Search results for " thoracic"

showing 10 items of 217 documents

Involvement of K+ channels in the relaxant effects of YC-1 in vascular smooth muscle

1999

This study addresses the question whether K(+) channels are involved in the vasorelaxant effects of 3-(5'-hydroxymethyl-2'-furyl)-1-benzyl-indazole (YC-1 ). In rat aorta, guinea pig aorta, and guinea pig a. carotis, YC-1 inhibited contractions induced by phenylephrine (3 microM) more potently than those induced by K(+)(48 mM). In rat aorta, tetraethylammonium (10 mM), charybdotoxin (0.2 microM), and iberiotoxin (0.1 microM), but not glibenclamide (10 microM), attenuated the relaxant effects of YC-1. In guinea pig a. carotis, YC-1 (30 microM) induced a hyperpolarisation which was antagonised by 1H-[1,2,4]oxadiazolo[4, 3-a]quinoxalin-1-one (ODQ; 50 microM). In rat aorta, YC-1 (30 microM) incr…

MaleIndazolesPotassium ChannelsTime FactorsVascular smooth muscleCharybdotoxinMuscle RelaxationGuinea PigsAorta ThoracicIn Vitro TechniquesPharmacologyMuscle Smooth VascularMembrane PotentialsRats Sprague-DawleyGlibenclamidePhenylephrinechemistry.chemical_compoundmedicine.arterymedicineAnimalsDrug InteractionsPhenylephrinePharmacologyAortaTetraethylammoniumDose-Response Relationship DrugChemistryAnatomyIberiotoxinRatsVasodilationCarotid ArteriesPotassiumFemaleZaprinastmedicine.drugEuropean Journal of Pharmacology
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Inflammation and Aortic Pulse Wave Velocity: A Multicenter Longitudinal Study in Patients With Inflammatory Bowel Disease

2019

Background Inflammatory bowel disease ( IBD ) is characterized by a low prevalence of traditional risk factors, an increased aortic pulse‐wave velocity ( aPWV ), and an excess of cardiovascular events. We have previously hypothesized that the cardiovascular risk excess reported in these patients could be explained by chronic inflammation. Here, we tested the hypothesis that chronic inflammation is responsible for the increased aPWV previously reported in IBD patients and that anti‐TNFa (anti‐tumor necrosis factor‐alpha) therapy reduce aPWV in these patients. Methods and Results This was a multicenter longitudinal study. We enrolled 334 patients: 82 patients with ulcerative colitis, 85 pati…

MaleLongitudinal studyTime FactorsSettore MED/09 - Medicina InternaAorta ThoracicBlood Pressure030204 cardiovascular system & hematologyGastroenterologyInflammatory bowel diseaseulcerative coliti0302 clinical medicineRisk Factorsarterial stiffneVascular DiseaseTumor necrosis factor-alphaEndoscopy Digestive SystemProspective Studies030212 general & internal medicinePulse wave velocityOriginal ResearchCrohn diseasetumor necrosis factor‐alphaUlcerative colitisC-Reactive Proteinarterial stiffnessCardiovascular DiseasesHypertensionFemaleTumor necrosis factor alphamedicine.symptomCardiology and Cardiovascular MedicineAdultmedicine.medical_specialtypulse wave velocityInflammationPulse Wave Analysis03 medical and health sciencesVascular StiffnessInternal medicinemedicineHumansIn patientulcerative colitisbusiness.industryInflammatory Bowel Diseasesmedicine.diseasearterial stiffness; Crohn disease; inflammation; pulse wave velocity; tumor necrosis factor‐alpha; ulcerative colitis; Cardiology and Cardiovascular MedicineinflammationArterial stiffnessbusinessBiomarkersFollow-Up Studies
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Long-Term Results After Repair of Type A Acute Aortic Dissection According to False Lumen Patency

2009

Background Late survival and freedom from retreatment on the descending aorta was evaluated after ascending aortic repair for type A acute aortic dissection (TAAAD). Methods Between March 1992 and January 2006, 189 TAAAD patients (mean age, 52 ± 11; range, 17 to 83 years) were included; of these, 58 had a patent false lumen, and 49 had Marfan syndrome. The descending aorta was evaluated postoperatively with computed tomography (CT). Late outcomes were assessed by Cox regression analysis and actuarial survival and freedom from retreatment by the Kaplan-Meier method. Mean follow-up was 88 ± 44 months. Results There were 38 (20%) late deaths. At 10 years, survival was 89.8% ± 2.1% for patients…

MaleMarfan syndromeTime FactorsThoracicAortic aneurysm80 and overHospital MortalityTomographyAged 80 and overAortic dissectionMiddle AgedhumanitiesAcute Disease; Adolescent; Aged; Aged 80 and over; Aneurysm Dissecting; Aortic Aneurysm Thoracic; Blood Vessel Prosthesis Implantation; Female; Follow-Up Studies; Hospital Mortality; Humans; Italy; Male; Middle Aged; Retrospective Studies; Survival Rate; Time Factors; Tomography X-Ray Computed; Treatment Outcome; Young AdultAortic AneurysmX-Ray ComputedSurvival RateTreatment OutcomeItalyCardiothoracic surgeryDescending aortaAcute DiseaseCirculatory systemCardiologyFemaleCardiology and Cardiovascular MedicinePulmonary and Respiratory Medicinemedicine.medical_specialtyAdolescentBlood Vessel Prosthesis ImplantationYoung AdultAneurysmmedicine.arteryInternal medicinemedicineHumansAgedRetrospective StudiesAortaAortic Aneurysm Thoracicbusiness.industrynutritional and metabolic diseasesSettore MED/23 - Chirurgia Cardiacamedicine.diseaseAneurysmSurgeryAortic DissectionSurgeryTomography X-Ray ComputedbusinessDissectingFollow-Up StudiesThe Annals of Thoracic Surgery
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Acute onset of esophageal duplication cyst in adult. Case report.

2009

Esophageal duplication (ED) cyst is unusual congenital disorder of the foregut, accounting for 10% to 15% of duplications of all foregut cysts. We report a case of esophageal duplication with acute clinical presentation, treated successfully with surgical resection.46-year-old man with acute dysphagia and gastroesophageal reflux of 2 weeks duration, showed submucosal bulging mass in the posterior wall of the middle third of oesophagus, fluid-filled cystic structure, dyshomogeneous, low-attenuation mass with smooth borders compatible with an hemorrhagic esophageal duplication cyst. With the suspect of acute presentation of a complicated esophageal cyst, the patient underwent surgery by right…

MaleMiddle AgedEsophageal DiseasesRadiographyEsophageal duplication cyst thoracic surgeryEsophagusThoracotomyEsophagoplastyAcute DiseaseGastroesophageal RefluxHumansEsophageal CystAge of OnsetDeglutition DisordersGastrointestinal Hemorrhage
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Redox Regulation of Dihydrofolate Reductase: Friend or Troublemaker?

2015

Oxidative stress is a hallmark of cardiovascular diseases1 and a major contributor to vascular dysfunction.2 On the basis on recent concepts, vascular oxidative stress is caused mainly by infiltrating inflammatory cells such as monocytes/macrophages or leucocytes,3,4 producing so-called kindling radicals that lead to the activation of secondary, vascular enzymatic sources of reactive oxygen species (mainly superoxide).2,5 A prominent example is the uncoupled nitric oxide (NO) synthase, which means that an NO-producing antiatherosclerotic enzyme is getting switched to a superoxide-producing proatherosclerotic enzyme.2 Molecular mechanisms causing endothelial NO synthase (eNOS) uncoupling or …

MaleNitric Oxide Synthase Type IIIAorta ThoracicOxidative phosphorylationBiologymedicine.disease_causeNitric OxideArticleNitric oxidechemistry.chemical_compoundEnosmedicineAnimalschemistry.chemical_classificationReactive oxygen speciesSuperoxideNitric Oxide Synthase Type IIIEndothelial CellsTetrahydrobiopterinbiology.organism_classificationTetrahydrofolate DehydrogenasechemistryBiochemistryCardiology and Cardiovascular MedicineOxidative stressmedicine.drugArteriosclerosis, thrombosis, and vascular biology
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Mediastinal syndrome from plasmablastic lymphoma in human immunodeficiency virus and human herpes virus 8 negative patient with polycythemia vera: a …

2017

Background: Plasmoblastic lymphoma is a rare and aggressive subtype of diffuse large B cell lymphoma, which occurs usually in the jaw of immunocompromised subjects. Case presentation: We describe the occurrence of plasmoblastic lymphoma in the mediastinum and chest wall skin of an human immunodeficiency virus-negative 63-year-old Caucasian man who had had polycytemia vera 7 years before. At admission, the patient showed a superior vena cava syndrome, with persistent dyspnoea, cough, and distension of the jugular veins. Imaging findings showed a 9.7 × 8 × 5.7 cm mediastinal mass. A chest wall neoformation biopsy and ultrasound-guided fine-needle aspiration biopsy of the mediastinal mass allo…

MalePathologySettore MED/21 - Chirurgia ToracicaCase ReportSettore MED/15 - Malattie Del Sangue0302 clinical medicinePolycythemia veraPolycythemia VeraCase report; Fine-needle aspiration biopsy; Hematology; Rare clinical case; Thoracic surgery; Medicine (all)UltrasonographyMedicine(all)Rare clinical caseSuperior vena cava syndromeHematologymedicine.diagnostic_testMedicine (all)MediastinumMediastinumGeneral MedicineHematologyHerpesviridae InfectionsSyndromeMiddle AgedThoracic surgerymedicine.anatomical_structureTreatment Outcome030220 oncology & carcinogenesisHerpesvirus 8 HumanPlasmablastic Lymphomamedicine.symptommedicine.medical_specialtyBiopsy Fine-NeedleMediastinal Neoplasms03 medical and health sciencesInternal medicineHIV SeronegativityBiopsymedicineHumansbusiness.industryThrombosisFine-needle aspiration biopsymedicine.diseaseLymphomaSettore MED/18 - Chirurgia GeneraleDyspneaCoughJugular VeinsbusinessDiffuse large B-cell lymphomaPlasmablastic lymphoma030215 immunology
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Role of TGF-β Pathway Polymorphisms in Sporadic Thoracic Aortic Aneurysm: rs900 TGF-β2 Is a Marker of Differential Gender Susceptibility

2013

Thoracic aortic aneurysm (TAA) is a progressive disorder involving gradual dilation of ascending and/or descending thoracic aorta with dissection or rupture as complications. It occurs as sporadic or defined syndromes/familial forms.Genetic, molecular and cellular mechanims of sporadic TAA forms are poorly characterized and known. Thus, our interest has been focused on investigating the role of genetic variants of transforming growth factor-β(TGF-β) pathways in TAA risk. On the other hand, no data on the role of genetic variants of TGF-βpathway in sporadic TAA exist until now. In addition, other cytokines, including IL-10, orchestrate TAA pathophysiology. Their balance determines the ultima…

MalePathologyThoracicGene FrequencyProtein IsoformsThoracic aortaReceptorSingle Nucleotidesporadic TAAAdult; Aged; Aortic Aneurysm Thoracic; Female; Gene Frequency; Genotype; Humans; Interleukin-10; Male; Middle Aged; Protein Isoforms; Regression Analysis; Sex Factors; Transforming Growth Factor beta2; Genetic Predisposition to Disease; Polymorphism Single NucleotideMiddle AgedPathophysiologyAortic AneurysmInterleukin-10Interleukin 10Regression AnalysisFemaleInflammationResearch Articlelcsh:RB1-214TGF-beta SNPAdultmedicine.medical_specialtyArticle SubjectGenotypeImmunologyBiologyPolymorphism Single NucleotideThoracic aortic aneurysmcomplex mixturesTransforming Growth Factor beta2Sex Factorsmedicine.arteryparasitic diseasesmedicinelcsh:PathologyHumansSNPSettore MED/05 - Patologia ClinicaGenetic Predisposition to DiseasePolymorphismAllele frequencyAgedSettore MED/04 - Patologia GeneraleAortic Aneurysm ThoracicSettore MED/23 - Chirurgia CardiacaCell Biologymedicine.diseasedigestive system diseasesTransforming growth factorTGF-beta SNPs; sporadic TAA; InflammationMediators of Inflammation
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Identification of Three Particular Morphological Phenotypes in Sporadic Thoracic Aortic Aneurysm: Phenotype III As Sporadic Thoracic Aortic Aneurysm …

2014

Abstract Aging has a striking impact on the heart and the vascular system, particularly on the large elastic arteries (i.e., aorta), resulting in a multitude of changes at different structural and functional levels. As result, medial degeneration (MD) occurs. A characteristic example of MD is sporadic thoracic aortic aneurysm (S-TAA), whose patho-physiological mechanisms remain unclear. In this study, typical MD morphological phenotypes were researched in S-TAA cases and control aorta specimens by histopathological and immunohistochemical analyses. Three phenotypes (I, II, and III) were detected, but mainly the phenotype III was observed. Elevated cystic MD, plurifocal medial apoptosis, and…

MalePathologymedicine.medical_specialtyAgingThoracicAging; Aorta; Aortic Aneurysm Thoracic; Biomarkers; Female; Humans; Male; Middle Aged; PhenotypeDissection (medical)Settore MED/08 - Anatomia PatologicaThoracic aortic aneurysmAneurysmmedicine.arteryMedicineHumansSettore MED/05 - Patologia ClinicaAortaSettore MED/04 - Patologia GeneraleAortaSurgical approachAortic Aneurysm Thoracicbusiness.industrySettore MED/23 - Chirurgia CardiacaMiddle Agedmedicine.diseaseTAA phenotype IIIPhenotypeAortic AneurysmPhenotypeImmunohistochemistryBiomarker (medicine)FemaleGeriatrics and GerontologybusinessBiomarkers
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Investigation of Sudan IV staining areas in aortas of infants and children: Possible prelesional stages of atherogenesis

2009

Although atherosclerosis in infants and children is generally acknowledged, the temporal and spatial sequence of LDL insudation, modification and intimal monocyte accumulation has not been systematically studied. We have investigated herein very early stages of lesion formation in human aortas of individuals up to the age of 15 years. Aortic specimens from 61 cases (37 male, 24 female) were examined. 34 cases were1 year old, 16 cases were between 1 and 5 years old, and 11 cases were between 6 and 15 years old. Areas preselected under a dissection microscope after Sudan IV staining were investigated in depth by immunohistochemical staining for apolipoprotein B, monocytes/macrophages, smooth …

MalePathologymedicine.medical_specialtyLipoprotein modificationAdolescentAorta ThoracicMonocytesmedicine.arterymedicineHumansMacrophageChildAortaApolipoproteins BAortabiologyVascular diseaseMacrophagesC-reactive proteinInfant NewbornInfantAtherosclerosismedicine.diseaseC-Reactive Proteinmedicine.anatomical_structureChild PreschoolImmunologybiology.proteinFemaleTunica IntimaCardiology and Cardiovascular MedicineAzo CompoundsBlood vesselArteryLipoproteinAtherosclerosis
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Familial thoracic aortic aneurysm/dissection with patent ductus arteriosus: genetic arguments for a particular pathophysiological entity.

2004

International audience; Thoracic aortic aneurysm and aortic dissection (TAA and AD) are an important cause of sudden death. Familial cases could account for 20% of all cases. A genetic heterogeneity with two identified genes (FBN1 and COL3A1) and three loci (3p24-25 or MFS2/TAAD2, 5q13-q14 and 11q23.2-24) has been shown previously. Study of a single family composed of 179 members with an abnormally high occurrence of TAA/AD disease. A total of 40 subjects from three generations were investigated. In addition to five cases of stroke and three cases of sudden death, there were four cases of AD and four cases of TAA in adults. In all, 11 cases of patent ductus arteriosus (PDA) were observed, t…

MalePathologymedicine.medical_specialtycongenital hereditary and neonatal diseases and abnormalitiesGenetic Linkage030204 cardiovascular system & hematologyThoracic aortic aneurysmSudden deathFamilial thoracic aortic aneurysm03 medical and health sciencesAortic aneurysmDeath Sudden0302 clinical medicineDuctus arteriosusGenetic modelGeneticsmedicine[INFO.INFO-IM]Computer Science [cs]/Medical ImagingHumansDuctus Arteriosus PatentGenetics (clinical)030304 developmental biologyAortic dissection0303 health sciences[ INFO.INFO-IM ] Computer Science [cs]/Medical ImagingAortic Aneurysm ThoracicGenetic heterogeneitybusiness.industryAnatomymedicine.disease3. Good healthPedigreeStrokeAortic Dissectionmedicine.anatomical_structureFemaleFrancebusinessMicrosatellite RepeatsEuropean journal of human genetics : EJHG
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