Search results for "ACTIVATION"

showing 10 items of 2079 documents

Human Epidermal Langerhans Cells Replenish Skin Xenografts and Are Depleted by Alloreactive T Cells In Vivo

2011

Abstract Epidermal Langerhans cells (LC) are potent APCs surveying the skin. They are crucial regulators of T cell activation in the context of inflammatory skin disease and graft-versus-host disease (GVHD). In contrast to other dendritic cell subtypes, murine LC are able to reconstitute after local depletion without the need of peripheral blood-derived precursors. In this study, we introduce an experimental model of human skin grafted to NOD-SCID IL2Rγnull mice. In this model, we demonstrate that xenografting leads to the transient loss of LC from the human skin grafts. Despite the lack of a human hematopoietic system, human LC repopulated the xenografts 6 to 9 wk after transplantation. By…

MalePathologymedicine.medical_specialtyT cellCellular differentiationTransplantation HeterologousImmunologyGraft vs Host DiseaseMice TransgenicHuman skinMice SCIDCD8-Positive T-LymphocytesBiologyLymphocyte ActivationMiceCell MovementMice Inbred NODIn vivomedicineAnimalsHumansImmunology and AllergyCells CulturedCell ProliferationMice KnockoutCell Deathintegumentary systemEpidermis (botany)Cell DifferentiationSkin TransplantationDendritic cellTransplantationDisease Models AnimalHaematopoiesismedicine.anatomical_structureLangerhans CellsCancer researchFemaleEpidermisThe Journal of Immunology
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Experimental autoimmune hepatitis: Disease induction, time course and t-cell reactivity

1990

This study describes a murine model of autoimmune hepatitis: experimental autoimmune hepatitis. Experimental autoimmune hepatitis could be induced most effectively in male C57BL/6 mice by intraperitoneal immunization with the 100,000 g supernatant of syngeneic liver homogenate (S-100) in complete Freund's adjuvant. BALB/C and C3H mice were less susceptible than C57BL/6 mice. Experimental autoimmune hepatitis could not be induced in Lewis rats. Intraperitoneal immunization was more effective than intramuscular or subcutaneous injections, and the amount of protein administered above a threshold was of little influence. A single intraperitoneal injection of S-100 in complete Freund's adjuvant …

MalePathologymedicine.medical_specialtyTime FactorsNecrosisT-Lymphocytesmedicine.medical_treatmentFreund's AdjuvantIntraperitoneal injectionAutoimmune hepatitisLymphocyte ActivationAutoimmune DiseasesHepatitisPathogenesisMiceNecrosisSpecies SpecificitymedicineAnimalsAutoimmune diseaseHepatitisMice Inbred BALB CMice Inbred C3HHepatologybusiness.industryLiver cellS100 Proteinsmedicine.diseaseMice Inbred C57BLLiverFreund's adjuvantImmunologyFemalemedicine.symptombusinessHepatology
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A 7-Year-Old Boy and a 14-Year-Old Girl Initially Diagnosed with Toxic Shock Syndrome and Tested Positive for SARS-CoV-2 Infection, Supporting a Diag…

2021

Case series Patients: Male, 7-year-old • Female, 14-year-old Final Diagnosis: Multisystem inflammatory syndrome in children (MIS-C) Symptoms: Muscular weakness • shock Medication: — Clinical Procedure: — Specialty: Critical Care Medicine • Infectious Diseases • Pediatrics and Neonatology • Rheumatology Objective: Unusual clinical course Background: Multisystem inflammatory syndrome in children (MIS-C) has recently been described in children infected with severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). This report describes 2 children with MIS-C who were initially diagnosed with toxic shock syndrome but who tested positive for SARS-CoV-2 infection on reverse transcription-polym…

MalePediatricsmedicine.medical_specialtyCOVID-19 Related Immunoglobulins Intravenous Mucocutaneous Lymph Node Syndrome Pediatric Multisystem Inflammatory Disease Adolescent COVID-19 Testing Child Female Humans Male Pandemics SARS-CoV-2 Systemic Inflammatory Response Syndrome COVID-19 Shock SepticAdolescentMucocutaneous Lymph Node SyndromeTachypneaPericardial effusionCOVID-19 TestingOliguriamedicineHumansPediatric Multisystem Inflammatory Disease COVID-19 RelatedChildPandemicsbusiness.industrySARS-CoV-2Toxic shock syndromeImmunoglobulins IntravenousCOVID-19General MedicineArticlesmedicine.diseaseShock SepticSystemic Inflammatory Response SyndromeSystemic inflammatory response syndromePneumoniaMethylprednisoloneMacrophage activation syndromeFemalemedicine.symptombusinessmedicine.drugThe American Journal of Case Reports
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NEXMIF encephalopathy: an X-linked disorder with male and female phenotypic patterns

2021

Contains fulltext : 231688.pdf (Publisher’s version ) (Closed access) PURPOSE: Pathogenic variants in the X-linked gene NEXMIF (previously KIAA2022) are associated with intellectual disability (ID), autism spectrum disorder, and epilepsy. We aimed to delineate the female and male phenotypic spectrum of NEXMIF encephalopathy. METHODS: Through an international collaboration, we analyzed the phenotypes and genotypes of 87 patients with NEXMIF encephalopathy. RESULTS: Sixty-three females and 24 males (46 new patients) with NEXMIF encephalopathy were studied, with 30 novel variants. Phenotypic features included developmental delay/ID in 86/87 (99%), seizures in 71/86 (83%) and multiple comorbidi…

MalePediatricsmedicine.medical_specialtyINTELLECTUAL DISABILITYAutism Spectrum DisorderEncephalopathyNerve Tissue ProteinsILAE COMMISSIONMOSAICISMEpilepsy/geneticsCLASSIFICATIONEpilepsyBrain Diseases/geneticsGenes X-LinkedSeizuresIntellectual disabilityGenotypemedicineHumansdevelopmental and epileptic encephalopathyMYOCLONIAAtonic seizureGenetics (clinical)Brain Diseasesddc:618Neurodevelopmental disorders Donders Center for Medical Neuroscience [Radboudumc 7]KIAA2022business.industryMUTATIONSmedicine.diseasePhenotypeAutism Spectrum Disorder/geneticsGenes X-Linked/geneticsAutism spectrum disorderintellectual disabilityNEXMIFAutismepilepsyFemaleINACTIVATIONHuman medicineSeizures/geneticsbusinessPOSITION PAPERGenetics in Medicine
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RAPID AND EFFICIENT ANTIGEN PROCESSING AND PRESENTATION OF A PROTECTIVE AND IMMUNODOMINANT HLA-B*27-RESTRICTED HEPATITIS C VIRUS-SPECIFIC CD8+T CELL …

2012

HLA-B*27 exerts protective effects in hepatitis C virus (HCV) and human immunodeficiency virus (HIV) infections. While the immunological and virological features of HLA-B*27-mediated protection are not fully understood, there is growing evidence that the presentation of specific immunodominant HLA-B*27-restricted CD8+ T-cell epitopes contributes to this phenomenon in both infections. Indeed, protection can be linked to single immunodominant CD8+ T-cell epitopes and functional constraints on escape mutations within these epitopes. To better define the immunological mechanisms underlying HLA-B*27-mediated protection in HCV infection, we analyzed the functional avidity, functional profile, ant…

MaleProteasome Endopeptidase ComplexQH301-705.5Immune CellsAntigen presentationImmunologyAntigen-Presenting CellsAntigen Processing and RecognitionHepacivirusBiologyAdaptive ImmunityCD8-Positive T-LymphocytesMicrobiologyEpitopeImmune ActivationMajor Histocompatibility ComplexEpitopesImmune systemVirologyGeneticsCytotoxic T cellHumansAvidityBiology (General)Antigen-presenting cellMolecular BiologyBiologyAntigen PresentationLinear epitopeAntigen processingT CellsImmunityRC581-607VirologyHepatitis CHLA-B AntigensImmunologyProteolysisQR180ParasitologyFemaleImmunologic diseases. AllergyHepatitis C AntigensResearch Article
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Association of increased CCL5 and CXCL7 chemokine expression with neutrophil activation in severe stable COPD

2009

BACKGROUND: Increased numbers of activated neutrophils have been reported in the bronchial mucosa of patients with stable chronic obstructive pulmonary disease (COPD), particularly in severe disease. OBJECTIVES: To investigate the expression of neutrophilic chemokines and adhesion molecules in bronchial biopsies from patients with stable COPD of different severity (GOLD stages I-IV) compared with age-matched control subjects, smokers with normal lung function and never smokers. METHODS: The expression of CCL5, CXCL1, 5, 6, 7 and 8, CXCR1, CXCR2, CD11b and CD44 was measured in the bronchial mucosa using immunohistochemistry, confocal immunofluorescence, real-time quantitative polymerase chai…

MalePulmonary and Respiratory MedicineChemokinePathologymedicine.medical_specialtyCOPD neutrophils bronchial mucosa CCL5 CXCL7BronchiRespiratory MucosaGranulocyteNeutrophil ActivationCCL5Pulmonary Disease Chronic ObstructiveneutrophilsSubmucosaCOPDHumansMedicineCXC chemokine receptorsChemokine CCL5AgedCOPDbronchial mucosaCCL5biologySettore BIO/16 - Anatomia UmanaCD11 Antigensbusiness.industryCD44Epithelial CellsMiddle Agedrespiratory systemmedicine.diseaseRespiratory Function Testsrespiratory tract diseasesCXCL1Hyaluronan Receptorsmedicine.anatomical_structureAcute DiseaseImmunologyCXCL7biology.proteinFemaleLeukocyte ElastasebusinessCOPD; neutrophils; bronchial mucosa; CCL5; CXCL7Chemokines CXCCOPD CCL5CXCL7NEUTROPHILThorax
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β2Integrin deficiency yields unconventional double-negative T cells distinct from mature classical natural killer T cells in mice

2009

Expressed on leucocytes, beta(2) integrins (CD11/CD18) are specifically involved in leucocyte function. Using a CD18-deficient (CD18(-/-)) mouse model, we here report on their physiological role in lymphocyte differentiation and trafficking. CD18(-/-) mice present with a defect in the distribution of lymphocytes with highly reduced numbers of naïve B and T lymphocytes in inguinal and axillary lymph nodes. In contrast, cervical lymph nodes were fourfold enlarged harbouring unconventional T-cell receptor-alphabeta (TCR-alphabeta) and TCR-gammadelta CD3(+) CD4(-) CD8(-) (double-negative; DN) T cells that expanded in situ. Using adoptive transfer experiments, we found that these cells did not h…

MaleReceptors Antigen T-Cell alpha-betaT cellImmunologyCD1chemical and pharmacologic phenomenaBiologyLymphocyte ActivationImmunophenotypingMiceInterleukin 21T-Lymphocyte SubsetsImmune TolerancemedicineAnimalsImmunology and AllergyCytotoxic T cellIL-2 receptorAntigen-presenting cellLungLymphatic DiseasesMice KnockoutB-LymphocytesZAP70Receptors Antigen T-Cell gamma-deltahemic and immune systemsOriginal ArticlesNatural killer T cellAdoptive TransferMolecular biologyCoculture TechniquesChemotaxis Leukocytemedicine.anatomical_structureLiverCD18 AntigensImmunologyNatural Killer T-CellsFemaleLymph NodesLymphocyte Culture Test MixedImmunology
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Analysis of memory and effector CD8+ T cell subsets in chronic graft-versus-host disease.

2009

In humans, the selective depletion of CD8+ cells may prevent GVHD after allogeneic transplantation. These cells can infiltrate and damage target tissues. It is of interest to investigate the phenotypical characteristics and cytotoxic properties of the different CD8+ subsets in cGVHD patients. In a preliminary study we found that patients with cGVHD had a markedly elevated percentage of peripheral blood CCR7−/CD45RA+ cells compared to patients without cGVHD; conversely, the CCR7+/CD45RA+ subsets of CD8+ cells was significantly decreased. In this study, we report in depth on the phenotype of effector T cell subsets in cGVHD patients, as well as their proliferative capability, cytotoxic prope…

MaleReceptors CCR7T cellImmunologyGraft vs Host DiseaseC-C chemokine receptor type 7CD8-Positive T-LymphocytesLymphocyte ActivationGranzymesimmune system diseasesmedicineImmunology and AllergyCytotoxic T cellHumansAgedPharmacologybiologyEffectorChemistryPerforinMiddle Agedmedicine.diseaseGraft-versus-host diseasemedicine.anatomical_structureGranzymePerforinImmunologyChronic Diseasebiology.proteinLeukocyte Common AntigensFemaleImmunologic MemoryCD8International journal of immunopathology and pharmacology
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Disulfide bridge formation between C1q and IgG in vitro.

1990

The globular heads of C1q are known to possess free-SH groups. Here we show that these groups, which are concealed in the native molecule, are exposed by interaction of C1q with dialysis membrane. During iodination, I+ and I2 oxidize these sulfhydryls to produce disulfide-linked C1q aggregates. Approximately 15% of C1q bound to immunoglobulin aggregates is resistant to high conductivity elution and reducing agent is required to release it. These data show that dialysis, adsorption to Ig and iodination of C1q result in structural and functional changes in the molecule, and suggest a mechanism by which these changes occur. Disulfide bridging between C1q and IgG in vitro suggests that this may…

MaleReducing agentImmunologyGuinea Pigschemical and pharmacologic phenomenaBiologyIn Vitro Techniquesurologic and male genital diseasesDialysis tubingfluids and secretionsimmune system diseasesImmunology and AllergyAnimalsSulfhydryl Compoundsskin and connective tissue diseasesComplement C1qComplement ActivationGel electrophoresisComplement C1qIn vitroBiochemistryImmunoglobulin Gbiology.proteinElectrophoresis Polyacrylamide GelFemaleAntibodyDialysis (biochemistry)CysteineEuropean journal of immunology
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Retinal oxidation, apoptosis and age- and sex-differences in the mnd mutant mouse, a model of neuronal ceroid lipofuscinosis

2004

Retinal degeneration is an early and progressive event in many forms of neuronal ceroid lipofuscinoses (NCLs), a heterogeneous group of neurodegenerative disorders with unknown pathogenesis. We here used the mutant motor neuron degeneration (mnd) mouse, a late-infantile NCL variant, to investigate the retinal oxidative state and apoptotic cell death as a function of age and sex. Total superoxide dismutase (SOD) activities and thiobarbituric acid-reactive substance (TBARS) levels revealed progressive increases in retinal oxyradicals and lipid peroxides of mnd mice of both sexes. Female mnd retinas showed a higher oxidation rate and consistently exhibited the 4-hydroxy-2-nonenal (4-HNE)-adduc…

MaleRetinal degenerationPathologymedicine.medical_specialtyApoptosisBiologymedicine.disease_causeThiobarbituric Acid Reactive SubstancesRetinaMiceMice Neurologic Mutantschemistry.chemical_compoundSex FactorsNeuronal Ceroid-LipofuscinosesIn Situ Nick-End LabelingmedicineAnimalsOuter nuclear layerMolecular BiologyAldehydesRetinaTUNEL assayLipid peroxideCaspase 3Superoxide DismutaseGeneral NeuroscienceRetinal DegenerationRetinalmedicine.diseaseImmunohistochemistryEnzyme ActivationMice Inbred C57BLDisease Models AnimalOxidative Stressmedicine.anatomical_structureBiochemistrychemistryCaspasesFemaleNeuronal ceroid lipofuscinosisNeurology (clinical)Oxidation-ReductionOxidative stressDevelopmental BiologyBrain Research
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