Search results for " virus"

showing 10 items of 1897 documents

APOBEC3-mediated restriction of RNA virus replication

2018

AbstractAPOBEC3 family members are cytidine deaminases with roles in intrinsic responses to infection by retroviruses and retrotransposons, and in the control of other DNA viruses, such as herpesviruses, parvoviruses and hepatitis B virus. Although effects of APOBEC3 members on viral DNA have been demonstrated, it is not known whether they edit RNA genomes through cytidine deamination. Here, we investigated APOBEC3-mediated restriction of Coronaviridae. In experiments in vitro, three human APOBEC3 proteins (A3C, A3F and A3H) inhibited HCoV-NL63 infection and limited production of progeny virus, but did not cause hypermutation of the coronaviral genome. APOBEC3-mediated restriction was parti…

0301 basic medicineHepatitis B virusviruseslcsh:MedicineGenome Viralmedicine.disease_causeVirus ReplicationVirusArticleCell LineCytosine Deaminase03 medical and health scienceschemistry.chemical_compoundCytidine deaminationCytidine DeaminasemedicineCoronaviridaeHumansRNA VirusesAPOBEC Deaminaseslcsh:ScienceCoronavirusMultidisciplinarybiology630 Agriculturelcsh:RDNA VirusesRNARNA virusbiochemical phenomena metabolism and nutritionbiology.organism_classificationVirology3. Good health030104 developmental biologyNucleoproteinschemistryViral replicationRNA570 Life sciences; biologylcsh:QDNA
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Failure on voxilaprevir, velpatasvir, sofosbuvir and efficacy of rescue therapy

2021

Background & Aims There are limited data on patients with chronic HCV infection in whom combination voxilaprevir (VOX), velpatasvir (VEL), sofosbuvir (SOF) retreatment fails. Thus, we aimed to assess treatment failure and rescue treatment options in these patients. Methods Samples from 40 patients with HCV genotypes (GT) 1-4 in whom VOX/VEL/SOF retreatment failed were collected within the European Resistance Study Group. Population-based resistance analyses were conducted and clinical parameters and retreatment efficacies were evaluated retrospectively in 22 patients. Results Most VOX/VEL/SOF failure patients were infected with HCV GT3a (n = 18, 45%) or GT1a (n = 11, 28%) and had cirrhosis …

0301 basic medicineHepatitis C Virusmedicine.medical_specialtySofosbuvirVoxilaprevirPopulationresistance-associated substitutionsDirect-acting antiviralVoxilaprevir/velpatasvir/sofosbuvir.GastroenterologySettore MED/07Telaprevir03 medical and health scienceschemistry.chemical_compound0302 clinical medicineVoxilaprevir/Velpatasvir/SofosbuvirInternal medicineBoceprevirRescue therapymedicineResistance-associated substitutioneducationdirect-acting antiviralsDAAeducation.field_of_studyHepatologybusiness.industryvirus diseasesGlecaprevirDAA; HCV; Hepatitis C Virus; Voxilaprevir/Velpatasvir/Sofosbuvir; direct-acting antivirals; rescue therapy; resistance-associated substitutionsdigestive system diseasesPibrentasvirRegimen030104 developmental biologychemistryHCV030211 gastroenterology & hepatologyHepatitis C virubusinessmedicine.drugJournal of Hepatology
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Molecular targets in inhibition of hepatitis C virus replication

1997

Hepatitis C virus (HCV) is the major cause of transfusion-associated hepatitis and accounts for a significant proportion of hepatitis cases worldwide. Most, if not all, infections become persistent and about 60% of cases develop chronic liver disease with various outcomes ranging from an asymptomatic carrier state to chronic active hepatitis and liver cirrhosis, which is strongly associated with the development of hepatocellular carcinoma. Since the initial cloning of the viral genome in 1989, our knowledge of the molecular biology of HCV has increased rapidly and led to the identification of several potential targets for antiviral intervention. In contrast, the low replication of the virus…

0301 basic medicineHepatitisCirrhosisbiologyHepatitis C virus030106 microbiologyGeneral Medicinebiology.organism_classificationmedicine.diseaseChronic liver diseasemedicine.disease_cause01 natural sciencesVirologyVirus0104 chemical sciences010404 medicinal & biomolecular chemistry03 medical and health sciencesFlaviviridaeDrug developmentHepatocellular carcinomaImmunologymedicine
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Anti-herpetic and anti-dengue activity of abietane ferruginol analogues synthesized from (+)-dehydroabietylamine

2015

The abietane-type diterpenoid (+)-ferruginol (1), a bioactive compound isolated from several plants, has attracted much attention as consequence of its pharmacological properties, which includes antibacterial, antifungal, antimicrobial, cardioprotective, anti-oxidative, anti-plasmodial, leishmanicidal, anti-ulcerogenic, anti-inflammatory and antitumor actions. In this study, we report on the antiviral evaluation of ferruginol (1) and several analogues synthesized from commercial (+)-dehydroabietylamine. Thus, the activity against Human Herpesvirus type 1, Human Herpesvirus type 2 and Dengue Virus type 2, was studied. Two ferruginol analogues showed high antiviral selectivity index and reduc…

0301 basic medicineHerpesvirus 2 HumanDehydroabietylamineHerpesvirus 1 HumanMicrobial Sensitivity TestsDengue virusmedicine.disease_causeAntiviral AgentsArticleDengue feverDengueStructure-Activity Relationship03 medical and health scienceschemistry.chemical_compoundDrug DiscoverymedicineHumansStructure–activity relationshipAntiviralFerruginolAbietanePharmacologyDose-Response Relationship DrugMolecular StructureChemistryOrganic ChemistryStereoisomerismGeneral MedicineDengue VirusHerpesmedicine.diseaseAntimicrobialBioactive compoundFerruginol030104 developmental biologyBiochemistryAbietanesAbietaneDiterpeneDiterpeneEuropean Journal of Medicinal Chemistry
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Linear biocompatible glyco-polyamidoamines as dual action mode virus infection inhibitors with potential as broad-spectrum microbicides for sexually …

2016

AbstractThe initial steps of viral infections are mediated by interactions between viral proteins and cellular receptors. Blocking the latter with high-affinity ligands may inhibit infection. DC-SIGN, a C-type lectin receptor expressed by immature dendritic cells and macrophages, mediates human immunodeficiency virus (HIV) infection by recognizing mannose clusters on the HIV-1 gp120 envelope glycoprotein. Mannosylated glycodendrimers act as HIV entry inhibitors thanks to their ability to block this receptor. Previously, an amphoteric, but prevailingly cationic polyamidoamine named AGMA1 proved effective as infection inhibitor for several heparan sulfate proteoglycan-dependent viruses, such …

0301 basic medicineHerpesvirus 2 HumanSexually Transmitted DiseasesMannoseBiocompatible MaterialsHIV Infections010402 general chemistrymedicine.disease_causeAntiviral Agents01 natural sciencesantivirals polymers glyco-conjugates click-chemistry HIV HPVArticleVirus03 medical and health scienceschemistry.chemical_compoundPolyaminesmedicineHumansReceptorchemistry.chemical_classificationHuman papillomavirus 16MultidisciplinarybiologyLectinHeparan sulfateVirology0104 chemical sciencesMolecular WeightMicrobicides for sexually transmitted diseases030104 developmental biologyHerpes simplex viruschemistryHIV-1biology.proteinBiological AssayGlycoproteinMannoseHeLa CellsScientific Reports
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ICTV Virus Taxonomy Profile: Iflaviridae

2017

Iflaviridae is a family of small non-enveloped viruses with monopartite, positive-stranded RNA genomes of approximately 9–11 kilobases. Viruses of all classified species infect arthropod hosts, with the majority infecting insects. Both beneficial and pest insects serve as hosts, and infections can be symptomless (Nilaparvatalugens honeydew virus 1) or cause developmental abnormalities (deformed wing virus), behavioural changes (sacbrood virus) and premature mortality (infectious flacherie virus). The host range has not been examined for most members. The most common route of infection for iflaviruses is the ingestion of virus-contaminated food sources. This is a summary of the Internation…

0301 basic medicineHoneydewInsectavirusesICTV Virus Taxonomy Profile030106 microbiologyInsect VirusesBiologyGenomeHost SpecificityVirus03 medical and health sciencesVirologyDeformed wing virusICTV ReportRNA VirusesAnimalsPhylogenyVirus classificationTaxonomyIflaviridaebiology.organism_classificationVirology3. Good health030104 developmental biologyIflaviridaeTaxonomy (biology)ArthropodInsectJournal of General Virology
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Autophagy interferes with human cytomegalovirus genome replication, morphogenesis, and progeny release.

2020

Viral infections are often accompanied by the induction of autophagy as an intrinsic cellular defense mechanism. Herpesviruses have developed strategies to evade autophagic degradation and to manipulate autophagy of the host cells to their benefit. Here we addressed the role of macroautophagy/autophagy in human cytomegalovirus replication and for particle morphogenesis. We found that proteins of the autophagy machinery localize to cytoplasmic viral assembly compartments and enveloped virions in the cytoplasm. Surprisingly, the autophagy receptor SQSTM1/p62 was also found to colocalize with HCMV capsids in the nucleus of infected cells. This finding indicates that the autophagy machinery int…

0301 basic medicineHuman cytomegalovirusCytoplasmEpstein-Barr Virus InfectionsvirusesCytomegalovirusBiology03 medical and health sciencesMultiplicity of infectionmedicineXenophagyAutophagyMorphogenesisHumansMolecular BiologyCytopathic effect030102 biochemistry & molecular biologyAutophagyCell BiologyBECN1biochemical phenomena metabolism and nutritionFibroblastsmedicine.diseaseVirus ReleaseCell biology030104 developmental biologyCytomegalovirus InfectionsMAP1LC3AResearch PaperAutophagy
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Inhibition of tetraspanin functions impairs human papillomavirus and cytomegalovirus infections

2018

Tetraspanins are suggested to regulate the composition of cell membrane components and control intracellular transport, which leaves them vulnerable to utilization by pathogens such as human papillomaviruses (HPV) and cytomegaloviruses (HCMV) to facilitate host cell entry and subsequent infection. In this study, by means of cellular depletion, the cluster of differentiation (CD) tetraspanins CD9, CD63, and CD151 were found to reduce HPV16 infection in HeLa cells by 50 to 80%. Moreover, we tested recombinant proteins or peptides of specific tetraspanin domains on their effect on the most oncogenic HPV type, HPV16, and HCMV. We found that the C-terminal tails of CD63 and CD151 significantly i…

0301 basic medicineHuman cytomegalovirusMaleTelomeraseTetraspaninsviruses610 MedizinCytomegalovirusIC50virus entrylcsh:ChemistryTetraspanin610 Medical scienceshuman papillomaviruslcsh:QH301-705.5SpectroscopyHuman papillomavirus 16virus diseasesGeneral MedicineBiología y Biomedicina / BiologíaEntry into hostComputer Science ApplicationsCytomegalovirus Infectionsembryonic structuresIC<sub>50</sub>HPV16BiologyCatalysisArticleInorganic Chemistry03 medical and health sciencesInhibitory Concentration 50AntigenViral entrymedicineHumansddc:610Physical and Theoretical ChemistryHumanes PapillomavirusMolecular BiologyCluster of differentiationOrganic ChemistryVirus internalizationCytomegalie-VirusIC 50Human papillomavirus virusesmedicine.diseaseVirologyHaCaT030104 developmental biologytetraspaninlcsh:Biology (General)lcsh:QD1-999human cytomegalovirusPeptidesDDC 610 / Medicine &amp; healthblocking peptideHeLa Cells
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Efficient Delivery of Human Cytomegalovirus T Cell Antigens by Attenuated Sendai Virus Vectors.

2018

ABSTRACT Human cytomegalovirus (HCMV) represents a major cause of clinical complications during pregnancy as well as immunosuppression, and the licensing of a protective HCMV vaccine remains an unmet global need. Here, we designed and validated novel Sendai virus (SeV) vectors delivering the T cell immunogens IE-1 and pp65. To enhance vector safety, we used a replication-deficient strain (rdSeV) that infects target cells in a nonproductive manner while retaining viral gene expression. In this study, we explored the impact that transduction with rdSeV has on human dendritic cells (DCs) by comparing it to the parental, replication-competent Sendai virus strain (rcSeV) as well as the poxvirus …

0301 basic medicineHuman cytomegalovirusModified vaccinia AnkaraT cellmedicine.medical_treatmentvirusesImmunologyGenetic VectorsAlpha interferonCytomegalovirusMice TransgenicMicrobiologySendai virusViral Matrix Proteins03 medical and health sciencesCytomegalovirus VaccinesMiceTransduction GeneticVirologyCricetinaeChlorocebus aethiopsVaccines and Antiviral AgentsmedicineCytotoxic T cellAnimalsHumansAntigens ViralVero Cellsbiologyvirus diseasesImmunotherapymedicine.diseasebiology.organism_classificationPhosphoproteinsVirologySendai virus030104 developmental biologymedicine.anatomical_structureViral replicationInsect ScienceJournal of virology
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ICTV Virus Taxonomy Profile: Pseudoviridae

2021

Pseudoviridae is a family of reverse-transcribing viruses with long terminal repeats (LTRs) belonging to the order Ortervirales. Pseudoviruses are commonly found integrated in the genomes of diverse plants, fungi and animals and are broadly known as Ty1/Copia LTR retrotransposons. Inside the cell, they form icosahedral virus particles, but unlike most other viruses, do not have an extracellular phase. This is a summary of the ICTV Report on the family Pseudoviridae, which is available at ictv.global/report/pseudoviridae.

0301 basic medicineINTRetroelementstaxonomy. Abbreviations: CPvirusesLTR030106 microbiologynucleocapsidRetrotransposonGenome ViralVirus Replicationvirus-like particlesGenomeVirusPRRTPPT03 medical and health sciencestaxonomyVirologyVLPRetrovirusesreverse transcriptaseICTV ReportcapsidRNA VirusesPBSVirus classificationbiologyAnimalfungiTerminal Repeat SequencesPseudoviridaeproteasepolypurine tractbiology.organism_classificationVirologyLong terminal repeatlong terminal repeat030104 developmental biology[SDV.MP.VIR]Life Sciences [q-bio]/Microbiology and Parasitology/VirologyRNA ViralintegraseRHNCIctv Virus Taxonomy Profileribonuclease HPseudoviridaeprimer binding site
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