Search results for "ACTIVATION"

showing 10 items of 2079 documents

Endotoxin accelerates atherosclerosis independent of complement activation

2008

a Central Laboratory Animal Facility b Institute of Clinical Chemistry and Laboratory Medicine c Institute of Medical Microbiology and Hygiene, Johannes Gutenberg University, Mainz, Germany d Klinik fur Gefaschirurgie und Nierentransplantation, Heinrich Heine Universitatsklinik, Dusseldorf, Germany e Zentrum fur Medizin und Biowissenschaften, Forschungszentrum, Borstel, Germany f Centre Hospitalier Universitaire Vaudois, Lausanne, Switzerland g Department of Molecular Pathology, Graduate School of Medicine and Engineering, University of Yamanashi, Japan

MaleTime FactorsLibrary scienceHyperlipidemiasHematologyBiologyAtherosclerosisComplement C6Central laboratoryEndotoxinsC-Reactive ProteinCholesterolImmunologyAnimalsHumansFemaleRabbitsComplement ActivationTriglyceridesAnimal facilityThrombosis Research
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Ipsilateral corticomotor responses are confined to the homologous muscle following cross-education of muscular strength

2017

Cross-education of strength occurs when strength-training 1 limb increases the strength of the untrained limb and is restricted to the untrained homologous muscle. Cortical circuits located ipsilateral to the trained limb might be involved. We used transcranial magnetic stimulation (TMS) to determine the corticomotor responses from the untrained homologous (biceps brachii) and nonhomologous (flexor carpi radialis) muscle following strength-training of the right elbow flexors. Motor evoked potentials were recorded from the untrained left biceps brachii and flexor carpi radialis during a submaximal contraction from 20 individuals (10 women, 10 men; aged 18–35 years; training group, n = 10; c…

MaleTime FactorsPhysiologyEndocrinology Diabetes and MetabolismCross-activationPhysical strengthRandom Allocation0302 clinical medicineCross activationMedicineInhibitionMotor NeuronsNutrition and DieteticsMotor CortexGeneral MedicineAnatomymusculoskeletal systemTranscranial Magnetic StimulationestotTreatment OutcomeFemalelihaskuntoMuscle ContractionAdultAgonistmedicine.medical_specialtyAdolescentmedicine.drug_classAgonistCross educationYoung Adult03 medical and health sciencesPhysical medicine and rehabilitationPhysiology (medical)Homologous chromosomeHumansMuscle StrengthMuscle SkeletalSynergistCortical circuitsExcitabilityElectromyographybusiness.industryNeural InhibitionResistance Training030229 sport sciencesEvoked Potentials MotorC600raajatbody regionsbusiness030217 neurology & neurosurgerylihasvoimaApplied Physiology, Nutrition, and Metabolism
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Human somatic cells subjected to genetic induction with six germ line-related factors display meiotic germ cell-like features

2016

AbstractThe in vitro derivation of human germ cells has attracted interest in the last years, but their direct conversion from human somatic cells has not yet been reported. Here we tested the ability of human male somatic cells to directly convert into a meiotic germ cell-like phenotype by inducing them with a combination of selected key germ cell developmental factors. We started with a pool of 12 candidates that were reduced to 6, demonstrating that ectopic expression of the germ line-related genes PRDM1, PRDM14, LIN28A, DAZL, VASA and SYCP3 induced direct conversion of somatic cells (hFSK (46, XY) and hMSC (46, XY)) into a germ cell-like phenotype in vitro. Induced germ cell-like cells …

MaleTranscriptional Activation0301 basic medicineSomatic cellCellular differentiationForeskinCell Culture TechniquesMice NudeEmbryoid bodyHaploidyBiologyArticleGermline03 medical and health sciences0302 clinical medicinemedicineAnimalsHumansBusulfanCells CulturedGerm plasmGeneticsMultidisciplinaryCell DifferentiationDiploidySpermatogoniaCell biologyMeiosisGerm Cells030104 developmental biologymedicine.anatomical_structureGerm line developmentTranscriptomeReprogramming030217 neurology & neurosurgeryGerm cell
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Downregulation of β2-microglobulin in human cord blood somatic stem cells after transplantation into livers of SCID-mice: an escape mechanism of stem…

2002

Adherently growing, non-hematopoietic somatic stem cells isolated from human cord blood were stained with the fluorescent dye PKH26 and transplanted into livers of SCID-mice to examine a possible cell fate transition. Already 7 days after transplantation stem cells were well integrated into the liver tissue. Human albumin that was not expressed by the stem cells before transplantation was detectable in the host's livers after injection of cord blood stem cells. Human alpha1-antitrypsin was detectable in stem cells already before transplantation and remained positive in the mouse liver. The most interesting observation in this study was the downregulation of human beta2-microglobulin (beta2M…

MaleTranscriptional ActivationBiophysicsDown-RegulationMice SCIDBiologyBiochemistryMiceAlbuminsAnimalsHumansGene SilencingRNA MessengerMolecular BiologyAgedStem cell transplantation for articular cartilage repairInduced stem cellsStem CellsHematopoietic Stem Cell TransplantationAmniotic stem cellsCell BiologyFetal BloodImmunohistochemistryMolecular biologyEndothelial stem cellLiverAmniotic epithelial cellsCord bloodTransplantation ToleranceStem cellbeta 2-MicroglobulinStem Cell TransplantationAdult stem cellBiochemical and Biophysical Research Communications
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Chronic lithium salt treatment reduces CRE/CREB-directed gene transcription and reverses its upregulation by chronic psychosocial stress in transgeni…

2007

The molecular mechanism of action of the mood stabilizer lithium is assumed to involve changes in gene expression leading to neuronal adaptation. The transcription factor CREB (cAMP-responsive element binding protein) regulates the expression of many genes and has been implicated in important brain functions and the action of psychogenic agents. We here investigated the effect of lithium on cAMP-responsive element (CRE)/CREB-mediated gene transcription in the brain, using transgenic reporter mice that express the luciferase reporter gene under the control of four copies of the rat somatostatin gene promoter CRE. Chronic (21 days) but not acute (24 h) treatment with lithium (7.5 mmol/kg) sig…

MaleTranscriptional ActivationBipolar DisorderTransgeneDown-RegulationMice TransgenicCREBDrug Administration Schedule03 medical and health sciencesGlycogen Synthase Kinase 3Mice0302 clinical medicineGSK-3Transcription (biology)Antimanic AgentsGenes ReporterGene expressionAnimalsPhosphorylationProtein kinase ACyclic AMP Response Element-Binding ProteinSocial BehaviorTranscription factor030304 developmental biologyPharmacology0303 health sciencesReporter genebiologyBehavior AnimalBrainMolecular biologyUp-RegulationPsychiatry and Mental healthDisease Models AnimalGene Expression RegulationChronic Diseasebiology.proteinLithium Compounds030217 neurology & neurosurgeryStress PsychologicalAdenylyl CyclasesSignal TransductionNeuropsychopharmacology : official publication of the American College of Neuropsychopharmacology
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Frequent nonrandom activation of germ-line genes in human cancer.

2004

Abstract The growing class of cancer/germ-line genes is characterized by a unique expression pattern with transcription restricted to germ cells and cancer cells. It is not known which fraction of germ-line genes is ectopically activated in tumor cells and whether this fraction displays common features as compared with strictly germ-line genes remaining silent in cancer. Using an unbiased genome-wide scanning approach, representative samples of both cancer/germ-line genes as well as strictly germ-line-specific genes were determined. Comparative analysis disclosed highly significant diametric characteristics for these two categories of genes with regard to sex specificity, developmental stag…

MaleTranscriptional ActivationCancer ResearchBiologyGermlinechemistry.chemical_compoundTranscription (biology)NeoplasmsTestismedicineHumansEpigeneticsGeneRegulation of gene expressionGeneticsReverse Transcriptase Polymerase Chain ReactionOvaryCancerGene Expression Regulation Developmentalmedicine.diseaseGene Expression Regulation NeoplasticGerm CellsOncologychemistryCancer cellFemaleDNACancer research
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The human X chromosome is enriched for germline genes expressed in premeiotic germ cells of both sexes.

2006

The role of X-chromosomal genes in spermatogenesis has been subject to a number of studies in different organisms. Recently, it was proposed that the X chromosome has a predominant role in premeiotic stages of mammalian spermatogenesis. We analyzed the expression of a representative set of 17 X-linked and 48 autosomal germline-restricted genes in different stages of human germ cell development. In accordance with data from other species, we show that the human X chromosome is indeed significantly enriched for genes activated in premeiotic stages of spermatogenesis. In contrast to recent studies, however, we found that expression of these genes is not restricted to spermatogenesis, but is ac…

MaleTranscriptional ActivationGene DosageBiologyChromatin remodelingGametogenesisOogenesisGeneticsmedicineChromosomes HumanCluster AnalysisHumansSpermatogenesisMolecular BiologyGeneSkewed X-inactivationGenetics (clinical)X chromosomeCells CulturedRegulation of gene expressionGeneticsChromosomes Human XDosage compensationChromosome MappingGeneral MedicineDNA MethylationMeiosismedicine.anatomical_structureGerm CellsGene Expression RegulationDNA methylationFemaleGerm cellHuman molecular genetics
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Mandibular-pelvic-patellar syndrome (mpp) is a novel pitx1-related disorder due to alteration of pitx1 transactivation ability

2020

International audience; PITX1 is a homeobox transcription factor essential for hindlimb morphogenesis. Two PITX1-related human disorders have been reported to date: PITX1 ectopic expression causes Liebenberg syndrome, characterized by malformation of upper limbs showing a "lower limb" appearance; PITX1 deletions or missense variation cause a syndromic picture including clubfoot, tibial hemimelia, and preaxial polydactyly. We report two novel PITX1 missense variants, altering PITX1 transactivation ability, in three individuals from two unrelated families showing a distinct recognizable autosomal dominant syndrome, including first branchial arch, pelvic, patellar, and male genital abnormaliti…

MaleTranscriptional ActivationPathologymedicine.medical_specialtyHindlimb morphogenesis[SDV]Life Sciences [q-bio]Mutation MissensepelvisBiologyPierre-Robin03 medical and health sciencesTransactivationGeneticsmedicineMissense mutationAnimalsHumansPaired Box Transcription FactorsChildPITX1Genetics (clinical)030304 developmental biologyMice Knockoutcleft palate0303 health sciencesBone Diseases Developmental030305 genetics & heredityPreaxial polydactylyInfant NewbornLiebenberg syndromemedicine.disease3. Good healthgenitalpatella[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsChild PreschoolHomeoboxEctopic expressionHaploinsufficiency
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Estradiol induces endothelial cell migration and proliferation through estrogen receptor-enhanced RhoA/ROCK pathway

2010

Migration and proliferation of endothelial cells are involved in re-endothelialization and angiogenesis, two important cardiovascular processes that are increased in response to estrogens. RhoA, a small GTPase which controls multiple cellular processes, is involved in the control of cell migration and proliferation. Our aim was to study the role of RhoA on estradiol-induced migration and proliferation and its dependence on estrogen receptors activity. Human umbilical vein endothelial cells were stimulated with estradiol, in the presence or absence of ICI 182780 (estrogen receptors antagonist) and Y-27632 (Rho kinase inhibitor). Estradiol increased Rho GEF-1 gene expression and RhoA (gene an…

MaleTranscriptional ActivationRHOAAngiogenesismedicine.drug_classEstrogen receptorCell Cycle ProteinsBiochemistryUmbilical CordEndocrinologyCell MovementmedicineHumansReceptorMolecular BiologyCells CulturedCell ProliferationEnzyme Assaysrho-Associated KinasesEstradiolbiologyChemistryEndothelial CellsCell migrationUp-RegulationCell biologyEndothelial stem cellReceptors EstrogenRho kinase inhibitorEstrogenCancer researchbiology.proteinFemalerhoA GTP-Binding Proteinhormones hormone substitutes and hormone antagonistsSignal TransductionMolecular and Cellular Endocrinology
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Xanthine oxidase-induced oxidative stress causes activation of NF-κB and inflammation in the liver of type I diabetic rats

2009

We previously showed that xanthine oxidase activity increases in type I diabetic animals and that this is a significant cause of the oxidative stress which occurs in the disease. The aim of this work was to search for molecular links between xanthine oxidase-induced oxidative stress and inflammation in Type I diabetes and to assess the ability of allopurinol, a drug widely used in clinical practice, to prevent both processes. 3-month-old male Wistar rats were made diabetic by injection (i.p.) of either streptozotocin or alloxan. Allopurinol (32 mg/Kg) was administered (i.p) to diabetic rats after they had shown clear signs of diabetes such as glucosuria and polyuria. Hepatic phospho-IKKbeta…

MaleTranscriptional ActivationXanthine Oxidasemedicine.medical_specialtyNeutrophilsAllopurinolAllopurinolInterleukin 6Free radicalsInflammationmedicine.disease_causeBiochemistryStreptozocinDiabetes Mellitus ExperimentalDiabetic complicationsProinflammatory cytokineInterleukin 1βchemistry.chemical_compoundCell MovementPhysiology (medical)Internal medicineDiabetes mellitusAlloxanmedicineAnimalsRats WistarXanthine oxidasePolyuriabusiness.industryAllopurinol; Interleukin 1β; Interleukin 6; Diabetic complications; Free radicalsNF-kappa BXanthineStreptozotocinmedicine.diseaseRatsOxidative StressEndocrinologyLiverchemistryCytokinesInflammation Mediatorsmedicine.symptombusinessOxidative stressmedicine.drugFree Radical Biology and Medicine
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