Search results for " treatment."

showing 10 items of 3290 documents

Adrenal Gland and Gastric Malignant Melanoma without Evidence of Skin Lesion Treated with the Oncolytic Virus Rigvir

2020

Adrenal gland melanoma is an extremely rare diagnosis with less than 20 cases reported. The criteria for diagnosing adrenal gland melanoma include involvement of only one adrenal gland, presence of melanin pigment in the histological examination of the tumor tissue, no primary melanoma tumor in any other organ, and no history of resection of pigmented lesions. However, it is complicated to rule out melanoma of unknown primary origin. Here we report a female patient who at the age of 75 years was admitted to hospital due to suspicion of adrenal and gastric tumor. The largest tumor was found in the adrenal gland, thus leading to the diagnosis of primary adrenal gland melanoma presenting metas…

0301 basic medicinePathologymedicine.medical_specialtyCase ReportDiseaseMetastatic melanomalcsh:RC254-28203 medical and health sciences0302 clinical medicinemedicineOncolytic virotherapyAdrenal glandbusiness.industryMelanomaStomachStandard treatmentlcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseaseAdrenal gland melanomaOncolytic virus030104 developmental biologymedicine.anatomical_structureOncologyTolerability030220 oncology & carcinogenesisSkin lesionbusinessCase Reports in Oncology
researchProduct

Home infusion program with enzyme replacement therapy for Fabry disease: The experience of a large Italian collaborative group

2017

Fabry disease (FD) [OMIM 301500] is an X-linked lysosomal storage disorder caused by a deficiency of the lysosomal enzyme alpha-galactosidase A, resulting in progressive multisystem accumulation of globotriaosylceramide (Gb3). Although the introduction of Enzyme Replacement Therapy (ERT) resulted in a variety of clinical benefits, life-long intravenous (IV) treatment with ERT with an every other week schedule, may interfere with daily life activities and impact on QoL. We report here a multicentric, observational, longitudinal data analysis on a large cohort of 85 Italian FD patients (45 males, 40 females) from 11 out of 20 Italian regions, who received a cumulative number of 4269 home infu…

0301 basic medicinePediatricsmedicine.medical_specialtyQoLGlobotriaosylceramide03 medical and health scienceschemistry.chemical_compoundCollaborative group0302 clinical medicineEndocrinologyDisease severityGeneticGeneticsMedicine030212 general & internal medicinelcsh:QH301-705.5Molecular Biologylcsh:R5-920Fabry diseasebusiness.industrySettore BIO/14Home treatmentEnzyme replacement therapyAdherence; Enzyme replacement therapy; Fabry disease; Home treatment; QoLmedicine.diseaseFabry disease3. Good health030104 developmental biologylcsh:Biology (General)chemistryAdherenceEnzyme replacement therapyCohortarticle;congenital malformation; Fabry disease; enzyme replacement therapy; home treatment ; adherence; QoLObservational studyHome treatmentlcsh:Medicine (General)businessResearch Paper
researchProduct

Chemoresistance and chemosensitization in cholangiocarcinoma

2017

One of the main difficulties in the management of patients with advanced cholangiocarcinoma (CCA) is their poor response to available chemotherapy. This is the result of powerful mechanisms of chemoresistance (MOC) of quite diverse nature that usually act synergistically. The problem is often worsened by altered MOC gene expression in response to pharmacological treatment. Since CCA includes a heterogeneous group of cancers their genetic signature coding for MOC genes is also diverse; however, several shared traits have been defined. Some of these characteristics are shared with other types of liver cancer, namely hepatocellular carcinoma and hepatoblastoma. An important goal in modern onco…

0301 basic medicinePharmacologybile ductschemotherapydrug delivery systems0302 clinical medicineChemosensitizationantineoplastic agentsmolecular biologyReceptorhumansreceptor protein-tyrosine kinasesmedia_commonapoptosisgene expression regulationbile duct neoplasmsDrug Resistance Multipletargeted therapiesGene Expression Regulation Neoplasticmultiplebiliary cancer; chemotherapy; liver cancer; multidrug resistance; targeted therapies; antineoplastic agents; apoptosis; bile duct neoplasms; bile ducts; cell survival; cholangiocarcinoma; drug delivery systems; drug resistance multiple; drug resistance; neoplasm; epithelial cells; gene expression regulation neoplastic; genetic therapy; humans; protein kinase inhibitors; receptor protein-tyrosine kinases; signal transduction; treatment outcome; molecular medicine; molecular biology030220 oncology & carcinogenesisHepatocellular carcinomabiliary cancerLiver cancercholangiocarcinomaTyrosine kinasesignal transductionDrugHepatoblastomamedia_common.quotation_subjectcell survivalPharmacological treatmentliver cancer03 medical and health sciencesmultidrug resistancemedicinemolecular medicinedrug resistancebusiness.industrymedicine.diseaseepithelial cellsneoplasticprotein kinase inhibitors030104 developmental biologyDrug Resistance NeoplasmCancer researchtreatment outcomebusinessneoplasmgenetic therapy
researchProduct

Stimulation of natural killer cells with rhCD137 ligand enhances tumor-targeting antibody efficacy in gastric cancer

2018

Although many anticancer agents for gastric cancer have been developed, the prognosis for many patients remains poor. Recently, costimulatory immune molecules that reactivate antitumor immune responses by utilizing the host immune system have attracted attention as new therapeutic strategies. CD137 is a costimulatory molecule that reportedly potentiates the antitumor activity of tumor-targeting monoclonal antibodies (mAbs) by enhancing antibody-dependent cellular cytotoxicity. However, it remains unclear whether CD137 stimulates tumor-regulatory activity in gastric cancer. In this study, we investigated the antitumor effects of CD137 stimulation on gastric cancer cells administered tumor-ta…

0301 basic medicinePhysiologyCytotoxicityCancer Treatmentlcsh:MedicineNK cellsToxicologyPathology and Laboratory MedicineAntineoplastic Agents ImmunologicalSpectrum Analysis Techniques0302 clinical medicineImmune PhysiologyCellular typeslcsh:ScienceInnate Immune SystemCytotoxicity AssayMultidisciplinarybiologyChemistryImmune cellsCD137Drug SynergismFlow CytometryRecombinant ProteinsUp-RegulationGene Expression Regulation NeoplasticKiller Cells NaturalOncologySpectrophotometry030220 oncology & carcinogenesisCytokinesWhite blood cellsFemaleTumor necrosis factor alphaCytophotometryAntibodyResearch ArticleCell biologyBlood cellsCell Survivalmedicine.drug_classImmunologyAntibodies Monoclonal HumanizedResearch and Analysis MethodsMonoclonal antibody03 medical and health sciencesImmune systemStomach NeoplasmsCell Line TumorGastrointestinal TumorsmedicineHumansSecretionCell ProliferationMedicine and health sciencesBiology and life scienceslcsh:RCancers and NeoplasmsCancerTrastuzumabMolecular Developmentmedicine.diseaseGranzyme BGastric Cancer4-1BB Ligand030104 developmental biologyAnimal cellsImmune SystemCancer cellCancer researchbiology.proteinlcsh:QPhysiological ProcessesDevelopmental BiologyPLOS ONE
researchProduct

Chimeric proteins tagged with specific 3xHA cassettes may present instability and functional problems

2017

Epitope-tagging of proteins has become a widespread technique for the analysis of protein function, protein interactions and protein localization among others. Tagging of genes by chromosomal integration of PCR amplified cassettes is a widely used and fast method to label proteins in vivo. Different systems have been developed during years in the yeast Saccharomyces cerevisiae. In the present study, we analysed systematically a set of yeast proteins that were fused to different tags. Analysis of the tagged proteins revealed an unexpected general effect on protein level when some specific tagging module was used. This was due in all cases to a destabilization of the proteins and caused a red…

0301 basic medicinePhysiologyProtein Extractionlcsh:MedicineYeast and Fungal ModelsPolymerase Chain ReactionBiochemistryGreen fluorescent proteinEpitopesDatabase and Informatics MethodsGene Expression Regulation FungalImmune PhysiologyProtein purificationMacromolecular Structure AnalysisMedicine and Health SciencesProto-Oncogene Proteins c-myclcsh:ScienceStainingExtraction TechniquesImmune System ProteinsMultidisciplinarybiologyGene targetingProtein subcellular localization predictionMembrane StainingExperimental Organism SystemsGene TargetingArtifactsSequence AnalysisPlasmidsResearch ArticleProtein StructureSaccharomyces cerevisiae ProteinsBioinformaticsRecombinant Fusion ProteinsGenetic VectorsGreen Fluorescent ProteinsImmunologySaccharomyces cerevisiaeHemagglutinins ViralSaccharomyces cerevisiaeComputational biologyResearch and Analysis MethodsGreen Fluorescent ProteinGenomic InstabilityAntibodiesProtein–protein interactionProto-Oncogene Proteins c-mycSaccharomyces03 medical and health sciencesModel OrganismsAmino Acid Sequence AnalysisMolecular BiologyStaining and Labelinglcsh:ROrganismsFungiBiology and Life SciencesProteinsbiology.organism_classificationFusion proteinYeastLuminescent Proteins030104 developmental biologySpecimen Preparation and Treatmentlcsh:QProtein Structure NetworksPLOS ONE
researchProduct

Cell stimulation versus cell death induced by sequential treatments with pulsed electric fields and cold atmospheric pressure plasma

2018

Pulsed electric fields (PEFs) and cold atmospheric pressure plasma (CAP) are currently both investigated for medical applications. The exposure of cells to PEFs can induce the formation of pores in cell membranes and consequently facilitate the uptake of molecules. In contrast, CAP mainly acts through reactive species that are generated in the liquid environment. The objective of this study was to determine, if PEFs combined with plasma-treated cell culture medium can mutually reinforce effects on viability of mammalian cells. Experiments were conducted with rat liver epithelial WB-F344 cells and their tumorigenic counterpart WB-ras for a direct comparison of non-tumorigenic and tumorigenic…

0301 basic medicinePlasma GasesCell MembranesCancer Treatmentlcsh:MedicineMechanical Treatment of Specimens0302 clinical medicineElectricityNeoplasmsMedicine and Health SciencesEnzyme assaysColorimetric assayslcsh:ScienceBioassays and physiological analysisCells CulturedMTT assayMultidisciplinaryChemistryPhysicsElectroporationKetonesrespiratory systemCombined Modality TherapyChemistryElectroporationMembraneOncologySpecimen DisruptionElectric Field030220 oncology & carcinogenesisPhysical SciencesBiological CulturesCellular Structures and OrganellesResearch ArticlePyruvateCell typeProgrammed cell deathCell SurvivalElectric Stimulation TherapyAtmospheric-pressure plasmaResearch and Analysis Methods03 medical and health sciencesCell Line TumorAnimalsHumansMTT assayCell ProliferationCell growthlcsh:RChemical CompoundsBiology and Life SciencesEpithelial CellsCell BiologyCell CulturesCulture MediaRats030104 developmental biologyCytostaticsSpecimen Preparation and TreatmentCell cultureBiochemical analysisBiophysicslcsh:QAcidsPLOS ONE
researchProduct

Managing adult patients with infectious diseases in emergency departments: international ID-IRI study.

2021

We aimed to explore factors for optimizing antimicrobial treatment in emergency departments. A single-day point prevalence survey was conducted on January 18, 2020, in 53 referral/tertiary hospitals in 22 countries. 1957 (17%) of 11557 patients presenting to EDs had infections. The mean qSOFA score was 0.37 +/- 0.74. Sepsis (qSOFA >= 2) was recorded in 218 (11.1%) patients. The mean qSOFA score was significantly higher in low-middle (1.48 +/- 0.963) compared to upper-middle (0.17 +/- 0.482) and high-income (0.36 +/- 0.714) countries ( P < 0.001). Eight (3.7%) patients with sepsis were treated as outpatients. The most common diagnoses were upper-respiratory (n = 877, 43.3%), lower-respirator…

0301 basic medicinePoint prevalence surveymedicine.medical_specialtyUrologic NeoplasmsReferralinternational ID-IRI study- JOURNAL OF CHEMOTHERAPY 2021 [Erdem H. Hargreaves S. ANKARALI H. ÇAŞKURLU H. Ceviker S. A. Bahar-Kacmaz A. Meric-Koc M. ALTINDİŞ M. Yildiz-Kirazaldi Y. Kizilates F. et al. -Managing adult patients with infectious diseases in emergency departments]medicine.drug_classOrgan Dysfunction Scores030106 microbiologyAntibioticsPractice Patternsemergency ; antibiotic ; elderly ; infection ; sepsis ; treatmentGlobal HealthelderlyCommunicable Diseasestreatment.SepsisHospital03 medical and health sciences0302 clinical medicineantibioticSepsismedicineHumansPharmacology (medical)Practice Patterns Physicians'Developing CountriesRespiratory Tract InfectionsPharmacologyEmergency ServicePhysicians'Adult patientstreatmentbusiness.industryPatient AcuityAntimicrobialmedicine.diseasehumanitiesinfectionDrug UtilizationAnti-Bacterial AgentsInfectious DiseasesOncology030220 oncology & carcinogenesisEmergency medicineEmergencysepsibusinessEmergency Service HospitalJournal of chemotherapy (Florence, Italy)
researchProduct

Plasma modified PLA electrospun membranes for actinorhodin production intensification in Streptomyces coelicolor immobilized-cell cultivations

2017

Most of industrially relevant bioproducts are produced by submerged cultivations of actinomycetes. The immobilization of these Gram-positive filamentous bacteria on suitable porous supports may prevent mycelial cell-cell aggregation and pellet formation which usually negatively affect actinomycete submerged cultivations, thus, resulting in an improved biosynthetic capability. In this work, electrospun polylactic acid (PLA) membranes, subjected or not to O2-plasma treatment (PLA-plasma), were used as support for immobilized-cell submerged cultivations of Streptomyces coelicolor M145. This strain produces different bioactive compounds, including the blue-pigmented actinorhodin (ACT) and red-p…

0301 basic medicinePolyestersSegmented filamentous bacteriaS. coelicolor immobilizationAnthraquinonesStreptomyces coelicolor02 engineering and technologySecondary metaboliteSettore BIO/19 - Microbiologia GeneraleActinorhodinMicrobiology03 medical and health scienceschemistry.chemical_compoundColloid and Surface Chemistrystomatognathic systemPolylactic acidmedicinePlasma treatmentPhysical and Theoretical ChemistryIncubationMyceliumbiologyElectrospinningPhotoelectron SpectroscopyProdigiosinStreptomyces coelicolorActinorhodin productiontechnology industry and agricultureSettore ING-IND/34 - Bioingegneria IndustrialePLA membraneSurfaces and InterfacesGeneral Medicine021001 nanoscience & nanotechnologybiology.organism_classificationAnti-Bacterial Agents030104 developmental biologyMembraneSettore ING-IND/22 - Scienza E Tecnologia Dei MaterialichemistryBiochemistryMicroscopy Electron Scanning0210 nano-technologyBiotechnologymedicine.drug
researchProduct

Updated Results from the German Mpnsg-0212 Combination Trial: Ruxolitinib Plus Pomalidomide in Myelofibrosis with Anemia

2019

Background: Anemia remains one cardinal symptom associated with reduced quality of life (QoL) in patients (pts) with myelofibrosis (MF) which is normally not being addressed by ruxolitinib (RUX). In our previous MPNSG-0109 trial, single-agent pomalidomide (POM) improved cytopenia in 14% (POM 0.5 mg QD) and 29% (POM 2.0 mg QD) of MF pts, respectively. In the MPNSG-0212 study, we sought to investigate the potential synergism of RUX plus POM to improve anemia and QoL in MF pts. Study Design: MPNSG-0212 is an ongoing multicenter, open-label, single-arm phase-Ib/II trial with a target population of 90 pts following a two-stage design (NCT01644110). Pts 1-40 in cohort 1 (co1) were treated with RU…

0301 basic medicinePrior treatmentmedicine.medical_specialtyRuxolitinibbusiness.industryAnemiaImmunologyMedizinCell BiologyHematologyPomalidomidemedicine.diseaseBiochemistry03 medical and health sciences030104 developmental biology0302 clinical medicineBaseline characteristicsSustained responseInternal medicinemedicineIn patientbusinessBristol-Myers030215 immunologymedicine.drugBlood
researchProduct

Betulinic Acid Kills Colon Cancer Stem Cells

2016

Cancer stem cells (CSCs) are considered to be the origin of cancer and it is suggested that they are resistant to chemotherapy. Current therapies fail to eradicate CSCs and therefore selecting a resistant cell subset that is able to facilitate tumor recurrences. Betulinic acid (BetA) is a broad acting natural compound, shown to induce cell death via the inhibition of the stearoyl-CoA- desaturase (SCD- 1). This enzyme converts saturated fatty acids into unsaturated fatty acids and is over-expressed in tumor cells. Here we show that BetA induces rapid cell death in all colon CSCs tested and is able to affect the CSCs directly as shown, via the loss of clonogenic capacity. Similar results were…

0301 basic medicineProgrammed cell deathColorectal cancerMedicine (miscellaneous)Biology03 medical and health scienceschemistry.chemical_compound0302 clinical medicineCancer stem cellBetulinic acidCell Line TumormedicineHumansEnzyme InhibitorsClonogenic assayCell DeathCancer stem cellStearoyl CoA-desaturaseCancerGeneral Medicinemedicine.diseaseBetulinic acidTriterpenesClone CellsColon cancerTumor resistance030104 developmental biologychemistryBiochemistryCell culture030220 oncology & carcinogenesisCancer treatmentColonic NeoplasmsMutationCancer researchNeoplastic Stem CellsStem cellSettore MED/46 - Scienze Tecniche Di Medicina Di LaboratorioPentacyclic TriterpenesStearoyl-CoA DesaturaseCurrent stem cell research & therapy
researchProduct