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showing 10 items of 5782 documents

Reducing the initial number of rituximab maintenance-therapy infusions for ANCA-associated vasculitides: randomized-trial post-hoc analysis

2020

AbstractObjectiveThe randomized, controlled MAINRITSAN2 trial was designed to compare the capacity of an individually tailored therapy [randomization day 0 (D0)], with reinfusion only when CD19+ lymphocytes or ANCA had reappeared, or if the latter’s titre rose markedly, with that of five fixed-schedule 500-mg rituximab infusions [D0 + D14, then months (M) 6, 12 and 18] to maintain ANCA-associated vasculitis (AAV) remissions. Relapse rates did not differ at M28. This ancillary study was undertaken to evaluate the effect of omitting the D14 rituximab infusion on AAV relapse rates at M12.MethodsMAINRITSAN2 trial data were subjected to post-hoc analyses of M3, M6, M9 and M12 relapse-free surviv…

medicine.medical_specialtyRandomizationAntigens CD19Anti-Neutrophil Cytoplasmic Antibody-Associated VasculitisGastroenterologyDisease-Free SurvivalDrug Administration ScheduleAntibodies Antineutrophil CytoplasmicMaintenance Chemotherapylaw.invention03 medical and health sciences0302 clinical medicineRheumatologyMaintenance therapyRandomized controlled triallawInternal medicinePost-hoc analysismedicineHumansPharmacology (medical)030212 general & internal medicineSurvival rate030203 arthritis & rheumatologybusiness.industrymedicine.diseaseAntirheumatic AgentsRituximabRituximabMicroscopic polyangiitisGranulomatosis with polyangiitisbusinessmedicine.drugRheumatology
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Role of S-adenosyl-L-methionine in the treatment of intrahepatic cholestasis.

1990

Recent studies have established the clinical efficacy of S-adenosyl-L-methionine (SAMe) in the treatment of cholestasis associated with hepatic diseases, pregnancy and the administration of estrogen-containing oral contraceptives. In 4 clinical trials involving a total of 639 patients with cholestasis due to acute or chronic liver disease, SAMe in an intravenous dose of 800 mg/day or an oral regimen of 1.6 g/day for 2 weeks was superior to placebo in relieving the symptom of pruritus and in restoring serum total bilirubin and serum alkaline phosphatase towards normal. The drug is also effective in intrahepatic cholestasis of pregnancy (ICP), with intravenous administration of 800 mg/day for…

medicine.medical_specialtyS-Adenosylmethioninemedicine.drug_classmedicine.medical_treatmentCholestasis IntrahepaticPharmacologyChronic liver diseasePlaceboBile Acids and SaltsCholestasisPregnancyInternal medicinemedicineAnimalsHumansPharmacology (medical)ChlorpromazineChemotherapybusiness.industryBilirubinmedicine.diseasePregnancy ComplicationsEndocrinologymedicine.anatomical_structure1-NaphthylisothiocyanateEstrogenHepatocyteFemalebusinessCholestasis of pregnancymedicine.drugDrugs
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Modulation of hippocampal acetylcholine release after fimbria-fornix lesions and septal transplantation in rats

1997

Abstract Female Long–Evans rats sustained electrolytic lesions of the fimbria and the dorsal fornix causing a partial lesion of the septohippocampal pathway. Two weeks later, the rats received intra-hippocampal grafts of fetal septal cell suspensions. Nine to twelve months later, the release of acetylcholine (ACh) in the hippocampus of sham-operated, lesion-only and grafted rats was measured by microdialysis. The extent of cholinergic (re)innervation was determined by acetylcholinesterase (AChE) staining and densitometry. In both lesion-only and grafted rats, the ratio of ACh release to AChE staining intensity was increased as compared to sham-operated rats, indicating a loss of endogenous …

medicine.medical_specialtySciences du Vivant [q-bio]/Neurosciences [q-bio.NC]Microdialysis[SDV]Life Sciences [q-bio][SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyScopolamineMuscarinic AntagonistsHippocampal formationBiologySerotonergicHippocampus03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicineNeural PathwaysmedicineAnimalsBrain Tissue TransplantationCholinergic neuronNeurotransmitterComputingMilieux_MISCELLANEOUS030304 developmental biology0303 health sciences8-Hydroxy-2-(di-n-propylamino)tetralinGeneral NeuroscienceFornixMuscarinic antagonistRats Inbred StrainsAcetylcholineRatsEndocrinologychemistryCholinergic FibersAnesthesiaReceptors SerotoninCholinergicRaphe NucleiFemaleSeptal Nuclei[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Receptors Serotonin 5-HT1030217 neurology & neurosurgeryAcetylcholinemedicine.drug
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High Intestinal Cholesterol Absorption Is Associated With Cardiovascular Disease and Risk Alleles in ABCG8 and ABO

2013

Objectives This study sought to determine whether high intestinal cholesterol absorption represents a cardiovascular risk factor and to link ABCG8 and ABO variants to cardiovascular disease (CVD). Background Plant sterol–enriched functional foods are widely used for cholesterol lowering. Their regular intake yields a 2-fold increase in circulating plant sterol levels that equally represent markers of cholesterol absorption. Variants in ABCG8 and ABO have been associated with circulating plant sterol levels and CVD, thereby suggesting atherogenic effects of plant sterols or of cholesterol uptake. Methods The cholestanol-to-cholesterol ratio (CR) was used as an estimate of cholesterol absorpt…

medicine.medical_specialtySingle-nucleotide polymorphism030204 cardiovascular system & hematologyHigh cholesterolIntestinal absorption03 medical and health scienceschemistry.chemical_compound0302 clinical medicineEzetimibeInternal medicineABO blood group systemmedicineRisk factor030304 developmental biology2. Zero hunger0303 health sciencesCholesterolbusiness.industrymedicine.diseaseEndocrinologychemistryIntestinal cholesterol absorptionlipids (amino acids peptides and proteins)businessCardiology and Cardiovascular Medicinemedicine.drugJournal of the American College of Cardiology
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Uncoupling Protein 2 as genetic risk factor for systemic lupus erythematosus: association with malondialdehyde levels and intima media thickness

2020

BACKGROUND Increased oxidative stress potentially leads to accelerated atherosclerosis and, consequently, cardiovascular diseases, the main cause of death in systemic lupus erythematous (SLE). To gain insight into these mechanisms, we studied the association of uncoupling protein (UCP) 2 genetic variants, gene involved in the mitochondrial production of reactive oxygen species, and oxidative stress with SLE and the presence of atherosclerosis. METHODS Genetic analysis of the UCP2 -866G/A and UCP2 Ins/Del polymorphisms was performed in 45 SLE patients and 36 healthy controls by RFLP-PCR. Oxidation status was determined by measuring malondialdehyde (MDA) levels. Presence of subclinical athero…

medicine.medical_specialtySingle-nucleotide polymorphism030204 cardiovascular system & hematologymedicine.disease_causeCarotid Intima-Media Thickness03 medical and health scienceschemistry.chemical_compound0302 clinical medicineGeneticRisk FactorsMalondialdehydeInternal medicineGenotypemedicineHumansUncoupling proteinUncoupling Protein 2030212 general & internal medicineAlleleskin and connective tissue diseaseschemistry.chemical_classificationReactive oxygen speciesbusiness.industryMalondialdehydeLupus erythematosus systemicEndocrinologychemistryIntima-media thicknessCardiology and Cardiovascular MedicinebusinessCardiovascular diseases.Oxidative stressMinerva Cardioangiologica
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AB0427 Clinical and laboratory findings in patients with late-onset sle and correlations with il6 concentrations

2013

Background Systemic lupus erythematosus (SLE) is an autoimmune rheumatic disease that usually develops in women aged 18-50 years. It is known that age at onset modifies the clinical manifestations of SLE, and so the elderly may form a specific patient subgroup. It is now well established that the serum levels of the cytokines interleukin (IL) 6 and IL10 are increased in patients with SLE (1). Objectives The primary aim was to compare the type of clinical involvement and autoantibodies in patients with late-onset (LO) or early-onset (EO) SLE. The second aim was to compare IL6 levels in the two patient groups and their possible correlations with clinical and immunological manifestations. Meth…

medicine.medical_specialtySystemic lupus erythematosusAnti-nuclear antibodybusiness.industryImmunologyAutoantibodyArthritismedicine.diseaseGastroenterologyGeneral Biochemistry Genetics and Molecular BiologyRheumatologyRheumatologyimmune system diseasesInternal medicineImmunologymedicineImmunology and AllergyAge of onsetmedicine.symptomskin and connective tissue diseasesMalar rashbusinessSerositisAnnals of the Rheumatic Diseases
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Integrative transnational analysis to dissect tuberculosis transmission events along the migratory route from Africa to Europe

2021

páginas, 4 figuras, 3 tablas

medicine.medical_specialtyTuberculosisGenotypeDisease clusterlaw.inventionMycobacterium tuberculosis03 medical and health sciences0302 clinical medicinelawEpidemiologyTrans-nationalMedicineCluster AnalysisHumansTuberculosisTransmission030212 general & internal medicine0303 health sciencesbiology030306 microbiologybusiness.industryStrain (biology)transmissionGeneral MedicineMycobacterium tuberculosisbiology.organism_classificationmedicine.diseasetrans-nationalEuropeTransmission (mechanics)Horn of AfricaAfricabusinessDemography
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Vasoactive intestinal peptide stimulation of cyclic guanosine monophosphate formation: further evidence for a role of nitric oxide synthase and cytos…

1993

In the rat pineal gland vasoactive intestinal peptide (VIP) and beta-adrenergic agonists stimulate cyclic guanosine monophosphate (cGMP) formation and their action is amplified by alpha 1-adrenergic agonists. Since beta-adrenergic stimulation of cGMP is suggested to involve activation of nitric oxide (NO) synthase and NO-mediated activation of cytosolic guanylate cyclase (GC), we investigated the effects of the NO synthase inhibitor N-monomethyl-L-arginine (L-NMMA) and of the cytosolic GC inhibitor methylene blue (MB) on VIP receptor-stimulated cGMP formation. Both L-NMMA and MB depressed VIP-induced cGMP formation as well as alpha 1-adrenergic potentiation of VIP-stimulated cGMP formation …

medicine.medical_specialtyVasoactive intestinal peptideArgininePineal GlandPinealocyteNitric oxidechemistry.chemical_compoundPhenylephrineEndocrinologyCytosolInternal medicinemedicineAnimalsCyclic guanosine monophosphateCyclic GMPomega-N-MethylarginineATP synthasebiologyDrug SynergismRatsNitric oxide synthaseMethylene BlueEndocrinologychemistryGuanylate CyclaseSecond messenger systembiology.proteinOmega-N-MethylarginineAmino Acid OxidoreductasesNitric Oxide Synthasehormones hormone substitutes and hormone antagonistsVasoactive Intestinal PeptideEndocrinology
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Differential effects of potassium channel blockers on neurohypophysial release of oxytocin and vasopressin. Evidence for frequency-dependent interact…

1988

Isolated rat neurohypophyses were fixed by their stalks to a platinum wire electrode and superfused with Krebs-HEPES solution. Vasopressin and oxytocin released into the medium were determined by specific radioimmunoassays. Hormone secretion was increased by electrical stimulation of the pituitary stalk at different frequencies. The effects of several potassium channel blockers, tetraethylammonium (TEA) ions, 4-aminopyridine (4-AP) and 3,4-diaminopyridine (3,4-DAP) were tested. The release of vasopressin and oxytocin evoked by electrical stimulation with 900 pulses at 15 Hz (about 900 and 1,000 μU, respectively) was about 10 times higher than that evoked by 900 pulses at 3 Hz. Both 10 and 3…

medicine.medical_specialtyVasopressinCromakalimPotassium ChannelsVasopressinsRadioimmunoassayNeuropeptideAminopyridinesStimulation(+)-NaloxoneOxytocinPituitary Gland PosteriorInternal medicinemedicineAnimalsBenzopyransPyrroles4-AminopyridineEndogenous opioidPharmacologyChemistryNaloxoneTetraethylammoniumPotassium channel blockerRats Inbred StrainsGeneral MedicineTetraethylammonium CompoundsPotassium channelElectric StimulationRatsEndocrinologyOxytocinFemaleEndorphinsAmifampridinehormones hormone substitutes and hormone antagonistsmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Signaling through BMPR-IA regulates quiescence and long-term activity of neural stem cells in the adult hippocampus.

2010

SummaryNeural stem cells (NSCs) in the adult hippocampus divide infrequently, and the molecules that modulate their quiescence are largely unknown. Here, we show that bone morphogenetic protein (BMP) signaling is active in hippocampal NSCs, downstream of BMPR-IA. BMPs reversibly diminish proliferation of cultured NSCs while maintaining their undifferentiated state. In vivo, acute blockade of BMP signaling in the hippocampus by intracerebral infusion of Noggin first recruits quiescent NSCs into the cycle and increases neurogenesis; subsequently, it leads to decreased stem cell division and depletion of precursors and newborn neurons. Consistently, selective ablation of Bmpr1a in hippocampal …

medicine.medical_specialtyanimal structuresGenetic VectorsHippocampal formationBiologyBone morphogenetic proteinHippocampusModels BiologicalMOLNEUROCell LineMiceNeural Stem CellsInternal medicineGeneticsmedicineAnimalsHumansNogginBone Morphogenetic Protein Receptors Type ICells Culturedreproductive and urinary physiologySmad4 ProteinNeuronsReverse Transcriptase Polymerase Chain ReactionStem CellsCell CycleLentivirusNeurogenesisCentral-nervous-system; Bone morphogenetic protein; Dentate gyrus; Progenitor cells; Neurogenesis; Expression; Receptor; Noggin; Brain; DifferentiationCell BiologyFlow CytometrySTEMCELLRats Inbred F344BMPR1ANeural stem cellRatsCell biologyEndocrinologyStem cell divisionnervous systemembryonic structuresMolecular MedicineStem cellbiological phenomena cell phenomena and immunityCarrier ProteinsSignal Transduction
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