Search results for " transcription"

showing 10 items of 810 documents

Extracellular Vesicles from Neural Stem Cells Transfer IFN-γ via Ifngr1 to Activate Stat1 Signaling in Target Cells

2014

The idea that stem cell therapies work only via cell replacement is challenged by the observation of consistent intercellular molecule exchange between the graft and the host. Here we defined a mechanism of cellular signaling by which neural stem/precursor cells (NPCs) communicate with the microenvironment via extracellular vesicles (EVs), and we elucidated its molecular signature and function. We observed cytokine-regulated pathways that sort proteins and mRNAs into EVs. We described induction of interferon gamma (IFN-γ) pathway in NPCs exposed to proinflammatory cytokines that is mirrored in EVs. We showed that IFN-γ bound to EVs through Ifngr1 activates Stat1 in target cells. Finally, we…

Cell signalingCell CommunicationBiologyArticle3T3 cellsProinflammatory cytokineInterferon-gammaMiceTh2 CellsNeural Stem CellsPrecursor cellmedicineAnimalsInterferon gammaRNA MessengerTransport VesiclesMolecular BiologyReceptors InterferonInflammationBiological Transport3T3 CellsCell BiologyTh1 CellsNeural stem cellCell biologySTAT1 Transcription Factormedicine.anatomical_structureCellular MicroenvironmentSignal transductionStem cellSignal Transductionmedicine.drugMolecular Cell
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Endothelial transcriptomic changes induced by oxidized low density lipoprotein disclose an up-regulation of Jak-Stat pathway.

2015

Oxidized low density lipoproteins (oxLDLs) act as an etiological factor in the development of atherosclerosis by modifying the biological properties of endothelial cells through mechanisms of vascular inflammation. To deepen the oxLDL changes at cellular level, a transcriptomic analysis of human umbilical artery endothelial cells (HUAECs) treated with oxLDL was performed to identify the modified signaling pathways. Total RNA was isolated from HUAECs treated with oxLDL (100 μg/ml). Gene expression analysis was carried out using Affymetrix oligonucleotide microarrays. Biological pathway analysis was performed using Ingenuity Pathway Analysis software. Microarray assay demonstrated that oxLDL …

Cell signalingTime FactorsPhysiologyBlotting WesternBiologyReal-Time Polymerase Chain ReactionTransfectionGene Expression Regulation EnzymologicBiological pathwayTranscriptomeRNA interferenceGene expressionHuman Umbilical Vein Endothelial CellsHumansGene Regulatory NetworksProtein Kinase InhibitorsCells CulturedOligonucleotide Array Sequence AnalysisPharmacologyGene Expression ProfilingJAK-STAT signaling pathwaySTAT2 Transcription FactorJanus Kinase 1Janus Kinase 2Cell biologyEndothelial stem cellLipoproteins LDLSTAT1 Transcription FactorMolecular Medicinelipids (amino acids peptides and proteins)RNA InterferenceSignal transductionTranscriptomeSignal TransductionVascular pharmacology
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In Activated Murine Mast Cells, NFATc2 Is Critical for the Production of Autocrine IL-3, Thereby Promoting the Expression of IL-9

2019

Abstract IL-9 has lent its numerical designation to the Th9 subset of CD4+ Th cells, although it is also produced by additional cell types, including mast cells. It is a pleiotropic cytokine involved in allergic reactions, parasitic infections, autoimmune inflammation, and cancer immunity. In this article, we provide evidence that NFATc2 has contradictory functions in the expression of IL-9 in murine Th9 cells and bone marrow–derived mast cells (BMMC). The basis for this is our observation that the production of IL-9 in NFATc2-deficient Th9 cells is increased, whereas it is decreased in BMMC devoid of NFATc2. In addition, NFATc2 deficiency almost completely abrogates the expression of IL-3 …

Cell typeNFATC2medicine.medical_treatmentImmunologyCellAutocrine CommunicationMice03 medical and health sciences0302 clinical medicineDownregulation and upregulationSTAT5 Transcription FactormedicineAnimalsImmunology and AllergyMast CellsAutocrine signallingCells CulturedSTAT5Feedback PhysiologicalMice KnockoutMice Inbred BALB CNFATC Transcription FactorsbiologyChemistryInterleukin-9T-Lymphocytes Helper-InducerUp-RegulationCell biologyMice Inbred C57BLAutocrine CommunicationCytokinemedicine.anatomical_structurebiology.proteinInterleukin-3030215 immunologyThe Journal of Immunology
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Lineage-reprogramming of Pericyte-derived Cells of the Adult Human Brain into Induced Neurons

2014

Direct lineage-reprogramming of non-neuronal cells into induced neurons (iNs) may provide insights into the molecular mechanisms underlying neurogenesis and enable new strategies for in vitro modeling or repairing the diseased brain. Identifying brain-resident non-neuronal cell types amenable to direct conversion into iNs might allow for launching such an approach in situ, i.e. within the damaged brain tissue. Here we describe a protocol developed in the attempt of identifying cells derived from the adult human brain that fulfill this premise. This protocol involves: (1) the culturing of human cells from the cerebral cortex obtained from adult human brain biopsies; (2) the in vitro expansio…

Cell typePatch-Clamp TechniquesGeneral Chemical EngineeringCell Culture TechniquesBiologyGeneral Biochemistry Genetics and Molecular BiologySOX2Transduction GeneticmedicineHumansCell LineageCerebral CortexNeuronsGeneral Immunology and MicrobiologyGeneral NeuroscienceSOXB1 Transcription FactorsNeurogenesisHuman brainCell sortingCellular ReprogrammingFlow CytometryImmunohistochemistrymedicine.anatomical_structureRetroviridaeCell culturePericytePericytesNeuroscienceReprogrammingNeuroscience
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Differential expression levels of Sox9 in early neocortical radial glial cells regulate the decision between stem cell maintenance and differentiation

2021

ABSTRACTRadial glial progenitor cells (RGCs) in the dorsal forebrain directly or indirectly produce excitatory projection neurons and macroglia of the neocortex. Recent evidence shows that the pool of RGCs is more heterogeneous than originally thought and that progenitor subpopulations can generate particular neuronal cell types. Using single cell RNA sequencing, we have studied gene expression patterns of two subtypes of RGCs that differ in their neurogenic behavior. One progenitor type rapidly produces postmitotic neurons, whereas the second progenitor remains relatively quiescence before generating neurons. We have identified candidate genes that are differentially expressed between thes…

Cell typeTranscription GeneticNeurogenesisEpendymoglial CellsGenetic VectorsNeocortexNerve Tissue ProteinsBiologyMiceradial glia cellsprogenitors diversityGenes ReporterPregnancyGene expressionmedicineAnimalscortical developmentProgenitors diversityCell Self RenewalProgenitor cellPromoter Regions GeneticTranscription factorResearch ArticlesInjections IntraventricularProgenitorNeuronsNeocortexCortical developmentGeneral NeuroscienceCell CycleGene Expression Regulation DevelopmentalSOX9 Transcription FactorEmbryonic stem cellCell biologyMice Inbred C57BLCorticogenesisElectroporationmedicine.anatomical_structureCerebral cortexForebrainFemalesense organsSingle-Cell AnalysisStem cellNeuroscienceNeurogliaRadial glia cellsCellular/MolecularSox9
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Saccharomyces cerevisiae Rds2 transcription factor involvement in cell wall composition and architecture

2008

Although the cell wall is very important in yeasts, relatively little is known about the relationship between its structure and function. In Saccharomyces cerevisiae, a family of 55 transcription factor proteins unique to fungi, so-called zinc cluster proteins, has been described. Of these, Rds2 has been identified as an activator/inhibitor of gluconeogenesis. However, previous studies have pointed out additional roles for this protein, specifically, in the modulation of cell-wall architecture and drug sensitivity. In this work, evidence regarding the role of Rds2 as a regulator of cell-wall architecture and composition is presented based on phenotypical analysis of the cell walls prepared …

Cell wall:CIENCIAS DE LA VIDA::Microbiología [UNESCO]Gene RDS2Transcription factorsSaccharomyces cerevisiaeSaccharomyces cerevisiae; Transcription factors; Gene RDS2; Cell wallSaccharomyces cerevisiae ; Transcription factors ; Gene RDS2 ; Cell wallUNESCO::CIENCIAS DE LA VIDA::Microbiología
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PTHrP [67-86] regulates the expression of stress proteins in breast cancer cells inducing modifications in urokinase-plasminogen activator and MMP-1 …

2003

It was previously reported that a midregion domain of parathyroid hormone-related protein (PTHrP), that is, [67-86]-amide, is able to restrain growth and promote matrigel penetration by the 8701-BC cell line, derived from a biopsy fragment of a primary ductal infiltrating carcinoma of the human breast, and that cell invasion in vitro is drastically impaired by inactivation of urokinase-plasminogen activator (uPa). In this study we started a more detailed investigation of the possible effects on gene expression arising from the interaction between PTHrP [67-86]-amide and 8701-BC breast cancer cells by a combination of conventional-, differential display-and semi-quantitative multiplex-polyme…

CellBreast NeoplasmsBiologyHeat Shock Transcription FactorsDownregulation and upregulationCell Line TumorHeat shock proteinmedicineHumansNeoplasm InvasivenessHSP90 Heat-Shock ProteinsEnzyme InhibitorsHSF1Heat-Shock ProteinsMatrigelActivator (genetics)CarcinomaParathyroid Hormone-Related ProteinCell BiologyOligonucleotides AntisenseUrokinase-Type Plasminogen ActivatorMolecular biologyPeptide FragmentsProtein Structure TertiaryUp-RegulationDNA-Binding ProteinsGene Expression Regulation NeoplasticHeat shock factormedicine.anatomical_structureCell cultureCancer researchFemaleQuercetinMatrix Metalloproteinase 1Transcription FactorsJournal of Cell Science
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Hypoxia and HIF Signaling: One Axis with Divergent Effects

2020

The correct concentration of oxygen in all tissues is a hallmark of cellular wellness, and the negative regulation of oxygen homeostasis is able to affect the cells and tissues of the whole organism. The cellular response to hypoxia is characterized by the activation of multiple genes involved in many biological processes. Among them, hypoxia-inducible factor (HIF) represents the master regulator of the hypoxia response. The active heterodimeric complex HIF α/β, binding to hypoxia-responsive elements (HREs), determines the induction of at least 100 target genes to restore tissue homeostasis. A growing body of evidence demonstrates that hypoxia signaling can act by generating contrasting res…

CellInflammationReviewBiologyCatalysislcsh:ChemistryInorganic ChemistryImmune systemSettore BIO/13 - Biologia ApplicataOxygen homeostasisBasic Helix-Loop-Helix Transcription FactorsmedicineHumansRNA MessengerAcute and chronic diseasesPhysical and Theoretical ChemistryHypoxialcsh:QH301-705.5Molecular BiologySpectroscopyTissue homeostasisInflammationKidneyImmune cellsOrganic ChemistryHIF-αNuclear ProteinsGeneral MedicineHypoxia (medical)Cell HypoxiaComputer Science ApplicationsCell biologyDNA-Binding ProteinsOxygenmedicine.anatomical_structurelcsh:Biology (General)lcsh:QD1-999Hypoxia-Inducible Factor 1medicine.symptomSignal transductionSignal TransductionTranscription FactorsInternational Journal of Molecular Sciences
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Dihydrocucurbitacin B Inhibits Delayed Type Hypersensitivity Reactions by Suppressing Lymphocyte Proliferation

2007

We have studied the effects of dihydrocucurbitacin B, a triterpene isolated from Cayaponia tayuya roots, on different models of delayed type hypersensitivity (DTH) in mice, as well as on T-lymphocyte proliferation and the mediators involved. In experiments with mice, dihydrocucurbitacin B inhibited the inflammatory reactions induced by oxazolone, dinitrofluorobenzene, and sheep red blood cells, reducing both the edema and cell infiltration. Moreover, the analysis of inflamed tissues showed that dihydrocucurbitacin B reduced the presence of the most relevant cytokines implicated in these processes, including interleukin-1 beta, interleukin-4, and tumor necrosis factor-alpha. Dihydrocucurbita…

CellLymphocyte proliferationLymphocyte ActivationResting Phase Cell CycleOxazoloneMicechemistry.chemical_compoundCyclinsmedicineAnimalsHypersensitivity DelayedCyclinInflammationPharmacologyNFATC Transcription FactorsbiologyNFATCell cyclebiology.organism_classificationMolecular biologyTriterpenesCayaponia tayuyaDisease Models Animalmedicine.anatomical_structurechemistryImmunologyCytokinesMolecular MedicineTumor necrosis factor alphaJournal of Pharmacology and Experimental Therapeutics
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Differentiation of Type 1 ILCs from a Common Progenitor to All Helper-like Innate Lymphoid Cell Lineages

2014

SummaryInnate lymphoid cells (ILCs) are a recently recognized group of lymphocytes that have important functions in protecting epithelial barriers against infections and in maintaining organ homeostasis. ILCs have been categorized into three distinct groups, transcriptional circuitry and effector functions of which strikingly resemble the various T helper cell subsets. Here, we identify a common, Id2-expressing progenitor to all interleukin 7 receptor-expressing, “helper-like” ILC lineages, the CHILP. Interestingly, the CHILP differentiated into ILC2 and ILC3 lineages, but not into conventional natural killer (cNK) cells that have been considered an ILC1 subset. Instead, the CHILP gave rise…

Cellular differentiationLineage (evolution)Bone Marrow CellsGATA3 Transcription FactorBiologyGeneral Biochemistry Genetics and Molecular BiologyMicemedicineAnimalsLymphocytesskin and connective tissue diseasesProgenitorInhibitor of Differentiation Protein 2Receptors Interleukin-7Biochemistry Genetics and Molecular Biology(all)Intracellular parasiteStem CellsInnate lymphoid cellNFIL3Cell DifferentiationT helper cellImmunity InnateMice Inbred C57BLbody regionsmedicine.anatomical_structureImmunologyToxoplasmaIntracellularToxoplasmosisCell
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