Search results for "95"

showing 10 items of 2244 documents

MicroRNAs and Oxidative Stress: An Intriguing Crosstalk to Be Exploited in the Management of Type 2 Diabetes

2021

Type 2 diabetes is a chronic disease widespread throughout the world, with significant human, social, and economic costs. Its multifactorial etiology leads to persistent hyperglycemia, impaired carbohydrate and fat metabolism, chronic inflammation, and defects in insulin secretion or insulin action, or both. Emerging evidence reveals that oxidative stress has a critical role in the development of type 2 diabetes. Overproduction of reactive oxygen species can promote an imbalance between the production and neutralization of antioxidant defence systems, thus favoring lipid accumulation, cellular stress, and the activation of cytosolic signaling pathways, and inducing β-cell dysfunction, insul…

0301 basic medicinePhysiologymedicine.medical_treatmentClinical BiochemistryInflammationRM1-950Type 2 diabetesReviewBiologymedicine.disease_causeBiochemistry03 medical and health sciences0302 clinical medicineInsulin resistancemicroRNAmedicineoxidative stressredox signalingMolecular BiologymicroRNAInsulinCell Biologymedicine.diseaseCell biologyCrosstalk (biology)030104 developmental biology030220 oncology & carcinogenesisTherapeutics. Pharmacologytype 2 diabetesmedicine.symptomSignal transductionOxidative stressAntioxidants
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Mitoprotective Clinical Strategies in Type 2 Diabetes and Fanconi Anemia Patients: Suggestions for Clinical Management of Mitochondrial Dysfunction

2020

Oxidative stress (OS) and mitochondrial dysfunction (MDF) occur in a number of disorders, and several clinical studies have attempted to counteract OS and MDF by providing adjuvant treatments against disease progression. The present review is aimed at focusing on two apparently distant diseases, namely type 2 diabetes (T2D) and a rare genetic disease, Fanconi anemia (FA). The pathogenetic links between T2D and FA include the high T2D prevalence among FA patients and the recognized evidence for OS and MDF in both disorders. This latter phenotypic/pathogenetic feature—namely MDF—may be regarded as a mechanistic ground both accounting for the clinical outcomes in both diseases, and…

0301 basic medicinePhysiologymedicine.medical_treatmentClinical Biochemistrymitochondrial nutrientsDiseaseType 2 diabetesReviewBioinformaticsmedicine.disease_causeBiochemistry03 medical and health scienceschemistry.chemical_compound0302 clinical medicineIn vivoFanconi anemiamitochondrial dysfunctionmedicineoxidative stressMolecular Biologyfanconi anemiaCoenzyme Q10business.industrylcsh:RM1-950Mitochondrial nutrientCell Biologymedicine.diseasePhenotype030104 developmental biologylcsh:Therapeutics. Pharmacologychemistry030220 oncology & carcinogenesisOxidative stretype 2 diabetesbusinessAdjuvantOxidative stressAntioxidants
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Resveratrol shifts energy metabolism to increase lipid oxidation in healthy old mice.

2019

Abstract Objectives The objective of this work was to determine the specific mechanisms by which resveratrol inhibits lipogenesis and stimulates lipolysis. Methods Twelve male mice were individually introduced into a metabolic cage for 24 h to measure basal metabolic rate, prior to intervention. They were randomly divided into two groups, resveratrol (RSV) and control (C), and administered resveratrol intraperitoneally or vehicle, respectively, for two consecutive days. After 24 h, the metabolic energy expenditure was again determined for 24 h, before mice were sacrificed. Protein and gene expression of different enzymes related to metabolism in the hepatic tissue, adipose tissue and gastro…

0301 basic medicinePolyphenolMalemedicine.medical_specialtyAgingLipolysisAdipose tissueWhite adipose tissueRM1-950ResveratrolLipid catabolism03 medical and health scienceschemistry.chemical_compoundMice0302 clinical medicineInternal medicinemedicineAnimalsCarnitineBeta oxidationFatty acid synthesisRespiratory quotientPharmacologyLipogenesisFatty AcidsGeneral MedicineMice Inbred C57BL030104 developmental biologyEndocrinologyMalonyl-CoAchemistryAdipose TissueCarnitine AcyltransferasesLiverResveratrol030220 oncology & carcinogenesisLipogenesisTherapeutics. PharmacologyEnergy MetabolismOxidation-Reductionmedicine.drugAcetyl-CoA CarboxylaseBiomedicinepharmacotherapy = Biomedecinepharmacotherapie
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Ferroptosis and Its Potential Role in Human Diseases

2020

Ferroptosis is a novel regulated cell death pattern discovered when studying the mechanism of erastin-killing RAS mutant tumor cells in 2012. It is an iron-dependent programmed cell death pathway mainly caused by an increased redox imbalance but with distinct biological and morphology characteristics when compared to other known cell death patterns. Ferroptosis is associated with various diseases including acute kidney injury, cancer, and cardiovascular, neurodegenerative, and hepatic diseases. Moreover, activation or inhibition of ferroptosis using a variety of ferroptosis initiators and inhibitors can modulate disease progression in animal models. In this review, we provide a comprehensiv…

0301 basic medicineProgrammed cell deathReviewdegenerative diseasesBiologyHepatic Diseases03 medical and health sciences0302 clinical medicineRegulated cell deathmedicinePharmacology (medical)Pharmacologyreactive oxygen speciesMechanism (biology)Ferroptosislcsh:RM1-950Disease progressionCancermedicine.diseaseferroptosissignaling pathwayslcsh:Therapeutics. Pharmacology030104 developmental biologypharmacology design030220 oncology & carcinogenesisCancer researchSignal transductionFrontiers in Pharmacology
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Pekinenin E Inhibits the Growth of Hepatocellular Carcinoma by Promoting Endoplasmic Reticulum Stress Mediated Cell Death.

2017

Hepatocellular carcinoma (HCC) is a malignant primary liver cancer with poor prognosis. In the present study, we report that pekinenin E (PE), a casbane diterpenoid derived from the roots of Euphorbia pekinensis, has a strong antitumor activity against human HCC cells both in vitro and in vivo. PE suppressed the growth of human HCC cells Hep G2 and SMMC-7721. In addition, PE-mediated endoplasmic reticulum (ER) stress caused increasing expressions of C/EBP homologous protein (CHOP), leading to apoptosis in HCC cells both in vitro and in vivo. Inhibition of ER stress with CHOP small interfering RNA or 4-phenyl-butyric acid partially reversed PE-induced cell death. Furthermore, PE induced S ce…

0301 basic medicineProgrammed cell deathSmall interfering RNAPathologymedicine.medical_specialtyCell cycle checkpointCHOP03 medical and health sciencesmedicinePharmacology (medical)Original ResearchPharmacologybusiness.industryEndoplasmic reticulumlcsh:RM1-950apoptosisdigestive system diseasesHep G2030104 developmental biologylcsh:Therapeutics. PharmacologyApoptosishepatocarcinomacell cycle arrestUnfolded protein responseCancer researchbusinessER stresspekinenin EFrontiers in pharmacology
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Leaf and Root Extracts from Campomanesia adamantium (Myrtaceae) Promote Apoptotic Death of Leukemic Cells via Activation of Intracellular Calcium and…

2017

Phytochemical studies are seeking new alternatives to prevent or treat cancer, including different types of leukemias. Campomanesia adamantium, commonly known as guavira or guabiroba, exhibits pharmacological properties including antioxidant, antimicrobial, and antiproliferative activities. Considering the anticancer potential of this plant species, the aim of this study was to evaluate the antileukemic activity and the chemical composition of aqueous extracts from the leaves (AECL) and roots (AECR) of C. adamantium and their possible mechanisms of action. The extracts were analyzed by LC-DAD-MS, and their constituents were identified based on the UV, MS, and MS/MS data. The AECL and AECR s…

0301 basic medicineProgrammed cell deathnatural productsbioprospectingCaspase 3PharmacologyJurkat cellsCalcium in biology03 medical and health scienceschemistry.chemical_compound0302 clinical medicinemedicinal plantcancerPharmacology (medical)Propidium iodideCytotoxicityCaspasePharmacologybiologylcsh:RM1-950LC-MSlcsh:Therapeutics. Pharmacology030104 developmental biologychemistryBiochemistryApoptosis030220 oncology & carcinogenesisbiology.proteinFrontiers in Pharmacology
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Suicidal Erythrocyte Death in Metabolic Syndrome.

2021

Eryptosis is a coordinated, programmed cell death culminating with the disposal of cells without disruption of the cell membrane and the release of endocellular oxidative and pro-inflammatory milieu. While providing a convenient form of death for erythrocytes, dysregulated eryptosis may result in a series of detrimental and harmful pathological consequences highly related to the endothelial dysfunction (ED). Metabolic syndrome (MetS) is described as a cluster of cardiometabolic factors (hyperglycemia, dyslipidemia, hypertension and obesity) that increases the risk of cardiovascular complications such as those related to diabetes and atherosclerosis. In the light of the crucial role exerted …

0301 basic medicineProgrammed cell deathobesityhypertensionPhysiologyClinical BiochemistryReview030204 cardiovascular system & hematologyBioinformaticsmedicine.disease_causeBiochemistrymetabolic syndromeendothelial dysfunction03 medical and health sciences0302 clinical medicineDiabetes mellituseryptosisvascular damagemedicineoxidative stressEndothelial dysfunctionMolecular BiologyPathologicaldiabetesbusiness.industrylcsh:RM1-950dyslipidemiaCell Biologymedicine.diseaseObesitylcsh:Therapeutics. Pharmacology030104 developmental biologyMetabolic syndromeatherosclerosisbusinessDyslipidemiaOxidative stressAntioxidants (Basel, Switzerland)
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Alterations of perineuronal nets in the dorsolateral prefrontal cortex of neuropsychiatric patients

2019

Abstract Background Alterations in the structure and physiology of interneurons in the prefrontal cortex (PFC) are important factors in the etiopathology of different psychiatric disorders. Among the interneuronal subpopulations, parvalbumin (PV) expressing cells appear to be specially affected. Interestingly, during development and adulthood the connectivity of these interneurons is regulated by the presence of perineuronal nets (PNNs), specialized regions of the extracellular matrix, which are frequently surrounding PV expressing neurons. Previous reports have found anomalies in the density of PNNs in the PFC of schizophrenic patients. However, although some studies have described alterat…

0301 basic medicinePsychosisBipolar disorderPerineuronal netsPrefrontal cortexlcsh:RC321-57103 medical and health sciences0302 clinical medicinemental disordersNeuroplasticitymedicineMajor depressionPsiquiatriaBipolar disorderPrefrontal cortexlcsh:Neurosciences. Biological psychiatry. NeuropsychiatrySalut mentalBiological PsychiatryParvalbuminbiologyResearchPerineuronal netlcsh:QP351-495medicine.diseaseDorsolateral prefrontal cortexPsychiatry and Mental healthlcsh:Neurophysiology and neuropsychology030104 developmental biologymedicine.anatomical_structurenervous systemSchizophreniaSchizophreniabiology.proteinEsquizofrèniaNeuroscience030217 neurology & neurosurgeryParvalbuminInternational Journal of Bipolar Disorders
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Ethnopharmacology of Love

2018

Background: Elixirs conferring eternal youth or inducing amatory and erotic attraction have been searched for without success. Lovesickness is a widespread affliction resulting from unrequited love and/or the impossibility for physical and emotional union. The symptoms are reflections of altered dopamine, serotonin, noradrenaline, testosterone and cortisol levels and range from frenzy and intrusive thinking to despair and depression, sharing traits with the neurochemistry of addiction and compulsive behavior disorder. Although it can seriously impact the quality of life, lovesickness is currently not considered in official disease classification systems. Consequently, no official therapeuti…

0301 basic medicinePsychotherapistmedia_common.quotation_subjectethnomedicineReviewaphrodisiacs03 medical and health sciences0302 clinical medicinemedicinesexPharmacology (medical)LovesicknessFalling in lovemedia_commonPharmacologybeta-caryophylleneInfatuationinfatuationAddictionlcsh:RM1-950RomanceserotoninVentral tegmental area030104 developmental biologymedicine.anatomical_structurelcsh:Therapeutics. PharmacologylovesicknessCompulsive behaviormedicine.symptomdopaminePsychology030217 neurology & neurosurgeryUnrequited loveFrontiers in Pharmacology
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Addition of the Aldose Reductase Inhibitor Benzofuroxane Derivative BF-5m to Prolonged and Moderate Exercise Training Enhanced Protection of the Rat …

2019

Moderate exercise training may not be sufficient to exert beneficial effects on the cardiovascular system because of the long-term multifactorial etiology of diabetic complications. The addition of a proper pharmacological tool to the physical exercise should improve the outcomes of the diabetic damage. Here it is shown that 8 weeks exercise training of type 1 diabetic Sprague-Dawley (SD) rats resulted in a significantly increased heart rate, a 14% increase in the left ventricular ejection fraction (LVEF) increased plasma insulin levels and a 13% decrease in plasma glucose with respect to sedentary animals. The training also resulted in a 22% reduction in cardiac QT interval from a diabetic…

0301 basic medicineQT intervalmedicine.medical_specialtyPhysical exerciseDiabeteQT interval03 medical and health sciences0302 clinical medicineFibrosisInternal medicineDiabetes mellitusmedicinePharmacology (medical)Original ResearchPharmacologyAldose reductaseType 1 diabetesEjection fractiondiabetesexercisebusiness.industrylcsh:RM1-950medicine.diseaseAldose reductase inhibitor030104 developmental biologyEndocrinologylcsh:Therapeutics. Pharmacology030220 oncology & carcinogenesisbenzofuroxanebusinessaldose-reductasemedicine.drugFrontiers in Pharmacology
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