Search results for "030302 biochemistry & molecular biology"

showing 10 items of 238 documents

Cis autocatalytic cleavage of glycine-linked Zika virus NS2B-NS3 protease constructs.

2019

The flaviviral heterodimeric serine protease NS2B-NS3, consisting of the NS3 protease domain and the NS2B co-factor, is essential for ZIKA virus maturation and replication in cells. For in vitro studies a 'linked' construct, where a polyglycine linker connects NS2BCF and NS3pro , is often used. This construct undergoes autocatalytic cleavage. Here, we show that linked ZIKV NS2BCF -NS3pro is cleaved in cis in the NS2BCF exclusively at position R95 and not at the previously proposed alternate cleavage site at residue R29 in the NS3pro . Cleavage neither affects protease stability nor activity, despite some observed differences in spectroscopic behavior. This minimally modified construct may t…

Models MolecularProtein Conformationmedicine.medical_treatmentBiophysicsViral Nonstructural ProteinsCleavage (embryo)ArginineVirus ReplicationBiochemistryCatalysisZika virus03 medical and health sciencesViral ProteinsStructural BiologyGeneticsmedicineHomeostasisMolecular Biology030304 developmental biologySerine protease0303 health sciencesNS3ProteasebiologyChemistryCircular Dichroism030302 biochemistry & molecular biologySerine EndopeptidasesCell BiologyZika Virusbiology.organism_classificationIn vitroRecombinant ProteinsFlavivirusSpectrometry FluorescenceBiochemistrybiology.proteinProtein MultimerizationPeptidesLinkerPeptide HydrolasesFEBS lettersReferences
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Protein structure prediction assisted with sparse NMR data in CASP13

2019

CASP13 has investigated the impact of sparse NMR data on the accuracy of protein structure prediction. NOESY and 15 N-1 H residual dipolar coupling data, typical of that obtained for 15 N,13 C-enriched, perdeuterated proteins up to about 40 kDa, were simulated for 11 CASP13 targets ranging in size from 80 to 326 residues. For several targets, two prediction groups generated models that are more accurate than those produced using baseline methods. Real NMR data collected for a de novo designed protein were also provided to predictors, including one data set in which only backbone resonance assignments were available. Some NMR-assisted prediction groups also did very well with these data. CAS…

Models MolecularProtein FoldingMagnetic Resonance SpectroscopyProtein ConformationComputer scienceCrystallography X-RayBiochemistryArticle03 medical and health sciencesProtein structureStructural BiologyComputer SimulationCASPMolecular Biology030304 developmental biology0303 health sciences030302 biochemistry & molecular biologyProteinsReproducibility of ResultsRangingProtein structure predictionNmr dataData setResidual dipolar couplingTwo-dimensional nuclear magnetic resonance spectroscopyAlgorithmAlgorithmsProteins: Structure, Function, and Bioinformatics
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Structural Characterization of Set1 RNA Recognition Motifs and their Role in Histone H3 Lysine 4 Methylation

2006

Departament de Bioquimica iBiologia Molecular, Universitatde Valencia, C/Dr Moliner 50,46100, Burjassot, SpainThe yeast Set1 histone H3 lysine 4 (H3K4) methyltransferase contains, inaddition to its catalytic SET domain, a conserved RNA recognition motif(RRM1). We present here the crystal structure and the secondary structureassignment in solution of the Set1 RRM1. Although RRM1 has the expectedβαββαβ RRM-fold, it lacks the typical RNA-binding features of thesemodules. RRM1 is not able to bind RNA by itself in vitro, but a constructcombining RRM1 with a newly identified downstream RRM2 specificallybinds RNA. Invivo,H3K4 methylation isnot affectedbyapoint mutation inRRM2 that preserves Set1 s…

Models MolecularRiboswitchHistone H3 Lysine 4Saccharomyces cerevisiae ProteinsRNA-induced transcriptional silencingSurface Properties[SDV]Life Sciences [q-bio]Molecular Sequence DataSaccharomyces cerevisiae[SDV.BC.BC]Life Sciences [q-bio]/Cellular Biology/Subcellular Processes [q-bio.SC]BiologyMethylationHistonesStructure-Activity Relationship03 medical and health sciencesStructural BiologyHistone methylation[SDV.BC.BC] Life Sciences [q-bio]/Cellular Biology/Subcellular Processes [q-bio.SC]Amino Acid SequenceProtein Structure QuaternaryMolecular BiologyConserved Sequence030304 developmental biology0303 health sciencesRNA recognition motifLysine030302 biochemistry & molecular biologyRNARNA FungalHistone-Lysine N-MethyltransferaseNon-coding RNAMolecular biology[SDV] Life Sciences [q-bio]DNA-Binding ProteinsProtein SubunitsBiochemistryHistone methyltransferaseSequence AlignmentProtein BindingTranscription Factors
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Antibody inhibition of the transcriptase activity of the rotavirus DLP: a structural view.

2001

On entering the host cell the rotavirus virion loses its outer shell to become a double-layered particle (DLP). The DLP then transcribes the 11 segments of its dsRNA genome using its own transcriptase complex, and the mature mRNA emerges along the 5-fold axis. In order to better understand the transcription mechanism and the role of VP6 in transcription we have studied three monoclonal antibodies against VP6: RV-238 which inhibits the transcriptase activity of the DLP; and RV-133 and RV-138 which have no effect on transcription. The structures obtained by cryo-electron microscopy of the DLP/Fab complexes and by X-ray crystallography of the VP6 trimer and the VP6/Fab-238 complex have been co…

Models MolecularRotavirusConformational changeSTRUCTUREMature messenger RNAmedicine.drug_classProtein ConformationvirusesBiologyMonoclonal antibodyAntibodies ViralCrystallography X-RayEpitope03 medical and health sciencesEpitopesImmunoglobulin Fab FragmentsCapsidStructural BiologyTranscription (biology)medicine[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologyCRISTALLOGRAPHIE[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyRNA MessengerMolecular BiologyAntigens Viral030304 developmental biology0303 health sciencesMessenger RNA030302 biochemistry & molecular biologyCryoelectron Microscopyvirus diseasesRNADNA-Directed RNA PolymerasesMolecular biologyReverse transcriptase3. Good healthVIROLOGIECapsid ProteinsJournal of molecular biology
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Nucleoside Analogue Mutagenesis of a Single-Stranded DNA Virus: Evolution and Resistance

2012

ABSTRACT It has been well established that chemical mutagenesis has adverse fitness effects in RNA viruses, often leading to population extinction. This is mainly a consequence of the high RNA virus spontaneous mutation rates, which situate them close to the extinction threshold. Single-stranded DNA viruses are the fastest-mutating DNA-based systems, with per-nucleotide mutation rates close to those of some RNA viruses, but chemical mutagenesis has been much less studied in this type of viruses. Here, we serially passaged bacteriophage ϕX174 in the presence of the nucleoside analogue 5-fluorouracil (5-FU). We found that 5-FU was unable to trigger population extinction for the range of conce…

Mutation rateGenes ViralImmunologyBiologyMicrobiology03 medical and health scienceschemistry.chemical_compoundTranscription (biology)VirologyDrug Resistance ViralGenePolymerase030304 developmental biologyGenetics0303 health sciences030302 biochemistry & molecular biologyRNARNA virusDNAbiology.organism_classificationVirology3. Good healthGenetic Diversity and EvolutionchemistryInsect ScienceSingle Stranded DNA VirusMutagenesis Site-Directedbiology.proteinFluorouracilDirected Molecular EvolutionBacteriophage phi X 174DNAJournal of Virology
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Delayed lysis confers resistance to the nucleoside analogue 5-fluorouracil and alleviates mutation accumulation in the single-stranded DNA bacterioph…

2014

ABSTRACT Rates of spontaneous mutation determine viral fitness and adaptability. In RNA viruses, treatment with mutagenic nucleoside analogues selects for polymerase variants with increased fidelity, showing that viral mutation rates can be adjusted in response to imposed selective pressures. However, this type of resistance is not possible in viruses that do not encode their own polymerases, such as single-stranded DNA viruses. We previously showed that serial passaging of bacteriophage ϕX174 in the presence of the nucleoside analogue 5-fluorouracil (5-FU) favored substitutions in the lysis protein E (P. Domingo-Calap, M. Pereira-Gomez, and R. Sanjuán, J. Virol. 86: 9640–9646, 2012, doi:10…

Mutation rateImmunologyAdaptation BiologicalMutation MissenseDNA Single-Strandedmedicine.disease_causeMicrobiologyBacteriophage03 medical and health scienceschemistry.chemical_compoundViral ProteinsBacteriolysisMutation RateVirologymedicineBacteriophagesSelection GeneticPolymerase030304 developmental biologyGenetics0303 health sciencesMutationbiology030302 biochemistry & molecular biologyMutagenesisMutation AccumulationResistance mutationbiology.organism_classificationVirologychemistryGenetic Diversity and EvolutionInsect ScienceDNA Viralbiology.proteinMutant ProteinsFluorouracilDNAJournal of virology
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Correlation between mutation rate and genome size in riboviruses: mutation rate of bacteriophage Qβ.

2013

Abstract Genome sizes and mutation rates covary across all domains of life. In unicellular organisms and DNA viruses, they show an inverse relationship known as Drake’s rule. However, it is still unclear whether a similar relationship exists between genome sizes and mutation rates in RNA genomes. Coronaviruses, the RNA viruses with the largest genomes (∼30 kb), encode a proofreading 3′ exonuclease that allows them to increase replication fidelity. However, it is unknown whether, conversely, the RNA viruses with the smallest genomes tend to show particularly high mutation rates. To test this, we measured the mutation rate of bacteriophage Qβ, a 4.2-kb levivirus. Amber reversion-based Luria–D…

Mutation rate[SDE.MCG]Environmental Sciences/Global ChangesMutantGenome ViralInvestigationsGenomeEvolution Molecular03 medical and health scienceschemistry.chemical_compound[SDV.EE.ECO]Life Sciences [q-bio]/Ecology environment/EcosystemsGenome SizeMutation Rate[SDV.MHEP.MI]Life Sciences [q-bio]/Human health and pathology/Infectious diseasesGeneticsEscherichia coliGenome sizeComputingMilieux_MISCELLANEOUS030304 developmental biologyGenetics[SDV.EE.SANT]Life Sciences [q-bio]/Ecology environment/HealthAllolevivirus0303 health sciences[SDV.MHEP.ME]Life Sciences [q-bio]/Human health and pathology/Emerging diseasesbiology030302 biochemistry & molecular biologyRNAbiology.organism_classification[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriology3. Good healthchemistry[SDV.MP.VIR]Life Sciences [q-bio]/Microbiology and Parasitology/VirologyProofreading[SDE.BE]Environmental Sciences/Biodiversity and EcologyBacteriophage QβDNAGenetics
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Variability in the mutation rates of RNA viruses

2014

ABSTRACT:  It is well established that RNA viruses show extremely high mutation rates, but less attention has been paid to the fact that their mutation rates also vary strongly, from 10-6 to 10-4 substitutions per nucleotide per cell infection. The causes explaining this variability are still poorly understood, but candidate factors are the viral genome size and polarity, host-specific gene expression patterns, or the intracellular environment. Differences between animal and plant viruses, or between arthropod-borne and directly transmitted viruses have also been postulated. Finally, RNA viruses may be able to regulate the rate at which new mutations spread in the population by modifying f…

Mutation rate[SDE.MCG]Environmental Sciences/Global ChangesPopulationBiology03 medical and health sciences[SDV.EE.ECO]Life Sciences [q-bio]/Ecology environment/Ecosystems[SDV.MHEP.MI]Life Sciences [q-bio]/Human health and pathology/Infectious diseasesVirologyPlant virusGene expressioneducationGenome sizeComputingMilieux_MISCELLANEOUS030304 developmental biologyGenetics[SDV.EE.SANT]Life Sciences [q-bio]/Ecology environment/Health0303 health scienceseducation.field_of_study[SDV.MHEP.ME]Life Sciences [q-bio]/Human health and pathology/Emerging diseases030302 biochemistry & molecular biologyRNAVirology[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriology3. Good healthViral replicationViral evolution[SDV.MP.VIR]Life Sciences [q-bio]/Microbiology and Parasitology/Virology[SDE.BE]Environmental Sciences/Biodiversity and Ecology
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Effect of mismatch repair on the mutation rate of bacteriophage ϕX174

2015

Viral mutation rates vary widely in nature, yet the mechanistic and evolutionary determinants of this variability remain unclear. Small DNA viruses mutate orders of magnitude faster than their hosts despite using host-encoded polymerases for replication, which suggests these viruses may avoid post-replicative repair. Supporting this, the genome of bacteriophage ϕX174 is completely devoid of GATC sequence motifs, which are required for methyl-directed mismatch repair in Escherichia coli . Here, we show that restoration of the randomly expected number of GATC sites leads to an eightfold reduction in the rate of spontaneous mutation of the phage, without severely impairing its replicative capa…

Mutation ratemutation rateBase analogBiologymedicine.disease_causeMicrobiologyGenomeBacteriophage03 medical and health scienceschemistry.chemical_compoundVirologyevolutionmedicinestress-induced mutagenesisEscherichia coli030304 developmental biologyGeneticsbacteriophage ϕX1740303 health sciencesmethyl-directed mismatch repair030302 biochemistry & molecular biologyMutagenesisbiology.organism_classificationchemistryDNA mismatch repairDNAResearch ArticleVirus Evolution
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The external domains of the HIV-1 envelope are a mutational cold spot

2015

In RNA viruses, mutations occur fast and have large fitness effects. While this affords remarkable adaptability, it can also endanger viral survival due to the accumulation of deleterious mutations. How RNA viruses reconcile these two opposed facets of mutation is still unknown. Here we show that, in human immunodeficiency virus (HIV-1), spontaneous mutations are not randomly located along the viral genome. We find that the viral mutation rate experiences a threefold reduction in the region encoding the most external domains of the viral envelope, which are strongly targeted by neutralizing antibodies. This contrasts with the hypermutation mechanisms deployed by other, more slowly mutating …

Mutation ratevirusesGeneral Physics and AstronomyHIV InfectionsBiologymedicine.disease_causeArticleGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesCytidine deaminationMutation RateViral Envelope ProteinsViral envelopeViral entrymedicineViral structural proteinHumans030304 developmental biologyGenetics0303 health sciencesMutationMultidisciplinary030302 biochemistry & molecular biologyRNAGeneral ChemistryVirologyProtein Structure Tertiary3. Good healthViral evolutionHIV-1Nature Communications
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