Search results for " vivo"

showing 10 items of 1661 documents

An inherited deficiency of the third component of complement, C3, in guinea pigs

1986

Hereditary deficiency of the third component of complement, C3, is found very seldom in the human. C3 deficiency is associated with severe bacterial infections revealing the central role of C3 in complement activation via the classical or alternative pathway. We describe a new hereditary C3 deficiency in strain 2 guinea pigs. Serum from these animals had a markedly reduced lytic activity in a standard assay for complement-dependent, antibody-mediated cytotoxicity. In functional assays of individual components, the hemolytic activity of the components C4, C2, C5 and of factors B, D and H was in the normal range. The functional C3 titer, and similarly C3 antigenic activity in the serum of the…

Blood Bactericidal ActivityGuinea PigsImmunologyMacrophage-1 Antigenchemical and pharmacologic phenomenaBiologyHemolysisMajor Histocompatibility ComplexGuinea pigInbred strainAntigenIn vivoAnimalsImmunology and AllergyComplement ActivationRecombination GeneticComplement C3Molecular biologyIn vitroPedigreeReceptors ComplementComplement systemImmunologyAlternative complement pathwaybiology.proteinC3a receptorEuropean Journal of Immunology
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Postnatal overfeeding in rats leads to moderate overweight and to cardiometabolic and oxidative alterations in adulthood.

2011

In contrast to the masses of data on obesity, few data are available concerning the cardiometabolic and oxidative consequences of moderate overweight. The model of postnatal overfeeding (OF) induces an increase in body weight at weaning that remains during adult life. Litters of Wistar rats were either maintained at 12 pups (normal-fed group, NF), or reduced to 3 pups at birth in order to induce OF. At 6 months of age, metabolic parameters, circulating oxidative stress and aortic and coronary vasoreactivity were assessed. Cardiac susceptibility to ischemia-reperfusion injury was also evaluated ex vivo as were markers of cardiac remodeling. OF led to an increase in body weight at weaning (+5…

Blood GlucoseLeptinleft ventricular end-systolic pressuremedicine.medical_treatment030204 cardiovascular system & hematologyOverweight+dP/dtmedicine.disease_causeBiochemistryCardiovascular System0302 clinical medicineOvernutritionHRleft ventricular developed pressureheart rateInsulinhydroperoxidesworking modeComputingMilieux_MISCELLANEOUSmatrix metallo-proteinase-2W0303 health sciencesANOVAMMP-2OFLeptinROOHinternational unitsGeneral MedicineLsuperoxide dismutase[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemleft ventricular maximal pressure developmentFemalemedicine.symptomleft ventricular end-diastolic pressureanalysis of variancemedicine.medical_specialtyLDHNFleft ventricular minimal pressure developmentIschemiaSNPbody mass indexheartReal-Time Polymerase Chain Reactionoxidative stress AchBMI03 medical and health sciences[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemLangendorff modeoverfeedingInternal medicineRLUBKmedicineWeaningAnimalsLVEDPSODRats WistarVentricular remodeling030304 developmental biologyDNA PrimersPostnatal overfeedingBase Sequencebusiness.industryInsulinsodium nitroprussiatelactate dehydrogenaseLVDPLVESPOverweightrelative light unitsmedicine.diseaseacetylcholinearbitrary unitsRatsIUOxidative StressEndocrinology−dP/dtAUnormal-fedbradykininbusinessEx vivoOxidative stressBiochimie
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In vivo triglyceride synthesis in subcutaneous adipose tissue of humans correlates with plasma HDL parameters

2016

Backgrounds and aims: Low concentrations of plasma HDL-C are associated with the development of atherosclerotic cardiovascular diseases and type 2 diabetes. Here we aimed to explore the relationship between the in vivo fractional synthesis of triglycerides (fTG) in subcutaneous (s.q.) abdominal adipose tissue (AT), HDL-C concentrations and HDL particle size composition in non-diabetic humans. Methods: The fTG in s.q. abdominal AT was measured in 16 non-diabetic volunteers (7 women, 9 men; Age: 49 ± 20 years; BMI: 31 ± 5 kg/m; Fasting Plasma Glucose: 90 ± 10 mg/dl) after 2H2O labeling. HDL-C concentration and subclasses, large (L-HDL), intermediate (I-HDL) and small (S-HDL) were measured. Re…

Blood GlucoseMale0301 basic medicinemedicine.medical_specialtyAdipocyte; Atheroprotective; Gender-related; HDL-C metabolism; Mass spectrometry; Stable isotope tracer kinetics; Triglycerides; Cardiology and Cardiovascular MedicineAdipose tissueWhite adipose tissueType 2 diabetes030204 cardiovascular system & hematologyTriglycerideArticleAtheroprotective03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInsulin resistanceIn vivoInternal medicineAdipocytemedicineHumansObesityTriglyceridesAgedHDL-C metabolismAdipocyteStable isotope tracer kineticMass spectrometrybusiness.industryLipogenesisCholesterol HDLnutritional and metabolic diseasesMiddle AgedAtherosclerosismedicine.diseaseLipidsObesitySubcutaneous Fat AbdominalGender-related030104 developmental biologyEndocrinologychemistryLipogenesisFemalelipids (amino acids peptides and proteins)Insulin ResistanceCardiology and Cardiovascular MedicinebusinessAtherosclerosis
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Anti-inflammatory and analgesic activity of a novel inhibitor of microsomal prostaglandin E synthase-1 expression

2009

Abstract In a previous study, we reported a new γ-hydroxybutenolide derivative, 4-benzo[ b ]thiophen-2-yl-3-bromo-5-hydroxy-5 H -furan-2-one (BTH), as inhibitor of microsomal prostaglandin E synthase-1 (mPGES-1) expression in lypopolysaccharide (LPS) stimulated RAW 264.7 and TPH-1 cells, without affecting cyclooxygenase-2 (COX-2). In this study, we evaluated the in vivo effect of BTH on some acute and chronic inflammatory animal models in relation to its inhibitory profile on mPGES-1 expression. In the zymosan-induced mouse air pouch model, BTH produced a dose-dependent inhibition of prostaglandin E 2 (PGE 2 ) production and mPGES-1 protein expression in pouch exudates without any effect on…

Blood PlateletsMaleNeutrophilsmedicine.drug_classmedicine.medical_treatmentAnti-Inflammatory AgentsProstaglandinInflammationThiophenesAcetatesPharmacologyProstaglandin E synthaseLeukotriene B4Gene Expression Regulation EnzymologicAnti-inflammatoryMicechemistry.chemical_compound4-ButyrolactoneIn vivomedicineAnimalsHumansProstaglandin-E SynthasesInflammationPharmacologyAnalgesicsBehavior AnimalbiologyArthritis ExperimentalIntramolecular OxidoreductasesThromboxane B2BiochemistrychemistryHyperalgesiaChronic DiseaseHyperalgesiabiology.proteinCattlelipids (amino acids peptides and proteins)Arachidonic acidmedicine.symptomProstaglandin E
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In-vitro and Ex-vivo Inhibition of Blood Platelet Aggregation by Naftazone

1996

Abstract Because of the considerable interest in the role of platelets and antiplatelet therapy in cardiovascular disease, including the aggregation of platelets to each other during arterial thrombosis and atherogenesis, we have studied the effect of naftazone (Etioven), an original vasculotropic drug on platelet aggregation. Rat and human platelets were prepared and incubated in-vitro with different concentrations of naftazone. We found that naftazone inhibited both platelet secretion and aggregation in platelet-rich plasma (PRP) and washed platelets after stimulation with thrombin or ADP. Rats were also treated intraperitoneally for five days with various naftazone doses (0.125-10 mg kg−…

Blood PlateletsMaleSerotoninPlatelet AggregationPharmaceutical ScienceStimulationIn Vitro TechniquesPharmacologyRats Sprague-DawleyThrombinmedicineAnimalsHumansPlateletTiclopidinePharmacologyAspirinChemistryThrombinBiological activityRatsAdenosine DiphosphateDipyridamoleBiochemistryPlatelet Aggregation InhibitorsEx vivoNaphthoquinonesmedicine.drugJournal of Pharmacy and Pharmacology
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PHOSPHODIESTERASE INHIBITORS PIROXIMONE AND ENOXIMONE INHIBIT PLATELET AGGREGATION IN VIVO AND IN VITRO

1997

The phosphodiesterase type III inhibitors piroximone (PIR) and enoximone (ENO) exert positive inotropic and vasodilating effects in patients with severe heart failure. PIR and ENO raise cyclic AMP levels in cardiac and vascular smooth muscle cells. Platelet activity is also regulated by intracellular levels of cyclic AMP. In this study we have investigated the effects of PIR and ENO on platelet activity in vivo and in vitro. PIR and ENO inhibited ADP induced platelet aggregation in a time- and concentration-dependent manner with IC50-values of 67 +/- 14 mumol/l and 129 +/- 6 mumol/l, respectively. Coincubation of PIR with the adenylate cyclase activator iloprost resulted in a synergistic po…

Blood PlateletsMalemedicine.medical_specialtyCardiotonic AgentsVascular smooth musclePlatelet AggregationPhosphodiesterase InhibitorsVasodilationIn vivoInternal medicineCyclic AMPmedicineAnimalsHumansEnoximonePlateletPlatelet activationRats WistarEnoximonebiologyChemistryImidazolesPhosphodiesteraseHematologyRatsEndocrinologyEnzyme inhibitorbiology.proteinCalciumPlatelet Aggregation Inhibitorsmedicine.drugThrombosis Research
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Tumor-Specific Blood Serum Factors as Basis of Tumor Dormancy

2014

In the present review, we focus on the importance of blood serum factors for tumor growth in vivo. Data from mice experiments indicate the existence of serum factors, which decrease the dormancy of Ehrlich carcinoma cells from 85 to 20%. The impaired production of these factors increases the life span of tumor-bearing animals from 14 days to 120 days. Blocking the production of tumor-specific factors causes the complete regression of already developed Ehrlich carcinoma. These serum factors do not affect the malignant carcinoma cells in vitro. We identified serpins as tumor dormancy serum factors. Experimental evidence suggests that serpins are not only essential for tumor growth. Serpins ar…

Blood serumIn vivomedicine.medical_treatmentRegeneration (biology)ImmunologymedicineCarcinomaAdipose tissueDormancyLiver transplantationBiologymedicine.diseaseBlood proteinsInternational Journal of Biotechnology for Wellness Industries
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Meprin β: A novel regulator of blood–brain barrier integrity

2020

The metalloprotease meprin β (Mep1b) is capable of cleaving cell-adhesion molecules in different tissues (e.g. skin, kidney and intestine) and is dysregulated in several diseases associated with barrier breakdown (Alzheimer´s disease, kidney disruption, inflammatory bowel disease). In this study, we demonstrate that Mep1b is a novel regulator of tight junction (TJ) composition and blood–brain barrier (BBB) integrity in brain endothelium. In Mep1b-transfected mouse brain endothelial cells (bEnd.3), we observed a reduction of the TJ protein claudin-5, decreased transendothelial electrical resistance (TEER) and an elevated permeability to paracellular diffusion marker [14C]-inulin. Analysis o…

Blood–brain barrierOccludinMice03 medical and health sciences0302 clinical medicineCerebrospinal fluidIn vivomedicineAnimalsHumans030304 developmental biology0303 health sciencesMetalloproteinaseKidneyTight Junction ProteinsTight junctionChemistryBrainEndothelial CellsMetalloendopeptidasesOriginal ArticlesCell biologymedicine.anatomical_structureNeurologyBlood-Brain BarrierParacellular transportNeurology (clinical)Cardiology and Cardiovascular Medicine030217 neurology & neurosurgeryJournal of Cerebral Blood Flow & Metabolism
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Involvement of the Pseudomonas aeruginosa MexAB–OprM efflux pump in the secretion of the metallophore pseudopaline

2020

ABSTRACTThe ability for all organisms to acquire metals from their environment is essential for life. To overcome the metal restriction imposed by the host’s nutritional immunity, bacterial pathogens exploits the use of small high metal affinity molecules called metallophores. Metallophores are first synthetized in the cytoplasm, then secreted into the medium where they sequester the metal. The metal-metallophore complex is then imported into the bacterium following binding to dedicated cell surface receptors. Recently, a new family of metallophores has been discovered in pathogenic bacteria called staphylopine in Staphylococcus aureus and pseudopaline in Pseudomonas aeruginosa. Here, we ar…

Bodily Secretions[SDV]Life Sciences [q-bio]Microbial Sensitivity TestsBiologymedicine.disease_causeMicrobiology03 medical and health sciencesBacterial ProteinsIn vivoDrug Resistance Multiple BacterialpseudopalinemedicineInner membraneSecretionMolecular Biology030304 developmental biology0303 health sciencesMexAB–OprMBacteriametallophoreChemistry030306 microbiologyPseudomonas aeruginosaMembrane Transport Proteinsbiology.organism_classificationIn vitroCell biology[SDV.MP]Life Sciences [q-bio]/Microbiology and ParasitologyCytoplasmPseudomonas aeruginosaefflux pumpEffluxCell envelopeBacterial outer membraneOligopeptidesBacteriaBacterial Outer Membrane Proteins
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Osseous response on linear and cyclic RGD-peptides immobilized on titanium surfaces in vitro and in vivo

2017

Biomimetic surface modifications of titanium implants using the Arg-Gly-Asp-sequence (RGD) are promising to accelerate bone healing in cases of medical implants. Therefore, we compared the impact of linear and cyclic RGD (l- and c-RGD) covalently coupled onto titanium surfaces on the osseous response in vitro and in vivo. In vitro, osteoblasts' behaviour on different surfaces (unmodified, amino-silanized (APTES), l- and c-RGD) was analysed regarding adhesion (fluorescence microscopy), proliferation (resazurin stain) and differentiation (RT-PCR on alkaline phosphatase (AP) & osteocalcin (OC)). In vivo, osteosynthesis screws (unmodified n=8, l-RGD n=8, c-RGD n=8) were inserted into the proxim…

Bone growthMaterials sciencebiologyMetals and AlloysBiomedical Engineering030206 dentistry02 engineering and technologyBone healingAdhesion021001 nanoscience & nanotechnologyIn vitroBiomaterials03 medical and health sciences0302 clinical medicineIn vivoCeramics and CompositesFluorescence microscopeOsteocalcinbiology.proteinAlkaline phosphatase0210 nano-technologyBiomedical engineeringJournal of Biomedical Materials Research Part A
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