Search results for "ASCL1"

showing 6 items of 6 documents

Direct Reprogramming of Somatic Cells into Induced Neuronal Cells: Where We Are and Where We Want to Go

2014

Direct reprogramming of somatic cells has emerged as a novel strategy to generate neurons, including those of human origin. The method provides enormous potential for studying the mechanisms directing neuronal differentiation, the modeling of neurodegenerative diseases, and the development of potential novel approaches for brain repair. In this chapter, we briefly trace the history of reprogramming, as well as the strategies employed to date for converting different types of somatic cells, including fibroblasts, hepatocytes, astrocytes, and pericytes into induced neuronal cells of different subtype identity. Reprogramming involves particular emphasis on transcriptional and posttranscription…

ASCL1Somatic cellRegeneration (biology)NeurogenesismicroRNAEpigeneticsBiologyTranscription factorNeuroscienceReprogramming
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Direct pericyte-to-neuron reprogramming via unfolding of a neural stem cell-like program

2018

Ectopic expression of defined transcription factors can force direct cell-fate conversion from one lineage to another in the absence of cell division. Several transcription factor cocktails have enabled successful reprogramming of various somatic cell types into induced neurons (iNs) of distinct neurotransmitter phenotype. However, the nature of the intermediate states that drive the reprogramming trajectory toward distinct iN types is largely unknown. Here we show that successful direct reprogramming of adult human brain pericytes into functional iNs by Ascl1 and Sox2 encompasses transient activation of a neural stem cell-like gene expression program that precedes bifurcation into distinct…

AdultMale0301 basic medicineSomatic cellCellular differentiationBasic Helix-Loop-Helix Transcription FactorSOXB1 Transcription FactorBiologyArticleYoung Adult03 medical and health sciences0302 clinical medicineNeural Stem CellsSOX2Basic Helix-Loop-Helix Transcription FactorsHumansCell LineageNeural Stem CellAgedPericyteNeuronsSOXB1 Transcription FactorsGeneral NeuroscienceCell DifferentiationMiddle AgedNeuronCellular ReprogrammingNeural stem cellASCL1030104 developmental biologyGene Expression RegulationFemaleEctopic expressionPericytesNeural developmentReprogrammingNeuroscience030217 neurology & neurosurgeryHuman
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Sox2-Mediated Conversion of NG2 Glia into Induced Neurons in the Injured Adult Cerebral Cortex

2014

Summary The adult cerebral cortex lacks the capacity to replace degenerated neurons following traumatic injury. Conversion of nonneuronal cells into induced neurons has been proposed as an innovative strategy toward brain repair. Here, we show that retrovirus-mediated expression of the transcription factors Sox2 and Ascl1, but strikingly also Sox2 alone, can induce the conversion of genetically fate-mapped NG2 glia into induced doublecortin (DCX)+ neurons in the adult mouse cerebral cortex following stab wound injury in vivo. In contrast, lentiviral expression of Sox2 in the unlesioned cortex failed to convert oligodendroglial and astroglial cells into DCX+ cells. Neurons induced following …

Doublecortin ProteinGene ExpressionBiochemistryArticleMiceSOX2Cortex (anatomy)Basic Helix-Loop-Helix Transcription FactorsGeneticsmedicineAnimalslcsh:QH301-705.5Cell ProliferationCerebral CortexNeuronslcsh:R5-920biologySOXB1 Transcription FactorsCell BiologyAnatomySynaptic PotentialsCellular ReprogrammingDoublecortinASCL1medicine.anatomical_structurelcsh:Biology (General)nervous systemCerebral cortexCell Transdifferentiationbiology.proteinNeurogliaNeuNlcsh:Medicine (General)NeurogliaReprogrammingNeuroscienceDevelopmental BiologyStem Cell Reports
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Ascl1 Coordinately Regulates Gene Expression and the Chromatin Landscape during Neurogenesis

2015

Summary The proneural transcription factor Ascl1 coordinates gene expression in both proliferating and differentiating progenitors along the neuronal lineage. Here, we used a cellular model of neurogenesis to investigate how Ascl1 interacts with the chromatin landscape to regulate gene expression when promoting neuronal differentiation. We find that Ascl1 binding occurs mostly at distal enhancers and is associated with activation of gene transcription. Surprisingly, the accessibility of Ascl1 to its binding sites in neural stem/progenitor cells remains largely unchanged throughout their differentiation, as Ascl1 targets regions of both readily accessible and closed chromatin in proliferatin…

Genetics0303 health sciencesNeurogenesisNeurogenesisDNABiologyGeneral Biochemistry Genetics and Molecular BiologyChromatin remodelingArticleCell biologyChromatin03 medical and health sciences0302 clinical medicinelcsh:Biology (General)[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular Biology[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyAscl1Scaffold/matrix attachment regionEnhancerlcsh:QH301-705.5Transcription factor030217 neurology & neurosurgeryChIA-PET030304 developmental biologyBivalent chromatin
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Direct In Vitro Reprogramming of Astrocytes into Induced Neurons

2021

Spontaneous neuronal replacement is almost absent in the postnatal mammalian nervous system. However, several studies have shown that both early postnatal and adult astroglia can be reprogrammed in vitro or in vivo by forced expression of proneural transcription factors, such as Neurogenin-2 or Achaete-scute homolog 1 (Ascl1), to acquire a neuronal fate. The reprogramming process stably induces properties such as distinctly neuronal morphology, expression of neuron-specific proteins, and the gain of mature neuronal functional features. Direct conversion of astroglia into neurons thus possesses potential as a basis for cell-based strategies against neurological diseases. In this chapter, we …

Mammalian nervous systemASCL1medicine.anatomical_structurenervous systemIn vivoFunctional featuresCellmedicineBiologyTranscription factorReprogrammingIn vitroCell biology
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Transcriptional Mechanisms of Proneural Factors and REST in Regulating Neuronal Reprogramming of Astrocytes

2015

Summary Direct lineage reprogramming induces dramatic shifts in cellular identity, employing poorly understood mechanisms. Recently, we demonstrated that expression of Neurog2 or Ascl1 in postnatal mouse astrocytes generates glutamatergic or GABAergic neurons. Here, we take advantage of this model to study dynamics of neuronal cell fate acquisition at the transcriptional level. We found that Neurog2 and Ascl1 rapidly elicited distinct neurogenic programs with only a small subset of shared target genes. Within this subset, only NeuroD4 could by itself induce neuronal reprogramming in both mouse and human astrocytes, while co-expression with Insm1 was required for glutamatergic maturation. Cu…

Transcription GeneticRepressorNerve Tissue ProteinsCell fate determinationBiologyDNA-binding proteinArticleMiceGlutamatergicBasic Helix-Loop-Helix Transcription FactorsGeneticsAnimalsHumansPromoter Regions GeneticTranscription factorCells CulturedNeuronsCell BiologyCellular ReprogrammingMolecular biologyCell biologyDNA-Binding ProteinsRepressor ProteinsASCL1Astrocytesembryonic structuresMolecular MedicineGABAergicReprogrammingTranscription FactorsCell Stem Cell
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