculated. Polymerase chain reaction followed by restriction fragment length polymorphism analysis was used for genotyping CYP3A5*1 and *3 …

AdultMalemedicine.medical_specialtyATP Binding Cassette Transporter Subfamily BGenotypemedicine.medical_treatmentDNA Mutational AnalysisPopulationSingle-nucleotide polymorphismLiver transplantationBiologyKidneyPolymorphism Single NucleotideGastroenterologyBiomarkers PharmacologicalTacrolimusWhite PeopleGene FrequencyInternal medicineGeneticsmedicineCytochrome P-450 CYP3AHumansDrug Dosage CalculationsATP Binding Cassette Transporter Subfamily B Member 1educationAllele frequencyAllelesKidney transplantationAgededucation.field_of_studyKidney metabolismGeneral MedicineMiddle Agedmedicine.diseaseKidney TransplantationTacrolimusLiver TransplantationTransplantationsurgical procedures operativeItalyLiverImmunologySettore BIO/14 - FarmacologiaPharmacogenetics CYP3A5 ABCB1 TacrolimusTransplant patientsFemaleImmunosuppressive AgentsPolymorphism Restriction Fragment LengthInternational Journal of Molecular Medicine
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Alcohol Consumption in HealthyOPRM1G Allele Carriers and Its Association with Impulsive Behavior

2015

AIMS: A link between alcohol use disorders (AUD) and impulsivity is well established. As there is evidence for the heritability of AUD, the investigation of the underlying genetic disposition for both conditions is an important issue. An association between AUD and a coding single nucleotide polymorphism (SNP) (rs1799971 encoding an Asn40Asp amino acid substitution, A118G) within the µ-opioid receptor 1 gene (OPRM1) has been reported. Therefore we tested the association between the OPRM1 A118G polymorphism and drinking as well as impulsive behavior in social drinkers. METHODS: A total of 214 healthy male social drinkers were recruited. Each participant was genotyped for the OPRM1 A118G vari…

AdultMalemedicine.medical_specialtyAlcohol DrinkingGenotypePerseverationReceptors Opioid muPoison controlSingle-nucleotide polymorphismImpulsivityPolymorphism Single NucleotideYoung AdultPolymorphism (computer science)medicineGenetic predispositionHumansGenetic Predisposition to DiseaseAllelePsychiatryAllelesAlcohol Use Disorders Identification TestGeneral MedicineMiddle AgedImpulsive Behaviormedicine.symptomPsychologyClinical psychologyAlcohol and Alcoholism
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Genetic variation of the interleukin-1 family and nongenetic factors determining the interleukin-1 receptor antagonist phenotypes.

2009

The natural anti-inflammatory protein interleukin-1 receptor antagonist (IL-1Ra) inhibits the activity of IL-1 and is associated with vascular injury and metabolic disorders. We analyzed genetic and nongenetic determinants of the IL-1Ra phenotype. Fifteen haplotype-tagging single nucleotide polymorphisms (SNPs) in the IL-1α (IL1A), IL-1β (IL1B), and IL-1 receptor antagonist (IL-1RN) genes were determined in the Health 2000 survey (n = 6771) and European myocardial infarction (MI) survivors (n = 972). Three SNPs were genotyped in the FINRISK97 (FR97) study (n = 7222). We found 3 IL1RN variants that were associated with the IL-1Ra phenotype in the study populations and remained significant af…

AdultMalemedicine.medical_specialtyEndocrinology Diabetes and MetabolismMyocardial InfarctionSingle-nucleotide polymorphism030204 cardiovascular system & hematologyBiologyPolymorphism Single Nucleotide03 medical and health sciences0302 clinical medicineEndocrinologyGene FrequencyMeta-Analysis as TopicInternal medicineGenetic variationmedicineHumansLongitudinal StudiesSurvivorsAllele frequency030304 developmental biologyAged2. Zero hungerGenetics0303 health sciencesGenetic VariationMiddle AgedExplained variation3. Good healthMinor allele frequencyInterleukin 1 Receptor Antagonist ProteinInterleukin 1 receptor antagonistEndocrinologyPhenotypeIL1AMultigene FamilyFemaleBody mass indexInterleukin-1Metabolism: clinical and experimental
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Evidences of +896 A/G TLR4 Polymorphism as an Indicative of Prevalence of Complications in T2DM Patients

2014

T2DM is today considered as world-wide health problem, with complications responsible of an enhanced mortality and morbidity. Thus, new strategies for its prevention and therapy are necessary. For this reason, the research interest has focused its attention on TLR4 and its polymorphisms, particularly the rs4986790. However, no conclusive findings have been reported until now about the role of this polymorphism in development of T2DM and its complications, even if a recent meta-analysis showed its T2DM association in Caucasians. In this study, we sought to evaluate the weight of rs4986790 polymorphism in the risk of the major T2DM complications, including 367 T2DM patients complicated for th…

AdultMalemedicine.medical_specialtyGenotypeArticle SubjectT2DM TLR4 +896A/G SNP T2DM complicationsImmunologyPolymorphism Single NucleotideLower limbGene FrequencyDiabetes mellitusInternal medicineGenotypelcsh:PathologymedicineHumansSettore MED/05 - Patologia ClinicaGenetic Predisposition to DiseaseAllele frequencyAgedAged 80 and overSettore MED/04 - Patologia Generalebusiness.industryConfoundingTLR4 POLYMORPHISMCell BiologyMiddle Agedmedicine.diseaseSurgeryToll-Like Receptor 4Cumulative riskDiabetes Mellitus Type 2FemaleComplicationbusinessResearch Articlelcsh:RB1-214
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