Search results for "4 D"

showing 10 items of 15011 documents

Silicate modulates the cross-talk between osteoblasts (SaOS-2) and osteoclasts (RAW 264.7 cells): inhibition of osteoclast growth and differentiation

2012

It has been shown that inorganic monomeric and polymeric silica/silicate, in the presence of the biomineralization cocktail, increases the expression of osteoprotegerin (OPG) in osteogenic SaOS-2 sarcoma cells in vitro. In contrast, silicate does not affect the steady-state gene expression level of the osteoclastogenic ligand receptor activator of NF-κB ligand (RANKL). In turn it can be expected that the concentration ratio of the mediators OPG/RANKL increases in the presence of silicate. In addition, silicate enhances the growth potential of SaOS-2 cells in vitro, while it causes no effect on RAW 264.7 cells within a concentration range of 10-100 µM. Applying a co-cultivation assay system,…

musculoskeletal diseasesCell SurvivalCellular differentiationmedicine.medical_treatmentAcid PhosphataseMineralogyOsteoclastsCell Count02 engineering and technologyCell CommunicationBiochemistryCell Line03 medical and health sciencesMiceOsteoprotegerinOsteoclastOsteogenesismedicineAnimalsHumansMolecular BiologyRAW 264.7 Cells030304 developmental biologyTartrate-resistant acid phosphataseCell Proliferation0303 health sciencesOsteoblastsbiologyBone Density Conservation AgentsChemistryTartrate-Resistant Acid PhosphataseMacrophagesSilicatesRANK LigandCell DifferentiationCell Biology021001 nanoscience & nanotechnologyCoculture TechniquesCell biologyIsoenzymesmedicine.anatomical_structureCytokineCell cultureRANKLbiology.protein0210 nano-technologyJ. Cell. Biochem.
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Dystroglycan regulates structure, proliferation and differentiation of neuroepithelial cells in the developing vertebrate CNS.

2007

AbstractIn the developing CNS α- and β-dystroglycan are highly concentrated in the endfeet of radial neuroepithelial cells at the contact site to the basal lamina. We show that injection of anti-dystroglycan Fab fragments, knockdown of dystroglycan using RNAi, and overexpression of a dominant-negative dystroglycan protein by microelectroporation in neuroepithelial cells of the chick retina and optic tectum in vivo leads to the loss of their radial morphology, to hyperproliferation, to an increased number of postmitotic neurons, and to an altered distribution of several basally concentrated proteins. Moreover, these treatments also altered the oriented growth of axons from retinal ganglion c…

musculoskeletal diseasesCentral Nervous Systemcongenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtySuperior Colliculianimal structuresCellular differentiationNeuroepithelial CellsStem cellsDevelopmentDystrophin-associated protein complexRetinal ganglionAxonal growthMuscular DystrophiesRetina03 medical and health sciences0302 clinical medicineInternal medicineDystroglycanmedicineAnimalsDystroglycansMolecular BiologyCell Shape030304 developmental biologyCell Proliferation0303 health sciencesRetinabiologyfungiCell DifferentiationCell BiologyMuscular dystrophymusculoskeletal systemCell biologyNeuroepithelial cellmedicine.anatomical_structureEndocrinologyRNAiVertebratesbiology.proteinBasal laminaPikachurinStem cellChickens030217 neurology & neurosurgeryDevelopmental BiologyDevelopmental biology
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Bio-silica and bio-polyphosphate: applications in biomedicine (bone formation)

2012

Bio-silica represents the main mineral component of the sponge skeletal elements (siliceous spicules), while bio-polyphosphate (bio-polyP), a multifunctional polymer existing in microorganisms and animals acts, among others, as reinforcement for pores in cell membranes. These natural inorganic bio-polymers, which can be readily prepared, either by recombinant enzymes (bio-silica and bio-polyP) or chemically (polyP), are promising materials/substances for the amelioration and/or treatment of human bone diseases and dysfunctions. It has been demonstrated that bio-silica causes in vitro a differential effect on the expression of the genes OPG and RANKL, encoding two mediators that control the …

musculoskeletal diseasesSiliconAnabolismBiomedical EngineeringOsteoclastsBioengineering02 engineering and technologyBone morphogenetic protein 2Phosphates03 medical and health sciencesMediatorOsteogenesisAnimalsHumansProgenitor cell030304 developmental biology0303 health sciencesOsteoblastsbiologyCatabolismChemistry021001 nanoscience & nanotechnologySilicon DioxideIn vitro3. Good healthCell biologyPoriferaRANKLImmunologybiology.proteinOsteoporosisBone Diseases0210 nano-technologyFunction (biology)BiotechnologyCurr. Opin. Biotechnol.
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Development of the first marmoset-specific DNA microarray (EUMAMA): a new genetic tool for large-scale expression profiling in a non-human primate

2007

Abstract Background The common marmoset monkey (Callithrix jacchus), a small non-endangered New World primate native to eastern Brazil, is becoming increasingly used as a non-human primate model in biomedical research, drug development and safety assessment. In contrast to the growing interest for the marmoset as an animal model, the molecular tools for genetic analysis are extremely limited. Results Here we report the development of the first marmoset-specific oligonucleotide microarray (EUMAMA) containing probe sets targeting 1541 different marmoset transcripts expressed in hippocampus. These 1541 transcripts represent a wide variety of different functional gene classes. Hybridisation of …

musculoskeletal diseasesendocrine systemanimal structuresMicroarraylcsh:QH426-470Energy and redox metabolism [NCMLS 4]Bioinformaticslcsh:BiotechnologyMolecular Sequence DataComputational biologyBiologyHippocampus03 medical and health sciences0302 clinical medicinebiology.animallcsh:TP248.13-248.65Gene expressionGeneticsAnimalsBiotinylationTissue DistributionOligonucleotide Array Sequence Analysis030304 developmental biologyExpressed Sequence TagsGenetics0303 health sciencesExpressed sequence tagGenomeGene Expression ProfilingNucleic Acid HybridizationMarmosetCallithrixbiology.organism_classificationCallithrixGene expression profilinglcsh:GeneticsMitochondrial medicine [IGMD 8]Gene Expression RegulationGenetic TechniquesGenBankRNADNA microarrayCellular energy metabolism [UMCN 5.3]human activities030217 neurology & neurosurgeryResearch ArticleBiotechnology
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Combined effect of AAV-U7-induced dystrophin exon skipping and soluble activin Type IIB receptor in mdx mice.

2012

Adeno-associated virus (AAV)-U7-mediated skipping of dystrophin-exon-23 restores dystrophin expression and muscle function in the mdx mouse model of Duchenne muscular dystrophy. Soluble activin receptor IIB (sActRIIB-Fc) inhibits signaling of myostatin and homologous molecules and increases muscle mass and function of wild-type and mdx mice. We hypothesized that combined treatment with AAV-U7 and sActRIIB-Fc may synergistically improve mdx muscle function. Bioactivity of sActRIIB-Fc on skeletal muscle was first demonstrated in wild-type mice. In mdx mice we show that AAV-U7-mediated dystrophin restoration improved specific muscle force and resistance to eccentric contractions when applied a…

musculoskeletal diseasesmdx mousemedicine.medical_specialtycongenital hereditary and neonatal diseases and abnormalitiesDuchenne muscular dystrophyActivin Receptors Type IIGenetic VectorsMyostatinBiologyDystrophin03 medical and health sciencesMice0302 clinical medicineInternal medicineGeneticsmedicineMyocyteAnimalsMuscular dystrophyMuscle SkeletalMolecular Biology030304 developmental biology0303 health sciencesBody WeightSkeletal muscleExonsGenetic TherapyDependovirusMuscular Dystrophy Animalmedicine.diseasemusculoskeletal system3. Good healthMice Inbred C57BLEndocrinologymedicine.anatomical_structureImmunologybiology.proteinMice Inbred mdxMolecular MedicineITGA7Dystrophin030217 neurology & neurosurgeryMuscle ContractionHuman gene therapy
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Association between Serum Osteoprotegerin Levels and Severity of Coronary Artery Disease in Patients with Acute Myocardial Infarction

2021

International audience; Background. Osteoprotegerin (OPG), a glycoprotein of the tumour necrosis factor (TNF) superfamily, is one of the main biomarkers for vascular calcification. Aim. We aimed to evaluate the association between serum OPG levels and extent of coronary lesions in patients with acute myocardial infarction (MI). Methods. Consecutive patients hospitalized for an acute MI who underwent coronary angiography were included. SYNTAX score was calculated to assess the severity of coronary artery disease. The population was analysed in low (5 (3–6)), medium (11 (9–13)) and high (20 (18–23)) tertiles of SYNTAX score. Results. Among the 378 patients included, there was a gradual increa…

musculoskeletal diseasesmedicine.medical_specialtyNecrosisPopulationRenal function030204 cardiovascular system & hematologyCoronary artery disease03 medical and health sciences0302 clinical medicineOsteoprotegerin[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemDiabetes mellitusInternal medicinemedicineMyocardial infarction030212 general & internal medicineeducation030304 developmental biology0303 health scienceseducation.field_of_studyEjection fractionbusiness.industryBrief ReportRGeneral Medicinemedicine.diseaseSYNTAX scoremyocardial infarctionosteoprotegerinvascular calcificationCardiologyMedicinemedicine.symptombusinessCardiology and Cardiovascular Medicinecoronary artery diseaseJournal of Clinical Medicine
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Eight new mutations and the expanding phenotype variability in muscular dystrophy caused by ANO5.

2012

Objective: Description of 8 new ANO5 mutations and significant expansion of the clinical phenotype spectrum associated with previously known and unknown mutations to improve diagnostic accuracy. Methods: DNA samples of 101 patients in 95 kindreds at our quaternary referral center in Finland, who had undetermined limb-girdle muscular dystrophy (LGMD), calf distal myopathy, or creatine kinase (CK) elevations of more than 2,000 IU/L, were selected for ANO5 genetic evaluation, and the clinical findings of patients with mutations were retrospectively analyzed. Results: A total of 25 patients with muscular dystrophy caused by 11 different recessive mutations in the ANO5 gene were identified. The …

myalgiaMalePathologymedicine.disease_causeCohort Studies0302 clinical medicineMedicineMuscular dystrophyAge of OnsetCreatine KinaseFinland0303 health sciencesMutationMuscle WeaknessbiologyMiddle AgedPhenotypeMagnetic Resonance Imaging3. Good healthPhenotypeFemalemedicine.symptomAdultmedicine.medical_specialtyWeaknessGenotypeBlotting WesternAnoctaminsGenes RecessiveAsymptomatic03 medical and health sciencesChloride ChannelsHumansGenetic TestingMyopathyMuscle Skeletal030304 developmental biologyAgedbusiness.industryGenetic VariationReproducibility of ResultsDNAmedicine.diseaseMuscular Dystrophies Limb-GirdleMutationbiology.proteinCreatine kinaseNeurology (clinical)business030217 neurology & neurosurgeryNeurology
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Myoadenylate deaminase deficiency

1987

Myoadenylate deaminase (MAD) is the rate-limiting enzyme in the purine nucleotide cycle which is biochemically linked to glycolysis and the citric cycle and thereby providing energy during intense muscular activity. In muscle fibers, myoadenylate deaminase operates at considerably higher activity levels than in other organs. First detected using enzyme-histochemical methods, it now appears that deficiency of myoadenylate deaminase is one of the most frequent enzyme defects in muscle. The primary defect may occur as an isolated nosological entity or not infrequently it is also associated with a large spectrum of different neuromuscular conditions. It seems to be the primary unassociated MAD …

myalgiaWeaknessmedicine.medical_specialtyBiopsyElectromyographyMetabolic myopathyBiologyGastroenterologyAMP Deaminase03 medical and health sciences0302 clinical medicineInternal medicineDrug DiscoveryBiopsymedicineHumansGenetics (clinical)030304 developmental biology0303 health sciencesmedicine.diagnostic_testMusclesMuscle weaknessAMP deaminaseNeuromuscular DiseasesGeneral Medicinemedicine.diseaseEndocrinologyNucleotide DeaminasesMolecular MedicineSarcoidosismedicine.symptom030217 neurology & neurosurgeryKlinische Wochenschrift
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Communicative skills in Spanish children with Autism Spectrum Disorder and children with Attention Deficit Hyperactivity Disorder. Analysis through p…

2018

Background Communicative skills are one of the main deficits experienced by children with Autism Spectrum Disorder (ASD). The differential diagnosis is a complex issue that clinicians often face. For this reason, this study has two objectives: 1) to analyze the similarities and differences that children with Attention Deficit Hyperactivity Disorder (ADHD) and children with ASD present in their linguistic and communicative skills, through parentś perceptions and narrative production; 2) to identify the relative importance of the skills analyzed in discriminating children with ASD versus children with ADHD, as well as the classification power of each of the two measurement methods used. Metho…

narrativeAttention Deficit Hyperactivity DisorderAutism Spectrum Disordermedia_common.quotation_subjectContext (language use)behavioral disciplines and activitiesDevelopmental psychology03 medical and health sciences0302 clinical medicinePerceptionmental disordersDevelopmental and Educational PsychologymedicineAttention deficit hyperactivity disorder0501 psychology and cognitive sciencesNarrativemedia_commonCommunicative competencecommunication05 social sciencesmedicine.diseaseChecklistComprehensionPsychiatry and Mental healthClinical PsychologyAutism spectrum disorderPsychologypragmatics030217 neurology & neurosurgery050104 developmental & child psychology
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Narratives of children with high-functioning autism spectrum disorder: A meta-analysis

2016

Abstract Background The aim of this meta-analysis was to analyze the narrative performance of children and adolescents with high-functioning Autism Spectrum Disorders (ASD) in terms of microstructure, macrostructure and internal state language. Method A systematic literature search yielded 24 studies that met the predetermined inclusion criteria. Effect sizes for each study were calculated for eight variables and analyzed using a random effects model. Intellectual ability, age and type of narrative were considered as potential moderators. Results Results revealed that the children with ASD performed significantly worse than their peers on all the variables considered. Conclusions Findings a…

narrativeAutism Spectrum DisordermicrostructureautismDevelopmental psychology03 medical and health sciences0302 clinical medicineIntervention (counseling)macrostructureDevelopmental and Educational PsychologymedicineHumans0501 psychology and cognitive sciencesNarrativeSpectrum disorderChildLanguageNarration05 social sciencesinternal state languagemedicine.diseaseRandom effects modelmeta-analysisHigh-functioning autismClinical PsychologyCase-Control StudiesMeta-analysisAutismPsychologyInclusion (education)030217 neurology & neurosurgery050104 developmental & child psychologyResearch in Developmental Disabilities
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