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Characterization of non-expressed C4 genes in a case of complete C4 deficiency: identification of a novel point mutation leading to a premature stop …

1998

The genetic basis of complete C4 deficiency in a patient with SLE was investigated. Previous studies have demonstrated that this patient has two different major histocompatibility complex (MHC) haplotypes that each contain a major deletion and a non-expressed C4 gene. In the present study, non-expression of the C4 genes was explained by the finding of two distinct C4 gene mutations. A previously described two base pair insertion in exon 29 of the C4 gene was detected in the paternal MHC haplotype [HLA-A2, B40, SC00, DR6]. The maternal haplotype [HLA-A30, B18, F1C00, DR3] carried a C4 gene with a one base pair deletion in exon 20 generating a premature stop codon. This mutation was neither f…

AdultMaleHeterozygoteImmunologyGene mutationBiologymedicine.disease_causePolymerase Chain ReactionCell LineMajor Histocompatibility ComplexExonmedicineImmunology and AllergyHumansLupus Erythematosus SystemicPoint MutationGenePolymorphism Single-Stranded ConformationalGeneticsMutationPoint mutationHaplotypeC4AComplement C4General MedicineExonsSequence Analysis DNAMolecular biologyIsotypePedigreeHaplotypesCodon TerminatorFemalePolymorphism Restriction Fragment LengthHuman immunology
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Perilipin polymorphism interacts with saturated fat and carbohydrates to modulate insulin resistance

2010

Macronutrient intakes and genetic variants have been shown to interact to alter the risk of insulin resistance, but replication of gene-nutrient interactions across independent populations may be difficult to achieve. Saturated fat and carbohydrate were previously shown to interact with genotype for insulin resistance for a variant of perilipin (PLIN), the major adipocyte-associated protein and a regulator of adipocyte metabolism. We investigated the same interaction for PLIN 11482G>A (rs894160) on insulin resistance in US men(n=462) and women(n=508) (mean ±SD, 49±16 years). In multivariate linear regression models, we found a significant (PA for HOMA-IR (homeostasis model assessment of ins…

AdultMaleHeterozygotePerilipin-1medicine.medical_specialtyAdolescentMinnesotaEndocrinology Diabetes and MetabolismSaturated fatmedicine.medical_treatmentMedicine (miscellaneous)BiologyPolymorphism Single NucleotideWhite PeopleArticleYoung Adultchemistry.chemical_compoundNutrigenomicsInsulin resistanceUtahAdipocyteInternal medicineDietary CarbohydratesmedicineHumansInsulinGenetic Association StudiesAgedAged 80 and overSex CharacteristicsNutrition and DieteticsInsulinMetabolismMiddle AgedCarbohydratePhosphoproteinsmedicine.diseaseDietary FatsEndocrinologychemistryPerilipinFemaleInsulin ResistanceCarrier ProteinsCardiology and Cardiovascular MedicineHomeostasisNutrition, Metabolism and Cardiovascular Diseases
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Evaluation of clinical diagnosis criteria of familial ligand defective apoB 100 and lipoprotein phenotype comparison between LDL receptor gene mutati…

2007

Familial hypercholesterolemia (FH) and familial defective apoB 100 (FDB) are characterized by increased plasma low-density lipoprotein cholesterol (LDLc) levels and risk of coronary heart disease (CHD). FDB is clinically indistinguishable from FH. The aims of this study were to evaluate clinical diagnosis criteria for FDB and to compare the lipoprotein phenotype between carriers of LDL receptor (LDLR) gene mutations that affect the ligand-binding domain and subjects with the R3500Q mutation in apoB gene. We studied 213 subjects (113 probands) with FH and 19 heterozygous FDB subjects. Genetic diagnosis was determined by following a protocol based on Southern blot and polymerase chain reactio…

AdultMaleHeterozygotemedicine.medical_specialtyGenotypeApolipoprotein BPopulationMutation MissenseCoronary DiseaseFamilial hypercholesterolemiaGene mutationBiologyWhite PeopleHyperlipoproteinemia Type IIchemistry.chemical_compoundPhysiology (medical)Internal medicinemedicineHumansMissense mutationeducationPolymorphism Single-Stranded Conformationaleducation.field_of_studyBinding SitesCholesterolGenetic Carrier ScreeningBiochemistry (medical)Public Health Environmental and Occupational HealthCholesterol LDLGeneral MedicineMiddle Agedmedicine.diseaseFounder EffectProtein Structure TertiaryEuropePhenotypeEndocrinologyReceptors LDLchemistryApolipoprotein B-100LDL receptorbiology.proteinFemalelipids (amino acids peptides and proteins)LipoproteinTranslational Research
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Dietary polyunsaturated fatty acids may increase plasma LDL-cholesterol and plasma cholesterol concentrations in carriers of an ABCG1 gene single nuc…

2011

Abstract Background ABCG1 mediates cellular cholesterol transport, but there is very little known about the influence of ABCG1 polymorphisms on human plasma lipoprotein cholesterol concentrations or on the interactions of these polymorphisms with diet. Objective Our objective was to investigate whether interactions between PUFA intake and ABCG1 polymorphisms modulate associations with plasma total cholesterol (TC), LDL- and HDL-cholesterol in two Spanish populations. Methods We grounded our investigation on two general population-based studies: the Hortega study (population A) and the Pizarra study (population B). Participants included 1178 individuals (50.0% women, age range 21–85 years) a…

AdultMaleHeterozygotemedicine.medical_specialtyPopulationSingle-nucleotide polymorphismBiologyPolymorphism Single NucleotideYoung Adultchemistry.chemical_compoundGene FrequencySurveys and QuestionnairesInternal medicineGenotypemedicineHumansGene–environment interactionAlleleeducationAllele frequencyATP Binding Cassette Transporter Subfamily G Member 1AgedAged 80 and overchemistry.chemical_classificationGeneticseducation.field_of_studyChi-Square DistributionCholesterolHomozygoteCholesterol LDLMiddle AgedDietUp-RegulationCholesterolPhenotypeEndocrinologychemistrySpainFatty Acids UnsaturatedATP-Binding Cassette TransportersFemaleGene-Environment Interactionlipids (amino acids peptides and proteins)Cardiology and Cardiovascular MedicinePolyunsaturated fatty acidAtherosclerosis
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Maintenance of marginal bone support and soft tissue esthetics at immediately provisionalized OsseoSpeed implants placed into extraction sites: 2-yea…

2012

Background Placement of implants into extraction sockets targets the maintenance of peri-implant hard and soft tissue structures and the support of a natural and esthetic contour. The main advantages of immediate implant insertion in comparison with delayed implant placement protocols are as follows: a reduced treatment time, less number of sessions, and, thus, the less invasive procedure. This study examines the clinical performance (survival rate, marginal bone levels and Pink Esthetic Score [PES]) of OsseoSpeed™ implants placed into extraction sockets with immediate provisionalization in the anterior maxilla after a follow-up of at least 12 months. Methods Twenty patients received a tota…

AdultMaleImmediate Dental Implant LoadingFacial boneEstheticsmedicine.medical_treatmentAlveolar Bone LossDentistryProsthesisDental Implants Single-ToothAlveolar ProcessMedicineHumansProspective StudiesTooth SocketSurvival rateAgedbusiness.industryAlveolar processClinical performanceSoft tissueMiddle AgedImmediate provisionalizationmedicine.anatomical_structureTreatment OutcomeDental Prosthesis DesignFemaleImplantOral SurgerybusinessClinical oral implants research
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Clinical and esthetic outcome with immediate insertion and provisionalization with or without connective tissue grafting in presence of mucogingival …

2017

Background Recessions following immediate implant insertion are frequently reported in the literature. Data regarding implant installation in presence of mucogingival recessions are rare. Purpose This study observes soft tissue level changes following immediate implant insertion and provisionalization of implants with or without connective tissue grafts in the anterior maxilla in patients with initial mucogingival recession within a follow-up period between one and eight years. Materials and methods Twenty-six patients with marginal gingival recessions, which were designated for extraction and immediate implant insertion in the anterior zone of the maxilla (13-23), were included. Out of a l…

AdultMaleImmediate Dental Implant LoadingFacial boneGingivaDentistryConnective tissueEsthetics DentalTransplantation AutologousYoung Adult03 medical and health sciencesDental Implants Single-Tooth0302 clinical medicineGermanyMaxillaRetrospective analysisHumansPeriodontal PocketMedicineGingival RecessionGeneral DentistryAgedRetrospective StudiesDental ImplantsBone Transplantationbusiness.industrySoft tissue030206 dentistrySingle toothCone-Beam Computed TomographyMiddle AgedAutologous boneIncisorTreatment Outcomemedicine.anatomical_structureDental Prosthesis DesignConnective Tissue030220 oncology & carcinogenesisMaxillaTooth ExtractionFemaleDental Prosthesis Implant-SupportedImplantOral SurgerybusinessFollow-Up StudiesClinical Implant Dentistry and Related Research
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Survival rates and bone loss after immediate loading of implants in fresh extraction sockets (single gaps). A clinical prospective study with 4 year …

2016

Background The aim of this prospective study was to report the outcome of treatment with implants inserted after tooth extraction and immediately loaded. Material and Methods Fifty-six patients with single tooth loss were treated with 116 IPX Galimplant® implants with internal connections and a sandblasted, acid-etched surface. All implants were placed after tooth extraction using a flapless approach without bone regeneration, and they were then immediately loaded with cemented acrylic prostheses. After a period of three months, definitive cemented ceramic prostheses were placed. Patients were examined throughout a total of 4 years of follow-up. Marginal bone loss and survival rates were ev…

AdultMaleImmediate Dental Implant LoadingTime FactorsAlveolar Bone LossDentistry03 medical and health sciencesDental Implants Single-Tooth0302 clinical medicinemedicineHumansDental Restoration FailureProspective Studies030212 general & internal medicineTooth SocketProspective cohort studyBone regenerationGeneral DentistryDental alveolusPeriodontitisbusiness.industryResearchMandible030206 dentistrySingle toothMiddle Aged:CIENCIAS MÉDICAS [UNESCO]medicine.diseaseTreatment OutcomeOtorhinolaryngologyMaxillaUNESCO::CIENCIAS MÉDICASTooth ExtractionFemaleSurgeryImplantOral SurgerybusinessFollow-Up StudiesMedicina Oral Patología Oral y Cirugia Bucal
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Replication of interleukin 23 receptor and autophagy-related 16-like 1 association in adult- and pediatric-onset inflammatory bowel disease in Italy.

2008

AIM: To investigate gene variants in a large Italian inflammatory bowel disease (IBD) cohort, and to analyze the correlation of sub-phenotypes (including age at diagnosis) and epistatic interaction with other IBD genes. METHODS: Total of 763 patients with Crohn's disease (CD, 189 diagnosed at age < 19 years), 843 with ulcerative colitis (UC, 179 diagnosed < 19 years), 749 healthy controls, and 546 healthy parents (273 trios) were included in the study. The rs2241880 [autophagy-related 16-like 1 (ATG16L1)], rs11209026 and rs7517847 [interleukin 23 receptor (IL23R)], rs2066844, rs2066845, rs2066847 (CARD15), rs1050152 (OCTN1), and rs2631367 (OCTN2) gene variants were genotyped. RESULTS: The f…

AdultMaleInterleukin-23 receptorAdolescentGenotypeOrganic Cation Transport ProteinsIBDNod2 Signaling Adaptor ProteinAutophagy-Related Proteinsdigestive systemPolymorphism Single NucleotideInflammatory bowel diseaseYoung AdultSettore MED/38 - Pediatria Generale E SpecialisticaCrohn DiseaseIL23RClinical ResearchmedicineGenetic predispositionHumansGenetic Predisposition to DiseaseAge of OnsetYoung adultChildSolute Carrier Family 22 Member 5ReceptorAgedCrohn's diseaseSymportersbusiness.industryGastroenterologyInfantReceptors InterleukinGeneral MedicineMiddle AgedInflammatory Bowel Diseasesmedicine.diseaseUlcerative colitisdigestive system diseasesLogistic ModelsItalyCase-Control StudiesChild PreschoolImmunologyFemaleAge of onsetCarrier Proteinsbusiness
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Functional polymorphism in the neuropeptide Y gene promoter (rs16147) is associated with serum leptin levels and waist-hip ratio in women

2013

&lt;b&gt;&lt;i&gt;Objective:&lt;/i&gt;&lt;/b&gt; The neuropeptide-Y (NP-Y) gene is a strong candidate gene in the pathophysiology of obesity-linked behavior, and several single-nucleotide polymorphisms of NP-Y have already been linked to body weight and appetite. However, the results from current studies remain inconclusive. The aim of the present study was to test whether a certain functional genetic variant (SNP rs16147) in the NP-Y promoter gene is associated with serum leptin levels and body fat distribution. &lt;b&gt;&lt;i&gt;Method:&lt;/i&gt;&lt;/b&gt; We genotyped and measured the serum leptin levels of the NP-Y rs16147 polymorphism in 1,097 Caucasian subjects in the context of a pop…

AdultMaleLeptinCandidate genemedicine.medical_specialtyGenotypeblood [Leptin]Medicine (miscellaneous)610 Medicine & healthmacromolecular substancesPolymorphism Single NucleotideWhite PeopleStatistics NonparametricWaist–hip ratiogenetics [Obesity]Sex FactorsMedizinische FakultätInternal medicineGenotypemedicineHumansNeuropeptide YObesityddc:610Genephysiology [Neuropeptide Y]Nutrition and Dieteticsbusiness.industryWaist-Hip RatioLeptinCase-control study2701 Medicine (miscellaneous)Middle AgedNeuropeptide Y receptormedicine.diseaseObesitygenetics [European Continental Ancestry Group]EndocrinologyCase-Control Studies10054 Clinic for Psychiatry Psychotherapy and Psychosomatics2916 Nutrition and DieteticsFemalegenetics [Neuropeptide Y]businessphysiology [Polymorphism Single Nucleotide]
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Statistical colocalization of monocyte gene expression and genetic risk variants for type 1 diabetes

2012

One mechanism by which disease-associated DNA variation can alter disease risk is altering gene expression. However, linkage disequilibrium (LD) between variants, mostly single-nucleotide polymorphisms (SNPs), means it is not sufficient to show that a particular variant associates with both disease and expression, as there could be two distinct causal variants in LD. Here, we describe a formal statistical test of colocalization and apply it to type 1 diabetes (T1D)-associated regions identified mostly through genome-wide association studies and expression quantitative trait loci (eQTLs) discovered in a recently determined large monocyte expression data set from the Gutenberg Health Study (1…

AdultMaleLinkage disequilibriumGenotypeQuantitative Trait LociSingle-nucleotide polymorphismGenome-wide association studyQuantitative trait locusBiologyPolymorphism Single NucleotideLinkage DisequilibriumMonocytes03 medical and health sciences0302 clinical medicineRisk FactorsGeneticsHumansGenetic Predisposition to DiseaseMolecular BiologyGeneGenetics (clinical)Aged030304 developmental biologyGenetic associationGenetics0303 health sciencesModels GeneticAssociation Studies ArticlesColocalizationGeneral MedicineMiddle AgedDiabetes Mellitus Type 1Expression quantitative trait lociFemaleTranscriptomeAlgorithms030217 neurology & neurosurgeryGenome-Wide Association StudyHuman Molecular Genetics
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