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showing 10 items of 846 documents

RNA recognition by human TLR8 can lead to autoimmune inflammation

2013

High expression level of human TLR8 in mice results in spontaneous, multiorgan inflammation attributable in part to increased DC activation.

MaleT cellT-LymphocytesImmunologyArthritisInflammationMice TransgenicAutoimmunityTRL8AUTOIMMUNE INFLAMMATIONBiologymedicine.disease_causeArticleAutoimmunityProinflammatory cytokineMiceTRL8; AUTOIMMUNE INFLAMMATIONhemic and lymphatic diseasesmedicineImmunology and AllergyAnimalsHumansTransgenesChildRandomized Controlled Trials as TopicInflammationGene Expression ProfilingTLR7TLR8medicine.diseaseArthritis Experimentaldigestive system diseasesArthritis JuvenileMice Inbred C57BLmedicine.anatomical_structureToll-Like Receptor 7Toll-Like Receptor 8ImmunologyRNAFemaleCollagenSignal transductionmedicine.symptomSignal TransductionThe Journal of Experimental Medicine
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Partial Sleep Restriction Activates Immune Response-Related Gene Expression Pathways: Experimental and Epidemiological Studies in Humans

2013

Epidemiological studies have shown that short or insufficient sleep is associated with increased risk for metabolic diseases and mortality. To elucidate mechanisms behind this connection, we aimed to identify genes and pathways affected by experimentally induced, partial sleep restriction and to verify their connection to insufficient sleep at population level. The experimental design simulated sleep restriction during a working week: sleep of healthy men (N = 9) was restricted to 4 h/night for five nights. The control subjects (N = 4) spent 8 h/night in bed. Leukocyte RNA expression was analyzed at baseline, after sleep restriction, and after recovery using whole genome microarrays complem…

MaleTBX21NF-KAPPA-Blcsh:MedicineNK cellsBioinformaticskokeellinen tutkimusReceptors G-Protein-Coupled0302 clinical medicineCARDIOMETABOLIC RISKLeukocytesta319geeniekspressiolcsh:Scienceta515Sleep restrictionRegulation of gene expression0303 health sciencesMultidisciplinaryNATURAL-KILLERNF-kappa Bta3142Sleep in non-human animalsC-REACTIVE PROTEIN3. Good healthMACROPHAGE APOPTOSISINSUFFICIENT SLEEPSTAT1 Transcription FactorCARDIOVASCULAR-DISEASEimmuunivasteProteoglycansmedicine.symptomResearch ArticleAdulteducationENDOPLASMIC-RETICULUMMETABOLIC CONSEQUENCESSyntaxin 16Biologyepidemiologinen tutkimusuni (lepotila)03 medical and health sciencesImmune systemmedicineHumans030304 developmental biologyTOLL-LIKE RECEPTORSB cellsuniMicroarray analysis techniquesGene Expression Profilingsytokiinitlcsh:RMicroarray AnalysisGene expression profilingSleep deprivationGene Expression RegulationImmunologyRNASleep Deprivationlcsh:Q3111 BiomedicineT-Box Domain ProteinsReceptors Transforming Growth Factor beta030217 neurology & neurosurgery
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Transcriptional profiling of rat white adipose tissue response to 2,3,7,8-tetrachlorodibenzo-ρ-dioxin

2015

Polychlorinated dibenzodioxins are environmental contaminants commonly produced as a by-product of industrial processes. The most potent of these, 2,3,7,8-tetrachlorodibenzo-rho-dioxin (TCDD), is highly lipophilic, leading to bioaccumulation. White adipose tissue (WAT) is a major site for energy storage, and is one of the organs in which TCDD accumulates. In laboratory animals, exposure to TCDD causes numerous metabolic abnormalities, including a wasting syndrome. We therefore investigated the molecular effects of TCDD exposure on WAT by profiling the transcriptomic response of WAT to 100 mu g/kg of TCDD at 1 or 4 days in TCDD-sensitive Long-Evans (Turku/AB; L-E) rats. A comparative analysi…

MaleTCDDPolychlorinated DibenzodioxinsTime FactorsTranscription GeneticPolychlorinated dibenzodioxinsAHRAH GENE BATTERYAdipose tissueWhite adipose tissueRESISTANT413 Veterinary scienceToxicologyfeed restrictionTranscriptomechemistry.chemical_compoundGene Regulatory Networksheterocyclic compoundsreproductive and urinary physiologyta317biology3. Good healthPROBE LEVELLUNG-CANCER CELLSToxicityEnvironmental PollutantsMESSENGER-RNAARYL-HYDROCARBON RECEPTORSTRAINmedicine.medical_specialtyAdipose Tissue WhiteWEIGHT-LOSSta3111Immune systemSpecies Specificitytranscriptomic profilingwhite adipose tissueInternal medicinemedicineAnimalsHumansRats Long-EvansRats WistarCaloric RestrictionPharmacologyGene Expression Profilingta1184Lipid metabolismAryl hydrocarbon receptorstomatognathic diseasesEndocrinologyGene Expression RegulationchemistryDIOXIN-TREATED RATSbiology.proteinToxicology and Applied Pharmacology
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Controlled intermittent shortening contractions of a muscle-tendon complex: muscle fibre damage and effects on force transmission from a single head …

2005

This study was performed to examine effects of prolonged (3 h) intermittent shortening (amplitude 2 mm) contractions (muscles were excited maximally) of head III of rat extensor digitorum longus muscle (EDL III) on indices of muscle damage and on force transmission within the intact anterior crural compartment. Three hours after the EDL III exercise, muscle fibre damage, as assessed by immunohistochemical staining of structural proteins (i.e. dystrophin, desmin, titin, laminin-2), was found in EDL, tibialis anterior (TA) and extensor hallucis longus (EHL) muscles. The damaged muscle fibres were not uniformly distributed throughout the muscle cross-sections, but were located predominantly ne…

MaleTime Factorsanimal structuresPhysiologyMolecular Sequence DataMuscle Fibers SkeletalVesicular Transport ProteinsMuscle ProteinsBiochemistryDesminExtensor digitorum longus muscleTendonsSDG 3 - Good Health and Well-beingmedicineAnimalsRNA MessengerMuscle fibreRats WistarMuscle SkeletalbiologyChemistryReverse Transcriptase Polymerase Chain ReactionMETIS-229512Compartment (ship)Gene Expression Profilingmusculoskeletal neural and ocular physiologyMembrane ProteinsCell BiologyAnatomyOrgan Sizemusculoskeletal systemTendonRatsSlack lengthmedicine.anatomical_structureIR-104003Models Animalbiology.protein/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingTitinDesminStress MechanicalDystrophintissuesMuscle Contraction
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USH3A transcripts encode clarin-1, a four-transmembrane-domain protein with a possible role in sensory synapses.

2002

Usher syndrome type 3 (USH3) is an autosomal recessive disorder characterised by the association of post-lingual progressive hearing loss, progressive visual loss due to retinitis pigmentosa and variable presence of vestibular dysfunction. Because the previously defined transcripts do not account for all USH3 cases, we performed further analysis and revealed the presence of additional exons embedded in longer human and mouse USH3A transcripts and three novel USH3A mutations. Expression of Ush3a transcripts was localised by whole mount in situ hybridisation to cochlear hair cells and spiral ganglion cells. The full length USH3A transcript encodes clarin-1, a four-transmembrane-domain protein…

MaleUsher syndromeMolecular Sequence DataBiologyPhotoreceptor cellSynapse03 medical and health sciencesExonMice0302 clinical medicineSequence Analysis ProteinRetinitis pigmentosaHair Cells Auditoryotorhinolaryngologic diseasesGeneticsmedicineAnimalsHumansAmino Acid SequenceGenetics (clinical)Spiral ganglionIn Situ HybridizationPhylogeny030304 developmental biology0303 health sciencesGene Expression ProfilingChromosome MappingMembrane ProteinsSequence Analysis DNAmedicine.diseaseCell biologyPedigreeTransmembrane domainmedicine.anatomical_structureMutationSynapsesFemalesense organsHair cellCalcium ChannelsSequence Alignment030217 neurology & neurosurgeryEuropean journal of human genetics : EJHG
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Farnesoid X receptor activation increases cholesteryl ester transfer protein expression in humans and transgenic mice

2013

International audience; Cholesteryl ester transfer protein (CETP) activity results in a proatherogenic lipoprotein profile. In cholestatic conditions, farnesoid X receptor (FXR) signaling by bile acids (BA) is activated and plasma HDL cholesterol (HDL-C) levels are low. This study tested the hypothesis that FXR-mediated induction of CETP contributes to this phenotype. Patients with cholestasis and high plasma BA had lower HDL-C levels and higher plasma CETP activity and mass compared with matched controls with low plasma BA (each P < 0.01). BA feeding in APOE3*Leiden transgenic mice expressing the human CETP transgene controlled by its endogenous promoter increased cholesterol within apoB-c…

Male[SDV]Life Sciences [q-bio]Receptors Cytoplasmic and Nuclear030204 cardiovascular system & hematologyInbred C57BLBiochemistryTransgenicchemistry.chemical_compoundMice0302 clinical medicineEndocrinologyHigh-density lipoproteinLifeReceptorsnuclear receptorResearch ArticlesCells Cultured0303 health sciencesCulturedbiologyMiddle AgedUp-RegulationCytoplasmic and Nuclear/agonistslipids (amino acids peptides and proteins)FemaleEELS - Earth Environmental and Life SciencesMHR - Metabolic Health ResearchHealthy Livingmedicine.medical_specialtyTransgeneCellsMice TransgenicQD415-436macrophageReceptors Cytoplasmic and Nuclear/agonists03 medical and health sciencesDownregulation and upregulationInternal medicineCholesterylester transfer proteinmedicinehepatocyteFood and NutritionAnimalsHumans[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular Biology030304 developmental biologyNutritionbile acidsCholesterolGene Expression ProfilingCell BiologyCholesterol Ester Transfer Proteinscarbohydrates (lipids)Mice Inbred C57BLlipoproteinsEndocrinologyNuclear receptorchemistrybiology.proteinFarnesoid X receptor[SDV.AEN]Life Sciences [q-bio]/Food and NutritionLipoproteinCholesterol Ester Transfer Proteins/genetics
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Expression of transketolase TKTL1 predicts colon and urothelial cancer patient survival: Warburg effect reinterpreted

2006

Abstract Tumours ferment glucose to lactate even in the presence of oxygen (aerobic glycolysis; Warburg effect). The pentose phosphate pathway (PPP) allows glucose conversion to ribose for nucleic acid synthesis and glucose degradation to lactate. The nonoxidative part of the PPP is controlled by transketolase enzyme reactions. We have detected upregulation of a mutated transketolase transcript (TKTL1) in human malignancies, whereas transketolase (TKT) and transketolase-like-2 (TKTL2) transcripts were not upregulated. Strong TKTL1 protein expression was correlated to invasive colon and urothelial tumours and to poor patients outcome. TKTL1 encodes a transketolase with unusual enzymatic prop…

Maleaerobic glycolysiCancer ResearchAdenocarcinomanPentose phosphate pathwayTransketolaseBiologyMetastasispentose phosphate pathway (PPP)Downregulation and upregulationPredictive Value of TestsmedicineHumansNeoplasm InvasivenessGlycolysisNeoplasm MetastasisMolecular Diagnosticsaerobic glycolysisAgedtransketolase-like-1 (TKTL1)transketolase (TKT)Gene Expression ProfilingCancerMiddle AgedPrognosismedicine.diseaseSurvival AnalysisWarburg effectUp-RegulationUrinary Bladder NeoplasmsOncologyBiochemistryAnaerobic glycolysispharmacodiagnostic markerColonic NeoplasmsCancer researchFemaleWarburg effectTransketolaseGlycolysisBritish Journal of Cancer
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Expert witness in paternity testing in Germany.

2003

In Germany, paternity testing can be ordered by a judge as well as by private persons. In the case of private counselling, the expert has the duty to inform the parties on all medical, legal and ethical aspects. Informed consent must be given by each individual included in an exploration of family relationships. Due to the rapid progress of DNA typing in even minute amounts after polymerase chain reaction amplification, genotypes of short tandem repeat systems can be elicited by extraction from single cells. Therefore, the number of unlawful investigations of paternal relationships is steadily increasing. Here the requirements for paternity testing laboratories and sanctions for unlawful ex…

Malebusiness.industrymedia_common.quotation_subjectPaternityDNA FingerprintingFederal lawPathology and Forensic MedicineIssues ethics and legal aspectsDNA profilingExpert witnessInformed consentLawGermanyForensic engineeringMedicineSanctionsMicrosatelliteHumansbusinessDutyExpert Testimonymedia_commonLegal medicine (Tokyo, Japan)
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Disruption of TCBA1 associated with a de novo t(1;6)(q32.2;q22.3) presenting in a child with developmental delay and recurrent infections

2005

A boy with developmental delay, particularly of speech, a distinct face, antineutrophil cytoplasmic antibodies, and recurrent infections was found to have an apparently balanced de novo t(1;6)(q32.3;q22.3) translocation. Fluorescent in situ hybridisation with BAC/PAC clones and long range polymerase chain reaction products assessed in the human genome sequence localised the chromosome 1 breakpoint to a 9.8 kb segment within a hypothetical gene, LOC388735, and the chromosome 6 breakpoint to a 12.8 kb segment in intron 4 of the T-cell lymphoma breakpoint-associated target 1 (TCBA1) gene. Disruption and/or formation of TCBA1 fusion genes in T cell lymphoma and leukaemia cell lines suggests a r…

Malemedicine.medical_specialtyDevelopmental DisabilitiesMolecular Sequence DataShort ReportBiologyInfectionsTranslocation GeneticFusion geneExonMiceMolecular geneticsGeneticsmedicineAnimalsHumansAmino Acid SequenceChildGeneGenetics (clinical)GeneticsGenome HumanGene Expression ProfilingBreakpointChromosome MappingMembrane ProteinsChromosome BreakageExonsNull alleleMolecular biologyGene expression profilingChromosomes Human Pair 1Child PreschoolCytogenetic AnalysisChromosomes Human Pair 6Chromosome breakage
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Lasting downregulation of the lipid peroxidation enzymes in the prefrontal cortex of mice susceptible to stress-induced anhedonia

2015

International audience; Antioxidant enzymes and lipid peroxidation in the brain are involved in neuropsychiatric pathologies, including depression. 14- or 28-day chronic stress model induced a depressive syndrome defined by lowered reward sensitivity in C57BL/6J mice and changed gene expression of peroxidation enzymes as shown in microarray assays. We studied how susceptibility or resilience to anhedonia is related to lipid peroxidation in the prefrontal cortex (PFC). With 14-day stress, a comparison of the activities of catalase (CAT), superoxide dismutase (SOD), glutathione peroxidase (GPX) and accumulation of malondialdehyde (MDA) revealed a decrease of the first two measures in suscepti…

Malemedicine.medical_specialtyImipramineAnhedoniaLipid peroxidationDown-RegulationMotor ActivityMicroarrayHippocampusImipraminePrefrontal cortexGene Expression Regulation EnzymologicSuperoxide dismutaseLipid peroxidationFood PreferencesMiceBehavioral Neurosciencechemistry.chemical_compoundNeurochemicalMalondialdehydeInternal medicinemedicineAnimalsChronic stresschemistry.chemical_classificationGlutathione PeroxidasebiologySuperoxide Dismutasebusiness.industryGene Expression ProfilingGlutathione peroxidaseAnhedoniaResilience PsychologicalCatalaseMalondialdehydeAggressionEndocrinologychemistrybiology.protein[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]medicine.symptombusinessChronic stress depression modelStress Psychologicalmedicine.drug
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