Search results for "Abatacept"

showing 7 items of 7 documents

CD4(+) and CD8(+) anergic T cells induced by interleukin-10-treated human dendritic cells display antigen-specific suppressor activity.

2002

Interleukin-10 (IL-10)–treated dendritic cells (DCs) induce an alloantigen- or peptide-specific anergy in various CD4+ and CD8+ T-cell populations. In the present study, we analyzed whether these anergic T cells are able to regulate antigen-specific immunity. Coculture experiments revealed that alloantigen-specific anergic CD4+ and CD8+ T cells suppressed proliferation of syngeneic T cells in a dose-dependent manner. The same effect was observed when the hemagglutinin-specific CD4+T-cell clone HA1.7 or tyrosinase-specific CD8+ T cells were cocultured with anergic T cells of the same specificity. Anergic T cells did not induce an antigen-independent bystander inhibition. Suppression was depe…

CD4-Positive T-LymphocytesIsoantigensImmunoconjugatesImmunologyAntigen-Presenting Cellschemical and pharmacologic phenomenaBiologyCD8-Positive T-LymphocytesLymphocyte ActivationBiochemistryT-Lymphocytes RegulatoryAbataceptInterleukin 21Antigens CDAntigens NeoplasmCytotoxic T cellHumansCTLA-4 AntigenIL-2 receptorLeukapheresisAntigen-presenting cellMelanomaCells CulturedClonal AnergyImmunosuppression TherapyMonophenol MonooxygenaseCD28Cell BiologyHematologyDendritic cellT lymphocyteDendritic CellsNatural killer T cellAntigens DifferentiationCoculture TechniquesCell biologyInterleukin-10ImmunologyCD4 AntigensLeukocytes MononuclearCell DivisionBlood
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Different effects of biological drugs in rheumatoid arthritis

2013

Biological drugs have brought new hope to patients with rheumatoid arthritis (RA) in whom previously existing treatments could not control inflammation, joint destruction, or the progression of disability. The five currently available TNF blockers are approved for treating RA patients, but they have different structures, morphology, pharmacokinetic properties, and activity. Randomised clinical trials (RCTs) have shown that they improve the signs and symptoms of both early and long-standing RA and other inflammatory arthritides, prevent radiographic progression, and improve the patients' health-related quality of life. However, they are more effective in combination with methotrexate (MTX) t…

Cartilage Articularmedicine.medical_specialtyDrug-Related Side Effects and Adverse ReactionsImmunologyArthritisPharmacologyArthritis Rheumatoidchemistry.chemical_compoundChondrocytesPharmacotherapyTocilizumabDrug TherapyRheumatoidInternal medicinemedicineHumansImmunology and AllergyArthritis Rheumatoid; Cartilage Articular; Chondrocytes; Drug Therapy Combination; Drug-Related Side Effects and Adverse Reactions; Humans; Inflammation Mediators; Pharmaceutical Preparations; Randomized Controlled Trials as Topic; Tumor Necrosis Factor-alphaAdverse effectRandomized Controlled Trials as TopicTumor Necrosis Factor-alphabusiness.industryArthritisAbataceptmedicine.diseaseClinical trialCartilagePharmaceutical PreparationschemistryRheumatoid arthritisCombinationDrug Therapy CombinationRituximabInflammation MediatorsbusinessArticularmedicine.drugAutoimmunity Reviews
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Suppressor activity of anergic T cells induced by IL-10-treated human dendritic cells: association with IL-2- and CTLA-4-dependent G1 arrest of the c…

2003

We have previously shown that human IL-10-treated dendritic cells (DC) induce an antigen-specific anergy in CD4+ T lymphocytes. These anergic T cells are characterized by an inhibited proliferation, a reduced production of IL-2, and additionally display antigen-specific suppressor activity. In this study we investigated the mechanisms underlying the anergic state and regulatory function of these T cells. We did not observe enhanced rates of programmed cell death of anergic CD4+ suppressor T cells compared to T cells stimulated with mature DC. Cell cycle analysis by DNA staining and Western blot experiments revealed an arrest of anergic CD4+ T suppressor cells in the G1 phase. High levels of…

ImmunoconjugatesRegulatory T cellT-LymphocytesImmunologyApoptosisCell Cycle ProteinsAbataceptCyclin-dependent kinaseAntigens CDmedicineImmunology and AllergyHumansCTLA-4 AntigenIL-2 receptorClonal AnergybiologyTumor Suppressor ProteinsRetinoblastoma proteinDendritic cellDendritic CellsCell cycleAntigens DifferentiationCell biologyInterleukin-10Interleukin 10medicine.anatomical_structurebiology.proteinInterleukin-2CDK inhibitorCell DivisionCyclin-Dependent Kinase Inhibitor p27European journal of immunology
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Tuning tumor-specific T-cell activation: a matter of costimulation?

2002

Abstract The stimulation of a specific antitumor immune response, involving the recruitment of T cells and induction of T-cell effector functions, is an attractive possibility for cancer immunotherapy. In the past few years, advances in our understanding of the mechanisms of T-cell activation and costimulation have provided the basis for strategies to enhance antitumor immunity and break tolerance. These strategies include the equipment of tumor cells with costimulatory molecules such as B7, blockade of inhibitory signals on T cells (e.g. through cytotoxic T-lymphocyte antigen 4) and grafting of T cells with antigen-triggered, recombinant costimulatory receptors. Combining antigen-triggered…

ImmunoconjugatesT cellmedicine.medical_treatmentT-LymphocytesImmunologyBiologyLymphocyte ActivationImmunotherapy AdoptiveAbataceptCancer immunotherapyCD28 AntigensAntigens CDNeoplasmsmedicineImmunology and AllergyCytotoxic T cellHumansAntigens Tumor-Associated CarbohydrateCTLA-4 AntigenIL-2 receptorAntigen-presenting cellCD28ImmunotherapyAntigens Differentiationmedicine.anatomical_structureCTLA-4ImmunologyB7-1 AntigenTrends in immunology
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Identification and Functional Characterization of Human Cd4+Cd25+ T Cells with Regulatory Properties Isolated from Peripheral Blood

2001

A subpopulation of peripheral human CD4(+)CD25(+) T cells that expresses CD45RO, histocompatibility leukocyte antigen DR, and intracellular cytotoxic T lymphocyte-associated antigen (CTLA) 4 does not expand after stimulation and markedly suppresses the expansion of conventional T cells in a contact-dependent manner. After activation, CD4(+)CD25(+) T cells express CTLA-4 on the surface detectable for several weeks. These cells show a G1/G0 cell cycle arrest and no production of interleukin (IL)-2, IL-4, or interferon (IFN)-gamma on either protein or mRNA levels. The anergic state of CD4(+)CD25(+) T cells is not reversible by the addition of anti-CD28, anti-CTLA-4, anti-transforming growth fa…

Immunoconjugateshuman regulatory T cellsT cellCTLA-4 expressionImmunologychemical and pharmacologic phenomenaCell CommunicationBiologyLymphocyte ActivationAbataceptMiceInterleukin 21Antigens CDT-Lymphocyte SubsetsCD4+CD25+ T cellsImmune TolerancemedicineAnimalsHumansImmunology and AllergyCytotoxic T cellCTLA-4 AntigenIL-2 receptorAntigen-presenting cellInterleukin 3toleranceCD28Receptors Interleukin-2hemic and immune systemsNatural killer T cellAntigens DifferentiationMolecular biologymedicine.anatomical_structureT cell inhibitionCD4 AntigensCytokinesLeukocyte Common AntigensOriginal ArticleJournal of Experimental Medicine
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Human CD4+CD25+ T cells derived from the majority of atopic donors are able to suppress TH1 and TH2 cytokine production

2003

Abstract Background: Recently, it has been established that CD4 + CD25 + T cells with regulatory capacity are present in human peripheral blood, inhibiting allogeneic proliferation and cytokine production of preactivated CD4 + CD25 − respond-er T cells. Objective: The aim of this study was to analyze in an allergen-specific setting whether such regulatory CD4 + CD25 + T cells also exist and function normally in atopic individuals, especially concerning the inhibition of T H 2 cytokines. Methods: For this purpose, CD4 + CD25 − or CD4 + CD25 + T cells from donors allergic to grass or birch pollen (mainly with rhinitis) or from healthy nonatopic donors were stimulated in the presence of autolo…

Immunoconjugatesmedicine.medical_treatmentImmunologychemical and pharmacologic phenomenaBiologyLymphocyte ActivationImmunophenotypingAbataceptInterleukin 21Th2 CellsAntigenAntigens CDTransforming Growth Factor betaHypersensitivitymedicineHumansImmunology and AllergyCytotoxic T cellCTLA-4 AntigenIL-2 receptorGrowth factorReceptors Interleukin-2hemic and immune systemsT lymphocyteDendritic cellTh1 CellsAntigens DifferentiationInterleukin-10CytokineCD4 AntigensImmunologyCytokinesJournal of Allergy and Clinical Immunology
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Abatacept as Adjunctive Therapy in Refractory Polymyalgia Rheumatica.

2021

Glucocorticoids (GCs) are the mainstay of treatment for patients with polymyalgia rheumatica (PMR).1 Despite their efficacy, GCs are associated with well-known adverse events and a substantial proportion of patients with PMR do not respond adequately, or are refractory, to initial GC treatment. GC-sparing agents in PMR are limited to methotrexate (MTX).1.

musculoskeletal diseasesmedicine.medical_specialtyeducationImmunologyGiant Cell ArteritisPolymyalgia rheumaticaAbataceptDiagnosis DifferentialRheumatologyRefractoryimmune system diseasesInternal medicinemedicineImmunology and AllergyHumansskin and connective tissue diseasesAdverse effectbusiness.industryAbataceptmedicine.diseaseCombined Modality TherapyPolymyalgia RheumaticaMethotrexatebusinesshormones hormone substitutes and hormone antagonistsmedicine.drugThe Journal of rheumatology
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