Search results for "13b"

showing 10 items of 10 documents

Cohen Syndrome-Associated Cataract Is Explained by VPS13B Functions in Lens Homeostasis and Is Modified by Additional Genetic Factors

2020

International audience; Purpose: Cohen syndrome (CS) is a rare genetic disorder caused by variants of the VPS13B gene. CS patients are affected with a severe form of retinal dystrophy, and in several cases cataracts also develop. The purpose of this study was to investigate the mechanisms and risk factors for cataract in CS, as well as to report on cataract surgeries in CS patients.Methods: To understand how VPS13B is associated with visual impairments in CS, we generated the Vps13b∆Ex3/∆Ex3 mouse model. Mice from 1 to 3 months of age were followed by ophthalmoscopy and slit-lamp examinations. Phenotypes were investigated by histology, immunohistochemistry, and western blot. Literature anal…

0301 basic medicinegenetic structuresDevelopmental DisabilitiesVesicular Transport Proteins030105 genetics & hereditysurgerygenetic backgroundchemistry.chemical_compoundLensMyopiaHomeostasisMice KnockoutCohen syndrome[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologymedicine.diagnostic_testRetinal DegenerationGenetic disorderinflamma- tionVPS13BcataractKnockout mouseMicrocephalyMuscle Hypotoniamedicine.medical_specialtymouse modelBlotting WesternRetinitisFingersOphthalmoscopy03 medical and health sciencesCataractsIntellectual DisabilityOphthalmologyVPS13BLens CrystallinemedicineAnimalsObesityCohen syndromebusiness.industryfibrosisRetinalgenetic modifiersmedicine.diseaseeye diseasesMice Inbred C57BLDisease Models Animalophthalmology030104 developmental biologyGene Expression RegulationchemistryinflammationRNAsense organsbusiness[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyInvestigative Ophthalmology & Visual Science
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Congenital neutropenia with retinopathy, a new phenotype without intellectual deficiency or obesity secondary toVPS13Bmutations

2013

Over one hundred VPS13B mutations are reported in Cohen syndrome (CS). Most cases exhibit a homogeneous phenotype that includes intellectual deficiency (ID), microcephaly, facial dysmorphism, slender extremities, truncal obesity, progressive chorioretinal dystrophy, and neutropenia. We report on a patient carrying two VPS13B splicing mutations with an atypical phenotype that included microcephaly, retinopathy, and congenital neutropenia, but neither obesity nor ID. RNA analysis of the IVS34+2T_+3AinsT mutation did not reveal any abnormal splice fragments but mRNA quantification showed a significant decrease in VPS13B expression. RNA sequencing analysis up- and downstream from the IVS57+2T>C…

AdultPathologymedicine.medical_specialtyMicrocephalyNeutropeniaDNA Mutational AnalysisVesicular Transport ProteinsNeutropeniamedicine.disease_causeRetinal DiseasesIntellectual DisabilityGene OrderGeneticsmedicineCongenital Bone Marrow Failure SyndromesHumansObesityCongenital NeutropeniaGenetics (clinical)GeneticsMutationCohen syndromebusiness.industryFaciesSyndromemedicine.diseasePhenotypePedigreeVPS13BPhenotypeMutationFemalebusinessRetinopathyAmerican Journal of Medical Genetics Part A
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Cohen syndrome is associated with major glycosylation defects

2014

International audience; Cohen syndrome (CS) is a rare autosomal recessive disorder with multisytemic clinical features due to mutations in the VPS13B gene, which has recently been described encoding a mandatory membrane protein involved in Golgi integrity. As the Golgi complex is the place where glycosylation of newly synthesized proteins occurs, we hypothesized that VPS13B deficiency, responsible of Golgi apparatus disturbance, could lead to glycosylation defects and/or mysfunction of this organelle, and thus be a cause of the main clinical manifestations of CS. The glycosylation status of CS serum proteins showed a very unusual pattern of glycosylation characterized by a significant accum…

GlycanGlycosylationGlycosylationEndosomeDevelopmental Disabilities[SDV]Life Sciences [q-bio]Vesicular Transport ProteinsGolgi ApparatusFingers03 medical and health scienceschemistry.chemical_compoundsymbols.namesake0302 clinical medicineAntigens CDIntellectual DisabilityMyopiaGeneticsHumansObesityMolecular BiologyGenetics (clinical)030304 developmental biology0303 health sciencesbiology[ SDV ] Life Sciences [q-bio]Retinal DegenerationTransferrinGeneral MedicineFibroblastsBrefeldin AGolgi apparatusIntercellular Adhesion Molecule-1Cell biologyVPS13BchemistryMembrane proteinBiochemistryMicrocephalysymbolsO-linked glycosylationbiology.proteinMuscle HypotoniaElectrophoresis Polyacrylamide GelRNA InterferenceCell Adhesion Molecules030217 neurology & neurosurgery
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Investigating CRISPR-CAS13b as a tool for the RNA editing of CFTR mRNA with premature stop codon

2020

Background and Rationale Some CF patients are compound heterozygous or homozygous for nonsense mutations in the CFTR gene. Mutant CFTR gene coding for transcripts with premature termination codons (PTCs) is responsible for truncated CFTR protein and for a severe form of the disease. In a precision medicine framework the “REPAIRv2” (RNA Editing for Programmable A to I Replacement v2) tool, developed in the laboratory of Dr. Feng Zhang (USA), seems a good alternative to restore the full-length CFTR protein by editing its mRNA containing PTCs. This new approach is based on the possibility of targeting a deaminase enzyme (huADAR2) to a specific Adenosine, to be edited to Inosine (G analogue), o…

Settore BIO/18 - GeneticamRNA editing CFTR CRISPR-dCAS13b
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CCDC 1047644: Experimental Crystal Structure Determination

2017

Related Article: Michael Mirion, Lars Andernach, Caroline Stobe, Joaquin Barjau, Dieter Schollmeyer, Till Opatz, Arne Lützen, Siegfried R. Waldvogel|2015|Eur.J.Org.Chem.|2015|4876|doi:10.1002/ejoc.201500600

Space GroupCrystallographyCrystal SystemCrystal Structure(5aSR7RS8aRS8cRS13bSR)-Ethyl-247101213b15-heptamethyl-568-trioxa-(benzo[h]-(benzo[b]furo)[23-b]-[4.3.3]propellan)-14-eneCell ParametersExperimental 3D Coordinates
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CCDC 1047645: Experimental Crystal Structure Determination

2017

Related Article: Michael Mirion, Lars Andernach, Caroline Stobe, Joaquin Barjau, Dieter Schollmeyer, Till Opatz, Arne Lützen, Siegfried R. Waldvogel|2015|Eur.J.Org.Chem.|2015|4876|doi:10.1002/ejoc.201500600

Space GroupCrystallographyCrystal SystemCrystal Structure(5aSR8aRS8cRS13bSR)-24101213b15-Hexamethyl-568-trioxa-spiro[cyclohexakis(benzo[h](benzo[b]furo)[23-b]-[4.3.3]propellan)]-14-eneCell ParametersExperimental 3D Coordinates
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CCDC 1047647: Experimental Crystal Structure Determination

2017

Related Article: Michael Mirion, Lars Andernach, Caroline Stobe, Joaquin Barjau, Dieter Schollmeyer, Till Opatz, Arne Lützen, Siegfried R. Waldvogel|2015|Eur.J.Org.Chem.|2015|4876|doi:10.1002/ejoc.201500600

Space GroupCrystallographyCrystal SystemCrystal StructureCell Parameters(5aSR8aRS8cRS13bSR)-8-Aza-56-dioxa-24101213b15-hexamethyl-N-phenyl-(benzo[h]-(benzo[b]furo)[23-b]-[4.3.3]propellan)-14-ene-7-oneExperimental 3D Coordinates
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CCDC 1047646: Experimental Crystal Structure Determination

2017

Related Article: Michael Mirion, Lars Andernach, Caroline Stobe, Joaquin Barjau, Dieter Schollmeyer, Till Opatz, Arne Lützen, Siegfried R. Waldvogel|2015|Eur.J.Org.Chem.|2015|4876|doi:10.1002/ejoc.201500600

Space GroupCrystallographyCrystal SystemCrystal StructureCell Parametersrac-(5aRS7RS8aSR8cSR13bRS)-7-Ethyl-24101213b15-hexamethyl-568-trioxa-(benzo[h]-(benzo[b]furo)[23-b]-[4.3.3]propellan)-14-eneExperimental 3D Coordinates
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Investigating REPAIRv2 as a Tool to Edit CFTR mRNA with Premature Stop Codons

2020

Cystic fibrosis (CF) is caused by mutations in the gene encoding the transmembrane conductance regulator (CFTR) protein. Some CF patients are compound heterozygous or homozygous for nonsense mutations in the CFTR gene. This implies the presence in the transcript of premature termination codons (PTCs) responsible for a truncated CFTR protein and a more severe form of the disease. Aminoglycoside and PTC124 derivatives have been used for the read-through of PTCs to restore the full-length CFTR protein. However, in a precision medicine framework, the CRISPR/dCas13b-based molecular tool &ldquo

congenital hereditary and neonatal diseases and abnormalitiesRNA editingMutantNonsense mutationSettore BIO/11 - Biologia MolecolareBiologyCRISPR/dCas13bCatalysislcsh:Chemistrycystic fibrosisInorganic ChemistryGuide RNASettore BIO/06 - Anatomia Comparata E CitologiaPhysical and Theoretical Chemistrylcsh:QH301-705.5Molecular BiologyGeneSpectroscopyMessenger RNApremature termination codons (PTCs)Organic ChemistryGeneral Medicinerespiratory systemStop codonTransmembrane proteinrespiratory tract diseasesComputer Science ApplicationsCell biologySettore BIO/18 - Geneticalcsh:Biology (General)lcsh:QD1-999RNA editingInternational Journal of Molecular Sciences
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CCDC 942549: Experimental Crystal Structure Determination

2015

Related Article: Christina Lohoelter, Malte Brutschy, Daniel Lubczyk and Siegfried R. Waldvogel|2013|Beilstein J.Org.Chem.|9|2821|doi:10.3762/bjoc.9.317

tris(31013a-trimethyl-10-((pent-4-en-1-yloxy)carbonyl)-123899a1011121313a13b-dodecahydro-37b-methanonaphtho[1'2':67]cyclohepta[12-b]pyrazine-56-diyl)-15:2016:1917:18-triptyceneSpace GroupCrystallographyCrystal SystemCrystal StructureCell ParametersExperimental 3D Coordinates
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