Search results for "ACIDS"

showing 10 items of 3520 documents

Changes in carnitine octanoyltransferase activity induce alteration in fatty acid metabolism

2011

The peroxisomal beta oxidation of very long chain fatty acids (VLCFA) leads to the formation of medium chain acyl-CoAs such as octanoyl-CoA. Today, it seems clear that the exit of shortened fatty acids produced by the peroxisomal beta oxidation requires their conversion into acyl-carnitine and the presence of the carnitine octanoyltransferase (CROT). Here, we describe the consequences of an overexpression and a knock down of the CROT gene in terms of mitochondrial and peroxisomal fatty acids metabolism in a model of hepatic cells. Our experiments showed that an increase in CROT activity induced a decrease in MCFA and VLCFA levels in the cell. These changes are accompanied by an increase in …

CellBiophysicsOxidative phosphorylationBiochemistrychemistry.chemical_compoundPeroxisomesmedicineHumansCarnitineRNA Small InterferingMolecular Biologychemistry.chemical_classificationFatty acid metabolismFatty AcidsHep G2 CellsCell BiologyMetabolismPeroxisomeHEK293 Cellsmedicine.anatomical_structureEnzymeCarnitine AcyltransferaseschemistryBiochemistryGene Knockdown TechniquesHepatic stellate cellOxidation-Reductionmedicine.drugBiochemical and Biophysical Research Communications
researchProduct

Transport of resveratrol, a cancer chemopreventive agent, to cellular targets: plasmatic protein binding and cell uptake

2004

Resveratrol produced by several plants, berries and fruits, including grapes, is one of the best known natural food microcomponents with potent chemopreventive properties towards the most severe contemporary human diseases: cardiovascular sickness, cancer and neurodegenerative pathologies. Demonstration of its mechanism of action also implies the elucidation of the steps of bioavailability and bioabsorption in cells and tissues. In order to estimate the relationships between the amounts of resveratrol taken up by food or drink intake, and the several possible benefits illustrated from in vitro/in vivo experiments and from epidemiological studies, it is essential to demonstrate step by step …

CellPlasma protein bindingPharmacologyResveratrolBiologyBiochemistrychemistry.chemical_compoundIn vivoStilbenesTumor Cells CulturedmedicineAnimalsAnticarcinogenic AgentsHumansAnticarcinogenSerum AlbuminPharmacologyFatty Acidsfood and beveragesBiological TransportBlood ProteinsIn vitromedicine.anatomical_structureBiochemistryMechanism of actionchemistryResveratrolmedicine.symptomIntracellularProtein BindingBiochemical Pharmacology
researchProduct

The emergence of Vibrio pathogens in Europe : ecology, evolution, and pathogenesis (Paris, 11-12th March 2015)

2015

Global change has caused a worldwide increase in reports of Vibrio-associated diseases with ecosystem-wide impacts on humans and marine animals. In Europe, higher prevalence of human infections followed regional climatic trends with outbreaks occurring during episodes of unusually warm weather. Similar patterns were also observed in Vibrio-associated diseases affecting marine organisms such as fish, bivalves and corals. Basic knowledge is still lacking on the ecology and evolutionary biology of these bacteria as well as on their virulence mechanisms. Current limitations in experimental systems to study infection and the lack of diagnostic tools still prevent a better understanding of Vibrio…

Cell- och molekylärbiologilcsh:QR1-502NetworkPACIFIC OYSTERS[ SDV.MP.BAC ] Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriologyglobal warminghuman healthgenome plasticityHORIZONTAL GENE-TRANSFERlcsh:Microbiologyeuropean network/dk/atira/pure/sustainabledevelopmentgoals/zero_hungerOYSTERS CRASSOSTREA-GIGASApplied researchFood securitybiologyEcologyGenome plasticityMARINE PHOTOBACTERIUMHuman health risksaquacultureSECRETION SYSTEMPerspective/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingVibrio-host interactionVIRULENCE FACTORSMicrobiology (medical)570Ecology (disciplines)Social issuesMicrobiology/dk/atira/pure/sustainabledevelopmentgoals/life_below_waterSDG 3 - Good Health and Well-being[SDV.BID.EVO] Life Sciences [q-bio]/Biodiversity/Populations and Evolution [q-bio.PE]14. Life underwaterSDG 14 - Life Below WaterSDG 2 - Zero HungerBacterial diseaseanimal modelGlobal warmingOutbreakBiology and Life Sciencesgenome asticityD-AMINO ACIDSAnimal model; Aquaculture; Bacterial disease; Biotic-abiotic interactions; Genome plasticity; Global warming; Human health; Network; Vibrio-host interaction; Microbiology; Microbiology (medical)interactionsbiology.organism_classificationCLINICAL SOURCES[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriologybacterial diseaseBiotic-abiotic interactionsVibrioDAMSELAE SUBSP DAMSELAE13. Climate actionnetworksCell and Molecular BiologyPHOTOBACTERIUM-DAMSELAE
researchProduct

Novel Lipid and Polymeric Materials as Delivery Systems for Nucleic Acid Based Drugs

2015

Nucleic acid based drugs (NADBs) are short DNA/RNA molecules that include among others, antisense oligonucleotides, aptamers, small interfering RNAs and micro-interfering RNAs. Despite the different mechanisms of actions, NABDs have the ability to combat the effects of pathological gene expression in many experimental systems. Thus, nowadays, NABDs are considered to have a great therapeutic potential, possibly superior to that of available drugs. Unfortunately, however, the lack of effective delivery systems limits the practical use of NABDs. Due to their hydrophilic nature, NABDs cannot efficiently cross cellular membrane; in addition, they are subjected to fast degradation by cellular and…

Cellular membranePolymersAntisense oligonucleotides aptamers carbon nanotubes exososomes liposomes miRNA polymers siRNAAptamerClinical BiochemistryNanotechnologyAnimals; Humans; Lipids; Nanoparticles; Nanotubes Carbon; Nucleic Acids; Polymers; Drug Delivery SystemsBiologyNanoparticleDrug Delivery SystemsNucleic AcidsAnimalsHumansAvailable drugsPolymerPharmacologyNanotubesNucleic AcidAnimalNanotubes CarbonCarbon chemistryRNALipidLipidsCarbonSettore CHIM/09 - Farmaceutico Tecnologico ApplicativoAntisense oligonucleotidesNucleic acidNanoparticlesHuman
researchProduct

Cannabinoids mediate analgesia largely via peripheral type 1 cannabinoid receptors in nociceptors

2007

Although endocannabinoids constitute one of the first lines of defense against pain, the anatomical locus and the precise receptor mechanisms underlying cannabinergic modulation of pain are uncertain. Clinical exploitation of the system is severely hindered by the cognitive deficits, memory impairment, motor disturbances and psychotropic effects resulting from the central actions of cannabinoids. We deleted the type 1 cannabinoid receptor (CB1) specifically in nociceptive neurons localized in the peripheral nervous system of mice, preserving its expression in the CNS, and analyzed these genetically modified mice in preclinical models of inflammatory and neuropathic pain. The nociceptor-spec…

Central Nervous SystemCannabinoid receptorCannabinoid Receptor Modulatorsmedicine.medical_treatmentCentral nervous systemPharmacologyBiologyArticleMiceReceptor Cannabinoid CB1Ganglia SpinalCannabinoid Receptor ModulatorsPeripheral Nervous SystemmedicineAnimalsNeurons AfferentAllelesDNA PrimersMice KnockoutNerve Fibers UnmyelinatedCannabinoidsGeneral NeuroscienceNociceptorsPeripheral Nervous System DiseasesEndocannabinoid systemElectrophysiologyMice Inbred C57BLmedicine.anatomical_structurenervous systemPeripheral nervous systemNeuropathic painNociceptorlipids (amino acids peptides and proteins)CannabinoidAnalgesiaNeuroscience
researchProduct

Direct suppression of CNS autoimmune inflammation via the cannabinoid receptor CB1 on neurons and CB2 on autoreactive T cells.

2007

The cannabinoid system is immunomodulatory and has been targeted as a treatment for the central nervous system (CNS) autoimmune disease multiple sclerosis. Using an animal model of multiple sclerosis, experimental autoimmune encephalomyelitis (EAE), we investigated the role of the CB(1) and CB(2) cannabinoid receptors in regulating CNS autoimmunity. We found that CB(1) receptor expression by neurons, but not T cells, was required for cannabinoid-mediated EAE suppression. In contrast, CB(2) receptor expression by encephalitogenic T cells was critical for controlling inflammation associated with EAE. CB(2)-deficient T cells in the CNS during EAE exhibited reduced levels of apoptosis, a higher…

Central Nervous SystemCannabinoid receptorEncephalomyelitis Autoimmune Experimentalmedicine.medical_treatmentEncephalomyelitisT-LymphocytesInflammationApoptosisMice TransgenicBiologyGeneral Biochemistry Genetics and Molecular BiologyReceptor Cannabinoid CB2MiceReceptor Cannabinoid CB1medicineCannabinoid receptor type 2AnimalsCell ProliferationDNA PrimersAutoimmune diseaseNeuronsExperimental autoimmune encephalomyelitisGeneral Medicinemedicine.diseaseEndocannabinoid systemImmunohistochemistryImmunologyEncephalitislipids (amino acids peptides and proteins)Cannabinoidmedicine.symptomNature medicine
researchProduct

AAV vector-mediated overexpression of CB1 cannabinoid receptor in pyramidal neurons of the hippocampus protects against seizure-induced excitoxicity.

2010

The CB1 cannabinoid receptor is the most abundant G-protein coupled receptor in the brain and a key regulator of neuronal excitability. There is strong evidence that CB1 receptor on glutamatergic hippocampal neurons is beneficial to alleviate epileptiform seizures in mouse and man. Therefore, we hypothesized that experimentally increased CB1 gene dosage in principal neurons would have therapeutic effects in kainic acid (KA)-induced hippocampal pathogenesis. Here, we show that virus-mediated conditional overexpression of CB1 receptor in pyramidal and mossy cells of the hippocampus is neuroprotective and moderates convulsions in the acute KA seizure model in mice. We introduce a recombinant a…

Central Nervous SystemCannabinoid receptormedicine.medical_treatmentHippocampuslcsh:MedicineHippocampal formationHippocampuschemistry.chemical_compoundMiceReceptor Cannabinoid CB1Neurobiology of Disease and RegenerationTransgeneslcsh:ScienceNeuronsRecombination GeneticMultidisciplinaryBehavior AnimalNeuromodulationmusculoskeletal neural and ocular physiologyfood and beveragesNeurochemistryGenomicsGene TherapyDependovirusEndocannabinoid systemCell biologyFunctional GenomicsNeurologyHomeostatic MechanismsMedicinelipids (amino acids peptides and proteins)Viral VectorsNeurochemicalsGenetic EngineeringResearch ArticleBiotechnologyKainic acidGenetic VectorsGreen Fluorescent ProteinsNeurophysiologyBiologyMicrobiologyVector BiologyGlutamatergicGenomic MedicineSeizuresmedicineGeneticsAnimalsBiologyEpilepsyIntegrasesDentate gyruslcsh:RMolecular biologyMice Inbred C57BLchemistryGene Expression Regulationnervous systemGenetics of DiseaseSynapseslcsh:QCannabinoidGene FunctionMolecular NeuroscienceAnimal GeneticsTransgenicsNeuroscienceEndocannabinoidsPLoS ONE
researchProduct

Improved synthesis and in vitro evaluation of the cytotoxic profile of oxysterols oxidized at C4 (4α- and 4β-hydroxycholesterol) and C7 (7-ketocholes…

2013

Whereas the biological activities of oxysterols oxidized at C7 (7-ketocholesterol (7KC), 7β-hydroxycholesterol (7β-OHC), 7α-hydroxycholesterol (7α-OHC)) are well documented, those of oxysterols oxidized at C4 (4β-hydroxycholesterol (4β-OHC), 4α-hydroxycholesterol (4α-OHC)) are not well known, especially on the cells of the central nervous system. Therefore, an improved methodology has been validated for 4β-OHC and 4α-OHC synthesis, and the effects on cell viability and cell growth of these molecules were studied on immortalized, tumoral and normal brain cells (158N, C6 and SK-N-BE cells, and mixed primary cultures of astrocytes and oligodendrocytes). Whereas inhibition of cell growth with 7…

Central Nervous SystemCell SurvivalCentral nervous systemMolecular ConformationCell LineStructure-Activity RelationshipDrug Discoverypolycyclic compoundsmedicineHumansCytotoxic T cellViability assayKetocholesterolsCell ProliferationPharmacologyDose-Response Relationship DrugChemistryCell growthOrganic ChemistryGeneral MedicineHydroxycholesterolsIn vitroSterolsOn cellsmedicine.anatomical_structureBiochemistryToxicitylipids (amino acids peptides and proteins)sense organs4β hydroxycholesterolOxidation-ReductionEuropean Journal of Medicinal Chemistry
researchProduct

Polysialic acid is required for dopamine D2 receptor-mediated plasticity involving inhibitory circuits of the rat medial prefrontal cortex.

2011

Decreased expression of dopamine D2 receptors (D2R), dysfunction of inhibitory neurotransmission and impairments in the structure and connectivity of neurons in the medial prefrontal cortex (mPFC) are involved in the pathogenesis of schizophrenia and major depression, but the relationship between these changes remains unclear. The polysialylated form of the neural cell adhesion molecule (PSA-NCAM), a plasticity-related molecule, may serve as a link. This molecule is expressed in cortical interneurons and dopamine, via D2R, modulates its expression in parallel to that of proteins related to synapses and inhibitory neurotransmission, suggesting that D2R-targeted antipsychotics/antidepressants…

Central Nervous SystemMaleAnatomy and Physiologylcsh:MedicineRats Sprague-DawleyNeural PathwaysMolecular Cell BiologyNeurobiology of Disease and Regenerationlcsh:SciencePsychiatryMicroscopy ConfocalNeuronal PlasticityMultidisciplinaryNeuronal MorphologybiologyGlutamate Decarboxylasemusculoskeletal neural and ocular physiologyNeurotransmittersAnatomyImmunohistochemistryMental Healthmedicine.anatomical_structureNeurologyDopamine AgonistsMedicineNcamResearch Articlemedicine.drugNeural NetworksInterneuronSynaptophysinNeurophysiologyPrefrontal CortexNeuropsychiatric DisordersNeural Cell Adhesion Molecule L1NeurotransmissionNeurological SystemNeuropharmacologyDopamineDopamine receptor D2NeuroplasticityCell AdhesionNeuropilmedicineAnimalsBiologyMood DisordersReceptors Dopamine D2lcsh:RRatsNeuroanatomynervous systemCellular NeuroscienceSynapsesSchizophreniaSialic Acidsbiology.proteinNeural cell adhesion moleculelcsh:QNeuroscienceParvalbuminNeurosciencePLoS ONE
researchProduct

Host microbiota constantly control maturation and function of microglia in the CNS.

2015

As the tissue macrophages of the CNS, microglia are critically involved in diseases of the CNS. However, it remains unknown what controls their maturation and activation under homeostatic conditions. We observed substantial contributions of the host microbiota to microglia homeostasis, as germ-free (GF) mice displayed global defects in microglia with altered cell proportions and an immature phenotype, leading to impaired innate immune responses. Temporal eradication of host microbiota severely changed microglia properties. Limited microbiota complexity also resulted in defective microglia. In contrast, recolonization with a complex microbiota partially restored microglia features. We determ…

Central Nervous SystemMaleCellGut–brain axis610 Medicine & healthBiologydigestive systemReceptors G-Protein-CoupledMiceImmunitymedicineAnimalsHomeostasis10239 Institute of Laboratory Animal ScienceReceptorInnate immune systemMicrogliaGeneral NeuroscienceMicrobiota2800 General NeuroscienceFatty Acids VolatilePhenotypeImmunity InnateMice Inbred C57BLmedicine.anatomical_structurenervous systemImmunology570 Life sciences; biology590 Animals (Zoology)FemaleMicrogliaNeuroscienceHomeostasisNature neuroscience
researchProduct