Search results for " transcription factor"

showing 10 items of 656 documents

Recovery after Heavy Resistance Exercise and Skeletal Muscle Androgen Receptor and Insulin-Like Growth Factor-I Isoform Expression in Strength Traine…

2011

The effects of heavy resistance exercise on skeletal muscle androgen receptor (AR) protein concentration and mRNAs of AR, insulin-like growth factor-I (IGF)-IEa, and mechano growth factor (MGF) expression were examined from biopsies of vastus lateralis (VL) muscle before and 48 hours after heavy resistance exercise (5 × 10 repetition maximum [RM] leg press and 4 × 10RM squats) in 8 adult strength trained men. The present exercise induced an acute decrease in maximal isometric force and increased serum total testosterone (T) and free testosterone (FT) concentrations. During 2 recovery days, maximal isometric force and subjective perception of physical fitness remained significantly lowered, …

AdultMalemedicine.medical_specialtymedicine.medical_treatmentPhysical fitnessPhysical Therapy Sports Therapy and RehabilitationIsometric exerciseQuadriceps MuscleYoung AdultInsulin-like growth factorBasal (phylogenetics)Isometric ContractionInternal medicineSTAT5 Transcription FactormedicineHumansProtein IsoformsTestosteroneOrthopedics and Sports MedicineMuscle StrengthInsulin-Like Growth Factor ILeg pressCreatine KinaseTestosteroneUltrasonographybusiness.industryTumor Suppressor ProteinsSkeletal muscleResistance TrainingRecovery of FunctionGeneral MedicineAdaptation PhysiologicalAndrogen receptormedicine.anatomical_structureEndocrinologyPhysical FitnessReceptors AndrogenbusinessJournal of Strength and Conditioning Research
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The influence of bone marrow- and synovium-derived mesenchymal stromal cells from osteoarthritis patients on regulatory T cells in co-culture

2013

Summary There is increasing evidence that inflammation in the synovium plays a major role in the progression of osteoarthritis (OA). However, the immunogenic properties of mesenchymal stromal cells (MSCs), which are considered to regulate immunity in various diseases, remain largely unknown in OA. The purpose of this study was to determine the influence of MSCs from OA patients on regulatory T cells (Tregs) in an allogeneic co-culture model. Bone marrow (BM) and synovial membrane (SM) were harvested from hip joints of OA patients and co-cultured with lymphocytes enriched in CD4+CD25+CD127– regulatory T cells (Treg+LC) from healthy donors. Treg proportions and MSC markers were assessed by fl…

AdultMalemedicine.medical_treatmentLymphocyteT cellImmunologyBone Marrow Cellschemical and pharmacologic phenomenaT-Lymphocytes RegulatoryInterleukin-7 Receptor alpha SubunitYoung AdultOsteoarthritisHumansImmunology and AllergyMedicineIL-2 receptorInterleukin-7 receptorAgedAged 80 and overInterleukin-6business.industrySynovial MembraneMesenchymal stem cellForkhead Transcription FactorsMesenchymal Stem CellsMiddle AgedCoculture Techniquesmedicine.anatomical_structureCytokineImmunologyFemaleBone marrowSynovial membranebusinessBiomarkersClinical and Experimental Immunology
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Reprogramming of Pericyte-Derived Cells of the Adult Human Brain into Induced Neuronal Cells

2012

SummaryReprogramming of somatic cells into neurons provides a new approach toward cell-based therapy of neurodegenerative diseases. A major challenge for the translation of neuronal reprogramming into therapy is whether the adult human brain contains cell populations amenable to direct somatic cell conversion. Here we show that cells from the adult human cerebral cortex expressing pericyte hallmarks can be reprogrammed into neuronal cells by retrovirus-mediated coexpression of the transcription factors Sox2 and Mash1. These induced neuronal cells acquire the ability of repetitive action potential firing and serve as synaptic targets for other neurons, indicating their capability of integrat…

AdultNeurogenesisCellular differentiationInduced Pluripotent Stem CellsAction PotentialsBiologySynaptic TransmissionMiceNeural Stem CellsSOX2Basic Helix-Loop-Helix Transcription FactorsGeneticsmedicineAnimalsHumansInduced pluripotent stem cellCells CulturedCerebral CortexNeuronsSOXB1 Transcription FactorsNeurogenesisCell DifferentiationNeurodegenerative DiseasesCell BiologyCellular ReprogrammingNeural stem cellCell biologyRetroviridaemedicine.anatomical_structureImmunologyMolecular MedicineNeuronPericyteNerve NetPericytesReprogrammingStem Cell TransplantationCell Stem Cell
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The role of recent thymic emigrant-regulatory T-cell (RTE-Treg) differentiation during pregnancy.

2014

During pregnancy, regulatory T cells (Tregs) have a key role in maternal immune tolerance to the semi-allogeneic fetus. Our previous results showed that the naive CD45RA(+)-Treg pool is functionally improved in pregnant women compared with non-pregnant women. Therefore, we examined the thymic output and differentiation of CD45RA(+)CD31(+) recent thymic emigrant (RTE)-Tregs during normal pregnancy and in the presence of preeclampsia. With the onset of pregnancy, the composition of the total CD4(+)CD127(low+/-)FoxP3(+)-Treg pool changed in the way that its percentage of RTE- and CD45RA(-)CD31(+)-memory Tregs decreased strongly, whereas that of the CD45RA(+)CD31(-)-mature naive (MN)-Tregs did …

Adultmedicine.medical_specialtyAdolescentRegulatory T cellImmunologyRecent Thymic Emigrantchemical and pharmacologic phenomenaThymus GlandT-Lymphocytes RegulatoryPreeclampsiaImmune toleranceYoung AdultPre-EclampsiaPregnancyT-Lymphocyte SubsetsInternal medicineImmune ToleranceImmunology and AllergyMedicineAnimalsHumansInterleukin-7 receptorFetusPregnancybusiness.industryFOXP3hemic and immune systemsCell DifferentiationForkhead Transcription FactorsCell BiologyMiddle Agedmedicine.diseasemedicine.anatomical_structureEndocrinologyLeukocyte Common AntigensFemalebusinessImmunologic MemoryImmunology and cell biology
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The role of regulatory T cell (Treg) subsets in gestational diabetes mellitus.

2014

Physiological changes during normal pregnancy are characterized by an inflammatory immune response and insulin resistance. Therefore, we hypothesize that gestational diabetes mellitus (GDM) may be caused by an inappropriate adaption of the maternal immune system to pregnancy. In this study we examined the role of regulatory T cell (Treg) differentiation for the development of GDM during pregnancy. We used six-colour flow cytometric analysis to demonstrate that the total CD4(+) CD127(low+/-) CD25(+) forkhead box protein 3 (FoxP3(+)) T(reg) pool consists of four different T(reg) subsets: naive CD45RA(+) T(regs), HLA-DR(-) CD45RA(-) memory T(regs) (DR(-) T(regs)) and the highly differentiated …

Adultmedicine.medical_specialtyendocrine system diseasesRegulatory T cellImmunologychemical and pharmacologic phenomenaCell SeparationLymphocyte ActivationT-Lymphocytes RegulatoryImmunophenotypingYoung AdultImmune systemInsulin resistanceimmune system diseasesPregnancyT-Lymphocyte Subsetshemic and lymphatic diseasesInternal medicinemedicineImmune ToleranceImmunology and AllergyHomeostasisHumansIL-2 receptorInterleukin-7 receptorbusiness.industryFOXP3hemic and immune systemsCell DifferentiationForkhead Transcription FactorsT helper cellOriginal Articlesmedicine.diseaseFlow CytometryGestational diabetesDiabetes GestationalEndocrinologymedicine.anatomical_structureImmunologyCD4 AntigensFemalebusinessImmunologic MemoryClinical and experimental immunology
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A53T-Alpha-Synuclein Overexpression Impairs Dopamine Signaling and Striatal Synaptic Plasticity in Old Mice

2010

BACKGROUND: Parkinson's disease (PD), the second most frequent neurodegenerative disorder at old age, can be caused by elevated expression or the A53T missense mutation of the presynaptic protein alpha-synuclein (SNCA). PD is characterized pathologically by the preferential vulnerability of the dopaminergic nigrostriatal projection neurons. METHODOLOGY/PRINCIPAL FINDINGS: Here, we used two mouse lines overexpressing human A53T-SNCA and studied striatal dysfunction in the absence of neurodegeneration to understand early disease mechanisms. To characterize the progression, we employed young adult as well as old mice. Analysis of striatal neurotransmitter content demonstrated that dopamine (DA…

AgingDopaminelcsh:MedicineMicechemistry.chemical_compoundHomer Scaffolding ProteinsReceptor Cannabinoid CB1lcsh:ScienceLong-term depressionNeurotransmitterChromatography High Pressure LiquidIn Situ Hybridization FluorescenceOligonucleotide Array Sequence AnalysisMice KnockoutNeuronal PlasticityMultidisciplinaryReverse Transcriptase Polymerase Chain ReactionDopaminergicNeurodegenerationGenetics and Genomics/Gene ExpressionElectrophysiologyalpha-SynucleinResearch ArticleRadioimmunoprecipitation Assaymedicine.medical_specialtyNeuronal Calcium-Sensor ProteinsHOMER1Substantia nigraNeurotransmissionBiologyNeurological DisordersInternal medicinemedicineAnimalsHumansddc:610Cyclic Nucleotide Phosphodiesterases Type 7Activating Transcription Factor 2lcsh:RNeuropeptidesmedicine.diseaseMolecular biologyCorpus StriatumMice Mutant StrainsEndocrinologyGenetics and Genomics/Disease ModelschemistrySynaptic plasticitylcsh:QCarrier ProteinsPLoS ONE
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Negative Regulation of β Enolase Gene Transcription in Embryonic Muscle Is Dependent upon a Zinc Finger Factor That Binds to the G-rich Box within th…

1998

We have previously identified a muscle-specific enhancer within the first intron of the human beta enolase gene. Present in this enhancer are an A/T-rich box that binds MEF-2 protein(s) and a G-rich box (AGTGGGGGAGGGGGCTGCG) that interacts with ubiquitously expressed factors. Both elements are required for tissue-specific expression of the gene in skeletal muscle cells. Here, we report the identification and characterization of a Kruppel-like zinc finger protein, termed beta enolase repressor factor 1, that binds in a sequence-specific manner to the G-rich box and functions as a repressor of the beta enolase gene transcription in transient transfection assays. Using fusion polypeptides of b…

AgingTranscription GeneticMolecular Sequence DataDown-RegulationRepressorRegulatory Sequences Nucleic AcidBiologyBiochemistryDNA-binding proteinGene Expression Regulation EnzymologicMiceGene expressionAnimalsHumansAmino Acid SequenceCloning MolecularMuscle SkeletalEnhancerMolecular BiologyCell NucleusRegulation of gene expressionZinc fingerSp1 transcription factorBinding SitesSequence Homology Amino AcidZinc FingersCell BiologyMolecular biologyDNA-Binding ProteinsEnhancer Elements GeneticRegulatory sequencePhosphopyruvate HydrataseJournal of Biological Chemistry
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Effective treatment of allergic airway inflammation with Helicobacter pylori immunomodulators requires BATF3-dependent dendritic cells and IL-10

2014

The prevalence of allergic asthma and other atopic diseases has reached epidemic proportions in large parts of the developed world. The gradual loss of the human indigenous microbiota has been held responsible for this trend. The bacterial pathogen Helicobacter pylori is a constituent of the normal gastric microbiota whose presence has been inversely linked to allergy and asthma in humans and experimental models. Here we show that oral or i.p. tolerization with H. pylori extract prevents the airway hyperresponsiveness, bronchoalveolar eosinophilia, pulmonary inflammation, and Th2 cytokine production that are hallmarks of allergen-induced asthma in mice. Asthma protection is not conferred by…

Allergybacterial persistence determinantsT-Lymphocytes Regulatorybacterial immunomodulationImmune toleranceMiceBacterial ProteinsImmune TolerancemedicineAnimalsHumansImmunologic FactorsEosinophiliaAsthmaMice KnockoutAntigens Bacterial1000 MultidisciplinaryMultidisciplinaryHelicobacter pyloribiologyallergy and asthma prevention10061 Institute of Molecular Cancer ResearchInterleukin-18tolerogenic dendritic cellsDendritic Cellsgamma-GlutamyltransferaseDendritic cellBiological SciencesAllergensHelicobacter pyloribacterial infections and mycosesmedicine.diseasebiology.organism_classificationAsthmaInterleukin-10respiratory tract diseases3. Good healthMice Inbred C57BLRepressor ProteinsDisease Models AnimalInterleukin 10Basic-Leucine Zipper Transcription FactorsImmunology570 Life sciences; biologyInterleukin 18medicine.symptom
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Stabilization of hsp70 mRNA on prolonged cell exposure to hypertonicity

2002

AbstractProlonged exposure of 3T3 cells to 0.5 osM hypertonic medium induced the accumulation of hsp70 mRNAs. This increase in mRNA levels required active protein synthesis. A weak and transient activation of heat shock factor 1 (HSF1) was noted, but it was temporally uncoupled to the accumulation of the hsp70 mRNAs. Nuclear run-on assay and transfection experiments showed that hsp70 gene transcription was not affected by hypertonicity. ActD chase experiments showed that during hypertonic treatment, degradation of hsp70 mRNAs was markedly reduced. This effect did not appear to be a general phenomenon since the increase in mRNA level of another gene induced by hypertonicity (ATA2 transporter…

Amino Acid Transport System ATranscription GeneticBiologyTransfectionMiceHeat Shock Transcription FactorsTranscription (biology)Heat shock proteinATA2 mRNAAnimalsHSP70 Heat-Shock ProteinsRNA MessengerHSF1HypertonicityMolecular BiologySaline Solution HypertonicMessenger RNAHeat shock proteinMRNA stabilizationTransfection3T3 CellsCell Biologyhsp70 mRNAMolecular biologyHsp70DNA-Binding ProteinsProtein BiosynthesisRNA stabilizationmRNA stabilizationTranscription FactorsBiochimica et Biophysica Acta (BBA) - Molecular Cell Research
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Mast Cells and Th17 Cells Contribute to the Lymphoma-Associated Pro-Inflammatory Microenvironment of Angioimmunoblastic T-Cell Lymphoma

2010

Reports focusing on the immunological microenvironment of peripheral T-cell lymphomas (PTCL) are rare. Here we studied the reciprocal contribution of regulatory (Treg) and interleukin-17-producing (Th17) T-cells to the composition of the lymphoma-associated microenvironment of angioimmunoblastic T-cell lymphoma (AITL) and PTCL not otherwise specified on tissue microarrays from 30 PTCLs not otherwise specified and 37 AITLs. We found that Th17 but not Treg cells were differently represented in the two lymphomas and correlated with the amount of mast cells (MCs) and granulocytes, which preferentially occurred in the cellular milieu of AITL cases. We observed that MCs directly synthesized inter…

Angioimmunoblastic T-cell lymphomaLymphomaInflammationBiologymedicine.disease_causeCXCR3Lymphoma T-CellCXCR5Pathology and Forensic MedicineAutoimmunityAnimals Chemokine CXCL13; immunology Cytokines; genetics/immu/nology Forkhead Transcription Factors; immunology Gene Expression Profiling Humans Immunoblastic Lymphadenopathy; immunology/pathology Inflammation; immunology Interleukin-17; immunology Interleukin-6; immunology Lymphoma; T-Cell; immunology/pathology Mast Cells; immunology Microarray Analysis Th17 Cells; immunology Tumor MicroenvironmentimmunologymedicineTumor MicroenvironmentAnimalsHumansMast CellsInflammationTumor microenvironmentInterleukin-6Gene Expression ProfilingInterleukin-17Forkhead Transcription FactorsMast cellmedicine.diseaseT-CellMicroarray AnalysisChemokine CXCL13humanitiesgenetics/immu/nologyLymphomamedicine.anatomical_structureImmunoblastic LymphadenopathyImmunologyCytokinesimmunology/pathologyTh17 CellsMast Cell microenvironment angioimmunoblasticmedicine.symptomRegular Articles
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