Search results for " signaling."

showing 10 items of 1032 documents

Regulation of T-Cell Immune Responses by Pro-Resolving Lipid Mediators

2021

Both the initiation and the resolution of inflammatory responses are governed by the sequential activation, migration, and control/suppression of immune cells at the site of injury. Bioactive lipids play a major role in the fine-tuning of this dynamic process in a timely manner. During inflammation and its resolution, polymorphonuclear cells (PMNs) and macrophages switch from producing pro-inflammatory prostaglandins and leukotrienes to specialized pro-resolving lipid mediators (SPMs), namely, lipoxins, resolvins, protectins, and maresins, which are operative at the local level to limit further inflammation and tissue injury and restore homeostasis. Accumulating evidences expand now the rol…

chronic inflammationT cellT-LymphocytesImmunologyInflammationReviewBiologymedicine.disease_causeT-Lymphocytes RegulatoryAutoimmunity03 medical and health sciencesImmunomodulating Agents0302 clinical medicineImmune systemmedicineHumansImmunology and Allergy030304 developmental biology0303 health sciencestherapyEffectorautoimmunityresolutionT cellLipid signalingadaptive immunityRC581-607Acquired immune systemCellular Reprogramming3. Good healthLipoxinsmedicine.anatomical_structureEicosapentaenoic AcidImmunologyspecialized pro-resolving lipid mediators (SPMs)Eicosanoidsmedicine.symptomImmunologic diseases. AllergyInflammation MediatorsHomeostasis030215 immunologyFrontiers in Immunology
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The hedgehog receptor patched is involved in cholesterol transport.

2011

International audience; BACKGROUND: Sonic hedgehog (Shh) signaling plays a crucial role in growth and patterning during embryonic development, and also in stem cell maintenance and tissue regeneration in adults. Aberrant Shh pathway activation is involved in the development of many tumors, and one of the most affected Shh signaling steps found in these tumors is the regulation of the signaling receptor Smoothened by the Shh receptor Patched. In the present work, we investigated Patched activity and the mechanism by which Patched inhibits Smoothened. METHODOLOGY/PRINCIPAL FINDINGS: Using the well-known Shh-responding cell line of mouse fibroblasts NIH 3T3, we first observed that enhancement …

ciliumlcsh:MedicineyeastBiochemistryReceptors G-Protein-CoupledTransmembrane Transport ProteinsMicechemistry.chemical_compound0302 clinical medicineMolecular Cell Biology[SDV.IDA]Life Sciences [q-bio]/Food engineeringMembrane Receptor SignalingBiomacromolecule-Ligand InteractionsSonic hedgehoglcsh:ScienceComputingMilieux_MISCELLANEOUS0303 health sciencesMultidisciplinaryMechanisms of Signal TransductionVeratrum Alkaloids[SDV.IDA] Life Sciences [q-bio]/Food engineeringdrosophilaSmoothened ReceptorLipidsHedgehog signaling pathwayCell biologySterolsSmoothened ReceptorAlimentation et Nutritionembryonic structurescilMembranes and Sorting[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Signal transductionvesicular traffickingSignal TransductionResearch Articleprimary ciliumPatched ReceptorsPatchedsignal-transductionanimal structuresCyclopamine[SPI.GPROC] Engineering Sciences [physics]/Chemical and Process EngineeringBiophysicsReceptors Cell Surfacepathway activationSaccharomyces cerevisiaetransduction du signalBiology03 medical and health sciencessonic hedgehoglipidAnimalsHumansFood and NutritionHedgehog Proteins[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process EngineeringBiology030304 developmental biologyPatched Receptorsprotein signalsCell Membranelcsh:RProteinscholesterolBiological TransportTransmembrane Proteinssterol-sensing domainchemistry[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]NIH 3T3 Cellscholesterol;lipid;cell trafficking; yeast;drosophila;cells ; pathway activation; vesicular trafficking; signal-transduction; sonic hedgehog;sterol-sensing domain; primary cilium;protein signalsbiology.proteincellslcsh:Qcell traffickingSmoothened030217 neurology & neurosurgery
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ANKRD26-RET - A novel gene fusion involving RET in papillary thyroid carcinoma

2018

Abstract Background Rearrangements of RET are drivers of oncogenesis, traceable in different cancer types as papillary thyroid carcinoma (PTC), non-small cell lung cancer, colorectal or breast cancer. Anchored multiplex PCR based next-generation sequencing (NGS) can detect RET rearrangements involving previously unknown partner genes. Methods A sample of PTC underwent NGS, following detection of RET rearrangement by fluorescence in situ hybridization. Expression analysis of ANKRD26 and RET was performed for the tumor harboring ANKRD26-RET, for corresponding normal thyroid tissue and PTC tumors with representative genetic alterations (BRAFV600E, CCDC6-RET), complemented by a comparative sear…

congenital hereditary and neonatal diseases and abnormalitiesendocrine systemCancer Researchendocrine system diseasesBiologymedicine.disease_causeMetastasisThyroid carcinoma03 medical and health sciences0302 clinical medicineGeneticsmedicineHumansThyroid NeoplasmsneoplasmsMolecular BiologyGenemedicine.diagnostic_testProto-Oncogene Proteins c-retThyroidHigh-Throughput Nucleotide SequencingCancermedicine.diseaseSurvival Analysismedicine.anatomical_structureThyroid Cancer Papillary030220 oncology & carcinogenesisCancer researchIntercellular Signaling Peptides and ProteinsGene FusionCarcinogenesisTyrosine kinaseFluorescence in situ hybridizationCancer Genetics
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DIVIDEND CONTROVERSY: A THEORETICAL APPROACH

2011

One of the major financial decisions for a public company is the dividend policy - the proportion in which the company decides to distribute profits to shareholders. The difficulty of the decision comes from the implications on firm value. There are conflicting points of view on dividend policy. Even if under ideal conditions (perfect markets) dividend policy is irrelevant (the theory of Modigliani and Miller), still the way in which companies behave shows that the dividend policy is relevant in practice. Market imperfections (taxes, information asymmetry, transaction costs) influence dividend policy. The aim of this paper is to present the major theories and findings related to dividend po…

dividend policy value of the firm cost of equity clientele effect signaling effectStudies in Business and Economics
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Nuclear calcium signaling by inositol trisphosphate in GH3 pituitary cells

2008

It has been proposed that nuclear and cytosolic Ca2+ ([Ca2+]N and [Ca2+]C) may be regulated independently. We address here the issue of whether inositol trisphosphate (IP3) can, bypassing changes of [Ca2+]C, produce direct release of Ca2+ into the nucleoplasm. We have used targeted aequorins to selectively measure and compare the changes in [Ca2+]C and [Ca2+]N induced by IP3 in GH3 pituitary cells. Heparin, an IP3 inhibitor that does not permeate the nuclear pores, abolished the [Ca2+]C peaks but inhibited only partly the [Ca2+]N peaks. The permeant inhibitor 2-aminoethoxy-diphenyl-borate (2-APB) blocked both responses. Removal of ATP also inhibited more strongly the [Ca2+]C than [Ca2+]N pe…

endocrine systemCytoplasm[SDV.OT]Life Sciences [q-bio]/Other [q-bio.OT]PhysiologyAequorinNucleoplasmic reticulumaequorinInositol 145-TrisphosphateCell Line03 medical and health scienceschemistry.chemical_compound0302 clinical medicinenuclear signal transductionmedicineAnimalsInositol 145-Trisphosphate Receptorsinositol trisphosphate receptorsCalcium SignalingReceptorMolecular Biology030304 developmental biologyCell Nucleus0303 health sciencesNucleoplasmbiologypituitary cellsInositol trisphosphateCell Biologyherpes simplex virusMolecular biologyRatsCytosolmedicine.anatomical_structurechemistryCytoplasmPituitary Glandbiology.proteinnucleoplasmic reticulumNucleus030217 neurology & neurosurgery
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MiR-33a Controls hMSCS Osteoblast Commitment Modulating the Yap/Taz Expression Through EGFR Signaling Regulation

2019

Mesenchymal stromal cells (hMSCs) display a pleiotropic function in bone regeneration. The signaling involved in osteoblast commitment is still not completely understood, and that determines the failure of current therapies being used. In our recent studies, we identified two miRNAs as regulators of hMSCs osteoblast differentiation driving hypoxia signaling and cytoskeletal reorganization. Other signalings involved in this process are epithelial to mesenchymal transition (EMT) and epidermal growth factor receptor (EGFR) signalings through the regulation of Yes-associated protein (YAP)/PDZ-binding motif (TAZ) expression. In the current study, we investigated the role of miR-33a family as a (…

epithelial mesenchymal transitionregenerative medicinePDZ DomainsCell CommunicationArticlemicroRNAmedicineHumansEpidermal growth factor receptorEpithelial–mesenchymal transitionBone regenerationCells CulturedEGFR inhibitorsAdaptor Proteins Signal TransducingOsteoblastsmicroRNAbiologyMesenchymal stem cellComputational BiologyOsteoblastMesenchymal Stem CellsYAP-Signaling ProteinsGeneral MedicinePhenotypeCell biologymicroRNAsErbB Receptorsmedicine.anatomical_structureTranscriptional Coactivator with PDZ-Binding Motif Proteinsmesenchymal stromal cellbiology.proteinTrans-Activatorsmesenchymal stromal cellsEGFR signalingSignal TransductionTranscription FactorsCells
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Effects of combined hormone replacement therapy or its effective agents on the IGF-1 pathway in skeletal muscle.

2010

Objectives To investigate the effects of combined hormone replacement therapy (HRT) and its effective agents on the IGF-1 signaling pathway. Design and methods To examine the effects of HRT on skeletal muscle in vivo, we utilized pre- and post-intervention samples from a randomized double blinded trial with 50–57-year-old women. The intervention included the year-long use of either HRT preparation (2 mg 17β-estradiol, E2; 1 mg norethisterone acetate, NETA, n = 10) or placebo (CO, n = 9). Microarray technology and quantitative PCR (qPCR) were used to study the expression of insulin-like growth factor I (IGF-1) and its splice variants as well as IGF-1 receptor, Akt1, mTOR, FOXO1, FOXO3, atrog…

estradioliTranscription GeneticEndocrinology Diabetes and MetabolismMuscle Fibers SkeletalEstrogen receptorpostmenopausal womenMuscle ProteinsFOXO1Receptor IGF Type 10302 clinical medicineEndocrinologyProtein IsoformsTestosteroneInsulin-Like Growth Factor IReceptorRandomized Controlled Trials as Topic0303 health sciencesEstradiolMyogenesisForkhead Box Protein O1TOR Serine-Threonine KinasesEstrogen Replacement TherapyForkhead Box Protein O3Forkhead Transcription FactorsMiddle Agedmedicine.anatomical_structureReceptors EstrogenReceptors AndrogenFemalemedicine.medical_specialtynorethisterone acetate030209 endocrinology & metabolismBiologypostmenopausaalinen nainen03 medical and health sciencesInternal medicinemedicineHumansnoretisteroniasetaattiluurankolihasskeletal muscleMuscle SkeletalProtein kinase BPI3K/AKT/mTOR pathway030304 developmental biologyhormonikorvaushoitoSKP Cullin F-Box Protein LigasesSkeletal muscleAndrogen receptorNorethindrone AcetateEndocrinologyHormone replacement therapyIGF-1 signalointiNorethindroneIGF-1 signalingProto-Oncogene Proteins c-aktGrowth hormoneIGF research : official journal of the Growth Hormone Research Society and the International IGF Research Society
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Hormone replacement therapy enhances IGF-1 signaling in skeletal muscle by diminishing miR-182 and miR-233 expressions: A study on postmenopausal mon…

2014

MiRNAs are fine-tuning modifiers of skeletal muscle regulation, but knowledge of their hormonal control is lacking. We used a co-twin case–control study design, that is, monozygotic postmenopausal twin pairs discordant for estrogen-based hormone replacement therapy (HRT) to explore estrogen-dependent skeletal muscle regulation via miRNAs. MiRNA profiles were determined from vastus lateralis muscle of nine healthy 54–62- years-old monozygotic female twin pairs discordant for HRT (median 7 years). MCF-7 cells, human myoblast cultures and mouse muscle experiments were used to confirm estrogen’s causal role on the expression of specific miRNAs, their target mRNAs and proteins and finally the ac…

fosforylaatiovaihdevuodetAKTmiR-182agingmiR-142-3pmTORFOXO3AmiR-233IGF-1 signalingIGF-1R
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Brain glucose sensing and energy balance: role of mitochondrial reactive oxygen species

2012

Brain glucose sensing and energy balance: role of mitochondrial reactive oxygen species . 7. congrès de physiologie, de pharmacologie et de thérapeutique (P2T)

glucose sensing[ SDV.AEN ] Life Sciences [q-bio]/Food and Nutritionphysiologie du système nerveuxchaîne respiratoire mitochondrialeROS signaling[SDV.AEN] Life Sciences [q-bio]/Food and Nutritionmitochondrie[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]fissioncerveau[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC][SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]hypothalamusdynamique mitochondrialeglucose[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition
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'Good to Repeat': Making Random Access Near-Optimal with Repeated Contentions

2020

Recent advances on WLAN technology have been focused mostly on boosting network capacity by means of a more efficient and flexible physical layer. A new concept is required at MAC level to exploit fully the new capabilities of the PHY layer. In this article, we propose a contention mechanism based on Repeated Contentions (ReCo) in frequency domain. It provides a simple-to-configure, robust and short-term fair algorithm for the random contention component of the MAC protocol. The throughput efficiency of ReCo is not sensitive to the number of contending stations, so that ReCo does not require adaptive tuning of the access parameters for performance optimization. Efficiency and robustness is …

imperfect sensingbusiness.industryOrthogonal frequency-division multiplexingComputer scienceSettore ING-INF/03 - TelecomunicazioniApplied MathematicsComputerSystemsOrganization_COMPUTER-COMMUNICATIONNETWORKSPhysical layer020206 networking & telecommunicationsThroughput02 engineering and technologyIEEE 802.11ax; random access contention; frequency-domain signalling; imperfect sensingComputer Science ApplicationsPHYfrequency-domain signalling0202 electrical engineering electronic engineering information engineeringfrequency-domain signalingWirelessrandom access contentionSettore ICAR/19 - RestauroElectrical and Electronic EngineeringbusinessRandom accessIEEE 802.11axComputer network
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