Search results for "ACID"

showing 10 items of 13107 documents

The differential effects of thiazolidindiones on atherogenic dyslipidemia in type 2 diabetes: what is the clinical significance?

2008

Diabetic dyslipidemia is typically characterized by an increase in plasma triglycerides, a decrease in high-density lipoprotein cholesterol and a concomitant increase in atherogenic small dense low-density lipoproteins. Thiazolidindiones are able to lower the levels of fasting glucose and glycated hemoglobin significantly by improving insulin sensitivity, as well as improving some aspects of diabetic dyslipidemia: total cholesterol, low-density lipoprotein cholesterol and high-density lipoprotein cholesterol tend to increase while triglycerides are generally decreased.This paper reviewed the effects of pioglitazone and rosiglitazone on atherogenic diabetic dyslipidemia, in particular on sma…

medicine.medical_specialty10265 Clinic for Endocrinology and Diabetology610 Medicine & healthType 2 diabetesRosiglitazonechemistry.chemical_compoundDiabetes mellitusInternal medicinemedicine2736 Pharmacology (medical)HumansHypoglycemic AgentsPharmacology (medical)Clinical significancecardiovascular risk dense low-density lipoprotein diabetes low-density lipoprotein size pioglitazone rosiglitazone smallTriglyceridesDyslipidemiasPharmacologyAtherogenic dyslipidemiaPioglitazonebusiness.industryThiazolidindionesAtherogenic dyslipidemiaGeneral Medicinemedicine.diseaseLipoproteins LDLEndocrinology3004 PharmacologychemistryDiabetes Mellitus Type 2lipids (amino acids peptides and proteins)ThiazolidinedionesGlycated hemoglobinbusinessRosiglitazoneLipoproteins HDLPioglitazoneBiomarkersmedicine.drugLipoprotein
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Clearance and metabolism of arachidonic acid by C6 glioma cells and astrocytes.

1995

Effects of increased levels of arachidonic acid (AA) were analyzed in vitro by employment of C6 glioma cells and astrocytes from primary culture. The cells were suspended in a physiological medium added with arachidonic acid (AA) in a concentration range from 0.01 to 0.5 mM. The concentration profiles of the fatty acid and AA-metabolites were subsequently followed for 90 min. AA was measured by gas chromatography, whereas the AA-metabolites PGF2 alpha and LTB4 by radioimmunoassay (RIA). Following administration of AA at 0.05 or 0.1 mM the medium was completely cleared from the fatty acid within 10 to 15 min. However, when 0.5 mM were added, AA concentrations of 0.36 +/- 0.055 mM were found …

medicine.medical_specialty45-Dihydro-1-(3-(trifluoromethyl)phenyl)-1H-pyrazol-3-amineBiologyDinoprostBiochemistryLeukotriene B4Cellular and Molecular NeuroscienceLipoxygenasechemistry.chemical_compoundInternal medicinemedicineTumor Cells CulturedCyclooxygenase InhibitorsLipoxygenase Inhibitorschemistry.chemical_classificationArachidonic AcidFatty acidRadioimmunoassayGeneral MedicineMetabolismGliomaCulture MediaKineticsEndocrinologymedicine.anatomical_structurechemistryCell cultureAstrocytesbiology.proteinNeurogliaArachidonic acidAstrocyteNeurochemical research
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Oleic Acid-Injection in Pigs As a Model for Acute Respiratory Distress Syndrome

2018

The acute respiratory distress syndrome is a relevant intensive care disease with an incidence ranging between 2.2% and 19% of intensive care unit patients. Despite treatment advances over the last decades, ARDS patients still suffer mortality rates between 35 and 40%. There is still a need for further research to improve the outcome of patients suffering from ARDS. One problem is that no single animal model can mimic the complex pathomechanism of the acute respiratory distress syndrome, but several models exist to study different parts of it. Oleic acid injection (OAI)-induced lung injury is a well-established model for studying ventilation strategies, lung mechanics and ventilation/perfus…

medicine.medical_specialtyARDSSwineGeneral Chemical EngineeringAcute Lung InjuryDiseaseAcute respiratory distressLung injuryGeneral Biochemistry Genetics and Molecular Biologylaw.invention03 medical and health sciences0302 clinical medicinelawIntensive caremedicineAnimalsHumansIntensive care medicineRespiratory Distress SyndromeGeneral Immunology and Microbiologybusiness.industryGeneral NeuroscienceMortality rate030208 emergency & critical care medicinemedicine.diseaseRespiration ArtificialIntensive care unitRespiratory Function TestsDisease Models Animal030228 respiratory systemBreathingMedicinebusinessOleic AcidJournal of Visualized Experiments
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Stimulation with carbachol alters endomembrane distribution and plasma membrane expression of intracellular proteins in lacrimal acinar cells.

2000

The events that lead to Sjogren's autoimmune processes in the lacrimal gland remain poorly understood. The acinar cell's responses to acute cholinergic stimulation include release of secretory products across the apical plasma membrane (apm) and a number of processes related to traffic between endomembrane compartments and the basal-lateral plasma membranes (blm), such as recruitment of Na, K-ATPase, accelerated recycling, and accelerated transcytosis of secretory IgA. We tested the hypothesis that stimulation-induced acceleration of endomembrane traffic is accompanied by changes in compartmentation and increased blm expression of proteins that are normally sequestered in endomembrane compa…

medicine.medical_specialtyAcid PhosphataseImmunoblottingGolgi ApparatusStimulationBiologyCholinergic AgonistsCathepsin BCathepsin BCellular and Molecular Neurosciencesymbols.namesakeInternal medicinemedicineAcinar cellAnimalsEndomembrane systemCells Culturedrab5 GTP-Binding ProteinsDifferential centrifugationEnzyme PrecursorsCell MembraneHistocompatibility Antigens Class IIMembrane Proteinsalpha-GlucosidasesGolgi apparatusGalactosyltransferasesCathepsinsSensory SystemsStimulation Chemicalbeta-N-AcetylhexosaminidasesCell biologyOphthalmologyEndocrinologySjogren's SyndromeTranscytosisrab GTP-Binding ProteinssymbolsCarbacholElectrophoresis Polyacrylamide GelFemaleRabbitsSodium-Potassium-Exchanging ATPaseIntracellularExperimental eye research
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Diagnostic and prognostic value of H-FABP in acute coronary syndrome: Still evidence to bring

2018

Abstract The assessment of chest pain patients presenting to the emergency area (EA) is still a clinical challenge, as the majority of patients are not diagnosed with acute coronary syndrome (ACS). New generation high sensitivity c-Tn (hs-cTn) assays have showed better performances compared to the standard c-Tn. However, hs-Tn still presents some limitations. Hence, novel, early biomarkers are needed in this setting. Among all, heart-type fatty acid binding protein (H-FABP) has been largely investigated. This article reviews the studies evaluating H-FABP performance in diagnosing acute myocardial infarction (AMI) and stratifying chest pain patients by risk. H-FABP optimal performances in AC…

medicine.medical_specialtyAcute coronary syndromeClinical BiochemistryPredictive Value of Test030204 cardiovascular system & hematologyChest painClinical biochemistryAMIChest pain03 medical and health sciences0302 clinical medicinePredictive Value of TestsRule-inInternal medicinemedicineAnimalsHumans030212 general & internal medicineMyocardial infarctionAcute Coronary SyndromeRule-outAnimalbusiness.industrySmall sampleBiomarkerGeneral MedicineACSmedicine.diseasePredictive value of testsRisk stratificationH-FABPBiomarker (medicine)medicine.symptomTroponin CbusinessFatty Acid Binding Protein 3BiomarkersHumanClinical Biochemistry
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?-Carboline binding indicates the presence of benzodiazepine receptor subclasses in the bovine central nervous system

1982

Receptor binding studies were performed with tritiated propyl β-carboline-3-carboxylate ([3H]PrCC), tritiated ethyl β-carboline-3-carboxylate ([3H]ECC), and tritiated flunitrazepam ([3H]FNT) in membrane preparations from different regions of the bovine brain and retina. Specific binding in all regions investigated was associated with benzodiazepine receptor sites. However, not all benzodiazepine receptor sites. However, not all benzodiazepine receptors in the regions investigated as determined by the specific binding of tritiated flunitrazepam ([3H]FNT) are available for [3H]PrCC suggesting that specific [3H]PrCC binding labels only one subclass or subpopulation of the benzodiazepine recept…

medicine.medical_specialtyAdenosineIndolesmedicine.drug_classReceptors DrugCentral nervous systemHippocampusSubstrate Specificitychemistry.chemical_compoundInternal medicinemedicineAnimalsReceptorgamma-Aminobutyric AcidBrain ChemistryPharmacologyBenzodiazepineBinding SitesGABAA receptorChemistrybeta-CarbolineGeneral MedicineReceptors GABA-Amedicine.anatomical_structureEndocrinologyGABAergicCattleFlunitrazepamCarbolinesmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Adrenergic modulation of astroglial phospholipase D activity and cell proliferation.

1999

As phospholipase D (PLD) activation has been associated with mitogenic signalling in several cell types, we tested an association between adrenergic activation of PLD and cellular proliferation in primary cultures of rat cortical astrocytes. In 2-week old cultures, PLD activation by noradrenaline (EC50: 0.49 microM) was inhibited by prazosin, a specific antagonist at alpha1-adrenergic receptors (IC50: 0.23 microM). Adrenergic PLD activation was not affected by genistein, an inhibitor of tyrosine kinases, or by Ro 31-8220, an inhibitor of protein kinase C (PKC), but was dose-dependently depressed in the presence of brefeldin A (1-100 microg/ml), an inhibitor of ARF activation. In experiments…

medicine.medical_specialtyAdrenergic AntagonistsAdrenergicBiologyPharmacologychemistry.chemical_compoundNorepinephrineGTP-Binding ProteinsIsoprenalineInternal medicinemedicineAdrenergic antagonistPrazosinPhospholipase DPhospholipase D activityAnimalsMolecular BiologyProtein kinase CCells CulturedBrefeldin APhospholipase DGeneral NeurosciencePrazosinBrefeldin AAdrenergic AgonistsPropranololRatsReceptors AdrenergicEndocrinologychemistryAlcoholsAstrocyteslipids (amino acids peptides and proteins)Neurology (clinical)Cell DivisionDevelopmental Biologymedicine.drugSignal TransductionBrain research
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Glutamatergic activation of hippocampal phospholipase D: postnatal fading and receptor desensitization.

2002

Abstract: Phospholipase D (PLD) activity was determined in rat hippocampal slices between postnatal days 3 and 35. After birth, basal PLD activity was low and, within 2 weeks, increased to reach a plateau that was maintained up to the adult age. Likewise the response to glutamate developed postnatally to reach a maximum at day 8, but then faded rapidly and was almost absent at day 35. Activation of PLD by 4β-phorbol 12β,13α-dibutyrate (PDB) was independent of age, whereas the effect of aluminum fluoride (AlF4−) increased to a plateau within the first week. At day 8, PLD stimulation by glutamate via metabotropic receptors involved protein kinase C activation, but was independent of Ca2+ infl…

medicine.medical_specialtyAgingGlutamic AcidStimulationBiologyHippocampal formationIn Vitro TechniquesReceptors Metabotropic GlutamateBiochemistryHippocampusCellular and Molecular NeuroscienceGlutamatergicchemistry.chemical_compoundInternal medicinemedicinePhospholipase DCholineAnimalsRats WistarPhospholipase DGlutamate receptorRatsEnzyme Activationenzymes and coenzymes (carbohydrates)Metabotropic receptorEndocrinologychemistryAnimals NewbornMetabotropic glutamate receptorlipids (amino acids peptides and proteins)Journal of neurochemistry
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The metabolism and availability of essential fatty acids in animal and human tissues.

1994

Essential fatty acids (EFA), which are not synthesized in animal and human tissues, belong to the n-6 and n-3 families of polyunsaturated fatty acids (PUFA), derived from linoleic acid (LA, 18:2n-6) and alpha-linolenic acid (LNA, 18:3n-3). Optimal requirements are 3-6% of ingested energy for LA and 0.5-1% for LNA in adults. Requirements in LNA are higher in development. Dietary sources of LA and LNA are principally plants, while arachidonic acid (AA, 20:4n-6) is found in products from terrestrian animals, and eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA) are found in products from marine animals. EFA are principally present in dietary triacylglycerols, which should be hydrolyze…

medicine.medical_specialtyAgingLinolenic acidLinoleic acidBiological AvailabilityBiologyIntestinal absorptionchemistry.chemical_compoundInternal medicine[SDV.BDD] Life Sciences [q-bio]/Development BiologymedicineAnimalsHumansNutritional Physiological Phenomena[SDV.BDLR] Life Sciences [q-bio]/Reproductive Biologychemistry.chemical_classificationFatty Acids EssentialNutritional Requirementsfood and beveragesMetabolismEicosapentaenoic acidDietary FatsHormones[SDV.AEN] Life Sciences [q-bio]/Food and NutritionEndocrinologyBiochemistrychemistryIntestinal AbsorptionDocosahexaenoic acidOrgan Specificitylipids (amino acids peptides and proteins)Arachidonic acidDigestionPolyunsaturated fatty acidReproduction, nutrition, development
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Nutritional Status in Aging and Lung Disease

2016

Abstract Lung diseases exert a negative impact on nutritional status, especially among older patients where aging per se is already associated with relevant changes in nutrient intake, metabolism, and body composition. Both respiratory (increased respiratory work, hypoxia, and local oxidative stress and inflammation) and nonrespiratory (sarcopenia, reduced mitochondrial biogenesis and anabolic hormones, balance of orexigenic/anorexigenic neuropeptides, and systemic inflammation and oxidative stress) contribute to the association between lung diseases and malnutrition. For these reasons, nutritional assessments are of paramount importance in older patients with lung diseases. Current evidenc…

medicine.medical_specialtyAgingPhysiologySettore MED/10 - Malattie Dell'Apparato RespiratorioSystemic inflammationmedicine.disease_causeElderlyOrexigenicInternal medicineVitamin D and neurologyMedicinechemistry.chemical_classificationbusiness.industryMalnutritionHypoxia (medical)medicine.diseaseMalnutritionEndocrinologychemistrySarcopeniaLung diseasemedicine.symptombusinessOxidative stressmedicine.drugPolyunsaturated fatty acid
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