Search results for "ATPase"

showing 6 items of 196 documents

The effect of human proinsulin C-peptide on erythrocyte deformability in patients with Type I diabetes mellitus.

1999

Aims/hypothesis. In recent years, evidence has arisen that proinsulin C-peptide exerts biological effects especially on microcirculation, e. g. C-peptide has been shown to increase skin microcirculation in patients with Type I (insulin-dependent) diabetes mellitus and to activate endothelial nitric oxide synthase. This study aimed to investigate the influence of proinsulin C-peptide on erythrocyte deformability which was assessed by means of laser diffractoscopy. Methods. Blood samples from healthy control subjects (n = 10) and Type I diabetic patients (n = 15) completely deficient of C-peptide were analysed at shear stresses ranging from 0.3 to 30 Pa. Results. Erythrocyte deformability was…

medicine.medical_specialtyErythrocytesEndocrinology Diabetes and MetabolismProhormoneMicrocirculationBlood cellchemistry.chemical_compoundInternal medicineDiabetes mellitusErythrocyte DeformabilityInternal MedicineMedicineErythrocyte deformabilityHumansEnzyme InhibitorsOuabainProinsulinC-Peptidebusiness.industryC-peptideLasersmedicine.diseaseRed blood cellEndocrinologymedicine.anatomical_structureDiabetes Mellitus Type 1chemistrySodium-Potassium-Exchanging ATPasebusinessmedicine.drugDiabetologia
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Effects of chronic alcohol consumption on enzyme activities and active methionine absorption in the small intestine of pregnant rats.

1996

The present study evaluates the effect of chronic alcohol intake on the intestinal transport of methionine during pregnancy. For this purpose, we have used an in vitro technique that allows measurement of the unidirectional influx of the amino acids across the brush-border membrane of the rat mid-jejunum, and the basolateral membrane enzyme Na+, K+-ATPase was also evaluated in the duodenum and jejunum. For chronic alcohol treatment, the rats were fed a liquid diet containing ethanol (36% of calories) or an isocaloric diet-(pair-fed control) for 5 weeks before and during pregnancy. Animals were killed at 21 days of gestation. Results from the kinetic analysis revealed that chronic ethanol tr…

medicine.medical_specialtyLiquid dietMedicine (miscellaneous)BiologyToxicologyIntestinal absorptionMethionine transportJejunumchemistry.chemical_compoundMethionineIntestinal mucosaPregnancyInternal medicinemedicineAnimalsNa+/K+-ATPaseIntestinal MucosaRats WistarMethionineSmall intestineRatsPsychiatry and Mental healthEndocrinologymedicine.anatomical_structureJejunumchemistryIntestinal AbsorptionFetal Alcohol Spectrum DisordersFemaleSodium-Potassium-Exchanging ATPaseAlcoholism, clinical and experimental research
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Clinical and genetic characteristics of 21 Spanish patients with biallelic pathogenic SPG7 mutations.

2021

Spastic paraplegia type 7 (SPG7) is one of the most common hereditary spastic paraplegias. SPG7 mutations most often lead to spastic paraparesis (HSP) and/or hereditary cerebellar ataxia (HCA), frequently with mixed phenotypes. We sought to clinically and genetically characterize a Spanish cohort of SPG7 patients. Patients were recruited from our HCA and HSP cohorts. We identified twenty-one patients with biallelic pathogenic SPG7 mutations. Mean age at onset was 37.4 years (SD ± 14.3). The most frequent phenotype was spastic ataxia (57%), followed by pure spastic paraplegia (19%) and complex phenotypes (19%). Isolated patients presented with focal or multifocal dystonia, subclinical myopat…

medicine.medical_specialtyNeurogeneticsCompound heterozygosityGastroenterologyInternal medicinemedicineSpasticHumansMyopathySubclinical infectionDystoniaCerebellar ataxiabusiness.industrySpastic Paraplegia HereditaryMetalloendopeptidasesmedicine.diseasenervous system diseasesOptic AtrophyPhenotypeNeurologyMutationATPases Associated with Diverse Cellular ActivitiesNeurology (clinical)medicine.symptombusinessSpastic paraplegia type 7Journal of the neurological sciences
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Focal elevation of liver microsomal epoxide hydrolase in early preneoplastic stages and its behaviour in the further course of hepatocarcinogenesis.

1981

Abstract Treatment of rats with N-nitrosomorpholine (NNM) for 7 weeks led to a focal increase in liver microsomal epoxide hydrolase (EH) as early as 2 weeks after withdrawal of the carcinogen. This treatment also leads to hyperplastic nodules and liver tumors, but much later. At the same early time point, ATPase activity was decreased in the same islands. Most of these areas already had increased γ-glutamyltranspeptidase activity. The increase in EH at this early time point was more distinct than the decrease in ATPase which has thus far been considered a suitable marker of the earliest stages in hepatocarcinogenesis. The focal increase in EH was also observed in all benign hepatomas, but n…

medicine.medical_specialtyNitrosaminesATPaseBiophysicsBiochemistryLiver Neoplasms ExperimentalInternal medicinemedicineAtpase activityAnimalsMolecular BiologyCarcinogenAdenosine TriphosphatasesEpoxide HydrolasesbiologyLiver NeoplasmsCell Biologygamma-GlutamyltransferaseRatsEndocrinologyLiverMicrosomal epoxide hydrolasebiology.proteinMicrosomes LiverFemaleRabbitsHyperplastic nodulesPrecancerous ConditionsBiochemical and biophysical research communications
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The physiological impact of proinsulin C-peptide

1999

Abstract The proinsulin C-peptide fulfills an important function in the biosynthesis of insulin by facilitating the formation of the correct secondary and tertiary structure of the hormone. C-peptide and insulin are released in equimolar amounts to the circulation but C-peptide has generally been considered to be biologically inert. However, recent studies indicate that C-peptide administration to type 1 diabetes patients is accompanied by improved renal function, amelioration of autonomic dysfunction, stimulated tissue glucose utilization and augmented skin and muscle blood flow. The cellular mechanisms underlying these effects may be related to C-peptide's capacity to stimulate both Na + …

medicine.medical_specialtyType 1 diabetesbiologyC-peptideInsulinmedicine.medical_treatmentATPasebiology.organism_classificationmedicine.diseasePathology and Forensic Medicinechemistry.chemical_compoundEndocrinologychemistryEnosPhysiology (medical)Internal medicinemedicinebiology.proteinNa+/K+-ATPaseHormoneProinsulinPathophysiology
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Amplicon-based high-throughput pooled sequencing identifies mutations in CYP7B1 and SPG7 in sporadic spastic paraplegia patients

2011

Hereditary spastic paraplegia (HSP) is a neurodegenerative disorder defined clinically by progressive lower limb spasticity and weakness. HSP is a genetically highly heterogeneous condition with at least 46 gene loci identified so far, involving X-linked, autosomal recessive (AR) and autosomal dominant inheritance. For correct diagnosis, molecular testing is essential because clinical parameters by themselves are not reliable to differentiate HSP forms. The purpose of this study was to establish amplicon-based high-throughput genotyping for AR-HSP. A sample of 187 index cases with apparently sporadic or recessive spastic paraplegia were analyzed by applying an array-based amplification stra…

methods [High-Throughput Nucleotide Sequencing]GenotypeHereditary spastic paraplegiaDNA Mutational AnalysisMolecular Sequence DataSPG7 protein humanCytochrome P450 Family 7diagnosis [Paraplegia]Biologymedicine.disease_causegenetics [Paraplegia]Cohort Studiesgenetics [Metalloendopeptidases]03 medical and health sciences0302 clinical medicineGenetic variationGenotypeGeneticsmedicineHumansddc:610Genetic TestingGenotypingGenetics (clinical)CYP7B1 protein human030304 developmental biologyGenetic testingParaplegiaGenetics0303 health sciencesMutationBase SequenceParapleginmedicine.diagnostic_testgenetics [Steroid Hydroxylases]Genetic VariationHigh-Throughput Nucleotide SequencingMetalloendopeptidasesmethods [DNA Mutational Analysis]Ampliconmedicine.diseasegenetics [Genetic Variation]3. Good healthMutationSteroid HydroxylasesATPases Associated with Diverse Cellular Activities030217 neurology & neurosurgeryClinical Genetics
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