Search results for "ATS"

showing 10 items of 6881 documents

Phosphatidylserine counteracts physiological and pharmacological suppression of humoral immune response

1990

Phosphatidylserine (PS) is a necessary cofactor for protein kinase C (PKC) activation, and changes in the synthesis of PS have been shown to participate in the mechanism(s) involved in the transmembrane signaling of interleukin 1 (IL-1). In view of the age-associated defects in T-cell functions, in the present study we have addressed the question of whether an in vivo treatment with PS might interfere with such processes. Furthermore, the effect of an in vitro treatment with PS in human peripheral blood monocytes (PBMC) or splenocytes activated with a lectin mitogen, on the expression of IL-2 receptor, was assessed. While the process of ageing was accompanied by a marked decline of humoral …

Agingmedicine.medical_specialtyReceptor expressionPhosphatidylserinesIn Vitro Techniqueschemistry.chemical_compoundImmune systemInternal medicineImmune TolerancemedicineAnimalsHumansReceptorProtein Kinase CProtein kinase CPharmacologybiologyInterleukinRats Inbred StrainsReceptors Interleukin-2PhosphatidylserineRatsEndocrinologychemistryHumoral immunityLeukocytes Mononuclearbiology.proteinFemaleAntibodySpleenImmunopharmacology
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Serum basal hormone concentrations and muscle mass in aging women: Effects of strength training and diet

2006

This study examined the effects of strength training and diet on serum basal hormone concentrations and muscle mass in aging women. Fifty-one women age 49 to 74 y were divided into two groups: strength training and nutritional counseling (n = 25), and strength training (n = 26). Both groups performed strength training twice a week for 21 wk. Nutritional counseling was given to attain sufficient energy and protein intake and recommended intake of fat and fiber. We found that the cross-sectional area of the quadriceps femoris increased by 9.5 ± 4.1% in the nutritional counseling group versus 6.8 ± 3.5% in the strength training only group after training (P < 0.052). Nutritional counseling e…

Agingmedicine.medical_specialtyWeight LiftingGlobulinStrength trainingeducationMedicine (miscellaneous)Muscle massBasal (phylogenetics)Sex Hormone-Binding GlobulinInternal medicinemedicineHumansTestosteroneOrthopedics and Sports MedicineMuscle SkeletalTestosteroneAgedNutrition and DieteticsbiologyDehydroepiandrosterone Sulfatebusiness.industryGeneral MedicineMiddle AgedDietary FatsHormonesNutritional counselingEndocrinologyBody Compositionbiology.proteinFemaleDietary ProteinsEnergy IntakebusinessMuscle ContractionRecommended IntakeHormone
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Cardiac Micro–Computed Tomography Imaging of the Aging Coronary Vasculature

2012

Background— Alterations at the level of the coronary circulation with aging may play an important role in the evolution of age-associated changes in left ventricular (LV) fibrosis and function. However these age-associated changes in the coronary vasculature remain poorly defined primarily due to the lack of high resolution imaging technologies. The current study was designed to utilize cardiac micro–computed tomography (micro-CT) technology as a novel imaging strategy, to define the 3-dimensional coronary circulation in the young and aged heart and its relationship to LV fibrosis and function. Methods and Results— Young (2 months old; n=10) and aged (20 months old; n=10) Fischer rats unde…

Agingmedicine.medical_specialtyX-ray microtomographyBlood PressureArticleVentricular Dysfunction LeftCoronary circulationImaging Three-DimensionalFibrosisCoronary CirculationInternal medicineLv dysfunctionAnimalsMedicineRadiology Nuclear Medicine and imagingbusiness.industryMicro computed tomographyCoronary vasculatureImaging studyX-Ray Microtomographymedicine.diseaseFibrosisSettore MED/11 - Malattie Dell'Apparato CardiovascolareRats Inbred F344Ratsmedicine.anatomical_structureBlood pressureEchocardiographyHigh resolution imaging techinques.Linear ModelsCardiologyCardiology and Cardiovascular MedicinebusinessCirculation: Cardiovascular Imaging
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Age-associated oxidative damage leads to absence of γ-cystathionase in over 50% of rat lenses: Relevance in cataractogenesis

2004

Oxidative damage to lens proteins and glutathione depletion play a major role in the development of senile cataract. We previously found that a deficiency in gamma-cystathionase activity may be responsible for glutathione depletion in old lenses. The aims of this study were: (1) to investigate the mechanism that causes the age-related deficiency in gamma-cystathionase activity in the eye lens, and (2) to determine the role of gamma-cystathionase deficiency in cataractogenesis. Two populations of old rats were found, one (56%) whose lenses lacked gamma-cystathionase activity and the rest that exhibited detectable enzyme activity. gamma-Cystathionase protein was absent in lenses from old rats…

Agingmedicine.medical_specialtygenetic structuresGlycinemedicine.disease_causeBiochemistryCataractLens proteinchemistry.chemical_compoundPhysiology (medical)Internal medicineLens CrystallineGene expressionmedicineAnimalsRats WistarGlyceraldehyde 3-phosphate dehydrogenasechemistry.chemical_classificationbiologyCystathionine gamma-lyaseCystathionine gamma-LyaseGlutathioneGlutathioneeye diseasesEnzyme assayRatsOxidative StressEndocrinologyEnzymeBiochemistrychemistryAlkynesbiology.proteinsense organsOxidative stressFree Radical Biology and Medicine
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Cannabinoid and nitric oxide signaling interplay in the modulation of hippocampal hyperexcitability: study on electrophysiological and behavioral mod…

2015

A growing bulk of evidence suggests that cannabinoid system plays a pivotal role in the control of hyperexcitability phenomena. Notwithstanding, the anticonvulsant action of cannabinoids has not been fully addressed, in particular the involvement of potential cellular neuromodulators, for instance nitric oxide. In the current study, we focused on two distinct rat models of temporal lobe epilepsy, the Maximal Dentate Activation and the pilocarpine-induced acute seizures, providing both electrophysiological and behavioral data on cannabinoid and nitrergic system interplay. We evaluated the antiepileptic effects of WIN 55,212-2, (R)-(+)-[2,3-dihydro-5-methyl-3-(4- morpholinylmethyl) pyrrolo[1,…

AgonistAM251MaleCannabinoid receptorIndazolesmedicine.drug_classmedicine.medical_treatmentMorpholinesHippocampusPharmacologyNaphthalenesNitric OxideHippocampusSettore BIO/09 - FisiologiaEpilepsyPiperidinesReceptor Cannabinoid CB1medicineAnimalshippocampus temporal lobe epilepsy cannabinoids behavior percentage of protection electrophysiology.Rats WistarWIN 55212-2Cannabinoid Receptor AgonistsDose-Response Relationship DrugCannabinoidsGeneral NeurosciencePilocarpinemedicine.diseaseEndocannabinoid systemBenzoxazinesRatsDisease Models AnimalEpilepsy Temporal LobePyrazolesCannabinoidNitric Oxide SynthasePsychologyNeurosciencemedicine.drug
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Evidences of cannabinoids-induced modulation of paroxysmal events in an experimental model of partial epilepsy in the rat.

2009

The anticonvulsant effect of cannabinoids (CB) has been shown to be mediated by the activation of the CB(1) receptor. This study evaluates the anticonvulsant activity of (R)-(+)-[2,3-dihydro-5-methyl-3-(4-morpholinylmethyl) pyrrolo[1,2,3-de]-1,4-benzoxazin-6-Yl]-1-naphthalenylmethanone (WIN55,212-2, CB agonist) alone or preceded by the administration of N-(piperidin-1-yl)-5-(4-iodophenyl)-1-(2,4-dichlorophenyl)-4-methyl-1H-pyrazole-3-carboxamide (AM251, selective CB(1) antagonist) in an experimental in vivo model of complex partial seizures (maximal dentate gyrus activation - MDA) in the rat. WIN55,212-2 (21mgkg(-1)) exerted an anticonvulsant effect, significantly reduced by the pre-treatme…

AgonistAM251Malemedicine.medical_specialtyCannabinoid receptormedicine.drug_classmedicine.medical_treatmentMorpholinesNaphthalenesSettore BIO/09 - FisiologiaEpilepsyPiperidinesReceptor Cannabinoid CB1Internal medicineControlCannabinoid Receptor ModulatorsmedicineAnimalsRats WistarReceptorEpilepsyChemistryCannabinoidsGeneral NeuroscienceAntagonistBrainmedicine.diseaseCalcium Channel BlockersElectric StimulationBenzoxazinesRatsDisease Models AnimalMaximal dentate activationAnticonvulsantEndocrinologySettore BIO/14 - FarmacologiaRatPyrazolesAnticonvulsantsCannabinoidEpilepsies Partialmedicine.drugNeuroscience letters
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Induction of apoptosis in cardiac myocytes by an A3 adenosine receptor agonist.

1998

The effects of the selective adenosine (ADO) A3 receptor agonist IB-MECA (N6-(3-iodobenzyl)adenosine-5'-N-methylcarboxamide) on cultured newborn rat cardiomyocytes were examined in comparison with ADO, the ADO A1 receptor-selective agonist R-PIA (N6-R-phenylisopropyladenosine), or the ADO A3 selective antagonist MRS 1191 (3-ethyl-5-benzyl-2-methyl-6-phenyl-4-phenylethynyl-1, 4-(+/-)-dihydropyridine-3,5 dicarboxylate), using digital image analysis of Feulgen-stained nuclei. At high concentration, IB-MECA (/=10 microM ) and ADO (200 microM) induced apoptosis; however, R-PIA or MRS 1191 did not have any detectable effects on cardiac cells. In addition, DNA breaks in cardiomyocytes undergoing a…

AgonistContraction (grammar)TUNEL assayAdenosinemedicine.drug_classMyocardiumReceptor Adenosine A3ApoptosisHeartCell BiologyDNA FragmentationBiologyAdenosine receptorMolecular biologyAdenosineRatsApoptosismedicinePurinergic P1 Receptor AgonistsMyocyteAnimalsCalciumReceptorCells Culturedmedicine.drugExperimental cell research
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Neuronal nicotinic receptors in synaptic functions in humans and rats: physiological and clinical relevance.

2000

The present report describes the participation of nicotinic receptors (nAChRs) in controlling the excitability of local neuronal circuitries in the rat hippocampus and in the human cerebral cortex. The patch-clamp technique was used to record responses triggered by the non-selective agonist ACh and the alpha7-nAChR-selective agonist choline in interneurons of human cerebral cortical and rat hippocampal slices. Evidence is provided that functional alpha7- and alpha4beta2-like nAChRs are present on somatodendritic and/or preterminal/terminal regions of interneurons in the CA1 field of the rat hippocampus and in the human cerebral cortex and that activation of the different nAChR subtypes pres…

AgonistInterneuronmedicine.drug_classCentral nervous systemHippocampusBiologyHippocampal formationReceptors NicotinicHippocampusSynaptic TransmissionMembrane PotentialsRats Sprague-DawleyBehavioral NeuroscienceAlzheimer DiseaseInterneuronsCulture Techniquesmental disordersmedicineAnimalsHumansReceptorgamma-Aminobutyric AcidCerebral CortexNeuronsBrain Mappingmusculoskeletal neural and ocular physiologyBrainRatsmedicine.anatomical_structurenervous systemCerebral cortexSchizophreniasense organsNeuroscienceAcetylcholinemedicine.drugBehavioural brain research
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Na+ ions binding to the bradykinin B2 receptor suppress agonist-independent receptor activation.

1996

Control of the balance between receptor activation and inactivation is a prerequisite for seven transmembrane domain (7TM) receptor function. We asked for a mechanism to stabilize the inactive receptor conformation which prevents agonist-independent receptor activation. Na+ ions have reciprocal effects on agonist versus antagonist interaction with various 7TM receptors. To investigate the Na+ dependence of receptor activation we chose the bradykinin B2 receptor as a prototypic 7TM receptor. Decrease of the intracellular Na+ content from 40 mM to 10 mM of COS-1 cells transiently expressing rat B2 receptors activated the B2 receptor in the absence of agonist as shown by a 3-fold increase in t…

AgonistIntracellular FluidIntrinsic activityReceptor Bradykinin B2medicine.drug_classInositol PhosphatesBradykininIn Vitro TechniquesBradykininLigandsBiochemistryCell Linechemistry.chemical_compoundmedicineAnimalsHumansPoint MutationBradykinin receptorPhosphorylationReceptorG protein-coupled receptorReceptors BradykininSodiumRatschemistryCOS CellsBiophysicsMutagenesis Site-DirectedAlpha-4 beta-2 nicotinic receptorIntracellularBiochemistry
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Systemic administration of D-penicillamine prevents the locomotor activation after intra-VTA ethanol administration in rats.

2010

Although recently published studies seem to confirm the important role displayed by acetaldehyde (ACH), the main metabolite of ethanol, in the behavioral effects of ethanol, the origin of ACH is still a matter of debate. While some authors confer more importance to the central (brain metabolism) origin of ACH, others indicate that the hepatic origin could be more relevant. In this study we have addressed this topic using an experimental approach that combines local microinjections of ethanol into the ventral tegmental area (VTA) (which guarantees the brain origin of the ACH) to induce motor activation in rats together with systemic administration (i.p.) of several doses (0, 12.5, 25 and 50 …

AgonistLocomotor activityMalemedicine.drug_classMetaboliteCentral nervous systemAcetaldehydePharmacologyMotor Activitychemistry.chemical_compoundAlcohol-Induced Disorders Nervous SystemmedicineAnimalsRats WistarReceptorEthanolGeneral NeurosciencePenicillamineD-PenicillaminePenicillamineVentral Tegmental AreaCentral Nervous System DepressantsRatsVentral tegmental areaDAMGOBrain metabolism of ethanolDisease Models Animalmedicine.anatomical_structurechemistryBiochemistrySystemic administrationVTAmedicine.drugNeuroscience letters
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