Search results for "43"

showing 10 items of 1438 documents

CONNEXIN43 GAP JUNCTION LEVELS DURING DEVELOPMENT OF THE THORACIC AORTA ARE TEMPORALLY CORRELATED WITH ELASTIC LAMINAE DEPOSITION AND INCREASED BLOOD…

1997

A characteristic property of the vascular smooth muscle cell is its ability to modulate between a contractile phenotype, responsible for control of vascular tone, through to a synthetic phenotype, capable of migration and synthesis of extracellular matrix molecules. Smooth muscle cells are coupled by gap junctions, the membrane structures which permit direct intercelluar passage of ions and small molecules, and which play a role both in electrical coupling and intercellular communication during patterning and development. We have previously found that connexin43 type gap junction expression is upregulated in the synthetic phenotype smooth muscle cell in vitro and during atherosclerotic plaq…

MaleCell signalingVascular smooth muscleAorta ThoracicBlood PressureCell CommunicationMuscle Smooth VascularRats Sprague-Dawleymedicine.arterymedicineAnimalsThoracic aortaAortaChemistryCell growthGap junctionGap JunctionsCell BiologyGeneral MedicineAnatomyPhenotypeRatsCell biologyConnexin 43FemaleIntracellularCell Biology International
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Trend of MDR-microorganisms isolated from the biological samples of patients with HAI and from the surfaces around that patient.

2018

Healthcare-associated infections (HAI) continue to be a major public health concern. A number of epidemiologically relevant HAI microorganisms are multidrug-resistant (MDR) germs that can spread rapidly and/or carry multiple resistance to antibiotics. They are the cause of high mortality and possible nosocomial epidemics. For this reason, we implemented microbiological surveillance acquiring samples from patients with HAI and environmental samples from the surfaces surrounding those patients. A retrospective study was carried out from January 2014 to December 2016 in two departments of the University Hospital in Messina, Italy: the Microbiology and the Hygiene Laboratories. A comparison was…

MaleCross InfectionSurveillanceBacteriaDrug-resistant microorganismBacterial InfectionsSettore MED/42 - Igiene Generale E ApplicataAnti-Bacterial AgentsDrug-resistant microorganisms; Nosocomial infections; SurveillanceNosocomial infectionItalySettore MED/43 - Medicina LegaleDrug Resistance Multiple BacterialEnvironmental MicrobiologyHumansFemaleDrug-resistant microorganisms; Nosocomial infections; Surveillance; Microbiology (medical)Retrospective Studies
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Utility of post mortem computed tomography in clivus fracture diagnosis. Case illustration and literature review

2017

Clivus fractures are usually associated with head blunt trauma due to traffic accident and falls. A 23 - year-old man died immediately after a smash-up while he was stopping on his motorcycle. Post-mortem Computed tomography (PMCT), performed before autopsy, revealed a complex basilar skull base fractures associated with brainstem and cranio-vertebral junction injuries, improving the diagnostic performance of conventional autopsy. Imaging data were re-assessable and PMCT offers the possibility to perform multiplanar and volume rendered reconstructions, increasing forensic medicine knowledge related to traumatic injuries.

MaleForensic pathologymedicine.medical_specialtyPost-mortem CT - Forensic pathology - Clivus fracture - Traffic accident - Brainstem laceration -Cervical spineClivus fractureAutopsyPathology and Forensic MedicineYoung Adult03 medical and health sciences0302 clinical medicineSettore MED/43 - Medicina LegaleClivusmedicineHumans030216 legal & forensic medicinePost mortem computed tomographyForensic PathologySkull Fracturesbusiness.industryTraffic accidentAccidents TrafficSurgeryDeathIssues ethics and legal aspectsSkullmedicine.anatomical_structureBlunt traumaAutopsyRadiologyTomography X-Ray Computedbusiness030217 neurology & neurosurgeryBrain StemLegal Medicine
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Anti-B-50 (GAP-43) antibodies decrease exocytosis of glutamate in permeated synaptosomes.

1999

Abstract The involvement of the protein kinase C substrate, B-50 (GAP-43), in the release of glutamate from small clear-cored vesicles in streptolysin-O-permeated synaptosomes was studied by using anti-B-50 antibodies. Glutamate release was induced from endogenous as well as 3 H -labelled pools in a [Ca2+]-dependent manner. This Ca2+-induced release was partially ATP dependent and blocked by the light-chain fragment of tetanus toxin, demonstrating its vesicular nature. Comparison of the effects of anti-B-50 antibodies on glutamate and noradrenaline release from permeated synaptosomes revealed two major differences. Firstly, Ca2+-induced glutamate release was decreased only partially by anti…

MaleGlutamic AcidBiologyIn Vitro TechniquesSynaptic vesicleExocytosisExocytosischemistry.chemical_compoundNorepinephrineAdenosine TriphosphateGAP-43 ProteinAnimalsEnzyme InhibitorsRats WistarNeurotransmitterProtein kinase CProtein Kinase CPharmacologySynaptosomeVesicleGlutamate receptorAntibodies MonoclonalIntracellular MembranesRatschemistryBiochemistryStreptolysinsBiophysicsLiberationCalciumSynaptosomesEuropean journal of pharmacology
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Determination of propofol by GC/MS and fast GC/MS-TOF in two cases of poisoning

2017

Two cases of suspected acute and lethal intoxication caused by propofol were delivered by the judicial authority to the Department of Sciences for Health Promotion and Mother-Child Care in Palermo, Sicily. In the first case a female nurse was found in a hotel room, where she lived with her mother; four 10 mg/mL vials and two 20 mg/mL vials of propofol were found near the decedent along with syringes and needles. In the second case a male nurse was found in the operating room of a hospital, along with a used syringe. In both cases a preliminary systematic and toxicological analysis indicated the presence of propofol in the blood and urine. As a result, a method for the quantitative determina…

MaleHealth Toxicology and MutagenesisHypnotics and SedativeUrineToxicology01 natural sciencesVialGas Chromatography-Mass SpectrometryAnalytical Chemistry03 medical and health sciencesForensic Toxicology0302 clinical medicineSettore MED/43 - Medicina LegaleEnvironmental ChemistryMedicineHumansHypnotics and SedativesSettore CHIM/01 - Chimica Analitica030216 legal & forensic medicinePropofolSyringeChemical Health and Safetybusiness.industry010401 analytical chemistryForensic toxicologyQuantitative determination0104 chemical sciencesManner of deathSuicideAnesthesiaFemaleGas chromatography–mass spectrometryDrug OverdosebusinessPropofolHomicidemedicine.drugHuman
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Hypothalamic Astroglial Connexins are Required for Brain Glucose Sensing-Induced Insulin Secretion

2014

Supplementary Information accompanies the paper on the Journal of Cerebral Blood Flow & Metabolism website; Hypothalamic glucose detection participates in maintaining glycemic balance, food intake, and thermogenesis. Although hypothalamic neurons are the executive cells involved in these responses, there is increasing evidence that astrocytes participate in glucose sensing (GS); however, it is unknown whether astroglial networking is required for glucose sensitivity. Astroglial connexins 30 and 43 (Cx30 and Cx43) form hexameric channels, which are apposed in gap junctions, allowing for the intercellular transfer of small molecules such as glucose throughout the astroglial networks. Here, we…

MaleINVOLVEMENTHOMEOSTASISmedicine.medical_specialtymedicine.medical_treatmentNerve Tissue ProteinsCarbohydrate metabolismBiologyASTROCYTESConnexinsconnexin 43RATSastrocyteInternal medicineInsulin SecretionmedicineAnimalsInsulinTANYCYTESRats WistarhypothalamusIN-VIVOHEMICHANNELSglucose sensingInsulinARCUATE NUCLEUSGap junctionFasting[ SDV.MHEP.EM ] Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism[SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolismBARRIERconnexin 30NETWORKSGlucoseEndocrinologymedicine.anatomical_structureNeurologyHypothalamus[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]RNA InterferenceOriginal Article[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]sense organsNeurology (clinical)Cardiology and Cardiovascular MedicineThermogenesisIntracellularHomeostasisAstrocyteJournal of Cerebral Blood Flow & Metabolism
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Gene-environment interactions of CETP gene variation in a high cardiovascular risk Mediterranean population

2010

Genome-wide association studies show that cholesteryl ester transfer protein (CETP) single nucleotide polymorphisms (SNPs) are more strongly associated with HDL cholesterol (HDL-C) concentrations than any other loci across the genome. However, gene-environment interactions for clinical applications are still largely unknown. We studied gene-environment interactions between CETP SNPs and dietary fat intake, adherence to the Mediterranean diet, alcohol consumption, smoking, obesity, and diabetes on HDL-C in 4,210 high cardiovascular risk subjects from a Mediterranean population. We focused on the −4,502C>T and the TaqIB SNPs in partial linkage disequilibrium (D'= 0.88; P < 0.001). They were i…

MaleLinkage disequilibriumMediterranean dietGenome-wide association studyBiochemistryEndocrinologyRisk FactorsNutrigeneticsAged 80 and overGeneticseducation.field_of_studyBioquímica y tecnologíaMediterranean RegionMiddle AgedLipidsBiochemistry and technologyCardiovascular DiseasesFemalelipids (amino acids peptides and proteins)Alcoholmedicine.medical_specialtyPopulationSingle-nucleotide polymorphismQD415-436EnvironmentBiologyBioquímica i biotecnologiaPolymorphism Single NucleotideInternal medicineNutrició -- Aspectes genèticsMediterranean dietCholesterylester transfer proteinmedicineHumansGenetic Predisposition to DiseaseObesityeducationLife StyleAgedGenetic associationMediterrània Regió -- PoblacióCholesterol HDLCell Biologymedicine.diseaseDietary FatsObesityCholesterol Ester Transfer ProteinsDietSistema cardiovascular -- Malalties -- Factors de riscEndocrinologyFatbiology.proteinPatient-Oriented and Epidemiological Research0022-2275Genome-Wide Association Study
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A role for the peroxisomal 3-ketoacyl-CoA thiolase B enzyme in the control of PPARα-mediated upregulation of SREBP-2 target genes in the liver.: ThB …

2011

International audience; Peroxisomal 3-ketoacyl-CoA thiolase B (Thb) catalyzes the final step in the peroxisomal β-oxidation of straight-chain acyl-CoAs and is under the transcription control of the nuclear hormone receptor PPARα. PPARα binds to and is activated by the synthetic compound Wy14,643 (Wy). Here, we show that the magnitude of Wy-mediated induction of peroxisomal β-oxidation of radiolabeled (1-(14)C) palmitate was significantly reduced in mice deficient for Thb. In contrast, mitochondrial β-oxidation was unaltered in Thb(-/-) mice. Given that Wy-treatment induced Acox1 and MFP-1/-2 activity at a similar level in both genotypes, we concluded that the thiolase step alone was respons…

MaleMESH: HepatomegalyPalmitatesMESH : PyrimidinesMESH : Gene DeletionBiochemistryelement-binding proteinsMESH : Acetyl-CoA C-AcyltransferaseMiceMESH: Up-RegulationMESH: AnimalsMESH : Up-RegulationMESH: Lipid Metabolism0303 health sciencesMESH : Gene Expression RegulationThiolase030302 biochemistry & molecular biologyGeneral MedicineMESH : HepatomegalyUp-Regulationzellweger-syndromePeroxisome ProliferatorsMESH: Peroxisome ProliferatorsHepatomegalySterol Regulatory Element Binding Protein 2peroxisomal 3-ketoacyl-CoA thiolase BMESH: Mitochondria3-oxoacyl-coa thiolaseLathosterolfatty-acid oxidationrat-liverMESH: Sterol Regulatory Element Binding Protein 203 medical and health sciencesMESH : Sterol Regulatory Element Binding Protein 2HumansPPAR alphaMESH : Peroxisome Proliferators[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologyPPARaVLAGMESH : Oxidation-ReductionFatty Acid Oxidation.MESH: HumansCholesterolMESH : HumanscholesterolLipid MetabolismMESH: PeroxisomesSterol regulatory element-binding proteinchemistryMESH: PyrimidinesCholesterol; Micro-array analysis; Peroxisomal 3-ketoacyl-CoA thiolase B; PPARα and SREBP-2; Wy14643Fatty Acid OxidationGene DeletionMESH: LiverMESH: Oxidation-ReductionMESH: Signal TransductionMESH: Mice KnockoutVoeding Metabolisme en Genomicachemistry.chemical_compoundMESH: CholesterolMESH : Lipid MetabolismWy14MESH : PalmitatesMESH: PPAR alphaMESH : CholesterolMice Knockoutneuronal migration643PeroxisomeAcetyl-CoA C-AcyltransferaseMESH: Gene Expression RegulationMetabolism and GenomicsMitochondriaLiverBiochemistryMicro-array analysisMetabolisme en GenomicaACOX1Nutrition Metabolism and GenomicsMESH : MitochondriaOxidation-ReductionSignal Transductionacyl-coa oxidasecholesterol-synthesisMESH : MaleMESH : PPAR alphaPeroxisome ProliferationPPARα and SREBP-2Biologybeta-oxidationVoedingproliferator-activated receptorsMESH : MicePeroxisomesAnimals[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyMESH: Mice030304 developmental biologySCP2NutritionMESH : Signal TransductionMESH : LiverMESH: PalmitatesMESH: MalePyrimidinesMESH: Acetyl-CoA C-AcyltransferaseGene Expression RegulationMESH: Gene DeletionMESH : Mice KnockoutMESH : AnimalsMESH : Peroxisomes
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The Healthy Nordic Diet and Mediterranean Diet and Incidence of Disability 10 Years Later in Home-Dwelling Old Adults.

2019

Background/Objective: Diet has a major impact on a person's health. However, limited information exists on the long-term role of the whole diet on disability. We investigated the association of the healthy Nordic diet and the Mediterranean diet with incident disability 10 years later. Design: Longitudinal, with a follow-up of 10 years. Settings/Participants: A total of 962 home-dwelling men and women from the Helsinki Birth Cohort Study, mean age 61.6 years, who were free of disability at baseline. Measurements: At baseline, 2001-2004, the Nordic diet score (NDS) and modified Mediterranean diet score (mMDS) were calculated using a validated 128-item food-frequency questionnaire. Higher scor…

MaleMediterranean dietLogistic regressionruokavaliotDiet MediterraneanMUSCLE STRENGTH0302 clinical medicineRisk FactorsMedicinetoimintarajoitteet030212 general & internal medicineLongitudinal StudiesGeneral NursingFinlandRISKeducation.field_of_studyHealth PolicyIncidence (epidemiology)ConfoundingBALTIC SEA DIETliikuntarajoitteetGeneral Medicineta3142Middle Aged3142 Public health care science environmental and occupational health3. Good healthCohortFemale3143 NutritionDiet HealthyVälimeren ruokavalioikääntyneetPopulationLongevityHealth Promotion03 medical and health sciencesmobility limitationADHERENCEvammaisuusMediterranean dietHumansCOHORTMobility LimitationeducationLife StyleMETAANALYSISFOOD FREQUENCY QUESTIONNAIREbusiness.industryNordic dietMOBILITY LIMITATIONSmediterranean dietOdds ratioFUNCTIONAL LIMITATIONSConfidence intervalikääntyminendisabilityageing3121 General medicine internal medicine and other clinical medicineChronic DiseaseGeriatrics and GerontologyPHYSICAL PERFORMANCEbusiness030217 neurology & neurosurgeryDemographyJournal of the American Medical Directors Association
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Associations of rs3918242 and rs2285053 MMP-9 and MMP-2 polymorphisms with the risk, severity, and short- and long-term complications of degenerative…

2016

Abstract Background Degenerative forms of mitral valve diseases (MVDs) are very complex pathologies. Thus, it is difficult to make generalizations about the disease pathways or genetic risk factors contributing to these diseases. However, a key role of metalloproteinases (MMPs) in their pathophysiology is emerging. Thus, we performed for the first time a perspective study to assess eventual associations of some functional single nucleotide polymorphisms (SNPs) in MMP-2 and MMP-9 genes with the MVD risk, symptom severity, and short- and long-term (4.8 years) complications. Materials and methods For this purpose, 90 patients and two control groups were genotyped for rs3918242, rs243865, and r…

MalePathologyHeart Valve DiseasesDisease030204 cardiovascular system & hematologyMatrix metalloproteinasers3918242 rs243865 rs2285053 MMP-2 and MMP-9 gene SNPDegenerative forms of mitral valve diseases; Management and outcome; Metalloproteinases; rs3918242 rs243865 rs2285053 MMP-2 and MMP-9 gene SNPs; Aged; Cohort Studies; Female; Genetic Predisposition to Disease; Genotype; Heart Valve Diseases; Humans; Male; Matrix Metalloproteinase 2; Matrix Metalloproteinase 9; Middle Aged; Mitral Valve; Prospective Studies; Risk Factors; Polymorphism Single Nucleotide0403 veterinary scienceCohort Studies0302 clinical mediciners2285053 MMP-2 and MMP-9 gene SNPsRisk FactorsGenotypeManagement and outcomeNatriuretic peptideProspective StudiesProspective cohort studyMVDSMetalloproteinaseDegenerative forms of mitral valve diseases04 agricultural and veterinary sciencesGeneral MedicineSingle NucleotideMiddle AgedMetalloproteinasesPathophysiologyMatrix Metalloproteinase 9Matrix Metalloproteinase 2Mitral ValveFemalers3918242Cardiology and Cardiovascular MedicineDegenerative forms of mitral valve diseasemedicine.medical_specialtyGenotype040301 veterinary sciencesmedicine.drug_class2734Single-nucleotide polymorphismDegenerative forms of mitral valve diseases; Management and outcome; Metalloproteinases; rs3918242 rs243865 rs2285053 MMP-2 and MMP-9 gene SNPs; Cardiology and Cardiovascular Medicine; 2734Polymorphism Single NucleotidePathology and Forensic Medicine03 medical and health sciencesInternal medicinemedicineHumansGenetic Predisposition to Diseasers243865PolymorphismAgedbusiness.industrySettore MED/23 - Chirurgia Cardiacabusiness
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